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Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format Example of Frontiers in Aging Neuroscience format
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open access Open Access

Frontiers in Aging Neuroscience — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Cognitive Neuroscience #15 of 96 up up by 5 ranks
Aging #10 of 35 down down by 1 rank
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 1581 Published Papers | 11659 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 15/06/2020
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Related Journals

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Quality:  
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SJR: 1.395
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CiteRatio: 6.5
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open access Open Access

Frontiers Media

Quality:  
High
CiteRatio: 5.7
SJR: 2.036
SNIP: 1.066
open access Open Access

Springer

Quality:  
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CiteRatio: 5.1
SJR: 0.911
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.362

20% from 2018

Impact factor for Frontiers in Aging Neuroscience from 2016 - 2019
Year Value
2019 4.362
2018 3.633
2017 3.582
2016 4.504
graph view Graph view
table view Table view

7.4

17% from 2019

CiteRatio for Frontiers in Aging Neuroscience from 2016 - 2020
Year Value
2020 7.4
2019 6.3
2018 5.5
2017 6.5
2016 6.3
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 20% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 17% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.827

12% from 2019

SJR for Frontiers in Aging Neuroscience from 2016 - 2020
Year Value
2020 1.827
2019 1.635
2018 1.474
2017 1.638
2016 1.941
graph view Graph view
table view Table view

1.451

17% from 2019

SNIP for Frontiers in Aging Neuroscience from 2016 - 2020
Year Value
2020 1.451
2019 1.244
2018 1.103
2017 1.111
2016 1.197
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 12% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 17% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Aging Neuroscience

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Frontiers Media

Frontiers in Aging Neuroscience

Approved by publishing and review experts on SciSpace, this template is built as per for Frontiers in Aging Neuroscience formatting guidelines as mentioned in Frontiers Media author instructions. The current version was created on 14 Jun 2020 and has been used by 356 authors to write and format their manuscripts to this journal.

Neuroscience

i
Last updated on
14 Jun 2020
i
ISSN
1663-4365
i
Impact Factor
Medium - 0.94
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
frontiersinSCNS_ENG_HUMS
i
Citation Type
Numbered
[25]
i
Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FNAGI.2010.00012
Selective neuronal vulnerability to oxidative stress in the brain.
Xinkun Wang1, Elias K. Michaelis1

Abstract:

Oxidative stress (OS), caused by the imbalance between the generation and detoxification of reactive oxygen and nitrogen species (ROS/RNS), plays an important role in brain aging, neurodegenerative diseases, and other related adverse conditions, such as ischemia. While ROS/RNS serve as signaling molecules at physiological lev... Oxidative stress (OS), caused by the imbalance between the generation and detoxification of reactive oxygen and nitrogen species (ROS/RNS), plays an important role in brain aging, neurodegenerative diseases, and other related adverse conditions, such as ischemia. While ROS/RNS serve as signaling molecules at physiological levels, an excessive amount of these molecules leads to oxidative modification and, therefore, dysfunction of proteins, nucleic acids, and lipids. The response of neurons to this pervasive stress, however, is not uniform in the brain. While many brain neurons can cope with a rise in OS, there are select populations of neurons in the brain that are vulnerable. Because of their selective vulnerability, these neurons are usually the first to exhibit functional decline and cell death during normal aging, or in age-associated neurodegenerative diseases, such as Alzheimer's disease. Understanding the molecular and cellular mechanisms of selective neuronal vulnerability (SNV) to OS is important in the development of future intervention approaches to protect such vulnerable neurons from the stresses of the aging process and the pathological states that lead to neurodegeneration. In this review, the currently known molecular and cellular factors that contribute to SNV to OS are summarized. Included among the major underlying factors are high intrinsic OS, high demand for ROS/RNS-based signaling, low ATP production, mitochondrial dysfunction, and high inflammatory response in vulnerable neurons. The contribution to the selective vulnerability of neurons to OS by other intrinsic or extrinsic factors, such as deficient DNA damage repair, low calcium-buffering capacity, and glutamate excitotoxicity, are also discussed. read more read less

Topics:

Neurodegeneration (56%)56% related to the paper, Oxidative stress (52%)52% related to the paper, Excitotoxicity (52%)52% related to the paper, Glutamate receptor (51%)51% related to the paper
View PDF
791 Citations
open accessOpen access Journal Article DOI: 10.3389/FNAGI.2010.00032
Plasticity of Brain Networks in a Randomized Intervention Trial of Exercise Training in Older Adults

Abstract:

Research has shown the human brain is organized into separable functional networks during rest and varied states of cognition, and that aging is associated with specific network dysfunctions. The present study used functional magnetic resonance imaging (fMRI) to examine low-frequency (.008<.08 Hz) coherence of cognitively rel... Research has shown the human brain is organized into separable functional networks during rest and varied states of cognition, and that aging is associated with specific network dysfunctions. The present study used functional magnetic resonance imaging (fMRI) to examine low-frequency (.008<.08 Hz) coherence of cognitively relevant and sensory brain networks in older adults who participated in a one-year intervention trial, comparing the effects of aerobic and non-aerobic fitness training on brain function and cognition. Results showed that aerobic training improved the aging brain’s resting functional efficiency in higher-level cognitive networks. One year of walking increased functional connectivity between aspects of the frontal, posterior, and temporal cortices within the Default Mode Network and a Frontal Executive Network, two brain networks central to brain dysfunction in aging. Length of training was also an important factor. Effects in favor of the walking group were observed only after 12 months of training, compared to non-significant trends after six months. A non-aerobic stretching and toning group also showed increased functional connectivity in the DMN after six months and in a Frontal Parietal Network after 12 months, possibly reflecting experience-dependent plasticity. Finally, we found that changes in functional connectivity were behaviorally relevant. Increased functional connectivity was associated with greater improvement in executive function. Therefore the study provides the first evidence for exercise-induced functional plasticity in large-scale brain systems in the aging brain, using functional connectivity techniques, and offers new insight into the role of aerobic fitness in attenuating age-related brain dysfunction. read more read less

Topics:

Default mode network (56%)56% related to the paper, Functional magnetic resonance imaging (54%)54% related to the paper, Aging brain (54%)54% related to the paper, Human brain (53%)53% related to the paper
View PDF
715 Citations
open accessOpen access Journal Article DOI: 10.3389/FNAGI.2015.00052
Long-chain omega-3 fatty acids and the brain: a review of the independent and shared effects of EPA, DPA and DHA.
Simon C. Dyall1

Abstract:

Omega-3 polyunsaturated fatty acids (PUFAs) exhibit neuroprotective properties and represent a potential treatment for a variety of neurodegenerative and neurological disorders. However, traditionally there has been a lack of discrimination between the different omega-3 PUFAs and effects have been broadly accredited to series... Omega-3 polyunsaturated fatty acids (PUFAs) exhibit neuroprotective properties and represent a potential treatment for a variety of neurodegenerative and neurological disorders. However, traditionally there has been a lack of discrimination between the different omega-3 PUFAs and effects have been broadly accredited to series as a whole. Evidence for unique effects of eicosapentaenoic acid (EPA), docosahexaenoic acid (DHA) and more recently docosapentaenoic acid (DPA) is growing. For example, beneficial effects in mood disorders have more consistently been reported in clinical trials using EPA; whereas, with neurodegenerative conditions such as Alzheimer’s disease, the focus has been on DHA. DHA is quantitatively the most important omega-3 PUFA in the brain, and consequently the most studied, whereas the availability of high purity DPA preparations has been extremely limited until recently, limiting research into its effects. However, there is now a growing body of evidence indicating both independent and shared effects of EPA, DPA and DHA. The purpose of this review is to highlight how a detailed understanding of these effects is essential to improving understanding of their therapeutic potential. The review begins with an overview of omega-3 PUFA biochemistry and metabolism, with particular focus on the central nervous system, where DHA has unique and indispensable roles in neuronal membranes with levels preserved by multiple mechanisms. This is followed by a review of the different enzyme-derived anti-inflammatory mediators produced from EPA, DPA and DHA. Lastly, the relative protective effects of EPA, DPA and DHA in normal brain aging and the most common neurodegenerative disorders are discussed. With a greater understanding of the individual roles of EPA, DPA and DHA in brain health and repair it is hoped that appropriate dietary recommendations can be established and therapeutic interventions can be more targeted and refined. read more read less

Topics:

Docosapentaenoic acid (57%)57% related to the paper, Docosahexaenoic acid (53%)53% related to the paper
View PDF
609 Citations
open accessOpen access Journal Article DOI: 10.3389/FNAGI.2016.00256
Effect of Probiotic Supplementation on Cognitive Function and Metabolic Status in Alzheimer's Disease: A Randomized, Double-Blind and Controlled Trial.

Abstract:

Alzheimer's disease (AD) is associated with severe cognitive impairments as well as some metabolic defects. Scant studies in animal models indicate a link between probiotics and cognitive function. This randomized, double-blind and controlled clinical trial was conducted among 60 AD patients to assess the effects of probiotic... Alzheimer's disease (AD) is associated with severe cognitive impairments as well as some metabolic defects. Scant studies in animal models indicate a link between probiotics and cognitive function. This randomized, double-blind and controlled clinical trial was conducted among 60 AD patients to assess the effects of probiotic supplementation on cognitive function and metabolic status. The patients were randomly divided into two groups (n=30 in each group) treating with either milk (control group) or a mixture of probiotic (probiotic group). The probiotic supplemented group took 200 ml/day probiotic milk containing Lactobacillus acidophilus, Lactobacillus casei, Bifidobacterium bifidum and Lactobacillus fermentum (2×109 CFU/g for each) for 12 weeks. Mini-mental state examination (MMSE) score was recorded in all subjects before and after the treatment. Pre- and post-treatment fasting blood samples were obtained to determine the related markers. After 12 weeks intervention, compared with the control group (-5.03%±3.00), the probiotic treated (+27.90%±8.07) patients showed a significant improvement in the MMSE score (P<0.001). In addition, changes in plasma malondialdehyde (-22.01%±4.84 vs. +2.67%±3.86 µmol/L, P<0.001), serum high-sensitivity C-reactive protein (-17.61%±3.70 vs. +45.26%±3.50 µg/mL, P<0.001), homeostasis model of assessment-estimated insulin resistance (+28.84%±13.34 vs.+76.95%±24.60, P=0.002), Beta cell function (+3.45%±10.91 vs. +75.62%±23.18, P=0.001), serum triglycerides (-20.29%±4.49 vs. -0.16%±5.24 mg/dL, P=0.003) and quantitative insulin sensitivity check index (-1.83±1.26 vs. -4.66±1.70, P=0.006) in the probiotic group were significantly varied compared to the control group. We found that the probiotic treatment had no considerable effect on other biomarkers of oxidative stress and inflammation, fasting plasma glucose and other lipid profiles. Overall, the current study demonstrated that probiotic consumption for 12 weeks positively affects cognitive function and some metabolic statuses in the AD patients. read more read less

Topics:

Lactobacillus fermentum (65%)65% related to the paper, Bifidobacterium bifidum (54%)54% related to the paper, Lactobacillus casei (53%)53% related to the paper, Lactobacillus acidophilus (52%)52% related to the paper, Quantitative insulin sensitivity check index (51%)51% related to the paper
View PDF
573 Citations
open accessOpen access Journal Article DOI: 10.3389/FNAGI.2010.00027
Hundred Days of Cognitive Training Enhance Broad Cognitive Abilities in Adulthood: Findings from the COGITO Study.
Florian Schmiedek1, Martin Lövdén1, Ulman Lindenberger1

Abstract:

We examined whether positive transfer of cognitive training, which so far has been observed for individual tests only, also generalizes to cognitive abilities, thereby carrying greater promise for improving everyday intellectual competence in adulthood and old age. In the COGITO Study, 101 younger and 103 older adults practic... We examined whether positive transfer of cognitive training, which so far has been observed for individual tests only, also generalizes to cognitive abilities, thereby carrying greater promise for improving everyday intellectual competence in adulthood and old age. In the COGITO Study, 101 younger and 103 older adults practiced six tests of perceptual speed (PS), three tests of working memory (WM), and three tests of episodic memory (EM) for over 100 daily 1-h sessions. Transfer assessment included multiple tests of PS, WM, EM, and reasoning. In both age groups, reliable positive transfer was found not only for individual tests but also for cognitive abilities, represented as latent factors. Furthermore, the pattern of correlations between latent change factors of practiced and latent change factors of transfer tasks indicates systematic relations at the level of broad abilities, making the interpretation of effects as resulting from unspecific increases in motivation or self-concept less likely. read more read less

Topics:

Cognition (56%)56% related to the paper, Cognitive training (54%)54% related to the paper, Working memory (51%)51% related to the paper, Episodic memory (51%)51% related to the paper
View PDF
568 Citations
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Frequently asked questions

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Yes, the template is compliant with the Frontiers in Aging Neuroscience guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Aging Neuroscience?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Aging Neuroscience citation style.

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5. Can I use a manuscript in Frontiers in Aging Neuroscience that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Aging Neuroscience that you can download at the end.

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To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Aging Neuroscience?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Aging Neuroscience. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Aging Neuroscience?

The 5 most common citation types in order of usage for Frontiers in Aging Neuroscience are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Aging Neuroscience?

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16. Can I download Frontiers in Aging Neuroscience in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Aging Neuroscience Endnote style according to Elsevier guidelines.

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