Example of Frontiers in Neurology format
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Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format
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Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format Example of Frontiers in Neurology format
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open access Open Access

Frontiers in Neurology — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Neurology (clinical) #144 of 343 down down by 22 ranks
Neurology #78 of 156 down down by 7 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 4663 Published Papers | 18694 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 21/07/2020
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General info
Top papers
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FAQ

Related Journals

open access Open Access
recommended Recommended

SAGE

Quality:  
High
CiteRatio: 9.5
SJR: 1.729
SNIP: 1.739
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SAGE

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CiteRatio: 7.3
SJR: 1.684
SNIP: 1.763
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SJR: 1.651
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open access Open Access

SAGE

Quality:  
High
CiteRatio: 4.3
SJR: 1.395
SNIP: 2.063

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.0

29% from 2019

CiteRatio for Frontiers in Neurology from 2016 - 2020
Year Value
2020 4.0
2019 3.1
2018 3.3
2017 4.0
2016 5.5
graph view Graph view
table view Table view

1.23

16% from 2019

SJR for Frontiers in Neurology from 2016 - 2020
Year Value
2020 1.23
2019 1.062
2018 1.185
2017 1.402
2016 1.56
graph view Graph view
table view Table view

1.282

28% from 2019

SNIP for Frontiers in Neurology from 2016 - 2020
Year Value
2020 1.282
2019 1.004
2018 0.982
2017 1.153
2016 1.081
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 29% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 16% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 28% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Neurology

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Frontiers Media

Frontiers in Neurology

Approved by publishing and review experts on SciSpace, this template is built as per for Frontiers in Neurology formatting guidelines as mentioned in Frontiers Media author instructions. The current version was created on 20 Jul 2020 and has been used by 616 authors to write and format their manuscripts to this journal.

Clinical Neurology

Medicine

i
Last updated on
20 Jul 2020
i
ISSN
1664-2295
i
Impact Factor
Medium - 0.713
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
frontiersinSCNS_ENG_HUMS
i
Citation Type
Numbered
[25]
i
Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FNEUR.2012.00073
Behavioral and Psychological Symptoms of Dementia
07 May 2012 - Frontiers in Neurology

Abstract:

Behavioral and psychological symptoms of dementia (BPSD), also known as neuropsychiatric symptoms, represent a heterogeneous group of non-cognitive symptoms and behaviors occurring in subjects with dementia. BPSD constitute a major component of the dementia syndrome irrespective of its subtype. They are as clinically relevant... Behavioral and psychological symptoms of dementia (BPSD), also known as neuropsychiatric symptoms, represent a heterogeneous group of non-cognitive symptoms and behaviors occurring in subjects with dementia. BPSD constitute a major component of the dementia syndrome irrespective of its subtype. They are as clinically relevant as cognitive symptoms as they strongly correlate with the degree of functional and cognitive impairment. BPSD include agitation, aberrant motor behavior, anxiety, elation, irritability, depression, apathy, disinhibition, delusions, hallucinations, and sleep or appetite changes. It is estimated that BPSD affect up to 90% of all dementia subjects over the course of their illness, and is independently associated with poor outcomes, including distress among patients and caregivers, long-term hospitalization, misuse of medication, and increased health care costs. Although these symptoms can be present individually it is more common that various psychopathological features co-occur simultaneously in the same patient. Thus, categorization of BPSD in clusters taking into account their natural course, prognosis, and treatment response may be useful in the clinical practice. The pathogenesis of BPSD has not been clearly delineated but it is probably the result of a complex interplay of psychological, social, and biological factors. Recent studies have emphasized the role of neurochemical, neuropathological, and genetic factors underlying the clinical manifestations of BPSD. A high degree of clinical expertise is crucial to appropriately recognize and manage the neuropsychiatric symptoms in a patient with dementia. Combination of non-pharmacological and careful use of pharmacological interventions is the recommended therapeutic for managing BPSD. Given the modest efficacy of current strategies, there is an urgent need to identify novel pharmacological targets and develop new non-pharmacological approaches to improve the adverse outcomes associated with BPSD. read more read less

Topics:

Dementia (56%)56% related to the paper, Apathy (52%)52% related to the paper, Psychopathology (51%)51% related to the paper
View PDF
775 Citations
open accessOpen access Journal Article DOI: 10.3389/FNEUR.2012.00074
Cognitive dysfunction in multiple sclerosis.
Joana Guimarães1, Maria José Sá1
24 May 2012 - Frontiers in Neurology

Abstract:

In Multiple Sclerosis (MS) prevalence studies of community and clinical samples, indicate that 45–60% of patients are cognitively impaired. These cognitive dysfunctions have been traditionally described as heterogeneous, but more recent studies suggest that there is a specific pattern of MS-related cognitive dysfunctions. Wit... In Multiple Sclerosis (MS) prevalence studies of community and clinical samples, indicate that 45–60% of patients are cognitively impaired. These cognitive dysfunctions have been traditionally described as heterogeneous, but more recent studies suggest that there is a specific pattern of MS-related cognitive dysfunctions. With the advent of disease-modifying medications for MS and emphasis on early intervention and treatment, detection of cognitive impairment at its earliest stage becomes particularly important. In this review the authors address: the cognitive domains most commonly impaired in MS (memory, attention, executive functions, speed of information processing, and visual–spatial abilities); the pathophysiological mechanism implied in MS cognitive dysfunction and correlated brain MRI features; the importance of neuropsychological assessment of MS patients in different stages of the disease and the influence of its course on cognitive performance; the most used tests and batteries for neuropsychological assessment; therapeutic strategies to improve cognitive abilities. read more read less

Topics:

Cognitive neuropsychology (67%)67% related to the paper, Cognitive remediation therapy (64%)64% related to the paper, Neuropsychological assessment (60%)60% related to the paper, Cognition (57%)57% related to the paper, Executive functions (56%)56% related to the paper
View PDF
612 Citations
open accessOpen access Journal Article DOI: 10.3389/FNEUR.2019.00282
Traumatic Spinal Cord Injury: An Overview of Pathophysiology, Models and Acute Injury Mechanisms.
Arsalan Alizadeh1, Scott M. Dyck1, Soheila Karimi-Abdolrezaee1
22 Mar 2019 - Frontiers in Neurology

Abstract:

Traumatic spinal cord injury (SCI) is a life changing neurological condition with substantial socioeconomic implications for patients and their care-givers. Recent advances in medical management of SCI has significantly improved diagnosis, stabilization, survival rate and well-being of SCI patients. However, there has been sm... Traumatic spinal cord injury (SCI) is a life changing neurological condition with substantial socioeconomic implications for patients and their care-givers. Recent advances in medical management of SCI has significantly improved diagnosis, stabilization, survival rate and well-being of SCI patients. However, there has been small progress on treatment options for improving the neurological outcomes of SCI patients. This incremental success mainly reflects the complexity of SCI pathophysiology and the diverse biochemical and physiological changes that occur in the injured spinal cord. Therefore, in the past few decades, considerable efforts have been made by SCI researchers to elucidate the pathophysiology of SCI and unravel the underlying cellular and molecular mechanisms of tissue degeneration and repair in the injured spinal cord. To this end, a number of preclinical animal and injury models have been developed to more closely recapitulate the primary and secondary injury processes of SCI. In this review, we will provide a comprehensive overview of the recent advances in our understanding of the pathophysiology of SCI. We will also discuss the neurological outcomes of human SCI and the available experimental model systems that have been employed to identify SCI mechanisms and develop therapeutic strategies for this condition. read more read less

Topics:

Spinal cord injury (53%)53% related to the paper, Poison control (52%)52% related to the paper
View PDF
590 Citations
open accessOpen access Journal Article DOI: 10.3389/FNEUR.2013.00018
The role of markers of inflammation in traumatic brain injury.
04 Mar 2013 - Frontiers in Neurology

Abstract:

Within minutes of a traumatic impact, a robust inflammatory response is elicited in the injured brain. The complexity of this post-traumatic squeal involves a cellular component, comprising the activation of resident glial cells, microglia, and astrocytes, and the infiltration of blood leukocytes. The second component regards... Within minutes of a traumatic impact, a robust inflammatory response is elicited in the injured brain. The complexity of this post-traumatic squeal involves a cellular component, comprising the activation of resident glial cells, microglia, and astrocytes, and the infiltration of blood leukocytes. The second component regards the secretion immune mediators, which can be divided into the following sub-groups: the archetypal pro-inflammatory cytokines (Interleukin-1, Tumor Necrosis Factor, Interleukin-6), the anti-inflammatory cytokines (IL-4, Interleukin-10, and TGF-beta), and the chemotactic cytokines or chemokines, which specifically drive the accumulation of parenchymal and peripheral immune cells in the injured brain region. Such mechanisms have been demonstrated in animal models, mostly in rodents, as well as in human brain. Whilst the humoral immune response is particularly pronounced in the acute phase following Traumatic brain injury (TBI), the activation of glial cells seems to be a rather prolonged effect lasting for several months. The complex interaction of cytokines and cell types installs a network of events, which subsequently intersect with adjacent pathological cascades including oxidative stress, excitotoxicity, or reparative events including angiogenesis, scarring, and neurogenesis. It is well accepted that neuroinflammation is responsible of beneficial and detrimental effects, contributing to secondary brain damage but also facilitating neurorepair. Although such mediators are clear markers of immune activation, to what extent cytokines can be defined as diagnostic factors reflecting brain injury or as predictors of long term outcome needs to be further substantiated. In clinical studies some groups reported a proportional cytokine production in either the cerebrospinal fluid or intraparenchymal tissue with initial brain damage, mortality, or poor outcome scores. However, the validity of cytokines as biomarkers is not broadly accepted. This review article will discuss the evidence from both clinical and laboratory studies exploring the validity of immune markers as a correlate to classification and outcome following TBI. read more read less

Topics:

Neuroinflammation (62%)62% related to the paper, Brain damage (61%)61% related to the paper, Traumatic brain injury (55%)55% related to the paper, Inflammation (55%)55% related to the paper, Cytokine (55%)55% related to the paper
View PDF
579 Citations
open accessOpen access Journal Article DOI: 10.3389/FNEUR.2019.00325
EEG Source Imaging: A Practical Review of the Analysis Steps.
Christoph M. Michel1, Denis Brunet1
04 Apr 2019 - Frontiers in Neurology

Abstract:

The electroencephalogram (EEG) is one of the oldest technologies to measure neuronal activity of the human brain. It has its undisputed value in clinical diagnosis, particularly (but not exclusively) in the identification of epilepsy and sleep disorders and in the evaluation of dysfunctions in sensory transmission pathways. W... The electroencephalogram (EEG) is one of the oldest technologies to measure neuronal activity of the human brain. It has its undisputed value in clinical diagnosis, particularly (but not exclusively) in the identification of epilepsy and sleep disorders and in the evaluation of dysfunctions in sensory transmission pathways. With the advancement of digital technologies, the analysis of EEG has moved from pure visual inspection of amplitude and frequency modulations over time to a comprehensive exploration of the temporal and spatial characteristics of the recorded signals. Today, EEG is accepted as a powerful tool to capture brain function with the unique advantage of measuring neuronal processes in the time frame in which these processes occur, namely in the sub-second range. However, it is generally stated that EEG suffers from a poor spatial resolution that makes it difficult to infer to the location of the brain areas generating the neuronal activity measured on the scalp. This statement has challenged a whole community of biomedical engineers to offer solutions to localize more precisely and more reliably the generators of the EEG activity. High-density EEG systems combined with precise information of the head anatomy and sophisticated source localization algorithms now exist that convert the EEG to a true neuroimaging modality. With these tools in hand and with the fact that EEG still remains versatile, inexpensive and portable, electrical neuroimaging has become a widely used technology to study the functions of the pathological and healthy human brain. However, several steps are needed to pass from the recording of the EEG to 3-dimensional images of neuronal activity. This review explains these different steps and illustrates them in a comprehensive analysis pipeline integrated in a stand-alone freely available academic software: Cartool. The information about how the different steps are performed in Cartool is only meant as a suggestion. Other EEG source imaging software may apply similar or different approaches to the different steps. read more read less

Topics:

Electroencephalography (53%)53% related to the paper
341 Citations
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Frequently asked questions

1. Can I write Frontiers in Neurology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Frontiers in Neurology guidelines and auto format it.

2. Do you follow the Frontiers in Neurology guidelines?

Yes, the template is compliant with the Frontiers in Neurology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Neurology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Neurology citation style.

4. Can I use the Frontiers in Neurology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Neurology.

5. Can I use a manuscript in Frontiers in Neurology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Neurology that you can download at the end.

6. How long does it usually take you to format my papers in Frontiers in Neurology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Frontiers in Neurology.

7. Where can I find the template for the Frontiers in Neurology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Neurology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Frontiers in Neurology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Frontiers in Neurology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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11. What is the output that I would get after using Frontiers in Neurology?

After writing your paper autoformatting in Frontiers in Neurology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Neurology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Neurology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Neurology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Neurology?

The 5 most common citation types in order of usage for Frontiers in Neurology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Neurology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Neurology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Frontiers in Neurology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Neurology Endnote style according to Elsevier guidelines.

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