Example of Clinical and Experimental Gastroenterology format
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Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format Example of Clinical and Experimental Gastroenterology format
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open access Open Access

Clinical and Experimental Gastroenterology — Template for authors

Publisher: Dove Medical Press
Categories Rank Trend in last 3 yrs
Gastroenterology #50 of 136 down down by 27 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 173 Published Papers | 760 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 23/06/2020
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Related Journals

open access Open Access

SAGE

Quality:  
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CiteRatio: 6.2
SJR: 1.667
SNIP: 1.516
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Springer

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SJR: 1.203
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CiteRatio: 6.0
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SNIP: 1.34
open access Open Access
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Springer

Quality:  
High
CiteRatio: 12.0
SJR: 2.33
SNIP: 2.277

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.4

4% from 2019

CiteRatio for Clinical and Experimental Gastroenterology from 2016 - 2020
Year Value
2020 4.4
2019 4.6
2018 4.1
2017 6.4
2016 4.8
graph view Graph view
table view Table view

1.113

22% from 2019

SJR for Clinical and Experimental Gastroenterology from 2016 - 2020
Year Value
2020 1.113
2019 0.915
2018 1.026
2017 1.261
2016 1.226
graph view Graph view
table view Table view

1.827

46% from 2019

SNIP for Clinical and Experimental Gastroenterology from 2016 - 2020
Year Value
2020 1.827
2019 1.251
2018 1.132
2017 1.179
2016 0.971
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 4% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 22% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 46% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Clinical and Experimental Gastroenterology

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Dove Medical Press

Clinical and Experimental Gastroenterology

Clinical and Experimental Gastroenterology is an international, peer reviewed, open access, online journal publishing original research, reports, editorials, reviews, and commentaries on all aspects of gastroenterology in the clinic and laboratory including the following topic...... Read More

Gastroenterology

Medicine

i
Last updated on
23 Jun 2020
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ISSN
1178-7023
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Blue faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. Specular andreev reflection in graphene. Phys. Rev. Lett., 97(6):067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.2147/CEG.S62831
Molecular pathways in non-alcoholic fatty liver disease.
Alba Berlanga1, Esther Guiu-Jurado1, José Antonio Porras1, Teresa Auguet1

Abstract:

Non-alcoholic fatty liver disease (NAFLD) is a clinicopathological change characterized by the accumulation of triglycerides in hepatocytes and has frequently been associated with obesity, type 2 diabetes mellitus, hyperlipidemia, and insulin resistance. It is an increasingly recognized condition that has become the most comm... Non-alcoholic fatty liver disease (NAFLD) is a clinicopathological change characterized by the accumulation of triglycerides in hepatocytes and has frequently been associated with obesity, type 2 diabetes mellitus, hyperlipidemia, and insulin resistance. It is an increasingly recognized condition that has become the most common liver disorder in developed countries, affecting over one-third of the population and is associated with increased cardiovascular- and liver-related mortality. NAFLD is a spectrum of disorders, beginning as simple steatosis. In about 15% of all NAFLD cases, simple steatosis can evolve into non-alcoholic steatohepatitis, a medley of inflammation, hepatocellular injury, and fibrosis, often resulting in cirrhosis and even hepatocellular cancer. However, the molecular mechanism underlying NAFLD progression is not completely understood. Its pathogenesis has often been interpreted by the "double-hit" hypothesis. The primary insult or the "first hit" includes lipid accumulation in the liver, followed by a "second hit" in which proinflammatory mediators induce inflammation, hepatocellular injury, and fibrosis. Nowadays, a more complex model suggests that fatty acids (FAs) and their metabolites may be the true lipotoxic agents that contribute to NAFLD progression; a multiple parallel hits hypothesis has also been suggested. In NAFLD patients, insulin resistance leads to hepatic steatosis via multiple mechanisms. Despite the excess hepatic accumulation of FAs in NAFLD, it has been described that not only de novo FA synthesis is increased, but FAs are also taken up from the serum. Furthermore, a decrease in mitochondrial FA oxidation and secretion of very-low-density lipoproteins has been reported. This review discusses the molecular mechanisms that underlie the pathophysiological changes of hepatic lipid metabolism that contribute to NAFLD. read more read less

Topics:

Fatty liver (62%)62% related to the paper, Steatohepatitis (61%)61% related to the paper, Liver disorder (57%)57% related to the paper, Steatosis (56%)56% related to the paper, Insulin resistance (52%)52% related to the paper
View PDF
320 Citations
open accessOpen access Journal Article DOI: 10.2147/CEG.S27530
Systematic review of randomized controlled trials of probiotics, prebiotics, and synbiotics in inflammatory bowel disease.
Yezaz A. Ghouri1, David Richards1, Erik Rahimi1, Joseph T Krill1, Katherine A Jelinek1, Andrew W. Dupont1

Abstract:

Background Probiotics are microorganisms that are ingested either in combination or as a single organism in an effort to normalize intestinal microbiota and potentially improve intestinal barrier function. Recent evidence has suggested that inflammatory bowel disease (IBD) may result from an inappropriate immunologic respons... Background Probiotics are microorganisms that are ingested either in combination or as a single organism in an effort to normalize intestinal microbiota and potentially improve intestinal barrier function. Recent evidence has suggested that inflammatory bowel disease (IBD) may result from an inappropriate immunologic response to intestinal bacteria and a disruption in the balance of the gastrointestinal microbiota in genetically susceptible individuals. Prebiotics, synbiotics, and probiotics have all been studied with growing interest as adjuncts to standard therapies for IBD. In general, probiotics have been shown to be well-tolerated with few side effects, making them a potential attractive treatment option in the management of IBD. read more read less

Topics:

Synbiotics (69%)69% related to the paper, Crohn's disease (52%)52% related to the paper, Inflammatory bowel disease (51%)51% related to the paper
View PDF
251 Citations
open accessOpen access Journal Article DOI: 10.2147/CEG.S32368
Lactose intolerance: diagnosis, genetic, and clinical factors.
Rejane Mattar1, Daniel Ferraz de Campso Mazo, Flair José Carrilho

Abstract:

Most people are born with the ability to digest lactose, the major carbohydrate in milk and the main source of nutrition until weaning. Approximately 75% of the world's population loses this ability at some point, while others can digest lactose into adulthood. This review discusses the lactase-persistence alleles that have a... Most people are born with the ability to digest lactose, the major carbohydrate in milk and the main source of nutrition until weaning. Approximately 75% of the world's population loses this ability at some point, while others can digest lactose into adulthood. This review discusses the lactase-persistence alleles that have arisen in different populations around the world, diagnosis of lactose intolerance, and its symptomatology and management. read more read less

Topics:

Lactase persistence (66%)66% related to the paper, Lactose intolerance (65%)65% related to the paper, Lactose (56%)56% related to the paper, Population (53%)53% related to the paper
View PDF
172 Citations
open accessOpen access Journal Article DOI: 10.2147/CEG.S86798
Efficacy of the low FODMAP diet for treating irritable bowel syndrome: the evidence to date

Abstract:

This review summarizes the published clinical studies concerning the management of irritable bowel syndrome (IBS) using restriction of Fermentable Oligosaccharide, Disaccharide, Monosaccharide, and Polyols in the diet (low FODMAP diet). In recent years, the data supporting low FODMAP diet for the management of IBS symptoms ha... This review summarizes the published clinical studies concerning the management of irritable bowel syndrome (IBS) using restriction of Fermentable Oligosaccharide, Disaccharide, Monosaccharide, and Polyols in the diet (low FODMAP diet). In recent years, the data supporting low FODMAP diet for the management of IBS symptoms have emerged, including several randomized controlled trials, case-control studies, and other observational studies. Unlike most dietary manipulations tried in the past to alleviate gastrointestinal symptoms of IBS, all studies on low FODMAP diet have consistently shown symptomatic benefits in the majority of patients with IBS. However, dietary adherence by the patients and clear dietary intervention led by specialized dietitians appear to be vital for the success of the diet. Up to 86% of patients with IBS find improvement in overall gastrointestinal symptoms as well as individual symptoms such as abdominal pain, bloating, constipation, diarrhea, abdominal distention, and flatulence following the diet. FODMAP restriction reduces the osmotic load and gas production in the distal small bowel and the proximal colon, providing symptomatic relief in patients with IBS. Long-term health effects of a low FODMAP diet are not known; however, stringent FODMAP restriction is not recommended owing to risks of inadequate nutrient intake and potential adverse effects from altered gut microbiota. In conclusion, the evidence to date strongly supports the efficacy of a low FODMAP diet in the treatment of IBS. Further studies are required to understand any potential adverse effects of long-term restriction of FODMAPs. read more read less

Topics:

FODMAP (59%)59% related to the paper, Irritable bowel syndrome (56%)56% related to the paper, Symptomatic relief (53%)53% related to the paper, Bloating (53%)53% related to the paper
View PDF
143 Citations
open accessOpen access Journal Article DOI: 10.2147/CEG.S51902
Fecal calprotectin in inflammatory bowel disease
Natalie E Walsham, Roy Sherwood1

Abstract:

Inflammatory bowel disease (IBD) and irritable bowel syndrome share many symptoms. While irritable bowel syndrome is a functional bowel disorder for which no specific treatment is available, the range of effective therapies for IBD is evolving rapidly. Accurate diagnosis of IBD is therefore essential. Clinical assessment, tog... Inflammatory bowel disease (IBD) and irritable bowel syndrome share many symptoms. While irritable bowel syndrome is a functional bowel disorder for which no specific treatment is available, the range of effective therapies for IBD is evolving rapidly. Accurate diagnosis of IBD is therefore essential. Clinical assessment, together with various imaging modalities and endoscopy, has been the mainstay of diagnosis for many years. Fecal biomarkers of gastrointestinal inflammation have appeared in the past decade, of which calprotectin, a neutrophil cytosolic protein, has been studied the most. Crohn's disease and ulcerative colitis are chronic remitting and relapsing diseases, and objective assessment of disease activity and response to treatment are important. This review focuses on the use of fecal calprotectin measurements in the diagnosis and monitoring of patients with IBD. read more read less

Topics:

Calprotectin (69%)69% related to the paper, Crohn's disease (64%)64% related to the paper, Inflammatory bowel disease (64%)64% related to the paper, Irritable bowel syndrome (58%)58% related to the paper, Ulcerative colitis (55%)55% related to the paper
View PDF
142 Citations
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Clinical and Experimental Gastroenterology format uses unsrt citation style.

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Frequently asked questions

1. Can I write Clinical and Experimental Gastroenterology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Clinical and Experimental Gastroenterology guidelines and auto format it.

2. Do you follow the Clinical and Experimental Gastroenterology guidelines?

Yes, the template is compliant with the Clinical and Experimental Gastroenterology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Clinical and Experimental Gastroenterology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Clinical and Experimental Gastroenterology citation style.

4. Can I use the Clinical and Experimental Gastroenterology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Clinical and Experimental Gastroenterology.

5. Can I use a manuscript in Clinical and Experimental Gastroenterology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Clinical and Experimental Gastroenterology that you can download at the end.

6. How long does it usually take you to format my papers in Clinical and Experimental Gastroenterology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Clinical and Experimental Gastroenterology.

7. Where can I find the template for the Clinical and Experimental Gastroenterology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Clinical and Experimental Gastroenterology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Clinical and Experimental Gastroenterology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Clinical and Experimental Gastroenterology an online tool or is there a desktop version?

SciSpace's Clinical and Experimental Gastroenterology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Clinical and Experimental Gastroenterology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Clinical and Experimental Gastroenterology?”

11. What is the output that I would get after using Clinical and Experimental Gastroenterology?

After writing your paper autoformatting in Clinical and Experimental Gastroenterology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Clinical and Experimental Gastroenterology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Clinical and Experimental Gastroenterology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Clinical and Experimental Gastroenterology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Clinical and Experimental Gastroenterology?

The 5 most common citation types in order of usage for Clinical and Experimental Gastroenterology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Clinical and Experimental Gastroenterology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Clinical and Experimental Gastroenterology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Clinical and Experimental Gastroenterology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Clinical and Experimental Gastroenterology Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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