Example of Science Signaling format
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Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format
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Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format Example of Science Signaling format
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Science Signaling — Template for authors

Categories Rank Trend in last 3 yrs
Biochemistry #33 of 415 -
Molecular Biology #46 of 382 up up by 3 ranks
Cell Biology #39 of 279 down down by 2 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 724 Published Papers | 7655 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 24/06/2020
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Related Journals

open access Open Access

Springer

Quality:  
High
CiteRatio: 6.8
SJR: 1.329
SNIP: 1.08
open access Open Access

Portland Press

Quality:  
High
CiteRatio: 6.7
SJR: 1.706
SNIP: 1.117
open access Open Access

American Society for Biochemistry and Molecular Biology (ASBMB)

Quality:  
High
CiteRatio: 7.7
SJR: 2.361
SNIP: 1.18
open access Open Access

Elsevier

Quality:  
High
CiteRatio: 5.5
SJR: 0.998
SNIP: 0.777

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

6.467

0% from 2018

Impact factor for Science Signaling from 2016 - 2019
Year Value
2019 6.467
2018 6.481
2017 6.378
2016 6.494
graph view Graph view
table view Table view

10.6

14% from 2019

CiteRatio for Science Signaling from 2016 - 2020
Year Value
2020 10.6
2019 9.3
2018 8.6
2017 10.0
2016 11.6
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 0% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 14% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.659

18% from 2019

SJR for Science Signaling from 2016 - 2020
Year Value
2020 3.659
2019 3.101
2018 3.169
2017 3.812
2016 4.858
graph view Graph view
table view Table view

1.504

15% from 2019

SNIP for Science Signaling from 2016 - 2020
Year Value
2020 1.504
2019 1.311
2018 1.191
2017 1.315
2016 1.565
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 18% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 15% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Science Signaling

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American Association for the Advancement of Science

Science Signaling

Science Signaling is a weekly journal, publishing on Tuesdays, 51 issues a year. The overarching goal of Science Signaling is publish key findings of broad relevance in the field of cell signaling and to provide articles and online resources that enable readers to gain insight...... Read More

Biochemistry

Molecular Biology

Cell Biology

Biochemistry, Genetics and Molecular Biology

i
Last updated on
24 Jun 2020
i
ISSN
1937-9145
i
Acceptance Rate
Not provided
i
Frequency
Not provided
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
Science
i
Citation Type
Numbered
(25)
i
Bibliography Example
G. E. Blonder, M. Tinkham, T. M. Klapwijk, Phys Rev B 25, 4515 (1982).

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1126/SCISIGNAL.2004088
Integrative analysis of complex cancer genomics and clinical profiles using the cBioPortal
02 Apr 2013 - Science Signaling

Abstract:

The cBioPortal for Cancer Genomics (http://cbioportal.org) provides a Web resource for exploring, visualizing, and analyzing multidimensional cancer genomics data. The portal reduces molecular profiling data from cancer tissues and cell lines into readily understandable genetic, epigenetic, gene expression, and proteomic even... The cBioPortal for Cancer Genomics (http://cbioportal.org) provides a Web resource for exploring, visualizing, and analyzing multidimensional cancer genomics data. The portal reduces molecular profiling data from cancer tissues and cell lines into readily understandable genetic, epigenetic, gene expression, and proteomic events. The query interface combined with customized data storage enables researchers to interactively explore genetic alterations across samples, genes, and pathways and, when available in the underlying data, to link these to clinical outcomes. The portal provides graphical summaries of gene-level data from multiple platforms, network visualization and analysis, survival analysis, patient-centric queries, and software programmatic access. The intuitive Web interface of the portal makes complex cancer genomics profiles accessible to researchers and clinicians without requiring bioinformatics expertise, thus facilitating biological discoveries. Here, we provide a practical guide to the analysis and visualization features of the cBioPortal for Cancer Genomics. read more read less
10,947 Citations
Journal Article DOI: 10.1126/SCISIGNAL.2000475
Quantitative Phosphoproteomics Reveals Widespread Full Phosphorylation Site Occupancy During Mitosis
12 Jan 2010 - Science Signaling

Abstract:

Eukaryotic cells replicate by a complex series of evolutionarily conserved events that are tightly regulated at defined stages of the cell division cycle. Progression through this cycle involves a large number of dedicated protein complexes and signaling pathways, and deregulation of this process is implicated in tumorigenesi... Eukaryotic cells replicate by a complex series of evolutionarily conserved events that are tightly regulated at defined stages of the cell division cycle. Progression through this cycle involves a large number of dedicated protein complexes and signaling pathways, and deregulation of this process is implicated in tumorigenesis. We applied high-resolution mass spectrometry-based proteomics to investigate the proteome and phosphoproteome of the human cell cycle on a global scale and quantified 6027 proteins and 20,443 unique phosphorylation sites and their dynamics. Co-regulated proteins and phosphorylation sites were grouped according to their cell cycle kinetics and compared to publicly available messenger RNA microarray data. Most detected phosphorylation sites and more than 20% of all quantified proteins showed substantial regulation, mainly in mitotic cells. Kinase-motif analysis revealed global activation during S phase of the DNA damage response network, which was mediated by phosphorylation by ATM or ATR or DNA-dependent protein kinases. We determined site-specific stoichiometry of more than 5000 sites and found that most of the up-regulated sites phosphorylated by cyclin-dependent kinase 1 (CDK1) or CDK2 were almost fully phosphorylated in mitotic cells. In particular, nuclear proteins and proteins involved in regulating metabolic processes have high phosphorylation site occupancy in mitosis. This suggests that these proteins may be inactivated by phosphorylation in mitotic cells. read more read less

Topics:

Phosphoproteomics (62%)62% related to the paper, Phosphorylation (58%)58% related to the paper, Cyclin-dependent kinase 1 (58%)58% related to the paper, Proteomics (57%)57% related to the paper, Mitosis (57%)57% related to the paper
1,447 Citations
Journal Article DOI: 10.1126/SCISIGNAL.2000610
Delivery of microRNA-126 by apoptotic bodies induces CXCL12-dependent vascular protection
08 Dec 2009 - Science Signaling

Abstract:

Apoptosis is a pivotal process in embryogenesis and postnatal cell homeostasis and involves the shedding of membranous microvesicles termed apoptotic bodies. In response to tissue damage, the CXC chemokine CXCL12 and its receptor CXCR4 counteract apoptosis and recruit progenitor cells. Here, we show that endothelial cell-deri... Apoptosis is a pivotal process in embryogenesis and postnatal cell homeostasis and involves the shedding of membranous microvesicles termed apoptotic bodies. In response to tissue damage, the CXC chemokine CXCL12 and its receptor CXCR4 counteract apoptosis and recruit progenitor cells. Here, we show that endothelial cell-derived apoptotic bodies are generated during atherosclerosis and convey paracrine alarm signals to recipient vascular cells that trigger the production of CXCL12. CXCL12 production was mediated by microRNA-126 (miR-126), which was enriched in apoptotic bodies and repressed the function of regulator of G protein (heterotrimeric guanosine triphosphate-binding protein) signaling 16, an inhibitor of G protein-coupled receptor (GPCR) signaling. This enabled CXCR4, a GPCR, to trigger an autoregulatory feedback loop that increased the production of CXCL12. Administration of apoptotic bodies or miR-126 limited atherosclerosis, promoted the incorporation of Sca-1+ progenitor cells, and conferred features of plaque stability on different mouse models of atherosclerosis. This study highlights functions of microRNAs in health and disease that may extend to the recruitment of progenitor cells during other forms of tissue repair or homeostasis. read more read less

Topics:

Paracrine signalling (55%)55% related to the paper, Progenitor cell (55%)55% related to the paper, Microvesicles (54%)54% related to the paper, Heterotrimeric G protein (51%)51% related to the paper, G protein-coupled receptor (51%)51% related to the paper
1,234 Citations
open accessOpen access Journal Article DOI: 10.1126/SCISIGNAL.2005189
Signaling mechanisms of the epithelial-mesenchymal transition
David M. Gonzalez1, Damian Medici2, Damian Medici1
23 Sep 2014 - Science Signaling

Abstract:

The epithelial-mesenchymal transition (EMT) is an essential mechanism in embryonic development and tissue repair. EMT also contributes to the progression of disease, including organ fibrosis and cancer. EMT, as well as a similar transition occurring in vascular endothelial cells called endothelial-mesenchymal transition (EndM... The epithelial-mesenchymal transition (EMT) is an essential mechanism in embryonic development and tissue repair. EMT also contributes to the progression of disease, including organ fibrosis and cancer. EMT, as well as a similar transition occurring in vascular endothelial cells called endothelial-mesenchymal transition (EndMT), results from the induction of transcription factors that alter gene expression to promote loss of cell-cell adhesion, leading to a shift in cytoskeletal dynamics and a change from epithelial morphology and physiology to the mesenchymal phenotype. Transcription program switching in EMT is induced by signaling pathways mediated by transforming growth factor β (TGF-β) and bone morphogenetic protein (BMP), Wnt-β-catenin, Notch, Hedgehog, and receptor tyrosine kinases. These pathways are activated by various dynamic stimuli from the local microenvironment, including growth factors and cytokines, hypoxia, and contact with the surrounding extracellular matrix (ECM). We discuss how these pathways crosstalk and respond to signals from the microenvironment to regulate the expression and function of EMT-inducing transcription factors in development, physiology, and disease. Understanding these mechanisms will enable the therapeutic control of EMT to promote tissue regeneration, treat fibrosis, and prevent cancer metastasis. read more read less

Topics:

Epithelial–mesenchymal transition (60%)60% related to the paper, Bone morphogenetic protein (54%)54% related to the paper, Transcription factor (53%)53% related to the paper, Receptor tyrosine kinase (53%)53% related to the paper, Signal transduction (52%)52% related to the paper
1,194 Citations
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Time taken to format a paper and Compliance with guidelines

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Science Signaling format uses Science citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Science Signaling in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Science Signaling guidelines and auto format it.

2. Do you follow the Science Signaling guidelines?

Yes, the template is compliant with the Science Signaling guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Science Signaling?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Science Signaling citation style.

4. Can I use the Science Signaling templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Science Signaling.

5. Can I use a manuscript in Science Signaling that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Science Signaling that you can download at the end.

6. How long does it usually take you to format my papers in Science Signaling?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Science Signaling.

7. Where can I find the template for the Science Signaling?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Science Signaling's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Science Signaling's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Science Signaling an online tool or is there a desktop version?

SciSpace's Science Signaling is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Science Signaling?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Science Signaling?”

11. What is the output that I would get after using Science Signaling?

After writing your paper autoformatting in Science Signaling, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Science Signaling's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Science Signaling?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Science Signaling. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Science Signaling?

The 5 most common citation types in order of usage for Science Signaling are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Science Signaling?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Science Signaling's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Science Signaling in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Science Signaling Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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