Example of The Lancet Neurology format
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Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format
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Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format Example of The Lancet Neurology format
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open access Open Access
recommended Recommended

The Lancet Neurology — Template for authors

Publisher: Elsevier
Categories Rank Trend in last 3 yrs
Neurology (clinical) #1 of 343 -
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 378 Published Papers | 16942 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 19/07/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

30.039

4% from 2018

Impact factor for The Lancet Neurology from 2016 - 2019
Year Value
2019 30.039
2018 28.755
2017 27.138
2016 26.284
graph view Graph view
table view Table view

44.8

0% from 2019

CiteRatio for The Lancet Neurology from 2016 - 2020
Year Value
2020 44.8
2019 44.6
2018 44.7
2017 46.3
2016 42.0
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 4% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 0% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

12.776

13% from 2019

SJR for The Lancet Neurology from 2016 - 2020
Year Value
2020 12.776
2019 11.258
2018 12.285
2017 11.964
2016 11.788
graph view Graph view
table view Table view

12.555

22% from 2019

SNIP for The Lancet Neurology from 2016 - 2020
Year Value
2020 12.555
2019 10.251
2018 9.173
2017 9.44
2016 8.65
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 13% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 22% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
The Lancet Neurology

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Elsevier

The Lancet Neurology

The Lancet Neurology is a monthly journal, publishing original research articles, reviews, personal views, commentaries, and news in clinical neurology.... Read More

Clinical Neurology

Medicine

i
Last updated on
19 Jul 2020
i
ISSN
1474-4422
i
Impact Factor
Maximum - 8.239
i
Acceptance Rate
6%
i
Open Access
Yes
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
elsarticle-num
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Citation Type
Numbered
[25]
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Bibliography Example
G. E. Blonder, M. Tinkham, T. M. Klapwijk, Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion, Phys. Rev. B 25 (7) (1982) 4515–4532. URL 10.1103/PhysRevB.25.4515

Top papers written in this journal

Journal Article DOI: 10.1016/S1474-4422(03)00590-8
The International Classification of Headache Disorders: 2nd edition
Jes Olesen1, Peter J. Goadsby1, Timothy J. Steiner1
01 Dec 2003 - Lancet Neurology

Topics:

International Classification of Headache Disorders (83%)83% related to the paper, Post-Traumatic Headache (66%)66% related to the paper, New daily persistent headache (65%)65% related to the paper, Primary Exertional Headache (58%)58% related to the paper, Primary Cough Headache (57%)57% related to the paper
5,847 Citations
open accessOpen access Journal Article DOI: 10.1016/S1474-4422(09)70299-6
Hypothetical model of dynamic biomarkers of the Alzheimer's pathological cascade
01 Jan 2010 - Lancet Neurology

Abstract:

Summary Currently available evidence strongly supports the position that the initiating event in Alzheimer's disease (AD) is related to abnormal processing of β-amyloid (Aβ) peptide, ultimately leading to formation of Aβ plaques in the brain. This process occurs while individuals are still cognitively normal. Biomarkers of br... Summary Currently available evidence strongly supports the position that the initiating event in Alzheimer's disease (AD) is related to abnormal processing of β-amyloid (Aβ) peptide, ultimately leading to formation of Aβ plaques in the brain. This process occurs while individuals are still cognitively normal. Biomarkers of brain β-amyloidosis are reductions in CSF Aβ 42 and increased amyloid PET tracer retention. After a lag period, which varies from patient to patient, neuronal dysfunction and neurodegeneration become the dominant pathological processes. Biomarkers of neuronal injury and neurodegeneration are increased CSF tau and structural MRI measures of cerebral atrophy. Neurodegeneration is accompanied by synaptic dysfunction, which is indicated by decreased fluorodeoxyglucose uptake on PET. We propose a model that relates disease stage to AD biomarkers in which Aβ biomarkers become abnormal first, before neurodegenerative biomarkers and cognitive symptoms, and neurodegenerative biomarkers become abnormal later, and correlate with clinical symptom severity. read more read less

Topics:

Alzheimer's disease biomarkers (68%)68% related to the paper, Alzheimer's disease (56%)56% related to the paper, Senile plaques (53%)53% related to the paper, Neurodegeneration (53%)53% related to the paper, Alzheimer's Disease Neuroimaging Initiative (52%)52% related to the paper
View PDF
3,953 Citations
Journal Article DOI: 10.1016/S1474-4422(07)70178-3
Research criteria for the diagnosis of Alzheimer's disease: revising the NINCDS–ADRDA criteria
01 Aug 2007 - Lancet Neurology

Abstract:

The NINCDS-ADRDA and the DSM-IV-TR criteria for Alzheimer's disease (AD) are the prevailing diagnostic standards in research; however, they have now fallen behind the unprecedented growth of scientific knowledge. Distinctive and reliable biomarkers of AD are now available through structural MRI, molecular neuroimaging with PE... The NINCDS-ADRDA and the DSM-IV-TR criteria for Alzheimer's disease (AD) are the prevailing diagnostic standards in research; however, they have now fallen behind the unprecedented growth of scientific knowledge. Distinctive and reliable biomarkers of AD are now available through structural MRI, molecular neuroimaging with PET, and cerebrospinal fluid analyses. This progress provides the impetus for our proposal of revised diagnostic criteria for AD. Our framework was developed to capture both the earliest stages, before full-blown dementia, as well as the full spectrum of the illness. These new criteria are centred on a clinical core of early and significant episodic memory impairment. They stipulate that there must also be at least one or more abnormal biomarkers among structural neuroimaging with MRI, molecular neuroimaging with PET, and cerebrospinal fluid analysis of amyloid beta or tau proteins. The timeliness of these criteria is highlighted by the many drugs in development that are directed at changing pathogenesis, particularly at the production and clearance of amyloid beta as well as at the hyperphosphorylation state of tau. Validation studies in existing and prospective cohorts are needed to advance these criteria and optimise their sensitivity, specificity, and accuracy. read more read less

Topics:

Alzheimer's disease biomarkers (60%)60% related to the paper, Alzheimer's Disease Neuroimaging Initiative (58%)58% related to the paper, Dementia (50%)50% related to the paper
View PDF
3,951 Citations
open accessOpen access Journal Article DOI: 10.1016/S1474-4422(15)70016-5
Neuroinflammation in Alzheimer's disease
01 Apr 2015 - Lancet Neurology

Abstract:

Increasing evidence suggests that Alzheimer's disease pathogenesis is not restricted to the neuronal compartment, but includes strong interactions with immunological mechanisms in the brain. Misfolded and aggregated proteins bind to pattern recognition receptors on microglia and astroglia, and trigger an innate immune respons... Increasing evidence suggests that Alzheimer's disease pathogenesis is not restricted to the neuronal compartment, but includes strong interactions with immunological mechanisms in the brain. Misfolded and aggregated proteins bind to pattern recognition receptors on microglia and astroglia, and trigger an innate immune response characterised by release of inflammatory mediators, which contribute to disease progression and severity. Genome-wide analysis suggests that several genes that increase the risk for sporadic Alzheimer's disease encode factors that regulate glial clearance of misfolded proteins and the inflammatory reaction. External factors, including systemic inflammation and obesity, are likely to interfere with immunological processes of the brain and further promote disease progression. Modulation of risk factors and targeting of these immune mechanisms could lead to future therapeutic or preventive strategies for Alzheimer's disease. read more read less

Topics:

Neuroinflammation (56%)56% related to the paper, Pattern recognition receptor (53%)53% related to the paper, Innate immune system (51%)51% related to the paper
View PDF
3,947 Citations
open accessOpen access Journal Article DOI: 10.1016/S1474-4422(17)30470-2
Diagnosis of multiple sclerosis: 2017 revisions of the McDonald criteria
21 Dec 2017 - Lancet Neurology

Abstract:

The 2010 McDonald criteria for the diagnosis of multiple sclerosis are widely used in research and clinical practice. Scientific advances in the past 7 years suggest that they might no longer provide the most up-to-date guidance for clinicians and researchers. The International Panel on Diagnosis of Multiple Sclerosis reviewe... The 2010 McDonald criteria for the diagnosis of multiple sclerosis are widely used in research and clinical practice. Scientific advances in the past 7 years suggest that they might no longer provide the most up-to-date guidance for clinicians and researchers. The International Panel on Diagnosis of Multiple Sclerosis reviewed the 2010 McDonald criteria and recommended revisions. The 2017 McDonald criteria continue to apply primarily to patients experiencing a typical clinically isolated syndrome, define what is needed to fulfil dissemination in time and space of lesions in the CNS, and stress the need for no better explanation for the presentation. The following changes were made: in patients with a typical clinically isolated syndrome and clinical or MRI demonstration of dissemination in space, the presence of CSF-specific oligoclonal bands allows a diagnosis of multiple sclerosis; symptomatic lesions can be used to demonstrate dissemination in space or time in patients with supratentorial, infratentorial, or spinal cord syndrome; and cortical lesions can be used to demonstrate dissemination in space. Research to further refine the criteria should focus on optic nerve involvement, validation in diverse populations, and incorporation of advanced imaging, neurophysiological, and body fluid markers. read more read less

Topics:

McDonald criteria (70%)70% related to the paper, Clinically isolated syndrome (59%)59% related to the paper, Multiple sclerosis (53%)53% related to the paper
View PDF
3,945 Citations
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The Lancet Neurology format uses elsarticle-num citation style.

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Frequently asked questions

1. Can I write The Lancet Neurology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the The Lancet Neurology guidelines and auto format it.

2. Do you follow the The Lancet Neurology guidelines?

Yes, the template is compliant with the The Lancet Neurology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in The Lancet Neurology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the The Lancet Neurology citation style.

4. Can I use the The Lancet Neurology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for The Lancet Neurology.

5. Can I use a manuscript in The Lancet Neurology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper The Lancet Neurology that you can download at the end.

6. How long does it usually take you to format my papers in The Lancet Neurology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in The Lancet Neurology.

7. Where can I find the template for the The Lancet Neurology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per The Lancet Neurology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the The Lancet Neurology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. The Lancet Neurology an online tool or is there a desktop version?

SciSpace's The Lancet Neurology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like The Lancet Neurology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like The Lancet Neurology?”

11. What is the output that I would get after using The Lancet Neurology?

After writing your paper autoformatting in The Lancet Neurology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is The Lancet Neurology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for The Lancet Neurology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for The Lancet Neurology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In The Lancet Neurology?

The 5 most common citation types in order of usage for The Lancet Neurology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the The Lancet Neurology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per The Lancet Neurology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download The Lancet Neurology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in The Lancet Neurology Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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