Example of International Journal of Cell Biology format
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Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format
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Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format Example of International Journal of Cell Biology format
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open access Open Access

International Journal of Cell Biology — Template for authors

Publisher: Hindawi
Categories Rank Trend in last 3 yrs
Cell Biology #180 of 279 down down by 145 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 20 Published Papers | 77 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 06/06/2020
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Top papers
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.9

54% from 2019

CiteRatio for International Journal of Cell Biology from 2016 - 2020
Year Value
2020 3.9
2019 8.5
2018 8.2
2017 10.3
2016 10.4
graph view Graph view
table view Table view

0.587

57% from 2019

SJR for International Journal of Cell Biology from 2016 - 2020
Year Value
2020 0.587
2019 1.355
2018 1.162
2017 1.887
2016 2.0
graph view Graph view
table view Table view

0.938

39% from 2019

SNIP for International Journal of Cell Biology from 2016 - 2020
Year Value
2020 0.938
2019 1.532
2018 1.273
2017 1.416
2016 1.156
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 54% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 57% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 39% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

International Journal of Cell Biology

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Hindawi

International Journal of Cell Biology

International Journal of Cell Biology is a peer-reviewed, open access journal that publishes original research articles as well as review articles in all areas of cell biology.... Read More

Cell Biology

Biochemistry, Genetics and Molecular Biology

i
Last updated on
06 Jun 2020
i
ISSN
1687-8876
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Impact Factor
Medium - 0.828
i
Acceptance Rate
26%
i
Frequency
Not provided
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. “Specular andreev reflection in graphene”. Phys. Rev. Lett., vol. 97, no. 6, 067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1155/2010/214074
Cellular Stress Responses: Cell Survival and Cell Death
Simone Fulda1, Adrienne M. Gorman2, Osamu Hori3, Afshin Samali2

Abstract:

Cells can respond to stress in various ways ranging from the activation of survival pathways to the initiation of cell death that eventually eliminates damaged cells. Whether cells mount a protective or destructive stress response depends to a large extent on the nature and duration of the stress as well as the cell type. Als... Cells can respond to stress in various ways ranging from the activation of survival pathways to the initiation of cell death that eventually eliminates damaged cells. Whether cells mount a protective or destructive stress response depends to a large extent on the nature and duration of the stress as well as the cell type. Also, there is often the interplay between these responses that ultimately determines the fate of the stressed cell. The mechanism by which a cell dies (i.e., apoptosis, necrosis, pyroptosis, or autophagic cell death) depends on various exogenous factors as well as the cell's ability to handle the stress to which it is exposed. The implications of cellular stress responses to human physiology and diseases are manifold and will be discussed in this review in the context of some major world health issues such as diabetes, Parkinson's disease, myocardial infarction, and cancer. read more read less

Topics:

Pyroptosis (57%)57% related to the paper, Cell type (53%)53% related to the paper, Programmed cell death (52%)52% related to the paper, Cell (50%)50% related to the paper
View PDF
1,166 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/282041
Lipophagy: Connecting Autophagy and Lipid Metabolism
Rajat Singh1, Ana Maria Cuervo

Abstract:

Lipid droplets (LDs), initially considered “inert” lipid deposits, have gained during the last decade the classification of cytosolic organelles due to their defined composition and the multiplicity of specific cellular functions in which they are involved The classification of LD as organelles brings along the need for their... Lipid droplets (LDs), initially considered “inert” lipid deposits, have gained during the last decade the classification of cytosolic organelles due to their defined composition and the multiplicity of specific cellular functions in which they are involved The classification of LD as organelles brings along the need for their regulated turnover and recent findings support the direct contribution of autophagy to this turnover through a process now described as lipophagy This paper focuses on the characteristics of this new type of selective autophagy and the cellular consequences of the mobilization of intracellular lipids through this process Lipophagy impacts the cellular energetic balance directly, through lipid breakdown and, indirectly, by regulating food intake Defective lipophagy has been already linked to important metabolic disorders such as fatty liver, obesity and atherosclerosis, and the age-dependent decrease in autophagy could underline the basis for the metabolic syndrome of aging read more read less

Topics:

Lipid droplet (58%)58% related to the paper, Autophagy (53%)53% related to the paper, Lipid metabolism (53%)53% related to the paper
View PDF
431 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/676731
Cancer cell adhesion and metastasis: selectins, integrins, and the inhibitory potential of heparins.
Gerd Bendas1, Lubor Borsig2

Abstract:

Cell adhesion molecules play a significant role in cancer progression and metastasis. Cell-cell interactions of cancer cells with endothelium determine the metastatic spread. In addition, direct tumor cell interactions with platelets, leukocytes, and soluble components significantly contribute to cancer cell adhesion, extrava... Cell adhesion molecules play a significant role in cancer progression and metastasis. Cell-cell interactions of cancer cells with endothelium determine the metastatic spread. In addition, direct tumor cell interactions with platelets, leukocytes, and soluble components significantly contribute to cancer cell adhesion, extravasation, and the establishment of metastatic lesions. Clinical evidence indicates that heparin, commonly used for treatment of thromboembolic events in cancer patients, is beneficial for their survival. Preclinical studies confirm that heparin possesses antimetastatic activities that lead to attenuation of metastasis in various animal models. Heparin contains several biological activities that may affect several steps in metastatic cascade. Here we focus on the role of cellular adhesion receptors in the metastatic cascade and discuss evidence for heparin as an inhibitor of cell adhesion. While P- and L-selectin facilitation of cellular contacts during hematogenous metastasis is being accepted as a potential target of heparin, here we propose that heparin may also interfere with integrin activity and thereby affect cancer progression. This review summarizes recent findings about potential mechanisms of tumor cell interactions in the vasculature and antimetastatic activities of heparin. read more read less

Topics:

Cell adhesion (62%)62% related to the paper, Cell adhesion molecule (59%)59% related to the paper, Metastasis (55%)55% related to the paper, Heparin (55%)55% related to the paper, Selectin (55%)55% related to the paper
View PDF
418 Citations
open accessOpen access Journal Article DOI: 10.1155/2010/215158
The Role of cyclooxygenase-2 in Cell Proliferation and Cell Death in Human Malignancies

Abstract:

It is well admitted that the link between chronic inflammation and cancer involves cytokines and mediators of inflammatory pathways, which act during the different steps of tumorigenesis. The cyclooxygenases (COXs) are a family of enzymes, which catalyze the rate-limiting step of prostaglandin biosynthesis. This family contai... It is well admitted that the link between chronic inflammation and cancer involves cytokines and mediators of inflammatory pathways, which act during the different steps of tumorigenesis. The cyclooxygenases (COXs) are a family of enzymes, which catalyze the rate-limiting step of prostaglandin biosynthesis. This family contains three members: ubiquitously expressed COX-1, which is involved in homeostasis; the inducible COX-2 isoform, which is upregulated during both inflammation and cancer; and COX-3, expressed in brain and spinal cord, whose functions remain to be elucidated. COX-2 was described to modulate cell proliferation and apoptosis mainly in solid tumors, that is, colorectal, breast, and prostate cancers, and, more recently, in hematological malignancies. These findings prompt us to analyze here the effects of a combination of COX-2 inhibitors together with different clinically used therapeutic strategies in order to further improve the efficiency of future anticancer treatments. COX-2 modulation is a promising field investigated by many research groups. read more read less

Topics:

Cancer (53%)53% related to the paper, Carcinogenesis (51%)51% related to the paper
View PDF
404 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/736905
Aggrephagy: selective disposal of protein aggregates by macroautophagy.
Trond Lamark1, Terje Johansen1

Abstract:

Protein aggregation is a continuous process in our cells. Some proteins aggregate in a regulated manner required for different vital functional processes in the cells whereas other protein aggregates result from misfolding caused by various stressors. The decision to form an aggregate is largely made by chaperones and chapero... Protein aggregation is a continuous process in our cells. Some proteins aggregate in a regulated manner required for different vital functional processes in the cells whereas other protein aggregates result from misfolding caused by various stressors. The decision to form an aggregate is largely made by chaperones and chaperone-assisted proteins. Proteins that are damaged beyond repair are degraded either by the proteasome or by the lysosome via autophagy. The aggregates can be degraded by the proteasome and by chaperone-mediated autophagy only after dissolution into soluble single peptide species. Hence, protein aggregates as such are degraded by macroautophagy. The selective degradation of protein aggregates by macroautophagy is called aggrephagy. Here we review the processes of aggregate formation, recognition, transport, and sequestration into autophagosomes by autophagy receptors and the role of aggrephagy in different protein aggregation diseases. read more read less

Topics:

Aggrephagy (76%)76% related to the paper, Protein aggregation (58%)58% related to the paper
View PDF
373 Citations
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International Journal of Cell Biology format uses unsrt citation style.

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Frequently asked questions

1. Can I write International Journal of Cell Biology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the International Journal of Cell Biology guidelines and auto format it.

2. Do you follow the International Journal of Cell Biology guidelines?

Yes, the template is compliant with the International Journal of Cell Biology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in International Journal of Cell Biology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the International Journal of Cell Biology citation style.

4. Can I use the International Journal of Cell Biology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for International Journal of Cell Biology.

5. Can I use a manuscript in International Journal of Cell Biology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper International Journal of Cell Biology that you can download at the end.

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7. Where can I find the template for the International Journal of Cell Biology?

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SciSpace's International Journal of Cell Biology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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11. What is the output that I would get after using International Journal of Cell Biology?

After writing your paper autoformatting in International Journal of Cell Biology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is International Journal of Cell Biology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for International Journal of Cell Biology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for International Journal of Cell Biology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In International Journal of Cell Biology?

The 5 most common citation types in order of usage for International Journal of Cell Biology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the International Journal of Cell Biology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per International Journal of Cell Biology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download International Journal of Cell Biology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in International Journal of Cell Biology Endnote style according to Elsevier guidelines.

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