Example of Frontiers in Cellular Neuroscience format
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Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format
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Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format Example of Frontiers in Cellular Neuroscience format
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open access Open Access

Frontiers in Cellular Neuroscience — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Cellular and Molecular Neuroscience #28 of 88 down down by 14 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 1880 Published Papers | 13085 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 26/06/2020
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Quality:  
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CiteRatio: 8.4
SJR: 1.989
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.921

1% from 2018

Impact factor for Frontiers in Cellular Neuroscience from 2016 - 2019
Year Value
2019 3.921
2018 3.9
2017 4.3
2016 4.555
graph view Graph view
table view Table view

7.0

30% from 2019

CiteRatio for Frontiers in Cellular Neuroscience from 2016 - 2020
Year Value
2020 7.0
2019 5.4
2018 7.0
2017 8.1
2016 7.7
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 1% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 30% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.877

16% from 2019

SJR for Frontiers in Cellular Neuroscience from 2016 - 2020
Year Value
2020 1.877
2019 1.618
2018 2.036
2017 2.298
2016 2.493
graph view Graph view
table view Table view

1.212

27% from 2019

SNIP for Frontiers in Cellular Neuroscience from 2016 - 2020
Year Value
2020 1.212
2019 0.958
2018 1.074
2017 1.137
2016 1.1
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 16% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 27% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Cellular Neuroscience

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Frontiers Media

Frontiers in Cellular Neuroscience

Frontiers in Cellular Neuroscience is a Specialty Journal of Frontiers in Cell and Developmental Biology, Frontiers in Neuroscience and Frontiers in Physiology. Frontiers in Cellular Neuroscience is a first-tier electronic journal devoted to better understanding the cellular m...... Read More

Neuroscience

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Last updated on
26 Jun 2020
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ISSN
1662-5102
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Impact Factor
Medium - 0.776
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
frontiersinSCNS_ENG_HUMS
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FNCEL.2015.00392
Breaking down the barriers: the gut microbiome, intestinal permeability and stress-related psychiatric disorders
John R. Kelly1, Paul J. Kennedy1, John F. Cryan1, Timothy G. Dinan1, Gerard Clarke1, Niall P. Hyland1

Abstract:

The emerging links between our gut microbiome and the central nervous system (CNS) are regarded as a paradigm shift in neuroscience with possible implications for not only understanding the pathophysiology of stress-related psychiatric disorders, but also their treatment. Thus the gut microbiome and its influence on host barr... The emerging links between our gut microbiome and the central nervous system (CNS) are regarded as a paradigm shift in neuroscience with possible implications for not only understanding the pathophysiology of stress-related psychiatric disorders, but also their treatment. Thus the gut microbiome and its influence on host barrier function is positioned to be a critical node within the brain-gut axis. Mounting preclinical evidence broadly suggests that the gut microbiota can modulate brain development, function and behavior by immune, endocrine and neural pathways of the brain-gut-microbiota axis. Detailed mechanistic insights explaining these specific interactions are currently underdeveloped. However, the concept that a "leaky gut" may facilitate communication between the microbiota and these key signaling pathways has gained traction. Deficits in intestinal permeability may underpin the chronic low-grade inflammation observed in disorders such as depression and the gut microbiome plays a critical role in regulating intestinal permeability. In this review we will discuss the possible role played by the gut microbiota in maintaining intestinal barrier function and the CNS consequences when it becomes disrupted. We will draw on both clinical and preclinical evidence to support this concept as well as the key features of the gut microbiota which are necessary for normal intestinal barrier function. read more read less

Topics:

Intestinal permeability (60%)60% related to the paper, Gut–brain axis (59%)59% related to the paper, Gut flora (58%)58% related to the paper
View PDF
719 Citations
open accessOpen access Journal Article DOI: 10.3389/FNCEL.2013.00045
Origin and differentiation of microglia.
Florent Ginhoux1, Shawn Lim, Guillaume Hoeffel1, Donovan Low1, Tara Huber2

Abstract:

Microglia are the resident macrophage population of the central nervous system (CNS). Adequate microglial function is crucial for a healthy CNS. Microglia are not only the first immune sentinels of infection, contributing to both innate and adaptive immune responses locally, but are also involved in the maintenance of brain h... Microglia are the resident macrophage population of the central nervous system (CNS). Adequate microglial function is crucial for a healthy CNS. Microglia are not only the first immune sentinels of infection, contributing to both innate and adaptive immune responses locally, but are also involved in the maintenance of brain homeostasis. Emerging data are showing new and fundamental roles for microglia in the control of neuronal proliferation and differentiation, as well as in the formation of synaptic connections. While microglia have been studied for decades, a long history of experimental misinterpretation meant that their true origins remained debated. However, recent studies on microglial origin indicate that these cells in fact arise early during development from progenitors in the embryonic yolk sac (YS) that seed the brain rudiment and, remarkably, appear to persist there into adulthood. Here, we review the history of microglial cells and discuss the latest advances in our understanding of their origin, differentiation, and homeostasis, which provides new insights into their roles in health and disease. read more read less
View PDF
686 Citations
open accessOpen access Journal Article DOI: 10.3389/FNCEL.2019.00363
Brain-Derived Neurotrophic Factor: A Key Molecule for Memory in the Healthy and the Pathological Brain.
Magdalena Miranda1, Juan Facundo Morici1, María Belén Zanoni1, Pedro Bekinschtein1

Abstract:

Brain Derived Neurotrophic Factor (BDNF) is a key molecule involved in plastic changes related to learning and memory. The expression of BDNF is highly regulated, and can lead to great variability in BDNF levels in healthy subjects. Changes in BDNF expression are associated with both normal and pathological aging and also psy... Brain Derived Neurotrophic Factor (BDNF) is a key molecule involved in plastic changes related to learning and memory. The expression of BDNF is highly regulated, and can lead to great variability in BDNF levels in healthy subjects. Changes in BDNF expression are associated with both normal and pathological aging and also psychiatric disease, in particular in structures important for memory processes such as the hippocampus and parahippocampal areas. Some interventions like exercise or antidepressant administration enhance the expression of BDNF in normal and pathological conditions. In this review, we will describe studies from rodents and humans to bring together research on how BDNF expression is regulated, how this expression changes in the pathological brain and also exciting work on how interventions known to enhance this neurotrophin could have clinical relevance. We propose that, although BDNF may not be a valid biomarker for neurodegenerative/neuropsychiatric diseases because of its disregulation common to many pathological conditions, it could be thought of as a marker that specifically relates to the occurrence and/or progression of the mnemonic symptoms that are common to many pathological conditions. read more read less

Topics:

Brain-derived neurotrophic factor (59%)59% related to the paper, Alzheimer's disease (50%)50% related to the paper
View PDF
595 Citations
open accessOpen access Journal Article DOI: 10.3389/FNCEL.2014.00213
A molecular web: endoplasmic reticulum stress, inflammation, and oxidative stress.

Abstract:

Execution of fundamental cellular functions demands regulated protein folding homeostasis. Endoplasmic reticulum (ER) is an active organelle existing to implement this function by folding and modifying secretory and membrane proteins. Loss of protein folding homeostasis is central to various diseases and budding evidences sug... Execution of fundamental cellular functions demands regulated protein folding homeostasis. Endoplasmic reticulum (ER) is an active organelle existing to implement this function by folding and modifying secretory and membrane proteins. Loss of protein folding homeostasis is central to various diseases and budding evidences suggest ER stress as being a major contributor in the development or pathology of a diseased state besides other cellular stresses. The trigger for diseases may be diverse but, inflammation and/or ER stress may be basic mechanisms increasing the severity or complicating the condition of the disease. Chronic ER stress and activation of the unfolded-protein response (UPR) through endogenous or exogenous insults may result in impaired calcium and redox homeostasis, oxidative stress via protein overload thereby also influencing vital mitochondrial functions. Calcium released from the ER augments the production of mitochondrial Reactive Oxygen Species (ROS). Toxic accumulation of ROS within ER and mitochondria disturbs fundamental organelle functions. Sustained ER stress is known to potentially elicit inflammatory responses via UPR pathways. Additionally, ROS generated through inflammation or mitochondrial dysfunction could accelerate ER malfunction. Dysfunctional UPR pathways have been associated with a wide range of diseases including several neurodegenerative diseases, stroke, metabolic disorders, cancer, inflammatory disease, diabetes mellitus, cardiovascular disease, and others. In this review, we have discussed the UPR signaling pathways, and networking between ER stress-induced inflammatory pathways, oxidative stress, and mitochondrial signaling events, which further induce or exacerbate ER stress. read more read less

Topics:

Unfolded protein response (64%)64% related to the paper, Endoplasmic reticulum (54%)54% related to the paper, Mitochondrion (53%)53% related to the paper, Oxidative stress (53%)53% related to the paper
View PDF
479 Citations
open accessOpen access Journal Article DOI: 10.3389/FNCEL.2018.00386
Neurologic Alterations Due to Respiratory Virus Infections
Karen Bohmwald1, Nicolás M. S. Gálvez1, Mariana Ríos1, Alexis M. Kalergis1

Abstract:

Central Nervous System (CNS) infections are one of the most critical problems in public health, as frequently patients exhibit neurologic sequelae. Usually, CNS pathologies are caused by known neurotropic viruses such as measles virus (MV), herpes virus and human immunodeficiency virus (HIV), among others. However, nowadays r... Central Nervous System (CNS) infections are one of the most critical problems in public health, as frequently patients exhibit neurologic sequelae. Usually, CNS pathologies are caused by known neurotropic viruses such as measles virus (MV), herpes virus and human immunodeficiency virus (HIV), among others. However, nowadays respiratory viruses have placed themselves as relevant agents responsible for CNS pathologies. Among these neuropathological viruses are the human respiratory syncytial virus (hRSV), the influenza virus (IV), the coronavirus (CoV) and the human metapneumovirus (hMPV). These viral agents are leading causes of acute respiratory infections every year affecting mainly children under 5 years old and also the elderly. Up to date, several reports have described the association between respiratory viral infections with neurological symptoms. The most frequent clinical manifestations described in these patients are febrile or afebrile seizures, status epilepticus, encephalopathies and encephalitis. All these viruses have been found in cerebrospinal fluid (CSF), which suggests that all these pathogens, once in the lungs, can spread throughout the body and eventually reach the CNS. The current knowledge about the mechanisms and routes used by these neuro-invasive viruses remains scarce. In this review article, we describe the most recent findings associated to neurologic complications, along with data about the possible invasion routes of these viruses in humans and their various effects on the CNS, as studied in animal models. read more read less

Topics:

Respiratory virus (64%)64% related to the paper, Human metapneumovirus (56%)56% related to the paper, Coronavirus (54%)54% related to the paper, Virus (53%)53% related to the paper, Measles virus (51%)51% related to the paper
View PDF
478 Citations
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Frontiers in Cellular Neuroscience format uses frontiersinSCNS_ENG_HUMS citation style.

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Frequently asked questions

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Yes, the template is compliant with the Frontiers in Cellular Neuroscience guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Cellular Neuroscience?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Cellular Neuroscience citation style.

4. Can I use the Frontiers in Cellular Neuroscience templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Cellular Neuroscience.

5. Can I use a manuscript in Frontiers in Cellular Neuroscience that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Cellular Neuroscience that you can download at the end.

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7. Where can I find the template for the Frontiers in Cellular Neuroscience?

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Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Frontiers in Cellular Neuroscience is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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After writing your paper autoformatting in Frontiers in Cellular Neuroscience, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Cellular Neuroscience's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Cellular Neuroscience?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Cellular Neuroscience. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Cellular Neuroscience?

The 5 most common citation types in order of usage for Frontiers in Cellular Neuroscience are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Cellular Neuroscience?

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16. Can I download Frontiers in Cellular Neuroscience in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Cellular Neuroscience Endnote style according to Elsevier guidelines.

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