Example of Journal of Oncology format
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Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format
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Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format Example of Journal of Oncology format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Journal of Oncology — Template for authors

Publisher: Hindawi
Categories Rank Trend in last 3 yrs
Oncology #206 of 340 down down by 134 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 503 Published Papers | 1547 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 16/06/2020
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Quality:  
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SJR: 0.73
SNIP: 0.924
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

3.1

121% from 2019

CiteRatio for Journal of Oncology from 2016 - 2020
Year Value
2020 3.1
2019 1.4
2018 6.1
2017 6.9
2016 6.8
graph view Graph view
table view Table view

1.228

31% from 2019

SJR for Journal of Oncology from 2016 - 2020
Year Value
2020 1.228
2019 0.937
2018 1.087
2017 1.151
2016 1.203
graph view Graph view
table view Table view

0.995

14% from 2019

SNIP for Journal of Oncology from 2016 - 2020
Year Value
2020 0.995
2019 0.872
2018 1.37
2017 1.572
2016 1.367
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 121% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 31% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 14% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Journal of Oncology

Guideline source: View

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Hindawi

Journal of Oncology

Journal of Oncology is a peer-reviewed, open access journal that publishes original research articles, review articles, and clinical studies in all areas of oncology.... Read More

Oncology

Medicine

i
Last updated on
16 Jun 2020
i
ISSN
1687-8450
i
Impact Factor
Medium - 0.683
i
Acceptance Rate
44%
i
Frequency
Not provided
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. “Specular andreev reflection in graphene”. Phys. Rev. Lett., vol. 97, no. 6, 067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1155/2010/617421
Significance of Circulating Tumor Cells Detected by the CellSearch System in Patients with Metastatic Breast Colorectal and Prostate Cancer.
01 Jan 2010 - Journal of Oncology

Abstract:

The increasing number of treatment options for patients with metastatic carcinomas has created a concomitant need for new methods to monitor their use. Ideally, these modalities would be noninvasive, be independent of treatment, and provide quantitative real-time analysis of tumor activity in a variety of carcinomas. Assessme... The increasing number of treatment options for patients with metastatic carcinomas has created a concomitant need for new methods to monitor their use. Ideally, these modalities would be noninvasive, be independent of treatment, and provide quantitative real-time analysis of tumor activity in a variety of carcinomas. Assessment of circulating tumor cells (CTCs) shed into the blood during metastasis may satisfy this need. We developed the CellSearch System to enumerate CTC from 7.5 mL of venous blood. In this review we compare the outcomes from three prospective multicenter studies investigating the use of CTC to monitor patients undergoing treatment for metastatic breast (MBC), colorectal (MCRC), or prostate cancer (MPC) and review the CTC definition used in these studies. Evaluation of CTC at anytime during the course of disease allows assessment of patient prognosis and is predictive of overall survival. read more read less

Topics:

Circulating tumor cell (58%)58% related to the paper, Metastasis (53%)53% related to the paper, Prostate cancer (52%)52% related to the paper
View PDF
730 Citations
open accessOpen access Journal Article DOI: 10.1155/2011/396076
Quiescent, Slow-Cycling Stem Cell Populations in Cancer: A Review of the Evidence and Discussion of Significance
Nathan Moore1, Stephen Lyle1
01 Jan 2011 - Journal of Oncology

Abstract:

Long-lived cancer stem cells (CSCs) with indefinite proliferative potential have been identified in multiple epithelial cancer types. These cells are likely derived from transformed adult stem cells and are thought to share many characteristics with their parental population, including a quiescent slow-cycling phenotype. Vari... Long-lived cancer stem cells (CSCs) with indefinite proliferative potential have been identified in multiple epithelial cancer types. These cells are likely derived from transformed adult stem cells and are thought to share many characteristics with their parental population, including a quiescent slow-cycling phenotype. Various label-retaining techniques have been used to identify normal slow cycling adult stem cell populations and offer a unique methodology to functionally identify and isolate cancer stem cells. The quiescent nature of CSCs represents an inherent mechanism that at least partially explains chemotherapy resistance and recurrence in posttherapy cancer patients. Isolating and understanding the cell cycle regulatory mechanisms of quiescent cancer cells will be a key component to creation of future therapies that better target CSCs and totally eradicate tumors. Here we review the evidence for quiescent CSC populations and explore potential cell cycle regulators that may serve as future targets for elimination of these cells. read more read less

Topics:

Cancer stem cell (69%)69% related to the paper, Stem cell (59%)59% related to the paper, Adult stem cell (59%)59% related to the paper, Cancer cell (56%)56% related to the paper, Cancer (51%)51% related to the paper
View PDF
342 Citations
open accessOpen access Journal Article DOI: 10.1155/2010/214186
Anticancer Drugs from Marine Flora: An Overview
01 Jan 2010 - Journal of Oncology

Abstract:

Marine floras, such as bacteria, actinobacteria, cyanobacteria, fungi, microalgae, seaweeds, mangroves, and other halophytes are extremely important oceanic resources, constituting over 90% of the oceanic biomass. They are taxonomically diverse, largely productive, biologically active, and chemically unique offering a great s... Marine floras, such as bacteria, actinobacteria, cyanobacteria, fungi, microalgae, seaweeds, mangroves, and other halophytes are extremely important oceanic resources, constituting over 90% of the oceanic biomass. They are taxonomically diverse, largely productive, biologically active, and chemically unique offering a great scope for discovery of new anticancer drugs. The marine floras are rich in medicinally potent chemicals predominantly belonging to polyphenols and sulphated polysaccharides. The chemicals have displayed an array of pharmacological properties especially antioxidant, immunostimulatory, and antitumour activities. The phytochemicals possibly activate macrophages, induce apoptosis, and prevent oxidative damage of DNA, thereby controlling carcinogenesis. In spite of vast resources enriched with chemicals, the marine floras are largely unexplored for anticancer lead compounds. Hence, this paper reviews the works so far conducted on this aspect with a view to provide a baseline information for promoting the marine flora-based anticancer research in the present context of increasing cancer incidence, deprived of the cheaper, safer, and potent medicines to challenge the dreadful human disease. read more read less
View PDF
318 Citations
open accessOpen access Journal Article DOI: 10.1155/2010/932371
Ovarian Cancer Pathogenesis: A Model in Evolution
Alison M. Karst1, Ronny Drapkin1, Ronny Drapkin2
01 Jan 2010 - Journal of Oncology

Abstract:

Ovarian cancer is a deadly disease for which there is no effective means of early detection. Ovarian carcinomas comprise a diverse group of neoplasms, exhibiting a wide range of morphological characteristics, clinical manifestations, genetic alterations, and tumor behaviors. This high degree of heterogeneity presents a major ... Ovarian cancer is a deadly disease for which there is no effective means of early detection. Ovarian carcinomas comprise a diverse group of neoplasms, exhibiting a wide range of morphological characteristics, clinical manifestations, genetic alterations, and tumor behaviors. This high degree of heterogeneity presents a major clinical challenge in both diagnosing and treating ovarian cancer. Furthermore, the early events leading to ovarian carcinoma development are poorly understood, thus complicating efforts to develop screening modalities for this disease. Here, we provide an overview of the current models of ovarian cancer pathogenesis, highlighting recent findings implicating the fallopian tube fimbria as a possible site of origin of ovarian carcinomas. The ovarian cancer model will continue to evolve as we learn more about the genetics and etiology of this disease. read more read less

Topics:

Ovarian carcinoma (69%)69% related to the paper, Ovarian cancer (59%)59% related to the paper
View PDF
264 Citations
open accessOpen access Journal Article DOI: 10.1155/2015/865816
The Regulatory Role of MicroRNAs in EMT and Cancer
Apostolos Zaravinos1
25 Mar 2015 - Journal of Oncology

Abstract:

The epithelial to mesenchymal transition (EMT) is a powerful process in tumor invasion, metastasis, and tumorigenesis and describes the molecular reprogramming and phenotypic changes that are characterized by a transition from polarized immotile epithelial cells to motile mesenchymal cells. It is now well known that miRNAs ar... The epithelial to mesenchymal transition (EMT) is a powerful process in tumor invasion, metastasis, and tumorigenesis and describes the molecular reprogramming and phenotypic changes that are characterized by a transition from polarized immotile epithelial cells to motile mesenchymal cells. It is now well known that miRNAs are important regulators of malignant transformation and metastasis. The aberrant expression of the miR-200 family in cancer and its involvement in the initiation and progression of malignant transformation has been well demonstrated. The metastasis suppressive role of the miR-200 members is strongly associated with a pathologic EMT. This review describes the most recent advances regarding the influence of miRNAs in EMT and the control they exert in major signaling pathways in various cancers. The ability of the autocrine TGF-β/ZEB/miR-200 signaling regulatory network to control cell plasticity between the epithelial and mesenchymal state is further discussed. Various miRNAs are reported to directly target EMT transcription factors and components of the cell architecture, as well as miRNAs that are able to reverse the EMT process by targeting the Notch and Wnt signaling pathways. The link between cancer stem cells and EMT is also reported and the most recent developments regarding clinical trials that are currently using anti-miRNA constructs are further discussed. read more read less

Topics:

Epithelial–mesenchymal transition (59%)59% related to the paper, Cancer stem cell (55%)55% related to the paper, Wnt signaling pathway (52%)52% related to the paper, Autocrine signalling (52%)52% related to the paper, Malignant transformation (51%)51% related to the paper
View PDF
258 Citations
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Journal of Oncology format uses unsrt citation style.

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Frequently asked questions

1. Can I write Journal of Oncology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Oncology guidelines and auto format it.

2. Do you follow the Journal of Oncology guidelines?

Yes, the template is compliant with the Journal of Oncology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Oncology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Oncology citation style.

4. Can I use the Journal of Oncology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Oncology.

5. Can I use a manuscript in Journal of Oncology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Journal of Oncology that you can download at the end.

6. How long does it usually take you to format my papers in Journal of Oncology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Journal of Oncology.

7. Where can I find the template for the Journal of Oncology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Oncology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Journal of Oncology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Journal of Oncology an online tool or is there a desktop version?

SciSpace's Journal of Oncology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Journal of Oncology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Journal of Oncology?”

11. What is the output that I would get after using Journal of Oncology?

After writing your paper autoformatting in Journal of Oncology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Journal of Oncology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Oncology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Oncology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Oncology?

The 5 most common citation types in order of usage for Journal of Oncology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Journal of Oncology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Oncology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Journal of Oncology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Oncology Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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