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Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format
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Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format Example of Frontiers in Immunology format
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open access Open Access

Frontiers in Immunology — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Immunology and Allergy #38 of 182 up up by 4 ranks
Immunology #46 of 202 up up by 5 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 10789 Published Papers | 86957 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 09/07/2020
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Insights
General info
Top papers
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FAQ

Related Journals

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American Association for the Advancement of Science

Quality:  
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open access Open Access

Springer

Quality:  
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

5.085

8% from 2018

Impact factor for Frontiers in Immunology from 2016 - 2019
Year Value
2019 5.085
2018 4.716
2017 5.511
2016 6.429
graph view Graph view
table view Table view

8.1

50% from 2019

CiteRatio for Frontiers in Immunology from 2016 - 2020
Year Value
2020 8.1
2019 5.4
2018 4.5
2017 6.8
2016 9.0
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 8% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 50% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.646

25% from 2019

SJR for Frontiers in Immunology from 2016 - 2020
Year Value
2020 2.646
2019 2.116
2018 2.021
2017 2.803
2016 3.034
graph view Graph view
table view Table view

1.573

29% from 2019

SNIP for Frontiers in Immunology from 2016 - 2020
Year Value
2020 1.573
2019 1.224
2018 1.124
2017 1.517
2016 1.505
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 25% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 29% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Immunology

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Frontiers Media

Frontiers in Immunology

Approved by publishing and review experts on SciSpace, this template is built as per for Frontiers in Immunology formatting guidelines as mentioned in Frontiers Media author instructions. The current version was created on 09 Jul 2020 and has been used by 837 authors to write and format their manuscripts to this journal.

Immunology and Allergy

Medicine

i
Last updated on
09 Jul 2020
i
ISSN
1664-3224
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
frontiersinSCNS_ENG_HUMS
i
Citation Type
Numbered
[25]
i
Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FIMMU.2014.00461
Toll-like receptor signaling pathways.
Takumi Kawasaki1, Taro Kawai1
25 Sep 2014 - Frontiers in Immunology

Abstract:

Toll-like receptors (TLRs) play crucial roles in the innate immune system by recognizing pathogen-associated molecular patterns derived from various microbes. TLRs signal through the recruitment of specific adaptor molecules, leading to activation of the transcription factors NF-κB and IRFs, which dictate the outcome of innat... Toll-like receptors (TLRs) play crucial roles in the innate immune system by recognizing pathogen-associated molecular patterns derived from various microbes. TLRs signal through the recruitment of specific adaptor molecules, leading to activation of the transcription factors NF-κB and IRFs, which dictate the outcome of innate immune responses. During the past decade, the precise mechanisms underlying TLR signaling have been clarified by various approaches involving genetic, biochemical, structural, cell biological and bioinformatics studies. TLR signaling appears to be divergent and to play important roles in many aspects of the innate immune responses to given pathogens. In this review, we describe recent progress in our understanding of TLR signaling regulation and its contributions to host defense. read more read less

Topics:

Toll-like receptor (58%)58% related to the paper, Innate immune system (54%)54% related to the paper
View PDF
2,398 Citations
open accessOpen access Journal Article DOI: 10.3389/FIMMU.2014.00520
IgG subclasses and allotypes: from structure to effector functions.
Gestur Vidarsson1, Gillian Dekkers1, Theo Rispens1
20 Oct 2014 - Frontiers in Immunology

Abstract:

Of the five immunoglobulin isotypes, Immunoglobulin G (IgG) is most abundant in human serum. The four subclasses, IgG1, IgG2, IgG3 and IgG4 which are highly conserved, differ in their constant region, particularly in their hinges and upper CH2 domains. These regions are involved in binding to both IgG-Fc receptor (FcγR) and C... Of the five immunoglobulin isotypes, Immunoglobulin G (IgG) is most abundant in human serum. The four subclasses, IgG1, IgG2, IgG3 and IgG4 which are highly conserved, differ in their constant region, particularly in their hinges and upper CH2 domains. These regions are involved in binding to both IgG-Fc receptor (FcγR) and C1q. As a result, the different subclasses have different effector functions, both in terms of triggering FcγR-expressing cells, resulting in phagocytosis or Antibody-dependent cell-mediated cytotoxicity (ADCC), and activating complement. The Fc-regions also contain a binding epitope for the neonatal Fc-receptor (FcRn), responsible for the extended half-life, placental transport, and bidirectional transport of IgG to mucosal surfaces. However, FcRn is also expressed in myeloid cells, where it participates in both phagocytosis and antigen presentation together with classical FcγR and complement. How these properties, IgG-polymorphisms and post-translational modification of the antibodies in the form of glycosylation, affect IgG-function, will be the focus of the current review. read more read less

Topics:

Immunoglobulin G (56%)56% related to the paper, Neonatal Fc receptor (52%)52% related to the paper, Epitope (52%)52% related to the paper, Antibody (52%)52% related to the paper, Antibody-dependent cell-mediated cytotoxicity (52%)52% related to the paper
View PDF
1,834 Citations
open accessOpen access Journal Article DOI: 10.3389/FIMMU.2014.00491
Macrophage cytokines: involvement in immunity and infectious diseases.
Guillermo Arango Duque1, Albert Descoteaux1
07 Oct 2014 - Frontiers in Immunology

Abstract:

The evolution of macrophages has made them primordial for both development and immunity. Their functions range from the shaping of body plans to the ingestion and elimination of apoptotic cells and pathogens. Cytokines are small soluble proteins that confer instructions and mediate communication among immune and non-immune ce... The evolution of macrophages has made them primordial for both development and immunity. Their functions range from the shaping of body plans to the ingestion and elimination of apoptotic cells and pathogens. Cytokines are small soluble proteins that confer instructions and mediate communication among immune and non-immune cells. A portfolio of cytokines is central to the role of macrophages as sentries of the innate immune system that mediate the transition from innate to adaptive immunity. In concert with other mediators, cytokines bias the fate of macrophages into a spectrum of inflammation-promoting "classically activated," to anti-inflammatory or "alternatively activated" macrophages. Deregulated cytokine secretion is implicated in several disease states ranging from chronic inflammation to allergy. Macrophages release cytokines via a series of beautifully orchestrated pathways that are spatiotemporally regulated. At the molecular level, these exocytic cytokine secretion pathways are coordinated by multi-protein complexes that guide cytokines from their point of synthesis to their ports of exit into the extracellular milieu. These trafficking proteins, many of which were discovered in yeast and commemorated in the 2013 Nobel Prize in Physiology or Medicine, coordinate the organelle fusion steps that are responsible for cytokine release. This review discusses the functions of cytokines secreted by macrophages, and summarizes what is known about their release mechanisms. This information will be used to delve into how selected pathogens subvert cytokine release for their own survival. read more read less

Topics:

Cytokine secretion (67%)67% related to the paper, Innate immune system (59%)59% related to the paper, Proinflammatory cytokine (59%)59% related to the paper, Macrophage inflammatory protein (59%)59% related to the paper, Cytokine (57%)57% related to the paper
View PDF
1,829 Citations
open accessOpen access Journal Article DOI: 10.3389/FIMMU.2019.00277
Short Chain Fatty Acids (SCFAs)-Mediated Gut Epithelial and Immune Regulation and Its Relevance for Inflammatory Bowel Diseases.
11 Mar 2019 - Frontiers in Immunology

Abstract:

Ulcerative colitis (UC) and Crohn's disease (CD), collectively known as Inflammatory Bowel Diseases (IBD), are caused by a complex interplay between genetic, immunologic, microbial and environmental factors. Dysbiosis of the gut microbiome is increasingly considered to be causatively related to IBD and is strongly affected by... Ulcerative colitis (UC) and Crohn's disease (CD), collectively known as Inflammatory Bowel Diseases (IBD), are caused by a complex interplay between genetic, immunologic, microbial and environmental factors. Dysbiosis of the gut microbiome is increasingly considered to be causatively related to IBD and is strongly affected by components of a Western life style. Bacteria that ferment fibers and produce short chain fatty acids (SCFAs) are typically reduced in mucosa and feces of patients with IBD, as compared to healthy individuals. SCFAs, such as acetate, propionate and butyrate, are important metabolites in maintaining intestinal homeostasis. Several studies have indeed shown that fecal SCFAs levels are reduced in active IBD. SCFAs are an important fuel for intestinal epithelial cells and are known to strengthen the gut barrier function. Recent findings, however, show that SCFAs, and in particular butyrate, also have important immunomodulatory functions. Absorption of SCFAs is facilitated by substrate transporters like MCT1 and SMCT1 to promote cellular metabolism. Moreover, SCFAs may signal through cell surface G-protein coupled receptors (GPCRs), like GPR41, GPR43, and GPR109A, to activate signaling cascades that control immune functions. Transgenic mouse models support the key role of these GPCRs in controlling intestinal inflammation. Here, we present an overview of microbial SCFAs production and their effects on the intestinal mucosa with specific emphasis on their relevance for IBD. Moreover, we discuss the therapeutic potential of SCFAs for IBD, either applied directly or by stimulating SCFAs-producing bacteria through pre- or probiotic approaches. read more read less

Topics:

Intestinal mucosa (56%)56% related to the paper, Dysbiosis (53%)53% related to the paper
View PDF
1,732 Citations
open accessOpen access Journal Article DOI: 10.3389/FIMMU.2020.00827
Reduction and Functional Exhaustion of T Cells in Patients With Coronavirus Disease 2019 (COVID-19).
01 May 2020 - Frontiers in Immunology

Abstract:

Background: The outbreak of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has posed great threat to human health. T cells play a critical role in antiviral immunity but their numbers and functional state in COVID-19 patients remain largely unclear. Methods: We retro... Background: The outbreak of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has posed great threat to human health. T cells play a critical role in antiviral immunity but their numbers and functional state in COVID-19 patients remain largely unclear. Methods: We retrospectively reviewed the counts of T cells and serum cytokine concentration from data of 522 patients with laboratory-confirmed COVID-19 and 40 healthy controls. In addition, the expression of T cell exhaustion markers were measured in 14 COVID-19 cases. Results: The number of total T cells, CD4+ and CD8+ T cells were dramatically reduced in COVID-19 patients, especially in patients requiring Intensive Care Unit (ICU) care. Counts of total T cells, CD8+ T cells or CD4+ T cells lower than 800, 300, or 400/μL, respectively, were negatively correlated with patient survival. T cell numbers were negatively correlated to serum IL-6, IL-10, and TNF-α concentration, with patients in the disease resolution period showing reduced IL-6, IL-10, and TNF-α concentrations and restored T cell counts. T cells from COVID-19 patients had significantly higher levels of the exhausted marker PD-1. Increasing PD-1 and Tim-3 expression on T cells was seen as patients progressed from prodromal to overtly symptomatic stages. Conclusions: T cell counts are reduced significantly in COVID-19 patients, and the surviving T cells appear functionally exhausted. Non-ICU patients with total T cells counts lower than 800/μL may still require urgent intervention, even in the immediate absence of more severe symptoms due to a high risk for further deterioration in condition. read more read less

Topics:

T cell (69%)69% related to the paper, CD8 (60%)60% related to the paper
View PDF
1,676 Citations
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Frequently asked questions

1. Can I write Frontiers in Immunology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Frontiers in Immunology guidelines and auto format it.

2. Do you follow the Frontiers in Immunology guidelines?

Yes, the template is compliant with the Frontiers in Immunology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Immunology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Immunology citation style.

4. Can I use the Frontiers in Immunology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Immunology.

5. Can I use a manuscript in Frontiers in Immunology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Immunology that you can download at the end.

6. How long does it usually take you to format my papers in Frontiers in Immunology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Frontiers in Immunology.

7. Where can I find the template for the Frontiers in Immunology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Immunology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Frontiers in Immunology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Frontiers in Immunology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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11. What is the output that I would get after using Frontiers in Immunology?

After writing your paper autoformatting in Frontiers in Immunology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Immunology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Immunology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Immunology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Immunology?

The 5 most common citation types in order of usage for Frontiers in Immunology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Immunology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Immunology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Frontiers in Immunology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Immunology Endnote style according to Elsevier guidelines.

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