Example of American Journal of Respiratory Cell and Molecular Biology format
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Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format
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Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format Example of American Journal of Respiratory Cell and Molecular Biology format
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recommended Recommended

American Journal of Respiratory Cell and Molecular Biology — Template for authors

Categories Rank Trend in last 3 yrs
Pulmonary and Respiratory Medicine #10 of 133 -
Clinical Biochemistry #14 of 113 down down by 3 ranks
Molecular Biology #77 of 382 down down by 3 ranks
Cell Biology #58 of 279 down down by 3 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 524 Published Papers | 4381 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 03/06/2020
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Related Journals

open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 11.4
SJR: 2.182
SNIP: 1.902
open access Open Access

Springer

Quality:  
Good
CiteRatio: 5.5
SJR: 0.864
SNIP: 0.835
open access Open Access
recommended Recommended

Taylor and Francis

Quality:  
High
CiteRatio: 15.1
SJR: 3.934
SNIP: 2.351
open access Open Access

De Gruyter

Quality:  
High
CiteRatio: 6.5
SJR: 1.246
SNIP: 0.854

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

5.373

24% from 2018

Impact factor for American Journal of Respiratory Cell and Molecular Biology from 2016 - 2019
Year Value
2019 5.373
2018 4.34
2017 3.785
2016 4.1
graph view Graph view
table view Table view

8.4

4% from 2019

CiteRatio for American Journal of Respiratory Cell and Molecular Biology from 2016 - 2020
Year Value
2020 8.4
2019 8.1
2018 8.0
2017 8.1
2016 8.2
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 24% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 4% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.469

28% from 2019

SJR for American Journal of Respiratory Cell and Molecular Biology from 2016 - 2020
Year Value
2020 2.469
2019 1.932
2018 1.849
2017 1.519
2016 1.947
graph view Graph view
table view Table view

1.181

5% from 2019

SNIP for American Journal of Respiratory Cell and Molecular Biology from 2016 - 2020
Year Value
2020 1.181
2019 1.127
2018 1.02
2017 1.031
2016 1.05
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 28% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 5% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

American Journal of Respiratory Cell and Molecular Biology

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American Thoracic Society

American Journal of Respiratory Cell and Molecular Biology

The American Journal of Respiratory Cell and Molecular Biology publishes papers that report significant and original observations in the area of pulmonary biology. The focus of the Journal includes, but is not limited to, cellular, biochemical, molecular, developmental, geneti...... Read More

Medicine

i
Last updated on
03 Jun 2020
i
ISSN
1044-1549
i
Impact Factor
Very High - 4.1
i
Acceptance Rate
35%
i
Open Access
Not provided
i
Sherpa RoMEO Archiving Policy
Blue faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
Vancouver
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Citation Type
Numbered
(25)
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent con-version. Phys Rev B. 1982;25(7):4515–4532. Available from: 10.1103/PhysRevB.25.4515.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1165/RCMB.2009-0210ST
An Official American Thoracic Society Workshop Report: Features and Measurements of Experimental Acute Lung Injury in Animals

Abstract:

Acute lung injury (ALI) is well defined in humans, but there is no agreement as to the main features of acute lung injury in animal models. A Committee was organized to determine the main features that characterize ALI in animal models and to identify the most relevant methods to assess these features. We used a Delphi approa... Acute lung injury (ALI) is well defined in humans, but there is no agreement as to the main features of acute lung injury in animal models. A Committee was organized to determine the main features that characterize ALI in animal models and to identify the most relevant methods to assess these features. We used a Delphi approach in which a series of questionnaires were distributed to a panel of experts in experimental lung injury. The Committee concluded that the main features of experimental ALI include histological evidence of tissue injury, alteration of the alveolar capillary barrier, presence of an inflammatory response, and evidence of physiological dysfunction; they recommended that, to determine if ALI has occurred, at least three of these four main features of ALI should be present. The Committee also identified key "very relevant" and "somewhat relevant" measurements for each of the main features of ALI and recommended the use of least one "very relevant" measurement and preferably one or two additional separate measurements to determine if a main feature of ALI is present. Finally, the Committee emphasized that not all of the measurements listed can or should be performed in every study, and that measurements not included in the list are by no means "irrelevant." Our list of features and measurements of ALI is intended as a guide for investigators, and ultimately investigators should choose the particular measurements that best suit the experimental questions being addressed as well as take into consideration any unique aspects of the experimental design. read more read less

Topics:

Lung injury (60%)60% related to the paper
View PDF
1,243 Citations
Journal Article DOI: 10.1165/AJRCMB.15.1.8679227
Heme oxygenase-1: function, regulation, and implication of a novel stress-inducible protein in oxidant-induced lung injury.
Augustine M.K. Choi1, Jawed Alam

Abstract:

Accumulating evidence suggests that oxidative stress plays a central role in the pathogenesis of many pulmonary diseases including adult respiratory distress syndrome, emphysema, asthma, bronchopulmonary dysplasia, and interstitial pulmonary fibrosis. The morbidity and mortality of these diseases remain high even with optimal... Accumulating evidence suggests that oxidative stress plays a central role in the pathogenesis of many pulmonary diseases including adult respiratory distress syndrome, emphysema, asthma, bronchopulmonary dysplasia, and interstitial pulmonary fibrosis. The morbidity and mortality of these diseases remain high even with optimal medical management. In our attempts to devise new therapies for these disorders, it is crucial to improve our understanding of the basic mechanism(s) of oxidant-induced lung injury. A major line of investigation seeks to characterize the cellular and molecular responses of the lung to oxidant insults. Much progress has been made in our understanding of the role of the "classic" antioxidant enzymes (e.g., superoxide dismutase, catalase, glutathione peroxidase) in mediating the lung's resistance against oxidant lung injury. However, it is becoming clear that other oxidant-induced gene products may also play vital roles in the lung's adaptive and/or protective response to oxidative stress. One such stress-response protein is heme oxygenase-1, HO-1. Since the identification of HO-1 in 1968, many of the studies involving this enzyme were understandably focused on the regulation and function of HO-1 in heme metabolism. This emphasis is self-evident as HO-1 catalyzes the first and rate-limiting step in heme degradation. Interestingly, however, evidence accumulated over the past 25 years demonstrates that HO-1 is induced not only by the substrate heme but also by a variety of non-heme inducers such as heavy metals, endotoxin, heat shock, inflammatory cytokines, and prostaglandins. The chemical diversity of HO-1 inducers led to the speculation that HO-1, besides its role in heme degradation, may also play a vital function in maintaining cellular homeostasis. Further support for this hypothesis was provided by Tyrrell and colleagues who showed in 1989 that HO-1 is also highly induced by a variety of agents causing oxidative stress. Subsequently, many investigators have focused their attention on the function and regulation of HO-1 in various in vitro and in vivo models of oxidant-mediated cellular and tissue injury. The magnitude of HO-1 induction after oxidative stress and the wide distribution of this enzyme in systemic tissues coupled with the intriguing biological activities of the catalytic byproducts, carbon monoxide, iron, and bilirubin, makes HO-1 a highly attractive and interesting candidate stress-response protein which may play key role(s) in mediating protection against oxidant-mediated lung injury. This review will focus on the current understanding of the physiological significance of HO-1 induction and the molecular regulation of HO-1 gene expression in response to oxidative stress. We hope that this discussion will stimulate interest and investigations into a field which is still largely uncharted in the pulmonary research community. read more read less

Topics:

Lung injury (64%)64% related to the paper, Heme oxygenase (60%)60% related to the paper, Cellular homeostasis (53%)53% related to the paper, Heme (51%)51% related to the paper, Oxidative stress (51%)51% related to the paper
1,122 Citations
Journal Article DOI: 10.1165/AJRCMB.17.1.F132
The role of nuclear factor-kappa B in cytokine gene regulation.
Timothy S. Blackwell1, John W. Christman

Abstract:

Transcription factors are DNA-binding proteins that regulate gene expression. Nuclear factor-κB (NF-κB) is a critical transcription factor for maximal expression of many cytokines that are involved in the pathogenesis of inflammatory diseases, such as adult respiratory distress syndrome (ARDS) and sepsis syndrome. Activation ... Transcription factors are DNA-binding proteins that regulate gene expression. Nuclear factor-κB (NF-κB) is a critical transcription factor for maximal expression of many cytokines that are involved in the pathogenesis of inflammatory diseases, such as adult respiratory distress syndrome (ARDS) and sepsis syndrome. Activation and regulation of NF-κB are tightly controlled by a group of inhibitory proteins (IκB) that sequester NF-κB in the cytoplasm of immune/inflammatory effector cells. NF-κB activation involves signaled phosphorylation, ubiquitination, and proteolysis of IκB. Liberated NF-κB migrates to the nucleus, where it binds to specific promoter sites and activates gene transcription. The activation of NF-κB initiates both extracellular and intracellular regulatory events that result in autoregulation of the inflammatory cascade through modulation of NF-κB activation. Recently, activation of NF-κB has been linked to ARDS and has been shown to be a critical proximal step in the initiation of neutroph... read more read less

Topics:

Transcription factor (58%)58% related to the paper, Gene expression (55%)55% related to the paper, Effector (54%)54% related to the paper
958 Citations
Journal Article DOI: 10.1165/AJRCMB.10.1.7507342
CFTR expression and chloride secretion in polarized immortal human bronchial epithelial cells.

Abstract:

A major limitation in the study of vectorial ion transport, secretion, and differentiated function in the human airway epithelium has been the lack of suitable cell culture systems. Progress in this direction has been made through the transformation of primary cultured epithelial cells. However, these transformants tend to lo... A major limitation in the study of vectorial ion transport, secretion, and differentiated function in the human airway epithelium has been the lack of suitable cell culture systems. Progress in this direction has been made through the transformation of primary cultured epithelial cells. However, these transformants tend to lose differentiated properties with increasing serial passage, particularly following crisis. The suc­ cessful establishment of a postcrisis SV40 large T-antigen transformed epithelial cell line derived from human bronchial epithelium is described. This cell line, 16HBEI40-, retains differentiated epithelial mor­ phology and functions. Cell cultures show the presence of tight junctions and cilia, and monolayers gener­ ate transepithelial resistance, as measured in Ussing chambers, and retain iJ-adrenergic stimulation of cAMP-dependent chloride ion transport, measured either by ,6CI- efflux or as short-circuit current in Ussing chambers. The cells also increase chloride transport in response to bradykinin or calcium iono­ phore. In addition, 16HBE140-cells express levels of both the cystic fibrosis transmembrane conductance regulator (CFTR) mRNA and protein readily detectable by Northern and Western hybridization analysis, respectively. These cells provide a valuable resource for studying the modulation of CFTR and its role in regulation of chloride ion transport in human airway epithelium as well as other aspects of human airway cell biology. The human airway epithelium is pseudostratified, consisting of highly organized layers of polar cells with specific dif­ ferentiated functions. It includes ciliated columnar cells, basal cells, and secretory goblet cells that are linked by tight junctions. The tight junctions provide a barrier between the airway lumen and the underlying tissues and divide the epi­ thelial cells into apical and basolateral domains. Both of these plasma membrane compartments contain different populations of proteins that allow for directional flux of ions read more read less

Topics:

Respiratory epithelium (60%)60% related to the paper, Tight junction (55%)55% related to the paper, Epithelium (55%)55% related to the paper, Cystic fibrosis transmembrane conductance regulator (51%)51% related to the paper, Cell culture (51%)51% related to the paper
919 Citations
Journal Article DOI: 10.1165/AJRCMB.20.5.3494
A Polymorphism* in the 5 ′ Flanking Region of the CD14 Gene Is Associated with Circulating Soluble CD14 Levels and with Total Serum Immunoglobulin E

Abstract:

Total serum immunoglobulin (Ig)E levels are genetically regulated, but the mechanism of inheritance is not well understood. Cytokines produced by T-helper (Th)1 and Th2 lymphocytes control IgE synthesis. Bacterial antigens may favor the development of Th1 cells from naive CD4-positive T cells through a CD14-dependent pathway.... Total serum immunoglobulin (Ig)E levels are genetically regulated, but the mechanism of inheritance is not well understood. Cytokines produced by T-helper (Th)1 and Th2 lymphocytes control IgE synthesis. Bacterial antigens may favor the development of Th1 cells from naive CD4-positive T cells through a CD14-dependent pathway. CD14 is constitutively expressed on the surface of monocytes and macrophages, and is also present in serum in a soluble form (sCD14). The CD14 gene maps to chromosome 5q31.1, a candidate region for loci regulating total serum IgE. We hypothesized that genetic variants in the CD14 gene could influence Th-cell differentiation and thus total serum IgE. We identified a C-to-T transition at base pair −159 from the major transcription start site (CD14/−159). Among 481 children recruited from a general population sample, frequency of allele C was 51.4%. TT homozygotes had significantly higher sCD14 levels than did carriers of both the CC and CT genotypes (P = 0.01). TT homozygotes also had ... read more read less

Topics:

Immunoglobulin E (56%)56% related to the paper, Antibody (53%)53% related to the paper, CD14 (53%)53% related to the paper, 5' flanking region (51%)51% related to the paper, Bacterial antigen (51%)51% related to the paper
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856 Citations
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American Journal of Respiratory Cell and Molecular Biology format uses Vancouver citation style.

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Frequently asked questions

1. Can I write American Journal of Respiratory Cell and Molecular Biology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the American Journal of Respiratory Cell and Molecular Biology guidelines and auto format it.

2. Do you follow the American Journal of Respiratory Cell and Molecular Biology guidelines?

Yes, the template is compliant with the American Journal of Respiratory Cell and Molecular Biology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in American Journal of Respiratory Cell and Molecular Biology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the American Journal of Respiratory Cell and Molecular Biology citation style.

4. Can I use the American Journal of Respiratory Cell and Molecular Biology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for American Journal of Respiratory Cell and Molecular Biology.

5. Can I use a manuscript in American Journal of Respiratory Cell and Molecular Biology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper American Journal of Respiratory Cell and Molecular Biology that you can download at the end.

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It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in American Journal of Respiratory Cell and Molecular Biology.

7. Where can I find the template for the American Journal of Respiratory Cell and Molecular Biology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per American Journal of Respiratory Cell and Molecular Biology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the American Journal of Respiratory Cell and Molecular Biology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. American Journal of Respiratory Cell and Molecular Biology an online tool or is there a desktop version?

SciSpace's American Journal of Respiratory Cell and Molecular Biology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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11. What is the output that I would get after using American Journal of Respiratory Cell and Molecular Biology?

After writing your paper autoformatting in American Journal of Respiratory Cell and Molecular Biology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is American Journal of Respiratory Cell and Molecular Biology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for American Journal of Respiratory Cell and Molecular Biology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for American Journal of Respiratory Cell and Molecular Biology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In American Journal of Respiratory Cell and Molecular Biology?

The 5 most common citation types in order of usage for American Journal of Respiratory Cell and Molecular Biology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the American Journal of Respiratory Cell and Molecular Biology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per American Journal of Respiratory Cell and Molecular Biology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download American Journal of Respiratory Cell and Molecular Biology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in American Journal of Respiratory Cell and Molecular Biology Endnote style according to Elsevier guidelines.

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