Example of International Journal of Nephrology and Renovascular Disease format
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Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format
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Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format Example of International Journal of Nephrology and Renovascular Disease format
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open access Open Access

International Journal of Nephrology and Renovascular Disease — Template for authors

Publisher: Dove Medical Press
Categories Rank Trend in last 3 yrs
Nephrology #19 of 62 down down by 6 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 136 Published Papers | 547 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 14/07/2020
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Related Journals

open access Open Access

Springer

Quality:  
High
CiteRatio: 4.4
SJR: 0.831
SNIP: 1.249
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Elsevier

Quality:  
High
CiteRatio: 5.0
SJR: 0.92
SNIP: 1.158
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Elsevier

Quality:  
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CiteRatio: 10.7
SJR: 2.677
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open access Open Access

Elsevier

Quality:  
High
CiteRatio: 5.3
SJR: 1.256
SNIP: 1.563

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.0

5% from 2019

CiteRatio for International Journal of Nephrology and Renovascular Disease from 2016 - 2020
Year Value
2020 4.0
2019 4.2
2018 3.0
2017 4.5
2016 3.7
graph view Graph view
table view Table view

0.846

11% from 2019

SJR for International Journal of Nephrology and Renovascular Disease from 2016 - 2020
Year Value
2020 0.846
2019 0.76
2018 0.822
2017 0.903
2016 0.904
graph view Graph view
table view Table view

1.551

53% from 2019

SNIP for International Journal of Nephrology and Renovascular Disease from 2016 - 2020
Year Value
2020 1.551
2019 1.014
2018 1.126
2017 1.257
2016 1.267
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 11% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 53% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

International Journal of Nephrology and Renovascular Disease

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Dove Medical Press

International Journal of Nephrology and Renovascular Disease

International Journal of Nephrology and Renovascular Disease is an international, peer-reviewed, open-access journal focusing on the pathophysiology of the kidney and vascular supply. Epidemiology, screening, diagnosis, and treatment interventions are covered as well as basic ...... Read More

Nephrology

Medicine

i
Last updated on
14 Jul 2020
i
ISSN
1178-7058
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Blue faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. Specular andreev reflection in graphene. Phys. Rev. Lett., 97(6):067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.2147/IJNRD.S40172
Diabetic nephropathy - complications and treatment.

Abstract:

Diabetic nephropathy is a significant cause of chronic kidney disease and end-stage renal failure globally Much research has been conducted in both basic science and clinical therapeutics, which has enhanced understanding of the pathophysiology of diabetic nephropathy and expanded the potential therapies available This review... Diabetic nephropathy is a significant cause of chronic kidney disease and end-stage renal failure globally Much research has been conducted in both basic science and clinical therapeutics, which has enhanced understanding of the pathophysiology of diabetic nephropathy and expanded the potential therapies available This review will examine the current concepts of diabetic nephropathy management in the context of some of the basic science and pathophysiology aspects relevant to the approaches taken in novel, investigative treatment strategies read more read less

Topics:

Diabetic nephropathy (54%)54% related to the paper, Kidney disease (51%)51% related to the paper
View PDF
439 Citations
open accessOpen access Journal Article DOI: 10.2147/IJNRD.S39739
Obesity, hypertension, and chronic kidney disease.
Michael E. Hall1, Jussara M. do Carmo1, Alexandre A. da Silva1, Luis A. Juncos1, Zhen Wang1, John E. Hall1

Abstract:

Obesity is a major risk factor for essential hypertension, diabetes, and other comorbid conditions that contribute to development of chronic kidney disease. Obesity raises blood pressure by increasing renal tubular sodium reabsorption, impairing pressure natriuresis, and causing volume expansion via activation of the sympathe... Obesity is a major risk factor for essential hypertension, diabetes, and other comorbid conditions that contribute to development of chronic kidney disease. Obesity raises blood pressure by increasing renal tubular sodium reabsorption, impairing pressure natriuresis, and causing volume expansion via activation of the sympathetic nervous system and renin–angiotensin–aldosterone system and by physical compression of the kidneys, especially when there is increased visceral adiposity. Other factors such as inflammation, oxidative stress, and lipotoxicity may also contribute to obesity-mediated hypertension and renal dysfunction. Initially, obesity causes renal vasodilation and glomerular hyperfiltration, which act as compensatory mechanisms to maintain sodium balance despite increased tubular reabsorption. However, these compensations, along with increased arterial pressure and metabolic abnormalities, may ultimately lead to glomerular injury and initiate a slowly developing vicious cycle that exacerbates hypertension and worsens renal injury. Body weight reduction, via caloric restriction and increased physical activity, is an important first step for management of obesity, hypertension, and chronic kidney disease. However, this strategy may not be effective in producing long-term weight loss or in preventing cardiorenal and metabolic consequences in many obese patients. The majority of obese patients require medical therapy for obesity-associated hypertension, metabolic disorders, and renal disease, and morbidly obese patients may require surgical interventions to produce sustained weight loss. read more read less

Topics:

Pathophysiology of hypertension (62%)62% related to the paper, Essential hypertension (62%)62% related to the paper, Kidney disease (59%)59% related to the paper, Blood pressure (56%)56% related to the paper, Glomerular hyperfiltration (54%)54% related to the paper
View PDF
377 Citations
open accessOpen access Journal Article DOI: 10.2147/IJNRD.S46643
Vascular access for hemodialysis: current perspectives.

Abstract:

A well-functioning vascular access (VA) is a mainstay to perform an efficient hemodialysis (HD) procedure. There are three main types of access: native arteriovenous fistula (AVF), arteriovenous graft, and central venous catheter (CVC). AVF, described by Brescia and Cimino, remains the first choice for chronic HD. It is the b... A well-functioning vascular access (VA) is a mainstay to perform an efficient hemodialysis (HD) procedure. There are three main types of access: native arteriovenous fistula (AVF), arteriovenous graft, and central venous catheter (CVC). AVF, described by Brescia and Cimino, remains the first choice for chronic HD. It is the best access for longevity and has the lowest association with morbidity and mortality, and for this reason AVF use is strongly recommended by guidelines from different countries. Once autogenous options have been exhausted, prosthetic fistulae become the second option of maintenance HD access alternatives. CVCs have become an important adjunct in maintaining patients on HD. The preferable locations for insertion are the internal jugular and femoral veins. The subclavian vein is considered the third choice because of the high risk of thrombosis. Complications associated with CVC insertion range from 5% to 19%. Since an increasing number of patients have implanted pacemakers and defibrillators, usually inserted via the subclavian vein and superior vena cava into the right heart, a careful assessment of risk and benefits should be taken. Infection is responsible for the removal of about 30%-60% of HD CVCs, and hospitalization rates are higher among patients with CVCs than among AVF ones. Proper VA maintenance requires integration of different professionals to create a VA team. This team should include a nephrologist, radiologist, vascular surgeon, infectious disease consultant, and members of the dialysis staff. They should provide their experience in order to give the best options to uremic patients and the best care for their VA. read more read less

Topics:

Subclavian vein (53%)53% related to the paper, Central venous catheter (53%)53% related to the paper, Arteriovenous fistula (53%)53% related to the paper
View PDF
197 Citations
open accessOpen access Journal Article DOI: 10.2147/IJNRD.S103784
Rodent models of diabetic nephropathy: their utility and limitations.
Munehiro Kitada1, Yoshio Ogura1, Daisuke Koya1

Abstract:

Diabetic nephropathy is the most common cause of end-stage renal disease. Therefore, novel therapies for the suppression of diabetic nephropathy must be developed. Rodent models are useful for elucidating the pathogenesis of diseases and testing novel therapies, and many type 1 and type 2 diabetic rodent models have been esta... Diabetic nephropathy is the most common cause of end-stage renal disease. Therefore, novel therapies for the suppression of diabetic nephropathy must be developed. Rodent models are useful for elucidating the pathogenesis of diseases and testing novel therapies, and many type 1 and type 2 diabetic rodent models have been established for the study of diabetes and diabetic complications. Streptozotocin (STZ)-induced diabetic animals are widely used as a model of type 1 diabetes. Akita diabetic mice that have an Ins2+/C96Y mutation and OVE26 mice that overexpress calmodulin in pancreatic β-cells serve as a genetic model of type 1 diabetes. In addition, db/db mice, KK-Ay mice, Zucker diabetic fatty rats, Wistar fatty rats, Otsuka Long-Evans Tokushima Fatty rats and Goto-Kakizaki rats serve as rodent models of type 2 diabetes. An animal model of diabetic nephropathy should exhibit progressive albuminuria and a decrease in renal function, as well as the characteristic histological changes in the glomeruli and the tubulointerstitial lesions that are observed in cases of human diabetic nephropathy. A rodent model that strongly exhibits all these features of human diabetic nephropathy has not yet been developed. However, the currently available rodent models of diabetes can be useful in the study of diabetic nephropathy by increasing our understanding of the features of each diabetic rodent model. Furthermore, the genetic background and strain of each mouse model result in differences in susceptibility to diabetic nephropathy with albuminuria and the development of glomerular and tubulointerstitial lesions. Therefore, the validation of an animal model reproducing human diabetic nephropathy will significantly facilitate our understanding of the underlying genetic mechanisms that contribute to the development of diabetic nephropathy. In this review, we focus on rodent models of diabetes and discuss the utility and limitations of these models for the study of diabetic nephropathy. read more read less

Topics:

Diabetic nephropathy (61%)61% related to the paper, Diabetes mellitus (56%)56% related to the paper, Genetic model (54%)54% related to the paper, Type 1 diabetes (53%)53% related to the paper, Type 2 diabetes (52%)52% related to the paper
View PDF
178 Citations
open accessOpen access Journal Article DOI: 10.2147/IJNRD.S39747
Drug-induced impairment of renal function

Abstract:

Pharmaceutical agents provide diagnostic and therapeutic utility that are central to patient care. However, all agents also carry adverse drug effect profiles. While most of these are clinically insignificant, some drugs may cause unacceptable toxicity that impacts negatively on patient morbidity and mortality. Recognizing ad... Pharmaceutical agents provide diagnostic and therapeutic utility that are central to patient care. However, all agents also carry adverse drug effect profiles. While most of these are clinically insignificant, some drugs may cause unacceptable toxicity that impacts negatively on patient morbidity and mortality. Recognizing adverse effects is important for administering appropriate drug doses, instituting preventive strategies, and withdrawing the offending agent due to toxicity. In the present article, we will review those drugs that are associated with impaired renal function. By focusing on pharmaceutical agents that are currently in clinical practice, we will provide an overview of nephrotoxic drugs that a treating physician is most likely to encounter. In doing so, we will summarize risk factors for nephrotoxicity, describe clinical manifestations, and address preventive and treatment strategies. read more read less

Topics:

Adverse effect (53%)53% related to the paper, Drug (51%)51% related to the paper
View PDF
168 Citations
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Frequently asked questions

1. Can I write International Journal of Nephrology and Renovascular Disease in LaTeX?

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Yes, the template is compliant with the International Journal of Nephrology and Renovascular Disease guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in International Journal of Nephrology and Renovascular Disease?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the International Journal of Nephrology and Renovascular Disease citation style.

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5. Can I use a manuscript in International Journal of Nephrology and Renovascular Disease that I have written in MS Word?

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12. Is International Journal of Nephrology and Renovascular Disease's impact factor high enough that I should try publishing my article there?

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13. What is Sherpa RoMEO Archiving Policy for International Journal of Nephrology and Renovascular Disease?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for International Journal of Nephrology and Renovascular Disease. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In International Journal of Nephrology and Renovascular Disease?

The 5 most common citation types in order of usage for International Journal of Nephrology and Renovascular Disease are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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16. Can I download International Journal of Nephrology and Renovascular Disease in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in International Journal of Nephrology and Renovascular Disease Endnote style according to Elsevier guidelines.

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