Example of EMBO Reports format
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Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format
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Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format Example of EMBO Reports format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access
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EMBO Reports — Template for authors

Publisher: EMBO Press
Categories Rank Trend in last 3 yrs
Biochemistry #32 of 415 down down by 4 ranks
Genetics #30 of 325 down down by 6 ranks
Molecular Biology #45 of 382 down down by 7 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 739 Published Papers | 7835 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 13/07/2020
Related journals
Insights
General info
Top papers
Popular templates
Get started guide
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FAQ

Related Journals

open access Open Access

Elsevier

Quality:  
High
CiteRatio: 7.0
SJR: 1.329
SNIP: 1.627
open access Open Access

Wiley

Quality:  
High
CiteRatio: 8.5
SJR: 2.677
SNIP: 1.204
open access Open Access

Hindawi

Quality:  
Good
CiteRatio: 4.2
SJR: 0.705
SNIP: 0.721
open access Open Access

Bentham Science

Quality:  
Good
CiteRatio: 2.7
SJR: 0.306
SNIP: 0.383

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

7.497

11% from 2018

Impact factor for EMBO Reports from 2016 - 2019
Year Value
2019 7.497
2018 8.383
2017 8.749
2016 8.568
graph view Graph view
table view Table view

10.6

2% from 2019

CiteRatio for EMBO Reports from 2016 - 2020
Year Value
2020 10.6
2019 10.8
2018 11.1
2017 10.9
2016 9.8
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 11% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has decreased by 2% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.584

1% from 2019

SJR for EMBO Reports from 2016 - 2020
Year Value
2020 4.584
2019 4.557
2018 4.459
2017 4.724
2016 4.09
graph view Graph view
table view Table view

1.577

9% from 2019

SNIP for EMBO Reports from 2016 - 2020
Year Value
2020 1.577
2019 1.452
2018 1.325
2017 1.406
2016 1.18
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 1% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 9% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
EMBO Reports

Guideline source: View

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Use of these names, trademarks and brands does not imply endorsement or affiliation. Disclaimer Notice

EMBO Press

EMBO Reports

EMBO reports publishes scientific research in all areas of molecular and cell biology, as well as scientific reviews and commentary from leading experts that explore science & society.... Read More

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Last updated on
13 Jul 2020
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ISSN
1469-221X
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Impact Factor
Maximum - 7.7
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
Yellow faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
APA
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Citation Type
Numbered
[25]
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Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1038/SJ.EMBOR.7400731
The gut flora as a forgotten organ
Ann M O'Hara1, Fergus Shanahan1
01 Jul 2006 - EMBO Reports

Abstract:

The intestinal microflora is a positive health asset that crucially influences the normal structural and functional development of the mucosal immune system. Mucosal immune responses to resident intestinal microflora require precise control and an immunosensory capacity for distinguishing commensal from pathogenic bacteria. I... The intestinal microflora is a positive health asset that crucially influences the normal structural and functional development of the mucosal immune system. Mucosal immune responses to resident intestinal microflora require precise control and an immunosensory capacity for distinguishing commensal from pathogenic bacteria. In genetically susceptible individuals, some components of the flora can become a liability and contribute to the pathogenesis of various intestinal disorders, including inflammatory bowel diseases. It follows that manipulation of the flora to enhance the beneficial components represents a promising therapeutic strategy. The flora has a collective metabolic activity equal to a virtual organ within an organ, and the mechanisms underlying the conditioning influence of the bacteria on mucosal homeostasis and immune responses are beginning to be unravelled. An improved understanding of this hidden organ will reveal secrets that are relevant to human health and to several infectious, inflammatory and neoplastic disease processes. read more read less

Topics:

Gut flora (60%)60% related to the paper, Intestinal mucosa (57%)57% related to the paper, Flora (53%)53% related to the paper
View PDF
2,364 Citations
open accessOpen access Journal Article DOI: 10.1038/SJ.EMBOR.7400779
Mediators of endoplasmic reticulum stress-induced apoptosis.
Eva Szegezdi1, Susan E. Logue1, Adrienne M. Gorman1, Afshin Samali1
01 Sep 2006 - EMBO Reports

Abstract:

The efficient functioning of the endoplasmic reticulum (ER) is essential for most cellular activities and survival. Conditions that interfere with ER function lead to the accumulation and aggregation of unfolded proteins. ER transmembrane receptors detect the onset of ER stress and initiate the unfolded protein response (UPR)... The efficient functioning of the endoplasmic reticulum (ER) is essential for most cellular activities and survival. Conditions that interfere with ER function lead to the accumulation and aggregation of unfolded proteins. ER transmembrane receptors detect the onset of ER stress and initiate the unfolded protein response (UPR) to restore normal ER function. If the stress is prolonged, or the adaptive response fails, apoptotic cell death ensues. Many studies have focused on how this failure initiates apoptosis, as ER stress-induced apoptosis is implicated in the pathophysiology of several neurodegenerative and cardiovascular diseases. In this review, we examine the role of the molecules that are activated during the UPR in order to identify the molecular switch from the adaptive phase to apoptosis. We discuss how the activation of these molecules leads to the commitment of death and the mechanisms that are responsible for the final demise of the cell. read more read less

Topics:

Unfolded protein response (68%)68% related to the paper, Endoplasmic reticulum (59%)59% related to the paper, Signal transduction (53%)53% related to the paper
View PDF
2,128 Citations
open accessOpen access Journal Article DOI: 10.1038/EMBOR.2008.74
A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells
01 Jun 2008 - EMBO Reports

Abstract:

The embryonic programme 'epithelial-mesenchymal transition' (EMT) is thought to promote malignant tumour progression. The transcriptional repressor zinc-finger E-box binding homeobox 1 (ZEB1) is a crucial inducer of EMT in various human tumours, and was recently shown to promote invasion and metastasis of tumour cells. Here, ... The embryonic programme 'epithelial-mesenchymal transition' (EMT) is thought to promote malignant tumour progression. The transcriptional repressor zinc-finger E-box binding homeobox 1 (ZEB1) is a crucial inducer of EMT in various human tumours, and was recently shown to promote invasion and metastasis of tumour cells. Here, we report that ZEB1 directly suppresses transcription of microRNA-200 family members miR-141 and miR-200c, which strongly activate epithelial differentiation in pancreatic, colorectal and breast cancer cells. Notably, the EMT activators transforming growth factor beta2 and ZEB1 are the predominant targets downregulated by these microRNAs. These results indicate that ZEB1 triggers an microRNA-mediated feedforward loop that stabilizes EMT and promotes invasion of cancer cells. Alternatively, depending on the environmental trigger, this loop might switch and induce epithelial differentiation, and thus explain the strong intratumorous heterogeneity observed in many human cancers. read more read less

Topics:

Epithelial–mesenchymal transition (58%)58% related to the paper, Cancer cell (53%)53% related to the paper, Cellular differentiation (52%)52% related to the paper
View PDF
1,657 Citations
open accessOpen access Journal Article DOI: 10.15252/EMBR.201642195
The integrated stress response.
01 Oct 2016 - EMBO Reports

Abstract:

In response to diverse stress stimuli, eukaryotic cells activate a common adaptive pathway, termed the integrated stress response (ISR), to restore cellular homeostasis. The core event in this pathway is the phosphorylation of eukaryotic translation initiation factor 2 alpha (eIF2α) by one of four members of the eIF2α kinase ... In response to diverse stress stimuli, eukaryotic cells activate a common adaptive pathway, termed the integrated stress response (ISR), to restore cellular homeostasis. The core event in this pathway is the phosphorylation of eukaryotic translation initiation factor 2 alpha (eIF2α) by one of four members of the eIF2α kinase family, which leads to a decrease in global protein synthesis and the induction of selected genes, including the transcription factor ATF4, that together promote cellular recovery. The gene expression program activated by the ISR optimizes the cellular response to stress and is dependent on the cellular context, as well as on the nature and intensity of the stress stimuli. Although the ISR is primarily a pro‐survival, homeostatic program, exposure to severe stress can drive signaling toward cell death. Here, we review current understanding of the ISR signaling and how it regulates cell fate under diverse types of stress. read more read less

Topics:

Integrated stress response (74%)74% related to the paper, Unfolded protein response (57%)57% related to the paper, Cellular homeostasis (57%)57% related to the paper, EIF4A1 (53%)53% related to the paper, Transcription factor (51%)51% related to the paper
View PDF
1,480 Citations
open accessOpen access Journal Article DOI: 10.15252/EMBR.201439246
The extracellular matrix modulates the hallmarks of cancer
Michael W. Pickup1, Janna K. Mouw1, Valerie M. Weaver
01 Dec 2014 - EMBO Reports

Abstract:

The extracellular matrix regulates tissue development and homeostasis, and its dysregulation contributes to neoplastic progression. The extracellular matrix serves not only as the scaffold upon which tissues are organized but provides critical biochemical and biomechanical cues that direct cell growth, survival, migration and... The extracellular matrix regulates tissue development and homeostasis, and its dysregulation contributes to neoplastic progression. The extracellular matrix serves not only as the scaffold upon which tissues are organized but provides critical biochemical and biomechanical cues that direct cell growth, survival, migration and differentiation and modulate vascular development and immune function. Thus, while genetic modifications in tumor cells undoubtedly initiate and drive malignancy, cancer progresses within a dynamically evolving extracellular matrix that modulates virtually every behavioral facet of the tumor cells and cancer-associated stromal cells. Hanahan and Weinberg defined the hallmarks of cancer to encompass key biological capabilities that are acquired and essential for the development, growth and dissemination of all human cancers. These capabilities include sustained proliferation, evasion of growth suppression, death resistance, replicative immortality, induced angiogenesis, initiation of invasion, dysregulation of cellular energetics, avoidance of immune destruction and chronic inflammation. Here, we argue that biophysical and biochemical cues from the tumor-associated extracellular matrix influence each of these cancer hallmarks and are therefore critical for malignancy. We suggest that the success of cancer prevention and therapy programs requires an intimate understanding of the reciprocal feedback between the evolving extracellular matrix, the tumor cells and its cancer-associated cellular stroma. read more read less

Topics:

The Hallmarks of Cancer (55%)55% related to the paper, Extracellular matrix (54%)54% related to the paper, Angiogenesis (52%)52% related to the paper, Stromal cell (51%)51% related to the paper
View PDF
1,301 Citations
Author Pic

SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

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With SciSpace, you do not need a word template for EMBO Reports.

It automatically formats your research paper to EMBO Press formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

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Time taken to format a paper and Compliance with guidelines

Plagiarism Reports via Turnitin

SciSpace has partnered with Turnitin, the leading provider of Plagiarism Check software.

Using this service, researchers can compare submissions against more than 170 million scholarly articles, a database of 70+ billion current and archived web pages. How Turnitin Integration works?

Turnitin Stats
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Easy support from all your favorite tools

EMBO Reports format uses APA citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write EMBO Reports in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the EMBO Reports guidelines and auto format it.

2. Do you follow the EMBO Reports guidelines?

Yes, the template is compliant with the EMBO Reports guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in EMBO Reports?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the EMBO Reports citation style.

4. Can I use the EMBO Reports templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for EMBO Reports.

5. Can I use a manuscript in EMBO Reports that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper EMBO Reports that you can download at the end.

6. How long does it usually take you to format my papers in EMBO Reports?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in EMBO Reports.

7. Where can I find the template for the EMBO Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per EMBO Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the EMBO Reports's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. EMBO Reports an online tool or is there a desktop version?

SciSpace's EMBO Reports is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like EMBO Reports?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like EMBO Reports?”

11. What is the output that I would get after using EMBO Reports?

After writing your paper autoformatting in EMBO Reports, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is EMBO Reports's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for EMBO Reports?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for EMBO Reports. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In EMBO Reports?

The 5 most common citation types in order of usage for EMBO Reports are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the EMBO Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per EMBO Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download EMBO Reports in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in EMBO Reports Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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