Example of Comprehensive Physiology format
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Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format
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Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format Example of Comprehensive Physiology format
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open access Open Access
recommended Recommended

Comprehensive Physiology — Template for authors

Categories Rank Trend in last 3 yrs
Physiology (medical) #6 of 98 down down by 2 ranks
Physiology #11 of 169 down down by 2 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 186 Published Papers | 2472 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 04/06/2020
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Related Journals

open access Open Access

Taylor and Francis

Quality:  
High
CiteRatio: 6.2
SJR: 0.981
SNIP: 1.447
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 17.2
SJR: 1.596
SNIP: 1.811
open access Open Access

Springer

Quality:  
High
CiteRatio: 9.6
SJR: 0.752
SNIP: 1.75
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 3.7
SJR: 0.724
SNIP: 1.069

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

6.604

6% from 2018

Impact factor for Comprehensive Physiology from 2016 - 2019
Year Value
2019 6.604
2018 6.246
2017 5.797
2016 6.949
graph view Graph view
table view Table view

13.3

4% from 2019

CiteRatio for Comprehensive Physiology from 2016 - 2020
Year Value
2020 13.3
2019 12.8
2018 11.3
2017 11.8
2016 12.3
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 6% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 4% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.207

12% from 2019

SJR for Comprehensive Physiology from 2016 - 2020
Year Value
2020 3.207
2019 2.852
2018 2.769
2017 3.067
2016 3.455
graph view Graph view
table view Table view

2.903

6% from 2019

SNIP for Comprehensive Physiology from 2016 - 2020
Year Value
2020 2.903
2019 2.736
2018 2.568
2017 2.836
2016 2.756
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 12% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 6% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Comprehensive Physiology

Guideline source: View

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American Physiological Society

Comprehensive Physiology

Comprehensive Physiology is the most authoritative and comprehensive collection of physiology information that has ever been assembled. Its starting point is more than 30,000 pages of content from the American Physiological Society's renowned Handbook of Physiology (HoP) serie...... Read More

Medicine

i
Last updated on
04 Jun 2020
i
ISSN
2040-4603
i
Impact Factor
Maximum - 6.949
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
(25)
i
Bibliography Example
C. W. J. Beenakker. Specular andreev reflection in graphene. Phys. Rev. Lett., 97(6):067007, 2006.

Top papers written in this journal

Other DOI: 10.1002/CPHY.CP010509
Circuitry of Primate Prefrontal Cortex and Regulation of Behavior by Representational Memory
Patricia S. Goldman-Rakic1
01 Dec 1987 - Comprehensive Physiology

Abstract:

The sections in this article are: 1 Essence of Prefrontal Function: Regulation of Behavior by Representational Knowledge 11 Subdivisions of Prefrontal Cortex 12 Global Nature of Prefrontal Syndrome in Humans 13 Animal Model for Prefrontal Function in Humans 14 Delayed-Response Tests and Varying Interpretat... The sections in this article are: 1 Essence of Prefrontal Function: Regulation of Behavior by Representational Knowledge 11 Subdivisions of Prefrontal Cortex 12 Global Nature of Prefrontal Syndrome in Humans 13 Animal Model for Prefrontal Function in Humans 14 Delayed-Response Tests and Varying Interpretations of Their Functional Significance 15 Distractability and Perseveration: Secondary Consequences of Basic Defect in Representational Memory 16 Representational Memory in Wisconsin Card Sort and Other Diagnostic Tests of Prefrontal Function in Humans 17 Localization of Delayed-Response Function: Principal Sulcus 18 Circuit Basis of Visuospatial Functions 2 Accessing and “On-Line” Processing of Representations in Visuospatial Domain: Parietal-Prefrontal Connections 21 Visuospatial Representational Memory in Humans 22 Spatial-Mnemonic Nature of Delayed-Response Deficit: Domain-Specific Memory Loss 23 Topography of Representational Memory in Prefrontal Cortex 24 Electrophysiological Evidence of Spatial-Mnemonic Processes in Principal Sulcus 25 Parietal-Prefrontal Connectivity 26 Columnar and Laminar Framework for Feedforward and Feedback Mechanisms 27 Functional Significance of Parietal-Prefrontal Collaboration 3 Long-Term Memory and “Off-Line” Processing: Prefrontal-Limbic Connections 31 Role of Hippocampus in Spatial Memory 32 Multiple Connections Between Principal Sulcus and Hippocampal Formation 33 Quadripartite Neural Network: Parietal-Temporal-Cingulate-Prefrontal Circuit 34 Limbic Contribution to Spatial Memory 4 Response Initiation and Inhibition: Projections to Striatum, Tectum, Thalamus, and Premotor Cortex 41 Motor-Control Functions of Prefrontal Cortex 42 Cortical-Striatal Pathway and Related Feedback Loops 43 Cortical-Tectal Pathway 44 Thalamic-Cortical Systems 45 Prefrontal-Premotor Connections: Anterior Supplementary Motor Cortex Relays 46 Functional Speculations 5 Modulatory Mechanisms: Brain Stem Catecholamine Projections 51 Activation of Cognitive Machinery 52 Concentration and Synthesis of Catecholamines in Primate Cortex 53 Brain Stem Innervation of Prefrontal Cortex 54 Delayed-Response Deficits and Recovery Produced by Catecholamine Loss and Replacement in Prefrontal Cortex 55 Circuit Basis for Neuromodulation in Principal Sulcus 6 Multiple Subsystems of Prefrontal Cortex: Unity or Diversity of Function 61 Unity or Diversity of Prefrontal Function 62 Frontal Eye Fields 63 Inferior Convexity 64 Orbital Prefrontal Cortices 65 Problem of Integration 7 Diseases Affecting Prefrontal Cortex 71 Schizophrenia: Loss of Corticocortical Processing and Regulation of Behavior by Representational Knowledge 72 Wernicke-Korsakoff Syndrome: Loss of Thalamocortical and Brain Stem Modulatory Mechanisms 73 Huntington's Chorea and Parkinson's Disease: Loss of Prefrontal-Striatal Mechanisms and Initiation or Inhibition of Motor Response 74 Overview of Neurobiology of Disease 8 Summary read more read less

Topics:

Prefrontal cortex (69%)69% related to the paper, Working memory (69%)69% related to the paper, Consumer neuroscience (64%)64% related to the paper, Interference theory (63%)63% related to the paper, Premotor cortex (63%)63% related to the paper
1,923 Citations
open accessOpen access Other DOI: 10.1002/CPHY.C110025
Lack of exercise is a major cause of chronic diseases
Frank W. Booth1, Christian K. Roberts2, Matthew J. Laye3
01 Apr 2012 - Comprehensive Physiology

Abstract:

Chronic diseases are major killers in the modern era. Physical inactivity is a primary cause of most chronic diseases. The initial third of the article considers: activity and prevention definitions; historical evidence showing physical inactivity is detrimental to health and normal organ functional capacities; cause vs. trea... Chronic diseases are major killers in the modern era. Physical inactivity is a primary cause of most chronic diseases. The initial third of the article considers: activity and prevention definitions; historical evidence showing physical inactivity is detrimental to health and normal organ functional capacities; cause vs. treatment; physical activity and inactivity mechanisms differ; gene-environment interaction [including aerobic training adaptations, personalized medicine, and co-twin physical activity]; and specificity of adaptations to type of training. Next, physical activity/exercise is examined as primary prevention against 35 chronic conditions [Accelerated biological aging/premature death, low cardiorespiratory fitness (VO2max), sarcopenia, metabolic syndrome, obesity, insulin resistance, prediabetes, type 2 diabetes, non-alcoholic fatty liver disease, coronary heart disease, peripheral artery disease, hypertension, stroke, congestive heart failure, endothelial dysfunction, arterial dyslipidemia, hemostasis, deep vein thrombosis, cognitive dysfunction, depression and anxiety, osteoporosis, osteoarthritis, balance, bone fracture/falls, rheumatoid arthritis, colon cancer, breast cancer, endometrial cancer, gestational diabetes, preeclampsia, polycystic ovary syndrome, erectile dysfunction, pain, diverticulitis, constipation, and gallbladder diseases]. The article ends with consideration of deterioration of risk factors in longer-term sedentary groups; clinical consequences of inactive childhood/adolescence; and public policy. In summary, the body rapidly maladapts to insufficient physical activity, and if continued, results in substantial decreases in both total and quality years of life. Taken together, conclusive evidence exists that physical inactivity is one important cause of most chronic diseases. In addition, physical activity primarily prevents, or delays, chronic diseases, implying that chronic disease need not be an inevitable outcome during life. read more read less

Topics:

Polycystic ovary (57%)57% related to the paper, Disease (56%)56% related to the paper, Metabolic syndrome (55%)55% related to the paper, Physical fitness (54%)54% related to the paper, Type 2 diabetes (54%)54% related to the paper
View PDF
1,753 Citations
open accessOpen access Other DOI: 10.1002/CPHY.C130024
Energy metabolism in the liver
Liangyou Rui1
10 Jan 2014 - Comprehensive Physiology

Abstract:

The liver is an essential metabolic organ, and its metabolic function is controlled by insulin and other metabolic hormones. Glucose is converted into pyruvate through glycolysis in the cytoplasm, and pyruvate is subsequently oxidized in the mitochondria to generate ATP through the TCA cycle and oxidative phosphorylation. In ... The liver is an essential metabolic organ, and its metabolic function is controlled by insulin and other metabolic hormones. Glucose is converted into pyruvate through glycolysis in the cytoplasm, and pyruvate is subsequently oxidized in the mitochondria to generate ATP through the TCA cycle and oxidative phosphorylation. In the fed state, glycolytic products are used to synthesize fatty acids through de novo lipogenesis. Long-chain fatty acids are incorporated into triacylglycerol, phospholipids, and/or cholesterol esters in hepatocytes. These complex lipids are stored in lipid droplets and membrane structures, or secreted into the circulation as very low-density lipoprotein particles. In the fasted state, the liver secretes glucose through both glycogenolysis and gluconeogenesis. During pronged fasting, hepatic gluconeogenesis is the primary source for endogenous glucose production. Fasting also promotes lipolysis in adipose tissue, resulting in release of nonesterified fatty acids which are converted into ketone bodies in hepatic mitochondria though β-oxidation and ketogenesis. Ketone bodies provide a metabolic fuel for extrahepatic tissues. Liver energy metabolism is tightly regulated by neuronal and hormonal signals. The sympathetic system stimulates, whereas the parasympathetic system suppresses, hepatic gluconeogenesis. Insulin stimulates glycolysis and lipogenesis but suppresses gluconeogenesis, and glucagon counteracts insulin action. Numerous transcription factors and coactivators, including CREB, FOXO1, ChREBP, SREBP, PGC-1α, and CRTC2, control the expression of the enzymes which catalyze key steps of metabolic pathways, thus controlling liver energy metabolism. Aberrant energy metabolism in the liver promotes insulin resistance, diabetes, and nonalcoholic fatty liver diseases. read more read less

Topics:

Starvation response (66%)66% related to the paper, Lipogenesis (65%)65% related to the paper, Ketone bodies (64%)64% related to the paper, Ketogenesis (61%)61% related to the paper, Gluconeogenesis (61%)61% related to the paper
1,444 Citations
Other DOI: 10.1002/CPHY.CP010226
Control of Locomotion in Bipeds, Tetrapods, and Fish
Sten Grillner1
01 Dec 1981 - Comprehensive Physiology

Abstract:

The sections in this article are: 1 Biomechanical and Electromyographical Information 1.1 Single Limb During Locomotion 1.2 Interlimb Coordination 1.3 Treadmill Versus Overground Locomotion 1.4 Trunk Movements During Locomotion 1.5 Pathological Gaits 1.6 Summary 2 Neural Generation of “Ba... The sections in this article are: 1 Biomechanical and Electromyographical Information 1.1 Single Limb During Locomotion 1.2 Interlimb Coordination 1.3 Treadmill Versus Overground Locomotion 1.4 Trunk Movements During Locomotion 1.5 Pathological Gaits 1.6 Summary 2 Neural Generation of “Basic Locomotor Synergy” 2.1 Central Versus Peripheral 2.2 Parts of CNS of Primary Importance for Neural Control of Basic Locomotor Synergy 2.3 Spinal Centers for Locomotion—Behavioral Results 2.4 Reflex Control of Basic Locomotor Synergy 2.5 Activity in Certain Spinal, Cerebellar, and Brain Stem Neurons and in Certain Reflex Pathways During Locomotion 2.6 Cerebellum and Locomotion 2.7 Initiation of Locomotion—Brain Stem Circuitry 2.8 Central Organization of Spinal Pattern Generation 2.9 Possible Rhythm-Generating Mechanisms and Models—Facts and Fiction 2.10 Developmental Aspects 2.11 Summary 3 Adapting Basic Locomotor Synergy to Animal's Needs 3.1 Changing Speed 3.2 Goal-Directed Locomotion—Turning and Walking Along Curvatures 3.3 Modifications of “Locomotor Posture” 3.4 Positioning of Limb in Each Step 3.5 Reflex Adaptation of Step 3.6 Summary 4 Concluding Remarks read more read less

Topics:

Spinal locomotion (60%)60% related to the paper, Neural substrate of locomotor central pattern generators in mammals (60%)60% related to the paper, Reflex (51%)51% related to the paper
1,264 Citations
open accessOpen access Other DOI: 10.1002/CPHY.C150015
Regulation of the hypothalamic-pituitary-adrenocortical stress response
15 Mar 2016 - Comprehensive Physiology

Abstract:

The hypothalamo-pituitary-adrenocortical (HPA) axis is required for stress adaptation. Activation of the HPA axis causes secretion of glucocorticoids, which act on multiple organ systems to redirect energy resources to meet real or anticipated demand. The HPA stress response is driven primarily by neural mechanisms, invoking ... The hypothalamo-pituitary-adrenocortical (HPA) axis is required for stress adaptation. Activation of the HPA axis causes secretion of glucocorticoids, which act on multiple organ systems to redirect energy resources to meet real or anticipated demand. The HPA stress response is driven primarily by neural mechanisms, invoking corticotrophin releasing hormone (CRH) release from hypothalamic paraventricular nucleus (PVN) neurons. Pathways activating CRH release are stressor dependent: reactive responses to homeostatic disruption frequently involve direct noradrenergic or peptidergic drive of PVN neurons by sensory relays, whereas anticipatory responses use oligosynaptic pathways originating in upstream limbic structures. Anticipatory responses are driven largely by disinhibition, mediated by trans-synaptic silencing of tonic PVN inhibition via GABAergic neurons in the amygdala. Stress responses are inhibited by negative feedback mechanisms, whereby glucocorticoids act to diminish drive (brainstem) and promote transsynaptic inhibition by limbic structures (e.g., hippocampus). Glucocorticoids also act at the PVN to rapidly inhibit CRH neuronal activity via membrane glucocorticoid receptors. Chronic stress-induced activation of the HPA axis takes many forms (chronic basal hypersecretion, sensitized stress responses, and even adrenal exhaustion), with manifestation dependent upon factors such as stressor chronicity, intensity, frequency, and modality. Neural mechanisms driving chronic stress responses can be distinct from those controlling acute reactions, including recruitment of novel limbic, hypothalamic, and brainstem circuits. Importantly, an individual's response to acute or chronic stress is determined by numerous factors, including genetics, early life experience, environmental conditions, sex, and age. The context in which stressors occur will determine whether an individual's acute or chronic stress responses are adaptive or maladaptive (pathological). read more read less

Topics:

Chronic stress (55%)55% related to the paper, Amygdala (51%)51% related to the paper
1,009 Citations
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SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

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With SciSpace, you do not need a word template for Comprehensive Physiology.

It automatically formats your research paper to American Physiological Society formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

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Time taken to format a paper and Compliance with guidelines

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Comprehensive Physiology format uses unsrt citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Comprehensive Physiology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Comprehensive Physiology guidelines and auto format it.

2. Do you follow the Comprehensive Physiology guidelines?

Yes, the template is compliant with the Comprehensive Physiology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Comprehensive Physiology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Comprehensive Physiology citation style.

4. Can I use the Comprehensive Physiology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Comprehensive Physiology.

5. Can I use a manuscript in Comprehensive Physiology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Comprehensive Physiology that you can download at the end.

6. How long does it usually take you to format my papers in Comprehensive Physiology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Comprehensive Physiology.

7. Where can I find the template for the Comprehensive Physiology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Comprehensive Physiology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Comprehensive Physiology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Comprehensive Physiology an online tool or is there a desktop version?

SciSpace's Comprehensive Physiology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Comprehensive Physiology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Comprehensive Physiology?”

11. What is the output that I would get after using Comprehensive Physiology?

After writing your paper autoformatting in Comprehensive Physiology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Comprehensive Physiology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Comprehensive Physiology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Comprehensive Physiology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Comprehensive Physiology?

The 5 most common citation types in order of usage for Comprehensive Physiology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Comprehensive Physiology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Comprehensive Physiology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Comprehensive Physiology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Comprehensive Physiology Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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