Example of Chemical Research in Toxicology format
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Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format
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Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format Example of Chemical Research in Toxicology format
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open access Open Access

Chemical Research in Toxicology — Template for authors

Categories Rank Trend in last 3 yrs
Toxicology #41 of 122 down down by 26 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 871 Published Papers | 4650 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 14/07/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.184

3% from 2018

Impact factor for Chemical Research in Toxicology from 2016 - 2019
Year Value
2019 3.184
2018 3.274
2017 3.432
2016 3.278
graph view Graph view
table view Table view

5.3

5% from 2019

CiteRatio for Chemical Research in Toxicology from 2016 - 2020
Year Value
2020 5.3
2019 5.6
2018 6.5
2017 6.5
2016 5.6
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 3% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has decreased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.031

3% from 2019

SJR for Chemical Research in Toxicology from 2016 - 2020
Year Value
2020 1.031
2019 1.002
2018 1.149
2017 1.187
2016 1.187
graph view Graph view
table view Table view

1.088

3% from 2019

SNIP for Chemical Research in Toxicology from 2016 - 2020
Year Value
2020 1.088
2019 1.057
2018 1.064
2017 1.045
2016 1.055
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 3% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 3% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Chemical Research in Toxicology

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American Chemical Society

Chemical Research in Toxicology

The Journal is intended to provide a venue for presentation of research relevant to all aspects of the chemical basis of toxic responses. It emphasizes rigorous chemical standards and encourages application of modern techniques of chemical analysis to mechanisms of toxicity. I...... Read More

Medicine

i
Last updated on
14 Jul 2020
i
ISSN
0893-228X
i
Impact Factor
High - 1.28
i
Acceptance Rate
52%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
ACS Custom Citation (achemso)
i
Citation Type
Numbered (Superscripted)
25
i
Bibliography Example
Beenakker, C. W. J. Specular Andreev Reflection in Graphene. Phys. Rev. Lett. 2006, 97, 067007.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1021/TX00026A012
Evaluation of the probe 2',7'-dichlorofluorescin as an indicator of reactive oxygen species formation and oxidative stress.
Carl P. LeBel1, Harry Ischiropoulos, Stephen C. Bondy

Abstract:

The use of dichlorofluorescin (DCFH) as a measure of reactive oxygen species was studied in aqueous media. Hydrogen peroxide oxidized DCFH to fluorescent dichlorofluorescein (DCF), and the oxidation was amplified by the addition of ferrous iron. Hydrogen peroxide-induced DCF formation in the presence of ferrous iron was compl... The use of dichlorofluorescin (DCFH) as a measure of reactive oxygen species was studied in aqueous media. Hydrogen peroxide oxidized DCFH to fluorescent dichlorofluorescein (DCF), and the oxidation was amplified by the addition of ferrous iron. Hydrogen peroxide-induced DCF formation in the presence of ferrous iron was completely inhibited by deferoxamine and partially inhibited by ethylenediaminetetraacetic acid, but was augmented by diethylenetriaminepentaacetic acid. Iron-peroxide-induced oxidation of DCFH was partially inhibited by catalase but not by horseradish peroxidase. Nonchelated iron-peroxide oxidation of DCFH was partially inhibited by several hydroxyl radical scavengers, but was independent of the scavenger concentration, and this suggests that free hydroxyl radical is not involved in the oxidation of DCFH in this system. Superoxide anion did not directly oxidize DCFH. Data suggest that H2O2-Fe(2+)-derived oxidant is mainly responsible for the nonenzymatic oxidation of DCFH. In addition, peroxidase alone and oxidants formed during the reduction of H2O2 by peroxidase oxidize DCFH. Since DCFH oxidation may be derived from several reactive intermediates, interpretation of specific reactive oxygen species involved in biological systems should be approached with caution. However, DCFH remains an attractive probe as an overall index of oxidative stress in toxicological phenomena. read more read less

Topics:

Reactive oxygen species (52%)52% related to the paper, Hydroxyl radical (51%)51% related to the paper, Hydrogen peroxide (50%)50% related to the paper, Dichlorofluorescein (50%)50% related to the paper
View PDF
2,465 Citations
Journal Article DOI: 10.1021/TX9902082
Role of quinones in toxicology.
Judy L. Bolton1, Michael A. Trush2, Trevor M. Penning2, Glenn Dryhurst3, Terrence J. Monks2

Abstract:

Quinones represent a class of toxicological intermediates which can create a variety of hazardous effects in vivo, including acute cytotoxicity, immunotoxicity, and carcinogenesis. The mechanisms by which quinones cause these effects can be quite complex. Quinones are Michael acceptors, and cellular damage can occur through a... Quinones represent a class of toxicological intermediates which can create a variety of hazardous effects in vivo, including acute cytotoxicity, immunotoxicity, and carcinogenesis. The mechanisms by which quinones cause these effects can be quite complex. Quinones are Michael acceptors, and cellular damage can occur through alkylation of crucial cellular proteins and/or DNA. Alternatively, quinones are highly redox active molecules which can redox cycle with their semiquinone radicals, leading to formation of reactive oxygen species (ROS), including superoxide, hydrogen peroxide, and ultimately the hydroxyl radical. Production of ROS can cause severe oxidative stress within cells through the formation of oxidized cellular macromolecules, including lipids, proteins, and DNA. Formation of oxidatively damaged bases such as 8-oxodeoxyguanosine has been associated with aging and carcinogenesis. Furthermore, ROS can activate a number of signaling pathways, including protein kinase C and RAS. This review explore... read more read less

Topics:

Oxidative stress (55%)55% related to the paper, Reactive oxygen species (54%)54% related to the paper
1,499 Citations
Journal Article DOI: 10.1021/TX700079Z
Cytochrome P450 and Chemical Toxicology
F. Peter Guengerich1

Abstract:

The field of cytochrome P450 (P450) research has developed considerably over the past 20 years, and many important papers on the roles of P450s in chemical toxicology have appeared in Chemical Research in Toxicology. Today, our basic understanding of many of the human P450s is relatively well-established, in terms of the deta... The field of cytochrome P450 (P450) research has developed considerably over the past 20 years, and many important papers on the roles of P450s in chemical toxicology have appeared in Chemical Research in Toxicology. Today, our basic understanding of many of the human P450s is relatively well-established, in terms of the details of the individual genes, sequences, and basic catalytic mechanisms. Crystal structures of several of the major human P450s are now in hand. The animal P450s are still important in the context of metabolism and safety testing. Many well-defined examples exist for roles of P450s in decreasing the adverse effects of drugs through biotransformation, and an equally interesting field of investigation is the bioactivation of chemicals, including drugs. Unresolved problems include the characterization of the minor “orphan” P450s, ligand cooperativity and kinetic complexity of several P450s, the prediction of metabolism, the overall contribution of bioactivation to drug idiosyncratic probl... read more read less
1,392 Citations
Journal Article DOI: 10.1021/TX800064J
Copper Oxide Nanoparticles Are Highly Toxic: A Comparison between Metal Oxide Nanoparticles and Carbon Nanotubes
Hanna L. Karlsson1, Pontus Cronholm1, J. Gustafsson1, Lennart Möller1

Abstract:

Since the manufacture and use of nanoparticles are increasing, humans are more likely to be exposed occupationally or via consumer products and the environment. However, so far toxicity data for most manufactured nanoparticles are limited. The aim of this study was to investigate and compare different nanoparticles and nanotu... Since the manufacture and use of nanoparticles are increasing, humans are more likely to be exposed occupationally or via consumer products and the environment. However, so far toxicity data for most manufactured nanoparticles are limited. The aim of this study was to investigate and compare different nanoparticles and nanotubes regarding cytotoxicity and ability to cause DNA damage and oxidative stress. The study was focused on different metal oxide particles (CuO, TiO2, ZnO, CuZnFe2O4, Fe3O4, Fe2O3), and the toxicity was compared to that of carbon nanoparticles and multiwalled carbon nanotubes (MWCNT). The human lung epithelial cell line A549 was exposed to the particles, and cytotoxicity was analyzed using trypan blue staining. DNA damage and oxidative lesions were determined using the comet assay, and intracellular production of reactive oxygen species (ROS) was measured using the oxidation-sensitive fluoroprobe 2',7'-dichlorofluorescin diacetate (DCFH-DA). The results showed that there was a high variation among different nanoparticles concerning their ability to cause toxic effects. CuO nanoparticles were most potent regarding cytotoxicity and DNA damage. The toxicity was likely not explained by Cu ions released to the cell medium. These particles also caused oxidative lesions and were the only particles that induced an almost significant increase (p = 0.058) in intracellular ROS. ZnO showed effects on cell viability as well as DNA damage, whereas the TiO2 particles (a mix of rutile and anatase) only caused DNA damage. For iron oxide particles (Fe3O4, Fe2O3), no or low toxicity was observed, but CuZnFe2O4 particles were rather potent in inducing DNA lesions. Finally, the carbon nanotubes showed cytotoxic effects and caused DNA damage in the lowest dose tested. The effects were not explained by soluble metal impurities. In conclusion, this study highlights the in vitro toxicity of CuO nanoparticles. read more read less

Topics:

Nanotoxicology (64%)64% related to the paper, DNA damage (55%)55% related to the paper, Comet assay (53%)53% related to the paper, Nanoparticle (52%)52% related to the paper, Oxidative stress (50%)50% related to the paper
1,281 Citations
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Chemical Research in Toxicology format uses ACS Custom Citation (achemso) citation style.

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Frequently asked questions

1. Can I write Chemical Research in Toxicology in LaTeX?

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Yes, the template is compliant with the Chemical Research in Toxicology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Chemical Research in Toxicology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Chemical Research in Toxicology citation style.

4. Can I use the Chemical Research in Toxicology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Chemical Research in Toxicology.

5. Can I use a manuscript in Chemical Research in Toxicology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Chemical Research in Toxicology that you can download at the end.

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7. Where can I find the template for the Chemical Research in Toxicology?

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Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Chemical Research in Toxicology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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After writing your paper autoformatting in Chemical Research in Toxicology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Chemical Research in Toxicology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Chemical Research in Toxicology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Chemical Research in Toxicology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Chemical Research in Toxicology?

The 5 most common citation types in order of usage for Chemical Research in Toxicology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Chemical Research in Toxicology?

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16. Can I download Chemical Research in Toxicology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Chemical Research in Toxicology Endnote style according to Elsevier guidelines.

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