Example of Journal of Thyroid Research format
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Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format
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Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format Example of Journal of Thyroid Research format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Journal of Thyroid Research — Template for authors

Publisher: Hindawi
Categories Rank Trend in last 3 yrs
Endocrinology, Diabetes and Metabolism #156 of 219 down down by 44 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 49 Published Papers | 106 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 12/06/2020
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.2

4% from 2019

CiteRatio for Journal of Thyroid Research from 2016 - 2020
Year Value
2020 2.2
2019 2.3
2018 2.0
2017 3.3
2016 4.0
graph view Graph view
table view Table view

0.457

45% from 2019

SJR for Journal of Thyroid Research from 2016 - 2020
Year Value
2020 0.457
2019 0.316
2018 0.507
2017 0.573
2016 0.662
graph view Graph view
table view Table view

0.926

44% from 2019

SNIP for Journal of Thyroid Research from 2016 - 2020
Year Value
2020 0.926
2019 0.645
2018 0.867
2017 0.999
2016 1.046
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 4% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 45% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 44% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Journal of Thyroid Research

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Hindawi

Journal of Thyroid Research

Approved by publishing and review experts on SciSpace, this template is built as per for Journal of Thyroid Research formatting guidelines as mentioned in Hindawi author instructions. The current version was created on 12 Jun 2020 and has been used by 455 authors to write and format their manuscripts to this journal.

Endocrinology, Diabetes and Metabolism

Medicine

i
Last updated on
12 Jun 2020
i
ISSN
2090-8067
i
Impact Factor
High - 1.022
i
Acceptance Rate
Not provided
i
Frequency
Not provided
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. “Specular andreev reflection in graphene”. Phys. Rev. Lett., vol. 97, no. 6, 067007, 2006.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1155/2014/638747
Antitumor Activity of Lenvatinib (E7080): An Angiogenesis Inhibitor That Targets Multiple Receptor Tyrosine Kinases in Preclinical Human Thyroid Cancer Models

Abstract:

Inhibition of tumor angiogenesis by blockading the vascular endothelial growth factor (VEGF) signaling pathway is a promising therapeutic strategy for thyroid cancer. Lenvatinib mesilate (lenvatinib) is a potent inhibitor of VEGF receptors (VEGFR1–3) and other prooncogenic and prooncogenic receptor tyrosine kinases, including... Inhibition of tumor angiogenesis by blockading the vascular endothelial growth factor (VEGF) signaling pathway is a promising therapeutic strategy for thyroid cancer. Lenvatinib mesilate (lenvatinib) is a potent inhibitor of VEGF receptors (VEGFR1–3) and other prooncogenic and prooncogenic receptor tyrosine kinases, including fibroblast growth factor receptors (FGFR1–4), platelet derived growth factor receptor α (PDGFRα), KIT, and RET. We examined the antitumor activity of lenvatinib against human thyroid cancer xenograft models in nude mice. Orally administered lenvatinib showed significant antitumor activity in 5 differentiated thyroid cancer (DTC), 5 anaplastic thyroid cancer (ATC), and 1 medullary thyroid cancer (MTC) xenograft models. Lenvatinib also showed antiangiogenesis activity against 5 DTC and 5 ATC xenografts, while lenvatinib showed in vitro antiproliferative activity against only 2 of 11 thyroid cancer cell lines: that is, RO82-W-1 and TT cells. Western blot analysis showed that cultured RO82-W-1 cells overexpressed FGFR1 and that lenvatinib inhibited the phosphorylation of FGFR1 and its downstream effector FRS2. Lenvatinib also inhibited the phosphorylation of RET with the activated mutation C634W in TT cells. These data demonstrate that lenvatinib provides antitumor activity mainly via angiogenesis inhibition but also inhibits FGFR and RET signaling pathway in preclinical human thyroid cancer models. read more read less

Topics:

Lenvatinib (75%)75% related to the paper, Anaplastic thyroid cancer (59%)59% related to the paper, Thyroid cancer (58%)58% related to the paper, Medullary thyroid cancer (55%)55% related to the paper, Cancer (52%)52% related to the paper
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319 Citations
open accessOpen access Journal Article DOI: 10.4061/2011/439463
Thyroid disorders and diabetes mellitus.
Mirella P. Hage1, Mira S. Zantout1, Sami T. Azar1

Abstract:

Studies have found that diabetes and thyroid disorders tend to coexist in patients. Both conditions involve a dysfunction of the endocrine system. Thyroid disorders can have a major impact on glucose control, and untreated thyroid disorders affect the management of diabetes in patients. Consequently, a systematic approach to ... Studies have found that diabetes and thyroid disorders tend to coexist in patients. Both conditions involve a dysfunction of the endocrine system. Thyroid disorders can have a major impact on glucose control, and untreated thyroid disorders affect the management of diabetes in patients. Consequently, a systematic approach to thyroid testing in patients with diabetes is recommended. read more read less

Topics:

Thyroid (57%)57% related to the paper, Diabetes mellitus (54%)54% related to the paper, Endocrine system (51%)51% related to the paper
View PDF
227 Citations
open accessOpen access Journal Article DOI: 10.4061/2011/432890
Autoimmune Thyroid Disorders
Rosalind S. Brown1, Gary L. Francis2

Abstract:

Thyroid autoimmunity, as reflected by the presence in serum of autoantibodies directed against the thyroid autoantigens thyroglobulin (Tg) and thyroid peroxidase (TPO), is present in >10% of the US population over 12 years of age [1] and is the most common cause of endocrine dysfunction in iodine-sufficient populations [2]. T... Thyroid autoimmunity, as reflected by the presence in serum of autoantibodies directed against the thyroid autoantigens thyroglobulin (Tg) and thyroid peroxidase (TPO), is present in >10% of the US population over 12 years of age [1] and is the most common cause of endocrine dysfunction in iodine-sufficient populations [2]. The underlying mechanism is a failure of T-cell tolerance leading to lymphocytic infiltration of the thyroid gland [3] and to a complex sequence of humoral and cellular immune responses to thyroid antigens, presumably in response to an environmental trigger [4]. In chronic lymphocytic thyroiditis (CLT), the predominant immunologic mechanisms are T-cell- and cytokine-mediated thyroid cell damage and apoptotic cell death whereas in Graves' disease (GD) generation of thyrotropin (TSH) receptor autoantibodies leads to thyroid cell stimulation [5], but significant overlap exists. Seven susceptibility genes, in addition to the major histocompatibility gene (HLA-DR3), have now been identified [6]. Some of these genes affect the immune response in general (CD40, CTLA-4, and PTPN22), while others are thyroid specific (thyroglobulin, thyrotropin (TSH) receptor). Some are common to both CLT and GD, while others are specific for GD. In view of the importance of AITD as well as the diverse array of new information, it is only fitting that this special issue of the Journal of Thyroid Research is devoted entirely to this complex subject. Four of the papers we have selected are focused on clinical topics, including AITD in childhood, during pregnancy, in the postpartum period, and in patients with type 1 diabetes mellitus. The fifth paper addresses the potential role of NKT cells in an animal model of thyroiditis. We conclude this special edition with a discussion of thyroid autoimmunity in patients with papillary thyroid cancer (PTC). The association of AI and thyroid cancer was first reported by Dailey et al. [7]. In general, patients with AI appear more likely to have PTC than follicular thyroid cancer (FTC), but a lower frequency of extrathyroidal extension, nodal and distant metastases when compared with patients without AI. In some but not all series, patients with autoimmune thyroiditis (AT) and PTC have improved survival when compared to those with PTC alone, suggesting that thyroid autoimmunity might contribute to improved survival [8–10]. In contrast, other data suggest that AI might actually increase the risk to develop thyroid cancer [10–12]. Several theories have been proposed to explain how AI might increase the risk for thyroid malignancy. Thyrocyte apoptosis and proliferation are increased in AI suggesting that thyrocytes rapidly progressing through the cell cycle might accumulate increased DNA damage resulting in malignant transformation [13]. Russell et al. hypothesized that thyroid cells predestined to become cancers might secrete proinflammatory cytokines that affect immune cells [14]. They showed that thyrocytes of ret/PTC3 transgenic mice express increased levels of interleukins, tumor necrosis factor-α, and cyclooxygenase-2 [14] that could attract and/or activate cells of the immune system. Finally, the ret/PTC recombinant genes have been detected in samples of AI [15–17] suggesting that ret/PTC rearrangements might be present in AI and could be precursors to PTC. From these papers, it is clear that thyroid autoimmunity is a frequent problem in the population and that thyroid autoimmunity can lead to a variety of thyroid disorders including alterations in thyroid hormone synthesis and possibly even neoplasia. Focused research in this area is beginning to illuminate some of the molecular mechanisms that help to explain these associations. Rosalind Brown Gary L. Francis read more read less

Topics:

Thyroid cancer (71%)71% related to the paper, Follicular thyroid cancer (69%)69% related to the paper, Thyroid peroxidase (68%)68% related to the paper, Autoimmune thyroiditis (67%)67% related to the paper, Papillary thyroid cancer (66%)66% related to the paper
View PDF
164 Citations
open accessOpen access Journal Article DOI: 10.1155/2012/618985
Differentiated thyroid cancer: management of patients with radioiodine nonresponsive disease.
Naifa L. Busaidy1, Maria E. Cabanillas

Abstract:

Differentiated thyroid carcinoma (papillary and follicular) has a favorable prognosis with an 85% 10-year survival. The patients that recur often require surgery and further radioactive iodine to render them disease-free. Five percent of thyroid cancer patients, however, will eventually succumb to their disease. Metastatic th... Differentiated thyroid carcinoma (papillary and follicular) has a favorable prognosis with an 85% 10-year survival. The patients that recur often require surgery and further radioactive iodine to render them disease-free. Five percent of thyroid cancer patients, however, will eventually succumb to their disease. Metastatic thyroid cancer is treated with radioactive iodine if the metastases are radioiodine avid. Cytotoxic chemotherapies for advanced or metastatic noniodine avid thyroid cancers show no prolonged responses and in general have fallen out of favor. Novel targeted therapies have recently been discovered that have given rise to clinical trials for thyroid cancer. Newer aberrations in molecular pathways and oncogenic mutations in thyroid cancer together with the role of angiogenesis in tumor growth have been central to these discoveries. This paper will focus on the management and treatment of metastatic differentiated thyroid cancers that do not take up radioactive iodine. read more read less

Topics:

Thyroid cancer (74%)74% related to the paper, Thyroid carcinoma (65%)65% related to the paper, Thyroid (62%)62% related to the paper
View PDF
161 Citations
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Frequently asked questions

1. Can I write Journal of Thyroid Research in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Thyroid Research guidelines and auto format it.

2. Do you follow the Journal of Thyroid Research guidelines?

Yes, the template is compliant with the Journal of Thyroid Research guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Thyroid Research?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Thyroid Research citation style.

4. Can I use the Journal of Thyroid Research templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Thyroid Research.

5. Can I use a manuscript in Journal of Thyroid Research that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Journal of Thyroid Research that you can download at the end.

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7. Where can I find the template for the Journal of Thyroid Research?

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Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Journal of Thyroid Research is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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After writing your paper autoformatting in Journal of Thyroid Research, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Journal of Thyroid Research's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Thyroid Research?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Thyroid Research. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Thyroid Research?

The 5 most common citation types in order of usage for Journal of Thyroid Research are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Journal of Thyroid Research?

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16. Can I download Journal of Thyroid Research in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Thyroid Research Endnote style according to Elsevier guidelines.

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