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Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format
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Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format Example of Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy format
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open access Open Access

Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy — Template for authors

Publisher: Dove Medical Press
Categories Rank Trend in last 3 yrs
Internal Medicine #70 of 121 down down by 58 ranks
Pharmacology #196 of 297 down down by 147 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 856 Published Papers | 2064 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 06/06/2020
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Related Journals

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Taylor and Francis

Quality:  
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SJR: 1.864
SNIP: 1.641
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CiteRatio: 4.8
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open access Open Access

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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.4

11% from 2019

CiteRatio for Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy from 2016 - 2020
Year Value
2020 2.4
2019 2.7
2018 4.7
2017 7.1
2016 7.3
graph view Graph view
table view Table view

0.853

1% from 2019

SJR for Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy from 2016 - 2020
Year Value
2020 0.853
2019 0.862
2018 0.997
2017 1.342
2016 1.586
graph view Graph view
table view Table view

1.168

6% from 2019

SNIP for Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy from 2016 - 2020
Year Value
2020 1.168
2019 1.241
2018 1.319
2017 1.351
2016 1.481
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 11% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 1% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 6% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy

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Dove Medical Press

Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy

Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy is an international, peer-reviewed, open access, online journal. The journal is committed to the rapid publication of the latest laboratory and clinical findings in the fields of diabetes, metabolic syndrome and obe...... Read More

Medicine

i
Last updated on
06 Jun 2020
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ISSN
1178-7007
i
Impact Factor
Medium - 0.854
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Blue faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
unsrt
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in supercon- ducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys Rev B. 1982;25(7):4515_x0015_4532. Available from: 10.1103/PhysRevB.25.4515.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.2147/DMSO.S67400
Mechanism linking diabetes mellitus and obesity.
Abdullah S Al-Goblan1, Mohammed A Al-Alfi1, Muhammad Zia Khan

Abstract:

Body mass index has a strong relationship to diabetes and insulin resistance. In obese individuals, the amount of nonesterified fatty acids, glycerol, hormones, cytokines, proinflammatory markers, and other substances that are involved in the development of insulin resistance, is increased. The pathogenesis in the development... Body mass index has a strong relationship to diabetes and insulin resistance. In obese individuals, the amount of nonesterified fatty acids, glycerol, hormones, cytokines, proinflammatory markers, and other substances that are involved in the development of insulin resistance, is increased. The pathogenesis in the development of diabetes is based on the fact that the β-islet cells of the pancreas are impaired, causing a lack of control of blood glucose. The development of diabetes becomes more inevitable if the failure of β-islet cells of the pancreas is accompanied by insulin resistance. Weight gain and body mass are central to the formation and rising incidence of type 1 and type 2 diabetes. This literature review will demonstrate the facts that link obesity with insulin resistance and pancreatic β-cell dysfunction. In conclusion, new approaches in managing and preventing diabetes in obese individuals must be studied and investigated based on the facts. read more read less

Topics:

Insulin resistance (72%)72% related to the paper, Type 2 diabetes (66%)66% related to the paper, Diabetes mellitus (63%)63% related to the paper, Obesity (54%)54% related to the paper, Body mass index (51%)51% related to the paper
View PDF
630 Citations
open accessOpen access Journal Article DOI: 10.2147/DMSOTT.S7384
The economic impact of obesity in the United States
Ross A. Hammond1, Ruth Levine

Abstract:

Over the past several decades, obesity has grown into a major global epidemic. In the United States (US), more than two-thirds of adults are now overweight and one-third is obese. In this article, we provide an overview of the state of research on the likely economic impact of the US obesity epidemic at the national level. Re... Over the past several decades, obesity has grown into a major global epidemic. In the United States (US), more than two-thirds of adults are now overweight and one-third is obese. In this article, we provide an overview of the state of research on the likely economic impact of the US obesity epidemic at the national level. Research to date has identified at least four major categories of economic impact linked with the obesity epidemic: direct medical costs, productivity costs, transportation costs, and human capital costs. We review current evidence on each set of costs in turn, and identify important gaps for future research and potential trends in future economic impacts of obesity. Although more comprehensive analysis of costs is needed, substantial economic impacts of obesity are identified in all four categories by existing research. The magnitude of potential economic impact underscores the importance of the obesity epidemic as a focus for policy and a topic for future research. read more read less

Topics:

Economic impact analysis (59%)59% related to the paper, Economic cost (53%)53% related to the paper
View PDF
498 Citations
open accessOpen access Journal Article DOI: 10.2147/DMSO.S43731
AMPK activation: a therapeutic target for type 2 diabetes?
Kimberly A. Coughlan1, Rudy J. Valentine1, Neil B. Ruderman1, Asish K. Saha1

Abstract:

Type 2 diabetes (T2D) is a metabolic disease characterized by insulin resistance, β-cell dysfunction, and elevated hepatic glucose output. Over 350 million people worldwide have T2D, and the International Diabetes Federation projects that this number will increase to nearly 600 million by 2035. There is a great need for more ... Type 2 diabetes (T2D) is a metabolic disease characterized by insulin resistance, β-cell dysfunction, and elevated hepatic glucose output. Over 350 million people worldwide have T2D, and the International Diabetes Federation projects that this number will increase to nearly 600 million by 2035. There is a great need for more effective treatments for maintaining glucose homeostasis and improving insulin sensitivity. AMP-activated protein kinase (AMPK) is an evolutionarily conserved serine/threonine kinase whose activation elicits insulin-sensitizing effects, making it an ideal therapeutic target for T2D. AMPK is an energy-sensing enzyme that is activated when cellular energy levels are low, and it signals to stimulate glucose uptake in skeletal muscles, fatty acid oxidation in adipose (and other) tissues, and reduces hepatic glucose production. There is substantial evidence suggesting that AMPK is dysregulated in animals and humans with metabolic syndrome or T2D, and that AMPK activation (physiological or pharmacological) can improve insulin sensitivity and metabolic health. Numerous pharmacological agents, natural compounds, and hormones are known to activate AMPK, either directly or indirectly - some of which (for example, metformin and thiazolidinediones) are currently used to treat T2D. This paper will review the regulation of the AMPK pathway and its role in T2D, some of the known AMPK activators and their mechanisms of action, and the potential for future improvements in targeting AMPK for the treatment of T2D. read more read less

Topics:

AMPK (81%)81% related to the paper, Insulin resistance (57%)57% related to the paper, Glucose homeostasis (57%)57% related to the paper, Glucose uptake (52%)52% related to the paper, Metformin (51%)51% related to the paper
View PDF
354 Citations
open accessOpen access Journal Article DOI: 10.2147/DMSO.S216791
How Western Diet And Lifestyle Drive The Pandemic Of Obesity And Civilization Diseases.

Abstract:

Westernized populations are plagued by a plethora of chronic non-infectious degenerative diseases, termed as "civilization diseases", like obesity, diabetes, cardiovascular diseases, cancer, autoimmune diseases, Alzheimer's disease and many more, diseases which are rare or virtually absent in hunter-gatherers and other non-we... Westernized populations are plagued by a plethora of chronic non-infectious degenerative diseases, termed as "civilization diseases", like obesity, diabetes, cardiovascular diseases, cancer, autoimmune diseases, Alzheimer's disease and many more, diseases which are rare or virtually absent in hunter-gatherers and other non-westernized populations. There is a growing awareness that the cause of this amazing discrepancy lies in the profound changes in diet and lifestyle during recent human history. This paper shows that the transition from Paleolithic nutrition to Western diets, along with lack of corresponding genetic adaptations, cause significant distortions of the fine-tuned metabolism that has evolved over millions of years of human evolution in adaptation to Paleolithic diets. With the increasing spread of Western diet and lifestyle worldwide, overweight and civilization diseases are also rapidly increasing in developing countries. It is suggested that the diet-related key changes in the developmental process include an increased production of reactive oxygen species and oxidative stress, development of hyperinsulinemia and insulin resistance, low-grade inflammation and an abnormal activation of the sympathetic nervous system and the renin-angiotensin system, all of which play pivotal roles in the development of diseases of civilization. In addition, diet-related epigenetic changes and fetal programming play an important role. The suggested pathomechanism is also able to explain the well-known but not completely understood close relationship between obesity and the wide range of comorbidities, like type 2 diabetes mellitus, cardiovascular disease, etc., as diseases of the same etiopathology. Changing our lifestyle in accordance with our genetic makeup, including diet and physical activity, may help prevent or limit the development of these diseases. read more read less

Topics:

Paleolithic diet (57%)57% related to the paper, Disease (55%)55% related to the paper
View PDF
322 Citations
open accessOpen access Journal Article DOI: 10.2147/DMSO.S36368
Importance of family/social support and impact on adherence to diabetic therapy.
Tricia A. Miller1, M. Robin DiMatteo

Abstract:

Diabetes mellitus affects 24 million individuals in the US. In order to manage their diabetes successfully, patients must adhere to treatment regimens that include dietary restrictions, physical activity goals, and self-monitoring of glucose levels. Numerous factors affect patients’ ability to adhere properly, eg, self-effica... Diabetes mellitus affects 24 million individuals in the US. In order to manage their diabetes successfully, patients must adhere to treatment regimens that include dietary restrictions, physical activity goals, and self-monitoring of glucose levels. Numerous factors affect patients’ ability to adhere properly, eg, self-efficacy, treatment expectations, health beliefs, and lack of social support. Consequently, diabetes management can be quite complex, requiring lifelong commitment and drastic changes to the patient’s lifestyle. Empirical studies have shown positive and significant relationships between social support and treatment adherence among patients with diabetes. Social support from family provides patients with practical help and can buffer the stresses of living with illness. However, the exact mechanism by which social support affects patient adherence is not yet completely understood. Further research is needed to address how the differences in types of support, such as functional or emotional support, are linked to outcomes for patients. The purpose of this review is to summarize what is known of the impact of social and family support on treatment adherence in patients with diabetes and to explore the current methods and interventions used to facilitate family support for diabetic patients. read more read less

Topics:

Family support (58%)58% related to the paper, Social support (57%)57% related to the paper, Diabetes management (56%)56% related to the paper
View PDF
310 Citations
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SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy?

The 5 most common citation types in order of usage for Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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