Example of Physiological Reports format
Recent searches

Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
Look Inside
Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format Example of Physiological Reports format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Physiological Reports — Template for authors

Categories Rank Trend in last 3 yrs
Physiology (medical) #52 of 98 up up by 3 ranks
Physiology #95 of 169 up up by 20 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 1455 Published Papers | 5701 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 06/06/2020
Related journals
Insights
General info
Top papers
Popular templates
Get started guide
Why choose from SciSpace
FAQ

Related Journals

open access Open Access

Taylor and Francis

Quality:  
High
CiteRatio: 6.2
SJR: 0.981
SNIP: 1.447
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 17.2
SJR: 1.596
SNIP: 1.811
open access Open Access

Springer

Quality:  
High
CiteRatio: 9.6
SJR: 0.752
SNIP: 1.75
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 3.7
SJR: 0.724
SNIP: 1.069

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.9

8% from 2019

CiteRatio for Physiological Reports from 2016 - 2020
Year Value
2020 3.9
2019 3.6
2018 3.5
2017 3.0
2016 2.4
graph view Graph view
table view Table view

0.918

1% from 2019

SJR for Physiological Reports from 2016 - 2020
Year Value
2020 0.918
2019 0.909
2018 0.963
2017 0.948
2016 0.976
graph view Graph view
table view Table view

0.779

0% from 2019

SNIP for Physiological Reports from 2016 - 2020
Year Value
2020 0.779
2019 0.776
2018 0.818
2017 0.705
2016 0.674
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 8% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 1% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 0% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Physiological Reports

Guideline source: View

All company, product and service names used in this website are for identification purposes only. All product names, trademarks and registered trademarks are property of their respective owners.

Use of these names, trademarks and brands does not imply endorsement or affiliation. Disclaimer Notice

American Physiological Society

Physiological Reports

Physiological Reports is an online only, open access journal that will publish peer reviewed research across all areas of basic, translational and clinical physiology and allied disciplines. Physiological Reports is a collaboration between The Physiological Society and the Ame...... Read More

Molecular medicine

i
Last updated on
06 Jun 2020
i
ISSN
2051-817X
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
(25)
i
Bibliography Example
C. W. J. Beenakker. Specular andreev reflection in graphene. Phys. Rev. Lett., 97(6):067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.14814/PHY2.12147
Salmonella‐infected crypt‐derived intestinal organoid culture system for host–bacterial interactions
Yong Guo Zhang1, Shaoping Wu1, Yinglin Xia2, Jun Sun1, Jun Sun3
01 Sep 2014 - Physiological Reports

Abstract:

The in vitro analysis of bacterial–epithelial interactions in the intestine has been hampered by a lack of suitable intestinal epithelium culture systems. Here, we report a new experimental model using an organoid culture system to study pathophysiology of bacterial–epithelial interactions post Salmonella infection. Using cry... The in vitro analysis of bacterial–epithelial interactions in the intestine has been hampered by a lack of suitable intestinal epithelium culture systems. Here, we report a new experimental model using an organoid culture system to study pathophysiology of bacterial–epithelial interactions post Salmonella infection. Using crypt-derived mouse intestinal organoids, we were able to visualize the invasiveness of Salmonella and the morphologic changes of the organoids. Importantly, we reported bacteria-induced disruption of epithelial tight junctions in the infected organoids. In addition, we showed the inflammatory responses through activation of the NF-jB pathway in the organoids. Moreover, our western blot, PCR, and immunofluorescence data demonstrated that stem cell markers (Lgr5 and Bmi1) were significantly decreased by Salmonella infection (determined using GFP-labeled Lgr5 organoids). For the first time, we created a model system that recapitulated a number of observations from in vivo studies of the Salmonella-infected intestine, including bacterial invasion, altered tight junctions, inflammatory responses, and decreased stem cells. We have demonstrated that the Salmonella-infected organoid culture system is a new experimental model suitable for studying host–bacterial interactions. read more read less

Topics:

Organoid (63%)63% related to the paper, Intestinal epithelium (53%)53% related to the paper, Tight junction (50%)50% related to the paper
View PDF
182 Citations
open accessOpen access Journal Article DOI: 10.14814/PHY2.12327
Strengthening of the intestinal epithelial tight junction by Bifidobacterium bifidum
Chen Yu Hsieh1, Toshifumi Osaka1, Eri Moriyama1, Yasuhiro Date2, Jun Kikuchi3, Jun Kikuchi2, Satoshi Tsuneda1
01 Mar 2015 - Physiological Reports

Abstract:

Epithelial barrier dysfunction has been implicated as one of the major contributors to the pathogenesis of inflammatory bowel disease. The increase in intestinal permeability allows the translocation of luminal antigens across the intestinal epithelium, leading to the exacerbation of colitis. Thus, therapies targeted at speci... Epithelial barrier dysfunction has been implicated as one of the major contributors to the pathogenesis of inflammatory bowel disease. The increase in intestinal permeability allows the translocation of luminal antigens across the intestinal epithelium, leading to the exacerbation of colitis. Thus, therapies targeted at specifically restoring tight junction barrier function are thought to have great potential as an alternative or supplement to immunology-based therapies. In this study, we screened Bifidobacterium, Enterococcus, and Lactobacillus species for beneficial microbes to strengthen the intestinal epithelial barrier, using the human intestinal epithelial cell line (Caco-2) in an in vitro assay. Some Bifidobacterium and Lactobacillus species prevented epithelial barrier disruption induced by TNF-α, as assessed by measuring the transepithelial electrical resistance (TER). Furthermore, live Bifidobacterium species promoted wound repair in Caco-2 cell monolayers treated with TNF-α for 48 h. Time course 1H-NMR-based metabonomics of the culture supernatant revealed markedly enhanced production of acetate after 12 hours of coincubation of B. bifidum and Caco-2. An increase in TER was observed by the administration of acetate to TNF-α-treated Caco-2 monolayers. Interestingly, acetate-induced TER-enhancing effect in the coculture of B. bifidum and Caco-2 cells depends on the differentiation stage of the intestinal epithelial cells. These results suggest that Bifidobacterium species enhance intestinal epithelial barrier function via metabolites such as acetate. read more read less

Topics:

Bifidobacterium bifidum (58%)58% related to the paper, Intestinal epithelium (56%)56% related to the paper, Intestinal permeability (55%)55% related to the paper, Barrier function (55%)55% related to the paper, Bifidobacterium (54%)54% related to the paper
View PDF
172 Citations
open accessOpen access Journal Article DOI: 10.14814/PHY2.12356
Leaky intestine and impaired microbiome in an amyotrophic lateral sclerosis mouse model
Shaoping Wu1, Jianxun Yi2, Yong Guo Zhang1, Jingsong Zhou2, Jun Sun1
01 Apr 2015 - Physiological Reports

Abstract:

Emerging evidence has demonstrated that intestinal homeostasis and the microbiome play essential roles in neurological diseases, such as Parkinson's disease. Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease characterized by a progressive loss of motor neurons and muscle atrophy. Currently, there is no ... Emerging evidence has demonstrated that intestinal homeostasis and the microbiome play essential roles in neurological diseases, such as Parkinson's disease. Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease characterized by a progressive loss of motor neurons and muscle atrophy. Currently, there is no effective treatment. Most patients die within 3–5 years due to respiratory paralysis. Although the death of motor neurons is a hallmark of ALS, other organs may also contribute to the disease progression. We examined the gut of an ALS mouse model, G93A, which expresses mutant superoxide dismutase (SOD1G93A), and discovered a damaged tight junction structure and increased permeability with a significant reduction in the expression levels of tight junction protein ZO-1 and the adherens junction protein E-cadherin. Furthermore, our data demonstrated increased numbers of abnormal Paneth cells in the intestine of G93A mice. Paneth cells are specialized intestinal epithelial cells that can sense microbes and secrete antimicrobial peptides, thus playing key roles in host innate immune responses and shaping the gut microbiome. A decreased level of the antimicrobial peptides defensin 5 alpha was indeed found in the ALS intestine. These changes were associated with a shifted profile of the intestinal microbiome, including reduced levels of Butyrivibrio Fibrisolvens, Escherichia coli, and Fermicus, in G93A mice. The relative abundance of bacteria was shifted in G93A mice compared to wild-type mice. Principal coordinate analysis indicated a difference in fecal microbial communities between ALS and wild-type mice. Taken together, our study suggests a potential novel role of the intestinal epithelium and microbiome in the progression of ALS. read more read less

Topics:

Microbiome (58%)58% related to the paper, Intestinal permeability (57%)57% related to the paper, Intestinal epithelium (54%)54% related to the paper, Amyotrophic lateral sclerosis (52%)52% related to the paper, Adherens junction (52%)52% related to the paper
View PDF
169 Citations
open accessOpen access Journal Article DOI: 10.14814/PHY2.12780
Mitochondrial dysfunction and oxidative stress in patients with chronic kidney disease
01 May 2016 - Physiological Reports

Abstract:

Mitochondria abnormalities in skeletal muscle may contribute to frailty and sarcopenia, commonly present in patients with chronic kidney disease (CKD). Dysfunctional mitochondria are also a major source of oxidative stress and may contribute to cardiovascular disease in CKD We tested the hypothesis that mitochondrial structur... Mitochondria abnormalities in skeletal muscle may contribute to frailty and sarcopenia, commonly present in patients with chronic kidney disease (CKD). Dysfunctional mitochondria are also a major source of oxidative stress and may contribute to cardiovascular disease in CKD We tested the hypothesis that mitochondrial structure and function worsens with the severity of CKD Mitochondrial volume density, mitochondrial DNA (mtDNA) copy number, BNIP3, and PGC1α protein expression were evaluated in skeletal muscle biopsies obtained from 27 subjects (17 controls and 10 with CKD stage 5 on hemodialysis). We also measured mtDNA copy number in peripheral blood mononuclear cells (PBMCs), plasma isofurans, and plasma F2-isoprostanes in 208 subjects divided into three groups: non-CKD (eGFR>60 mL/min), CKD stage 3-4 (eGFR 60-15 mL/min), and CKD stage 5 (on hemodialysis). Muscle biopsies from patients with CKD stage 5 revealed lower mitochondrial volume density, lower mtDNA copy number, and higher BNIP3 content than controls. mtDNA copy number in PBMCs was decreased with increasing severity of CKD: non-CKD (6.48, 95% CI 4.49-8.46), CKD stage 3-4 (3.30, 95% CI 0.85-5.75, P = 0.048 vs. non-CKD), and CKD stage 5 (1.93, 95% CI 0.27-3.59, P = 0.001 vs. non-CKD). Isofurans were higher in patients with CKD stage 5 (median 59.21 pg/mL, IQR 41.76-95.36) compared to patients with non-CKD (median 49.95 pg/mL, IQR 27.88-83.46, P = 0.001), whereas F2-isoprostanes did not differ among groups. Severity of CKD is associated with mitochondrial dysfunction and markers of oxidative stress. Mitochondrial abnormalities, which are common in skeletal muscle from patients with CKD stage 5, may explain the muscle dysfunction associated with frailty and sarcopenia in CKD Further studies are required to evaluate mitochondrial function in vivo in patients with different CKD stages. read more read less

Topics:

Kidney disease (55%)55% related to the paper, Sarcopenia (52%)52% related to the paper
View PDF
162 Citations
open accessOpen access Journal Article DOI: 10.14814/PHY2.14098
The maximal metabolic steady state: redefining the 'gold standard'.
Andrew M. Jones1, Mark Burnley2, Matthew I. Black1, David C. Poole3, Anni Vanhatalo1
01 May 2019 - Physiological Reports

Abstract:

© 2019 The Authors. Published by Wiley. This is an open access article available under a Creative Commons licence. The published version can be accessed at the following link on the publisher’s website: https://doi.org/10.14814/phy2.14098 © 2019 The Authors. Published by Wiley. This is an open access article available under a Creative Commons licence. The published version can be accessed at the following link on the publisher’s website: https://doi.org/10.14814/phy2.14098 read more read less
154 Citations
Author Pic

SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

Get MS-Word and LaTeX output to any Journal within seconds
1
Choose a template
Select a template from a library of 40,000+ templates
2
Import a MS-Word file or start fresh
It takes only few seconds to import
3
View and edit your final output
SciSpace will automatically format your output to meet journal guidelines
4
Submit directly or Download
Submit to journal directly or Download in PDF, MS Word or LaTeX

(Before submission check for plagiarism via Turnitin)

clock Less than 3 minutes

What to expect from SciSpace?

Speed and accuracy over MS Word

''

With SciSpace, you do not need a word template for Physiological Reports.

It automatically formats your research paper to American Physiological Society formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

Time comparison

Time taken to format a paper and Compliance with guidelines

Plagiarism Reports via Turnitin

SciSpace has partnered with Turnitin, the leading provider of Plagiarism Check software.

Using this service, researchers can compare submissions against more than 170 million scholarly articles, a database of 70+ billion current and archived web pages. How Turnitin Integration works?

Turnitin Stats
Publisher Logos

Freedom from formatting guidelines

One editor, 100K journal formats – world's largest collection of journal templates

With such a huge verified library, what you need is already there.

publisher-logos

Easy support from all your favorite tools

Physiological Reports format uses unsrt citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Physiological Reports in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Physiological Reports guidelines and auto format it.

2. Do you follow the Physiological Reports guidelines?

Yes, the template is compliant with the Physiological Reports guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Physiological Reports?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Physiological Reports citation style.

4. Can I use the Physiological Reports templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Physiological Reports.

5. Can I use a manuscript in Physiological Reports that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Physiological Reports that you can download at the end.

6. How long does it usually take you to format my papers in Physiological Reports?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Physiological Reports.

7. Where can I find the template for the Physiological Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Physiological Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Physiological Reports's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Physiological Reports an online tool or is there a desktop version?

SciSpace's Physiological Reports is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Physiological Reports?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Physiological Reports?”

11. What is the output that I would get after using Physiological Reports?

After writing your paper autoformatting in Physiological Reports, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Physiological Reports's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Physiological Reports?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Physiological Reports. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Physiological Reports?

The 5 most common citation types in order of usage for Physiological Reports are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Physiological Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Physiological Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Physiological Reports in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Physiological Reports Endnote style according to Elsevier guidelines.

Fast and reliable,
built for complaince.

Instant formatting to 100% publisher guidelines on - SciSpace.

Available only on desktops 🖥

No word template required

Typset automatically formats your research paper to Physiological Reports formatting guidelines and citation style.

Verifed journal formats

One editor, 100K journal formats.
With the largest collection of verified journal formats, what you need is already there.

Trusted by academicians

I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

Andreas Frutiger
Researcher & Ex MS Word user
Use this template