Example of Frontiers in Genetics format
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Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format
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Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format Example of Frontiers in Genetics format
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open access Open Access

Frontiers in Genetics — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Genetics #182 of 325 down down by 135 ranks
Genetics (clinical) #57 of 87 down down by 40 ranks
Molecular Medicine #110 of 167 down down by 82 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 3736 Published Papers | 13743 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 12/07/2020
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Related Journals

open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 15.2
SJR: 5.564
SNIP: 2.245
open access Open Access

Bentham Science

Quality:  
Good
CiteRatio: 4.7
SJR: 0.704
SNIP: 0.589
open access Open Access

Springer

Quality:  
Good
CiteRatio: 4.6
SJR: 1.028
SNIP: 1.006
open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 9.0
SJR: 3.587
SNIP: 1.457

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.7

37% from 2019

CiteRatio for Frontiers in Genetics from 2016 - 2020
Year Value
2020 3.7
2019 2.7
2018 5.6
2017 8.0
2016 7.0
graph view Graph view
table view Table view

1.413

4% from 2019

SJR for Frontiers in Genetics from 2016 - 2020
Year Value
2020 1.413
2019 1.469
2018 1.888
2017 2.274
2016 2.067
graph view Graph view
table view Table view

1.045

7% from 2019

SNIP for Frontiers in Genetics from 2016 - 2020
Year Value
2020 1.045
2019 0.975
2018 1.105
2017 1.039
2016 0.897
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 37% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 4% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 7% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Genetics

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Frontiers Media

Frontiers in Genetics

Approved by publishing and review experts on SciSpace, this template is built as per for Frontiers in Genetics formatting guidelines as mentioned in Frontiers Media author instructions. The current version was created on 12 Jul 2020 and has been used by 534 authors to write and format their manuscripts to this journal.

Medicine

i
Last updated on
12 Jul 2020
i
ISSN
1664-8021
i
Impact Factor
Medium - 0.566
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
frontiersinSCNS_ENG_HUMS
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Citation Type
Numbered
[25]
i
Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1038/NG.940
Human polymorphism at microRNAs and microRNA target sites.
Liuqing Yang, Chunru Lin, Chunyu Jin, Joy C. Yang, Bogdan Tanasa, Wenbo Wen-Hsiung Li, Daria Merkurjev, Kenneth a. Ohgi, Jie Zhang, Christopher P. Evans, Michael G. Rosenfeld, Charles E Vejnar, Matthias Blum, Evgeny M. Zdobnov, A Turchinovich, T R Samatov, a G Tonevitsky, B Burwinkel, Georg Schett, Dirk Elewaut, Iain B McInnes, Jean-Michel Dayer, Markus F Neurath, Sachin Pundhir, Jan Gorodkin, Fabiola Olivieri, Maria Rita Rippo, Antonio Domenico Procopio, Francesca Fazioli, Lukas T Jeker, Xuyu Zhou, Robert Blelloch, Jeffrey a Bluestone, Claudiane Guay, Romano Regazzi, Roopali Gandhi, Brian Healy, Taha Gholipour, Svetlana Egorova, Alexander Musallam, Mohammad Shuja Hussain, Parham Nejad, Bonny Patel, Hillary Hei, Samia Khoury, Francisco Quintana, Pia Kivisakk, Tanuja Chitnis, Howard L Weiner, Yun Deng, Jian Zhao, Daisuke Sakurai, Kenneth M. Kaufman, Jeffrey C. Edberg, Robert P. Kimberly, Diane L. Kamen, Gary S. Gilkeson, Chaim O. Jacob, R. Hal Scofield, Carl D. Langefeld, Jennifer A. Kelly, Rosalind Ramsey-Goldman, Michelle Petri, John D. Reveille, Luis M. Vilá, Graciela S. Alarcón, Timothy J. Vyse, Bernardo A. Pons-Estel, Barry I. Freedman, Patrick M. Gaffney, Kathy Moser Sivils, Judith A. James, Peter K. Gregersen, Juan-Manuel Anaya1, Timothy B. Niewold, Joan T. Merrill, Lindsey A. Criswell, Anne M. Stevens, Susan A. Boackle, Rita M. Cantor, Weiling Chen, J Grossman, Bevra H. Hahn, John B. Harley, Marta E Alarcόn-Riquelme, Elizabeth E. Brown, Betty P. Tsao, Susan Carpenter, Maninjay Atianand, Daniel Aiello, Emiliano P Ricci, Pallavi Gandhi, Lisa L Hall, Meg Byron, Brian Monks, Meabh Henry-Bezy, Jeanne B Lawrence, Luke a J O'Neill, Melissa J Moore, Daniel R Caffrey, Katherine a Fitzgerald, Dan-Xia Zhu, Lei Fan, Rui-Nan Lu, Cheng Fang, Wen-Yi Shen, Zhi-Jian Zou, Yin-Hua Wang, Hua-Yuan Zhu, Kou-Rong Miao, Peng Liu, Wei Xu, Jian-Yong Li, Jing Gong, Yin Tong, Hong-Mei Zhang, Kai Wang, Tao Hu, Ge Shan, Jun Sun, An-Yuan Guo, V Davalos, C Moutinho, A Villanueva, R Boque, Pedro Silva, F Carneiro, M Esteller, R Stephanie Huang, Eric R. Gamazon, Dana Ziliak, Yujia Wen, Hae Kyung Im, Wei Zhang, Claudia Wing, Shiwei Duan, Wasim K Bleibel, Nancy J. Cox, M. Eileen Dolan, Patrick Brest, Pierre Lapaquette, Mouloud Souidi, Kevin Lebrigand, Annabelle Cesaro, Valérie Vouret-Craviari, Bernard Mari, Pascal Barbry, Jean-François Mosnier, Xavier Hébuterne, Annick Harel-Bellan, Baharia Mograbi, Arlette Darfeuille-Michaud, Paul Hofman, Jonathan Marchini, Bryan Howie, Lucia A. Hindorff, Praveen Sethupathy, Heather a Junkins, Erin M. Ramos, Jayashri P Mehta, Francis S. Collins, Teri A. Manolio, William O.C.M. Cookson, Liming Liang, Gonçalo R. Abecasis, Miriam F. Moffatt, Mark Lathrop, Matthew A. Saunders, Han Liang
01 Jan 2013 - Frontiers in Genetics
View PDF
1,514 Citations
open accessOpen access Journal Article DOI: 10.3389/FGENE.2014.00001
Cytoplasmic 5 -3 exonuclease Xrn1p is also a genome-wide transcription factor in yeast
06 Feb 2014 - Frontiers in Genetics

Abstract:

The 5’ to 3’ exoribonuclease Xrn1 is a large protein involved in cytoplasmatic mRNA degradation as a critical component of the major decaysome. Its deletion in the yeast Saccharomyces cerevisiae is not lethal, but it has multiple physiological effects. In a previous study, our group showed that deletion of all tested componen... The 5’ to 3’ exoribonuclease Xrn1 is a large protein involved in cytoplasmatic mRNA degradation as a critical component of the major decaysome. Its deletion in the yeast Saccharomyces cerevisiae is not lethal, but it has multiple physiological effects. In a previous study, our group showed that deletion of all tested components of the yeast major decaysome, including XRN1, results in a decrease in the synthetic rate and an increase in half-life of most mRNAs in a compensatory manner. Furthermore, the same study showed that the all tested decaysome components are also nuclear proteins that bind to the 5’ region of a number of genes. In the present work, we show that disruption of Xrn1 activity preferentially affects both the synthesis and decay of a distinct subpopulation of mRNAs. The most affected mRNAs are the transcripts of the highly transcribed genes, mainly those encoding ribosome biogenesis and translation factors. Previously, we proposed that synthegradases play a key role in regulating both mRNA synthesis and degradation. Evidently, Xrn1 functions as a synthegradase, whose selectivity might help coordinating the expression of the protein synthetic machinery. We propose to name the most affected genes “Xrn1 synthegradon”. read more read less

Topics:

Saccharomyces cerevisiae (56%)56% related to the paper, Ribosome biogenesis (56%)56% related to the paper, Translation (biology) (55%)55% related to the paper, Exoribonuclease (54%)54% related to the paper, Saccharomyces (53%)53% related to the paper
View PDF
775 Citations
open accessOpen access Journal Article DOI: 10.3389/FGENE.2012.00035
Using Drosophila melanogaster as a Model for Genotoxic Chemical Mutational Studies with a New Program, SnpSift
Pablo Cingolani1, Viral M. Patel1, M. Coon1, Tung T. Nguyen1, Susan Land, Douglas M. Ruden, Xiangyi Lu
15 Mar 2012 - Frontiers in Genetics

Abstract:

This paper describes a new program SnpSift for filtering differential DNA sequence variants between two or more experimental genomes after genotoxic chemical exposure. Here, we illustrate how SnpSift can be used to identify candidate phenotype-relevant variants including single nucleotide polymorphisms, multiple nucleotide po... This paper describes a new program SnpSift for filtering differential DNA sequence variants between two or more experimental genomes after genotoxic chemical exposure. Here, we illustrate how SnpSift can be used to identify candidate phenotype-relevant variants including single nucleotide polymorphisms, multiple nucleotide polymorphisms, insertions, and deletions (InDels) in mutant strains isolated from genome-wide chemical mutagenesis of Drosophila melanogaster. First, the genomes of two independently isolated mutant fly strains that are allelic for a novel recessive male-sterile locus generated by genotoxic chemical exposure were sequenced using the Illumina next-generation DNA sequencer to obtain 20- to 29-fold coverage of the euchromatic sequences. The sequencing reads were processed and variants were called using standard bioinformatic tools. Next, SnpEff was used to annotate all sequence variants and their potential mutational effects on associated genes. Then, SnpSift was used to filter and select differential variants that potentially disrupt a common gene in the two allelic mutant strains. The potential causative DNA lesions were partially validated by capillary sequencing of polymerase chain reaction-amplified DNA in the genetic interval as defined by meiotic mapping and deletions that remove defined regions of the chromosome. Of the five candidate genes located in the genetic interval, the Pka-like gene CG12069 was found to carry a separate pre-mature stop codon mutation in each of the two allelic mutants whereas the other four candidate genes within the interval have wild-type sequences. The Pka-like gene is therefore a strong candidate gene for the male-sterile locus. These results demonstrate that combining SnpEff and SnpSift can expedite the identification of candidate phenotype-causative mutations in chemically mutagenized Drosophila strains. This technique can also be used to characterize the variety of mutations generated by genotoxic chemicals. read more read less

Topics:

Candidate gene (58%)58% related to the paper, Locus (genetics) (57%)57% related to the paper, DNA sequencing (55%)55% related to the paper, Gene (55%)55% related to the paper, DNA sequencer (53%)53% related to the paper
View PDF
746 Citations
open accessOpen access Journal Article DOI: 10.3389/FGENE.2012.00120
miR-34 - a microRNA replacement therapy is headed to the clinic.
02 Jul 2012 - Frontiers in Genetics

Abstract:

MicroRNA-34 (miR-34) is a master regulator of tumor suppression. It is downregulated in numerous cancers and inhibits malignant growth by repressing genes involved in various oncogenic signaling pathways. Consequently, miR-34 antagonizes processes that are necessary for basic cancer cell viability as well as cancer stemness, ... MicroRNA-34 (miR-34) is a master regulator of tumor suppression. It is downregulated in numerous cancers and inhibits malignant growth by repressing genes involved in various oncogenic signaling pathways. Consequently, miR-34 antagonizes processes that are necessary for basic cancer cell viability as well as cancer stemness, metastasis, and chemoresistance. This broad anti-oncogenic activity holds the prospect of creating a new remedy that is effective against tumor heterogeneity. This review focuses on the molecular mechanisms of miR-34-mediated tumor suppression, pharmacologies in animal models of cancer, and a status update of a miR-34 therapy that may be among the first miRNA mimics to reach the clinic. read more read less

Topics:

Cancer (59%)59% related to the paper, Metastasis (57%)57% related to the paper, Cancer cell (52%)52% related to the paper, microRNA (51%)51% related to the paper
View PDF
673 Citations
open accessOpen access Journal Article DOI: 10.3389/FGENE.2015.00001
Hereditary hemorrhagic telangiectasia: genetics and molecular diagnostics in a new era.
26 Jan 2015 - Frontiers in Genetics

Abstract:

Hereditary hemorrhagic telangiectasia (HHT) is a vascular dysplasia characterized by telangiectases and arteriovenous malformations (AVMs) in particular locations described in consensus clinical diagnostic criteria published in 2000. Two genes in the transforming growth factor-beta (TGF-β) signaling pathway, ENG and ACVRL1, w... Hereditary hemorrhagic telangiectasia (HHT) is a vascular dysplasia characterized by telangiectases and arteriovenous malformations (AVMs) in particular locations described in consensus clinical diagnostic criteria published in 2000. Two genes in the transforming growth factor-beta (TGF-β) signaling pathway, ENG and ACVRL1, were discovered almost two decades ago, and mutations in these genes have been reported to cause up to 85% of HHT. In our experience, approximately 96% of individuals with HHT have a mutation in these two genes, when published (Curacao) diagnostic criteria for HHT are strictly applied. More recently, two additional genes in the same pathway, SMAD4 and GDF2, have been identified in a much smaller number of patients with a similar or overlapping phenotype to HHT. Yet families still exist with compelling evidence of a hereditary telangiectasia disorder, but no identifiable mutation in a known gene. Recent availability of whole exome and genome testing has created new opportunities to facilitate gene discovery, identify genetic modifiers to explain clinical variability, and potentially define an increased spectrum of hereditary telangiectasia disorders. An expanded approach to molecular diagnostics for inherited telangiectasia disorders that incorporates a multi-gene next generation sequencing (NGS) HHT panel is proposed. read more read less

Topics:

Telangiectases (54%)54% related to the paper, Exome (52%)52% related to the paper
View PDF
655 Citations
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Frontiers in Genetics format uses frontiersinSCNS_ENG_HUMS citation style.

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Frequently asked questions

1. Can I write Frontiers in Genetics in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Frontiers in Genetics guidelines and auto format it.

2. Do you follow the Frontiers in Genetics guidelines?

Yes, the template is compliant with the Frontiers in Genetics guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Genetics?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Genetics citation style.

4. Can I use the Frontiers in Genetics templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Genetics.

5. Can I use a manuscript in Frontiers in Genetics that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Genetics that you can download at the end.

6. How long does it usually take you to format my papers in Frontiers in Genetics?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Frontiers in Genetics.

7. Where can I find the template for the Frontiers in Genetics?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Genetics's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Frontiers in Genetics's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Frontiers in Genetics an online tool or is there a desktop version?

SciSpace's Frontiers in Genetics is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Frontiers in Genetics?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Frontiers in Genetics?”

11. What is the output that I would get after using Frontiers in Genetics?

After writing your paper autoformatting in Frontiers in Genetics, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Genetics's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Genetics?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Genetics. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Genetics?

The 5 most common citation types in order of usage for Frontiers in Genetics are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Genetics?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Genetics's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Frontiers in Genetics in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Genetics Endnote style according to Elsevier guidelines.

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