Example of Infection and Immunity format
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Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format
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Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format Example of Infection and Immunity format
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open access Open Access

Infection and Immunity — Template for authors

Categories Rank Trend in last 3 yrs
Parasitology #10 of 65 down down by 3 ranks
Infectious Diseases #73 of 288 down down by 40 ranks
Microbiology #50 of 150 down down by 28 ranks
Immunology #88 of 202 down down by 41 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 1170 Published Papers | 6780 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 27/06/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.201

1% from 2018

Impact factor for Infection and Immunity from 2016 - 2019
Year Value
2019 3.201
2018 3.16
2017 3.256
2016 3.593
graph view Graph view
table view Table view

5.8

12% from 2019

CiteRatio for Infection and Immunity from 2016 - 2020
Year Value
2020 5.8
2019 5.2
2018 6.2
2017 7.2
2016 6.9
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 1% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 12% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.508

5% from 2019

SJR for Infection and Immunity from 2016 - 2020
Year Value
2020 1.508
2019 1.581
2018 1.591
2017 1.954
2016 2.04
graph view Graph view
table view Table view

0.968

7% from 2019

SNIP for Infection and Immunity from 2016 - 2020
Year Value
2020 0.968
2019 0.908
2018 0.91
2017 0.958
2016 0.929
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 5% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 7% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Infection and Immunity

Guideline source: View

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Use of these names, trademarks and brands does not imply endorsement or affiliation. Disclaimer Notice

American Society for Microbiology

Infection and Immunity

The scope of Infection and Immunity includes: Molecular Pathogenesis,Cellular Microbiology: Pathogen-Host Cell Molecular Interactions, Bacterial Infections, Host Response and Inflammation, Fungal and Parasitic Infections, Microbial Immunity and Vaccines, and Molecular Genomics.... Read More

Parasitology

Infectious Diseases

Microbiology

Immunology

Immunology and Microbiology

i
Last updated on
27 Jun 2020
i
ISSN
0019-9567
i
Impact Factor
High - 1.162
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
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Bibliography Name
unsrt asm custom citation
i
Citation Type
Numbered
(25)
i
Bibliography Example
Blonder, G. E., Tinkham, M., and Klapwijk, T. M. 1982. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B, 25(7):4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1128/IAI.73.4.1907-1916.2005
Apoptosis, pyroptosis, and necrosis: mechanistic description of dead and dying eukaryotic cells
Susan L. Fink1, Brad T. Cookson1
01 Apr 2005 - Infection and Immunity

Abstract:

A wide variety of pathogenic microorganisms have been demonstrated to cause eukaryotic cell death, either as a consequence of infecting host cells or by producing toxic products. Pathogen-induced host cell death has been characterized as apoptosis in many of these systems. It is increasingly being A wide variety of pathogenic microorganisms have been demonstrated to cause eukaryotic cell death, either as a consequence of infecting host cells or by producing toxic products. Pathogen-induced host cell death has been characterized as apoptosis in many of these systems. It is increasingly being read more read less

Topics:

Pyroptosis (60%)60% related to the paper
1,911 Citations
open accessOpen access Journal Article DOI: 10.1128/IAI.66.11.5224-5231.1998
Resident enteric bacteria are necessary for development of spontaneous colitis and immune system activation in interleukin-10-deficient mice.
01 Nov 1998 - Infection and Immunity

Abstract:

Mice with targeted deletion of the gene for interleukin-10 (IL-10) spontaneously develop enterocolitis when maintained in conventional conditions but develop only colitis when kept in specific-pathogen-free (SPF) environments. This study tested the hypothesis that enteric bacteria are necessary for the development of spontane... Mice with targeted deletion of the gene for interleukin-10 (IL-10) spontaneously develop enterocolitis when maintained in conventional conditions but develop only colitis when kept in specific-pathogen-free (SPF) environments. This study tested the hypothesis that enteric bacteria are necessary for the development of spontaneous colitis and immune system activation in IL-10-deficient mice. IL-10-deficient mice were maintained in either SPF conditions or germfree conditions or were populated with bacteria known to cause colitis in other rodent models. IL-10-deficient mice kept in SPF conditions developed colitis in all segments of the colon (cecum and proximal and distal colon). These mice exhibited immune system activation as evidenced by increased expression of CD44 on CD4+ T cells; increased mesenteric lymph node cell numbers; and increased production of immunoglobulin A (IgA), IgG1, and IL-12 p40 from colon fragment cultures. Mice populated with bacterial strains, including Bacteroides vulgatus, known to induce colitis in other rodent models had minimal colitis. Germfree IL-10-deficient mice had no evidence of colitis or immune system activation. We conclude therefore that resident enteric bacteria are necessary for the development of spontaneous colitis and immune system activation in IL-10-deficient mice. read more read less

Topics:

Colitis (62%)62% related to the paper, Enterocolitis (54%)54% related to the paper, Immune system (53%)53% related to the paper, Cecum (52%)52% related to the paper
1,462 Citations
open accessOpen access Journal Article DOI: 10.1128/IAI.37.1.318-326.1982
Adherence of slime-producing strains of Staphylococcus epidermidis to smooth surfaces.
01 Jul 1982 - Infection and Immunity

Abstract:

Slime production is not a generally recognized feature of Staphylococcus epidermidis. In a recent outbreak of S. epidermidis intravascular catheter-associated sepsis, we noted that 63% of clinically implicated strains grew as a slimy film coating the culture tube walls when propagated in tryptic soy broth. Only 37% of randoml... Slime production is not a generally recognized feature of Staphylococcus epidermidis. In a recent outbreak of S. epidermidis intravascular catheter-associated sepsis, we noted that 63% of clinically implicated strains grew as a slimy film coating the culture tube walls when propagated in tryptic soy broth. Only 37% of randomly collected blood culture contaminants and skin isolates demonstrated a similar phenomenon (p less than 0.05). Transmission electron micrographs of these coating bacteria showed them to be encased in an extracellular matrix that stained with alcian blue. Slime production was most evident in autoclaved media containing Casamino Acids and glucose supplementation (0.25% wt/vol). There were strain and media preparation variability of slime production in the presence of other carbohydrates. Some strains were not able to produce slime under any of the tested conditions. The production or nonproduction of slime did not influence growth rate. When grown in vitro, slime producers accumulated on the surface of intravascular catheters as macrocolonies, whereas non-slime, producers did not. Transmission and scanning electron micrographs showed slime producers to be encased in an adhesive layer on the catheter surface, whereas nonproducers were not encased. These results suggest that slime-mediated adherence may be a critical factor in the pathogenesis of S. epidermidis infections of medical devices. read more read less

Topics:

Staphylococcus epidermidis (56%)56% related to the paper, Tryptic soy broth (51%)51% related to the paper
1,330 Citations
open accessOpen access Journal Article DOI: 10.1128/IAI.68.12.7010-7017.2000
Differential Alteration in Intestinal Epithelial Cell Expression of Toll-Like Receptor 3 (TLR3) and TLR4 in Inflammatory Bowel Disease
Elke Cario1, Daniel K. Podolsky1
01 Dec 2000 - Infection and Immunity

Abstract:

Initiation and perpetuation of the inflammatory intestinal responses in inflammatory bowel disease (IBD) may result from an exaggerated host defense reaction of the intestinal epithelium to endogenous lumenal bacterial flora. Intestinal epithelial cell lines constitutively express several functional Toll-like receptors (TLRs)... Initiation and perpetuation of the inflammatory intestinal responses in inflammatory bowel disease (IBD) may result from an exaggerated host defense reaction of the intestinal epithelium to endogenous lumenal bacterial flora. Intestinal epithelial cell lines constitutively express several functional Toll-like receptors (TLRs) which appear to be key regulators of the innate response system. The aim of this study was to characterize the expression pattern of TLR2, TLR3, TLR4, and TLR5 in primary intestinal epithelial cells from patients with IBD. Small intestinal and colonic biopsy specimens were collected from patients with IBD (Crohn's disease [CD], ulcerative colitis [UC]) and controls. Non-IBD specimens were assessed by immunofluorescence histochemistry using polyclonal antibodies specific for TLR2, TLR3, TLR4, and TLR5. Primary intestinal epithelial cells (IEC) of normal mucosa constitutively expressed TLR3 and TLR5, while TLR2 and TLR4 were only barely detectable. In active IBD, the expression of TLR3 and TLR4 was differentially modulated in the intestinal epithelium. TLR3 was significantly downregulated in IEC in active CD but not in UC. In contrast, TLR4 was strongly upregulated in both UC and CD. TLR2 and TLR5 expression remained unchanged in IBD. These data suggest that IBD may be associated with distinctive changes in selective TLR expression in the intestinal epithelium, implying that alterations in the innate response system may contribute to the pathogenesis of these disorders. read more read less

Topics:

Intestinal mucosa (66%)66% related to the paper, Intestinal epithelium (58%)58% related to the paper, Inflammatory bowel disease (56%)56% related to the paper, Toll-like receptor (54%)54% related to the paper, TLR5 (52%)52% related to the paper
1,290 Citations
open accessOpen access Journal Article DOI: 10.1128/IAI.18.3.775-779.1977
Vero response to a cytotoxin of Escherichia coli.
J Konowalchuk, J I Speirs, S Stavric
01 Dec 1977 - Infection and Immunity

Abstract:

A cytotoxin was found in culture filtrates of a number of Escherichia coli strains that differed from the known heat-stable and heat-labile enterotoxins of E. coli. It was cytotoxic for Vero but not for Y-1 or CHO cells, and its effect on Vero was distinctly different from that of heat-labile enterotoxin. It was labile to hea... A cytotoxin was found in culture filtrates of a number of Escherichia coli strains that differed from the known heat-stable and heat-labile enterotoxins of E. coli. It was cytotoxic for Vero but not for Y-1 or CHO cells, and its effect on Vero was distinctly different from that of heat-labile enterotoxin. It was labile to heat and antigenically different from heat-labile enterotoxin, and membrane filtration indicated a molecular weight of 10,000 to 30,000. read more read less

Topics:

Heat-stable enterotoxin (60%)60% related to the paper, Enterotoxin (57%)57% related to the paper, Shiga-like toxin (55%)55% related to the paper, Escherichia coli (53%)53% related to the paper
1,149 Citations
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Frequently asked questions

1. Can I write Infection and Immunity in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Infection and Immunity guidelines and auto format it.

2. Do you follow the Infection and Immunity guidelines?

Yes, the template is compliant with the Infection and Immunity guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Infection and Immunity?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Infection and Immunity citation style.

4. Can I use the Infection and Immunity templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Infection and Immunity.

5. Can I use a manuscript in Infection and Immunity that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Infection and Immunity that you can download at the end.

6. How long does it usually take you to format my papers in Infection and Immunity?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Infection and Immunity.

7. Where can I find the template for the Infection and Immunity?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Infection and Immunity's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Infection and Immunity's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Infection and Immunity an online tool or is there a desktop version?

SciSpace's Infection and Immunity is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Infection and Immunity?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Infection and Immunity?”

11. What is the output that I would get after using Infection and Immunity?

After writing your paper autoformatting in Infection and Immunity, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Infection and Immunity's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Infection and Immunity?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Infection and Immunity. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Infection and Immunity?

The 5 most common citation types in order of usage for Infection and Immunity are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Infection and Immunity?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Infection and Immunity's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Infection and Immunity in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Infection and Immunity Endnote style according to Elsevier guidelines.

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