Example of Genes format
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Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format
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Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format Example of Genes format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Genes — Template for authors

Categories Rank Trend in last 3 yrs
Genetics #144 of 325 up up by 42 ranks
Genetics (clinical) #41 of 87 up up by 21 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 3556 Published Papers | 15796 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 05/07/2020
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Related Journals

open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 9.0
SJR: 3.587
SNIP: 1.457
open access Open Access

Springer

Quality:  
High
CiteRatio: 7.7
SJR: 1.945
SNIP: 0.996
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 15.2
SJR: 5.564
SNIP: 2.245
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 9.5
SJR: 2.351
SNIP: 1.869

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.4

22% from 2019

CiteRatio for Genes from 2016 - 2020
Year Value
2020 4.4
2019 3.6
2018 3.1
2017 3.3
2016 3.7
graph view Graph view
table view Table view

1.337

15% from 2019

SJR for Genes from 2016 - 2020
Year Value
2020 1.337
2019 1.564
2018 1.592
2017 1.82
2016 1.951
graph view Graph view
table view Table view

1.057

3% from 2019

SNIP for Genes from 2016 - 2020
Year Value
2020 1.057
2019 1.029
2018 0.882
2017 0.921
2016 0.795
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 22% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 15% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 3% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Genes

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Multidisciplinary Digital Publishing Institute

Genes

Genes (ISSN 2073-4425) is an international, peer-reviewed open access journal which provides an advanced forum for studies related to genes, genetics and genomics. It publishes reviews, research articles, communications and technical notes. There is no restriction on the lengt...... Read More

DNA

i
Last updated on
05 Jul 2020
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ISSN
2073-4425
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Impact Factor
Very High - 3.6
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Acceptance Rate
Not provided
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Frequency
Not provided
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Open Access
No
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
MDPI Custom Citation
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder, G.E.; Tinkham, M.; Klapwijk, T.M. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 1982, 25, 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3390/GENES6010087
Genetics of Type 2 Diabetes—Pitfalls and Possibilities
Rashmi B. Prasad1, Leif Groop2, Leif Groop1
12 Mar 2015 - Genes

Abstract:

Type 2 diabetes (T2D) is a complex disease that is caused by a complex interplay between genetic, epigenetic and environmental factors. While the major environmental factors, diet and activity level, are well known, identification of the genetic factors has been a challenge. However, recent years have seen an explosion of gen... Type 2 diabetes (T2D) is a complex disease that is caused by a complex interplay between genetic, epigenetic and environmental factors. While the major environmental factors, diet and activity level, are well known, identification of the genetic factors has been a challenge. However, recent years have seen an explosion of genetic variants in risk and protection of T2D due to the technical development that has allowed genome-wide association studies and next-generation sequencing. Today, more than 120 variants have been convincingly replicated for association with T2D and many more with diabetes-related traits. Still, these variants only explain a small proportion of the total heritability of T2D. In this review, we address the possibilities to elucidate the genetic landscape of T2D as well as discuss pitfalls with current strategies to identify the elusive unknown heritability including the possibility that our definition of diabetes and its subgroups is imprecise and thereby makes the identification of genetic causes difficult. read more read less

Topics:

Genome-wide association study (55%)55% related to the paper
View PDF
359 Citations
open accessOpen access Journal Article DOI: 10.3390/GENES10100771
Transcription Factors Associated with Abiotic and Biotic Stress Tolerance and Their Potential for Crops Improvement
Elamin Hafiz Baillo1, Roy Njoroge Kimotho1, Zhengbin Zhang1, Ping Xu1
30 Sep 2019 - Genes

Abstract:

In field conditions, crops are adversely affected by a wide range of abiotic stresses including drought, cold, salt, and heat, as well as biotic stresses including pests and pathogens. These stresses can have a marked effect on crop yield. The present and future effects of climate change necessitate the improvement of crop st... In field conditions, crops are adversely affected by a wide range of abiotic stresses including drought, cold, salt, and heat, as well as biotic stresses including pests and pathogens. These stresses can have a marked effect on crop yield. The present and future effects of climate change necessitate the improvement of crop stress tolerance. Plants have evolved sophisticated stress response strategies, and genes that encode transcription factors (TFs) that are master regulators of stress-responsive genes are excellent candidates for crop improvement. Related examples in recent studies include TF gene modulation and overexpression approaches in crop species to enhance stress tolerance. However, much remains to be discovered about the diverse plant TFs. Of the >80 TF families, only a few, such as NAC, MYB, WRKY, bZIP, and ERF/DREB, with vital roles in abiotic and biotic stress responses have been intensively studied. Moreover, although significant progress has been made in deciphering the roles of TFs in important cereal crops, fewer TF genes have been elucidated in sorghum. As a model drought-tolerant crop, sorghum research warrants further focus. This review summarizes recent progress on major TF families associated with abiotic and biotic stress tolerance and their potential for crop improvement, particularly in sorghum. Other TF families and non-coding RNAs that regulate gene expression are discussed briefly. Despite the emphasis on sorghum, numerous examples from wheat, rice, maize, and barley are included. Collectively, the aim of this review is to illustrate the potential application of TF genes for stress tolerance improvement and the engineering of resistant crops, with an emphasis on sorghum. read more read less

Topics:

Biotic stress (65%)65% related to the paper
285 Citations
open accessOpen access Journal Article DOI: 10.3390/GENES4020152
Signaling Pathways in Exosomes Biogenesis, Secretion and Fate
28 Mar 2013 - Genes

Abstract:

Exosomes are small extracellular vesicles (30–100 nm) derived from the endosomal system, which have raised considerable interest in the last decade. Several studies have shown that they mediate cell-to-cell communication in a variety of biological processes. Thus, in addition to cell-to-cell direct interaction or secretion of... Exosomes are small extracellular vesicles (30–100 nm) derived from the endosomal system, which have raised considerable interest in the last decade. Several studies have shown that they mediate cell-to-cell communication in a variety of biological processes. Thus, in addition to cell-to-cell direct interaction or secretion of active molecules, they are now considered another class of signal mediators. Exosomes can be secreted by several cell types and retrieved in many body fluids, such as blood, urine, saliva and cerebrospinal fluid. In addition to proteins and lipids, they also contain nucleic acids, namely mRNA and miRNA. These features have prompted extensive research to exploit them as a source of biomarkers for several pathologies, such as cancer and neurodegenerative disorders. In this context, exosomes also appear attractive as gene delivery vehicles. Furthermore, exosome immunomodulatory and regenerative properties are also encouraging their application for further therapeutic purposes. Nevertheless, several issues remain to be addressed: exosome biogenesis and secretion mechanisms have not been clearly understood, and physiological functions, as well as pathological roles, are far from being satisfactorily elucidated. read more read less

Topics:

Exosome (67%)67% related to the paper, Microvesicles (54%)54% related to the paper
View PDF
283 Citations
open accessOpen access Journal Article DOI: 10.3390/GENES8060148
CpG and Non-CpG Methylation in Epigenetic Gene Regulation and Brain Function.
Hyun Sik Jang1, Woo Jung Shin1, Jeong Eon Lee1, Jeong Tae Do1
23 May 2017 - Genes

Abstract:

DNA methylation is a major epigenetic mark with important roles in genetic regulation. Methylated cytosines are found primarily at CpG dinucleotides, but are also found at non-CpG sites (CpA, CpT, and CpC). The general functions of CpG and non-CpG methylation include gene silencing or activation depending on the methylated re... DNA methylation is a major epigenetic mark with important roles in genetic regulation. Methylated cytosines are found primarily at CpG dinucleotides, but are also found at non-CpG sites (CpA, CpT, and CpC). The general functions of CpG and non-CpG methylation include gene silencing or activation depending on the methylated regions. CpG and non-CpG methylation are found throughout the whole genome, including repetitive sequences, enhancers, promoters, and gene bodies. Interestingly, however, non-CpG methylation is restricted to specific cell types, such as pluripotent stem cells, oocytes, neurons, and glial cells. Thus, accumulation of methylation at non-CpG sites and CpG sites in neurons seems to be involved in development and disease etiology. Here, we provide an overview of CpG and non-CpG methylation and their roles in neurological diseases. read more read less

Topics:

Epigenetics of physical exercise (74%)74% related to the paper, DNA methylation (69%)69% related to the paper, Differentially methylated regions (68%)68% related to the paper, CpG site (66%)66% related to the paper, RNA-Directed DNA Methylation (65%)65% related to the paper
View PDF
267 Citations
open accessOpen access Journal Article DOI: 10.3390/GENES8020065
Advances in Non-Viral DNA Vectors for Gene Therapy.
Cinnamon L Hardee1, Lirio Milenka Arevalo-Soliz1, Benjamin D Hornstein1, Lynn Zechiedrich
10 Feb 2017 - Genes

Abstract:

Uses of viral vectors have thus far eclipsed uses of non-viral vectors for gene therapy delivery in the clinic. Viral vectors, however, have certain issues involving genome integration, the inability to be delivered repeatedly, and possible host rejection. Fortunately, development of non-viral DNA vectors has progressed stead... Uses of viral vectors have thus far eclipsed uses of non-viral vectors for gene therapy delivery in the clinic. Viral vectors, however, have certain issues involving genome integration, the inability to be delivered repeatedly, and possible host rejection. Fortunately, development of non-viral DNA vectors has progressed steadily, especially in plasmid vector length reduction, now allowing these tools to fill in specifically where viral or other non-viral vectors may not be the best options. In this review, we examine the improvements made to non-viral DNA gene therapy vectors, highlight opportunities for their further development, address therapeutic needs for which their use is the logical choice, and discuss their future expansion into the clinic. read more read less

Topics:

Vectors in gene therapy (56%)56% related to the paper, Viral vector (52%)52% related to the paper
266 Citations
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SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

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With SciSpace, you do not need a word template for Genes.

It automatically formats your research paper to Multidisciplinary Digital Publishing Institute formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

Time comparison

Time taken to format a paper and Compliance with guidelines

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Genes format uses MDPI Custom Citation citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Genes in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Genes guidelines and auto format it.

2. Do you follow the Genes guidelines?

Yes, the template is compliant with the Genes guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Genes?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Genes citation style.

4. Can I use the Genes templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Genes.

5. Can I use a manuscript in Genes that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Genes that you can download at the end.

6. How long does it usually take you to format my papers in Genes?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Genes.

7. Where can I find the template for the Genes?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Genes's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Genes's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Genes an online tool or is there a desktop version?

SciSpace's Genes is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Genes?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Genes?”

11. What is the output that I would get after using Genes?

After writing your paper autoformatting in Genes, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Genes's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Genes?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Genes. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Genes?

The 5 most common citation types in order of usage for Genes are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Genes?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Genes's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Genes in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Genes Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

Andreas Frutiger
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