Example of Frontiers in Cardiovascular Medicine format
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Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format Example of Frontiers in Cardiovascular Medicine format
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open access Open Access

Frontiers in Cardiovascular Medicine — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Cardiology and Cardiovascular Medicine #40 of 317 down down by None rank
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 473 Published Papers | 3673 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 05/06/2020
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

7.8

111% from 2019

CiteRatio for Frontiers in Cardiovascular Medicine from 2016 - 2020
Year Value
2020 7.8
2019 3.7
graph view Graph view
table view Table view

1.711

43% from 2019

SJR for Frontiers in Cardiovascular Medicine from 2019 - 2020
Year Value
2020 1.711
2019 1.197
graph view Graph view
table view Table view

1.346

42% from 2019

SNIP for Frontiers in Cardiovascular Medicine from 2019 - 2020
Year Value
2020 1.346
2019 0.947
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 111% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 43% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 42% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Cardiovascular Medicine

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Frontiers Media

Frontiers in Cardiovascular Medicine

Frontiers in Cardiovascular Medicine is a global, peer-reviewed Open Access medical journal providing unrestricted, online access to publications on diagnostic and therapeutic advances in cardiology and cardiovascular medicine. Frontiers in Cardiovascular Medicine publishes pe...... Read More

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Last updated on
05 Jun 2020
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
frontiersinSCNS_ENG_HUMS
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FCVM.2020.00022
Adipose Tissue Distribution, Inflammation and Its Metabolic Consequences, Including Diabetes and Cardiovascular Disease.
Alan Chait1, Laura J. den Hartigh1

Abstract:

Adipose tissue plays essential roles in maintaining lipid and glucose homeostasis. To date several types of adipose tissue have been identified, namely white, brown, and beige, that reside in various specific anatomical locations throughout the body. The cellular composition, secretome, and location of these adipose depots de... Adipose tissue plays essential roles in maintaining lipid and glucose homeostasis. To date several types of adipose tissue have been identified, namely white, brown, and beige, that reside in various specific anatomical locations throughout the body. The cellular composition, secretome, and location of these adipose depots define their function in health and metabolic disease. In obesity, adipose tissue becomes dysfunctional, promoting a pro-inflammatory, hyperlipidemic and insulin resistant environment that contributes to type 2 diabetes mellitus (T2DM). Concurrently, similar features that result from adipose tissue dysfunction also promote cardiovascular disease (CVD) by mechanisms that can be augmented by T2DM. The mechanisms by which dysfunctional adipose tissue simultaneously promote T2DM and CVD, focusing on adipose tissue depot-specific adipokines, inflammatory profiles, and metabolism, will be the focus of this review. The impact that various T2DM and CVD treatment strategies have on adipose tissue function and body weight also will be discussed. read more read less

Topics:

Adipose tissue (74%)74% related to the paper, Brown adipose tissue (66%)66% related to the paper, Adipokine (57%)57% related to the paper, Glucose homeostasis (53%)53% related to the paper, Insulin resistance (51%)51% related to the paper
View PDF
476 Citations
open accessOpen access Journal Article DOI: 10.3389/FCVM.2018.00135
Cardiovascular Effects and Benefits of Exercise.
Matthew A. Nystoriak1, Aruni Bhatnagar1

Abstract:

It is widely accepted that regular physical activity is beneficial for cardiovascular health. Frequent exercise is robustly associated with a decrease in cardiovascular mortality as well as the risk of developing cardiovascular disease. Physically active individuals have lower blood pressure, higher insulin sensitivity, and a... It is widely accepted that regular physical activity is beneficial for cardiovascular health. Frequent exercise is robustly associated with a decrease in cardiovascular mortality as well as the risk of developing cardiovascular disease. Physically active individuals have lower blood pressure, higher insulin sensitivity, and a more favorable plasma lipoprotein profile. Animal models of exercise show that repeated physical activity suppresses atherogenesis and increases the availability of vasodilatory mediators such as nitric oxide. Exercise has also been found to have beneficial effects on the heart. Acutely, exercise increases cardiac output and blood pressure, but individuals adapted to exercise show lower resting heart rate and cardiac hypertrophy. Both cardiac and vascular changes have been linked to a variety of changes in tissue metabolism and signaling, although our understanding of the contribution of the underlying mechanisms remains incomplete. Even though moderate levels of exercise have been found to be consistently associated with a reduction in cardiovascular disease risk, there is evidence to suggest that continuously high levels of exercise (e.g., marathon running) could have detrimental effects on cardiovascular health. Nevertheless, a specific dose response relationship between the extent and duration of exercise and the reduction in cardiovascular disease risk and mortality remains unclear. Further studies are needed to identify the mechanisms that impart cardiovascular benefits of exercise in order to develop more effective exercise regimens, test the interaction of exercise with diet, and develop pharmacological interventions for those unwilling or unable to exercise. read more read less
View PDF
345 Citations
open accessOpen access Journal Article DOI: 10.3389/FCVM.2017.00048
The Effects of Serotonin in Immune Cells
Nadine Herr1, Christoph Bode1, Daniel Duerschmied1

Abstract:

Serotonin (5-Hydroxytryptamine, 5-HT) plays an important role in many organs as a peripheral hormone. Most of the body's serotonin is circulating in the bloodstream, transported by blood platelets and is released upon activation. The functions of serotonin are mediated by members of the 7 known mammalian serotonin receptor su... Serotonin (5-Hydroxytryptamine, 5-HT) plays an important role in many organs as a peripheral hormone. Most of the body's serotonin is circulating in the bloodstream, transported by blood platelets and is released upon activation. The functions of serotonin are mediated by members of the 7 known mammalian serotonin receptor subtype classes (15 known subtypes), the serotonin transporter (SERT) and by covalent binding of serotonin to different effector proteins. Almost all immune cells express at least one serotonin component. In recent years, a number of immunoregulatory functions have been ascribed to serotonin. In monocytes/macrophages for example serotonin modulates cytokine secretion. Serotonin can also suppress the release of TNF-α and IL-1β by activating serotonin receptors. Furthermore, neutrophil recruitment and T-Cell activation can both be mediated by serotonin. These are only a few of the known immunomodulatory roles of serotonin that we will review here. read more read less

Topics:

Serotonergic (69%)69% related to the paper, Serotonin transporter (63%)63% related to the paper, 5-HT receptor (63%)63% related to the paper, Endogenous agonist (61%)61% related to the paper, Serotonin (61%)61% related to the paper
View PDF
276 Citations
open accessOpen access Journal Article DOI: 10.3389/FCVM.2018.00012
Source of Chronic Inflammation in Aging.
Fumihiro Sanada1, Yoshiaki Taniyama1, Jun Muratsu1, Rei Otsu1, Hideo Shimizu1, Hiromi Rakugi1, Ryuichi Morishita1

Abstract:

Aging is a complex process that results from a combination of environmental, genetic, and epigenetic factors. A chronic pro-inflammatory status is a pervasive feature of aging. This chronic low-grade inflammation occurring in the absence of overt infection has been defined as “inflammaging” and represents a significant risk f... Aging is a complex process that results from a combination of environmental, genetic, and epigenetic factors. A chronic pro-inflammatory status is a pervasive feature of aging. This chronic low-grade inflammation occurring in the absence of overt infection has been defined as “inflammaging” and represents a significant risk factor for morbidity and mortality in the elderly. The low-grade inflammation persists even after reversing pro-inflammatory stimuli such as LDL cholesterol and the renin–angiotensin system (RAS). Recently, several possible sources of chronic low-grade inflammation observed during aging and age-related diseases have been proposed. Cell senescence and dysregulation of innate immunity is one such mechanism by which persistent prolonged inflammation occurs even after the initial stimulus has been removed. Additionally, the coagulation factor that activates inflammatory signaling beyond its role in the coagulation system has been identified. This signal could be a new source of chronic inflammation and cell senescence. Here, we summarized the factors and cellular pathways/processes that are known to regulate low-grade persistent inflammation in aging and age-related disease. read more read less

Topics:

Senescence (57%)57% related to the paper, Inflammation (53%)53% related to the paper
View PDF
235 Citations
open accessOpen access Journal Article DOI: 10.3389/FCVM.2018.00068
Loss of Metabolic Flexibility in the Failing Heart.
Qutuba G. Karwi1, Golam M. Uddin1, Kim L. Ho1, Gary D. Lopaschuk1

Abstract:

To maintain its high energy demand the heart is equipped with a highly complex and efficient enzymatic machinery that orchestrates ATP production using multiple energy substrates, namely fatty acids, carbohydrates (glucose and lactate), ketones and amino acids. The contribution of these individual substrates to ATP production... To maintain its high energy demand the heart is equipped with a highly complex and efficient enzymatic machinery that orchestrates ATP production using multiple energy substrates, namely fatty acids, carbohydrates (glucose and lactate), ketones and amino acids. The contribution of these individual substrates to ATP production can dramatically change, depending on such variables as substrate availability, hormonal status and energy demand. This "metabolic flexibility" is a remarkable virtue of the heart, which allows utilization of different energy substrates at different rates to maintain contractile function. In heart failure, cardiac function is reduced, which is accompanied by discernible energy metabolism perturbations and impaired metabolic flexibility. While it is generally agreed that overall mitochondrial ATP production is impaired in the failing heart, there is less consensus as to what actual switches in energy substrate preference occur. The failing heart shift toward a greater reliance on glycolysis and ketone body oxidation as a source of energy, with a decrease in the contribution of glucose oxidation to mitochondrial oxidative metabolism. The heart also becomes insulin resistant. However, there is less consensus as to what happens to fatty acid oxidation in heart failure. While it is generally believed that fatty acid oxidation decreases, a number of clinical and experimental studies suggest that fatty acid oxidation is either not changed or is increased in heart failure. Of importance, is that any metabolic shift that does occur has the potential to aggravate cardiac dysfunction and the progression of the heart failure. An increasing body of evidence shows that increasing cardiac ATP production and/or modulating cardiac energy substrate preference positively correlates with heart function and can lead to better outcomes. This includes increasing glucose and ketone oxidation and decreasing fatty acid oxidation. In this review we present the physiology of the energy metabolism pathways in the heart and the changes that occur in these pathways in heart failure. We also look at the interventions which are aimed at manipulating the myocardial metabolic pathways toward more efficient substrate utilization which will eventually improve cardiac performance. read more read less

Topics:

Heart failure (54%)54% related to the paper, Beta oxidation (53%)53% related to the paper, Glycolysis (51%)51% related to the paper, Ketone bodies (51%)51% related to the paper
223 Citations
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Frontiers in Cardiovascular Medicine format uses frontiersinSCNS_ENG_HUMS citation style.

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Frequently asked questions

1. Can I write Frontiers in Cardiovascular Medicine in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Frontiers in Cardiovascular Medicine guidelines and auto format it.

2. Do you follow the Frontiers in Cardiovascular Medicine guidelines?

Yes, the template is compliant with the Frontiers in Cardiovascular Medicine guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Cardiovascular Medicine?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Cardiovascular Medicine citation style.

4. Can I use the Frontiers in Cardiovascular Medicine templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Cardiovascular Medicine.

5. Can I use a manuscript in Frontiers in Cardiovascular Medicine that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Cardiovascular Medicine that you can download at the end.

6. How long does it usually take you to format my papers in Frontiers in Cardiovascular Medicine?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Frontiers in Cardiovascular Medicine.

7. Where can I find the template for the Frontiers in Cardiovascular Medicine?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Cardiovascular Medicine's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Frontiers in Cardiovascular Medicine's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Frontiers in Cardiovascular Medicine an online tool or is there a desktop version?

SciSpace's Frontiers in Cardiovascular Medicine is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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After writing your paper autoformatting in Frontiers in Cardiovascular Medicine, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Cardiovascular Medicine's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Cardiovascular Medicine?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Cardiovascular Medicine. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Cardiovascular Medicine?

The 5 most common citation types in order of usage for Frontiers in Cardiovascular Medicine are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Cardiovascular Medicine?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Cardiovascular Medicine's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Frontiers in Cardiovascular Medicine in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Cardiovascular Medicine Endnote style according to Elsevier guidelines.

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