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Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format
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Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format Example of Nature Reviews Neurology  format
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Nature Reviews Neurology — Template for authors

Publisher: Nature
Categories Rank Trend in last 3 yrs
Neurology (clinical) #2 of 343 -
Cellular and Molecular Neuroscience #1 of 88 -
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 303 Published Papers | 8931 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 08/06/2020
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General info
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Related Journals

open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 8.2
SJR: 3.131
SNIP: 1.18
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 23.3
SJR: 7.183
SNIP: 3.099
open access Open Access

Springer

Quality:  
High
CiteRatio: 6.0
SJR: 1.239
SNIP: 1.096
open access Open Access

Springer

Quality:  
High
CiteRatio: 5.7
SJR: 0.851
SNIP: 0.946

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

27.0

28% from 2018

Impact factor for Nature Reviews Neurology from 2016 - 2019
Year Value
2019 27.0
2018 21.155
2017 19.819
2016 20.257
graph view Graph view
table view Table view

29.5

22% from 2019

CiteRatio for Nature Reviews Neurology from 2016 - 2020
Year Value
2020 29.5
2019 24.1
2018 27.3
2017 27.9
2016 23.5
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 28% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 22% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

7.271

39% from 2019

SJR for Nature Reviews Neurology from 2016 - 2020
Year Value
2020 7.271
2019 5.247
2018 6.01
2017 5.507
2016 5.563
graph view Graph view
table view Table view

5.939

9% from 2019

SNIP for Nature Reviews Neurology from 2016 - 2020
Year Value
2020 5.939
2019 5.46
2018 4.823
2017 4.509
2016 3.984
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 39% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 9% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Nature Reviews Neurology

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Nature

Nature Reviews Neurology

Nature Reviews Neurology aims to be the premier source of reviews and commentaries for the scientific and clinical communities we serve. We strive to publish articles that are authoritative, accessible and enhanced with clearly understandable figures, tables and other display ...... Read More

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Last updated on
08 Jun 2020
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ISSN
1759-4766
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Acceptance Rate
Not Provided
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Frequency
Not Provided
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Open Access
Not Provided
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Sherpa RoMEO Archiving Policy
Yellow faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Naturemag Citation
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Citation Type
Numbered (Superscripted)
25
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Bibliography Example
Beenakker, C. W. J. Specular andreev reflection in graphene. Phys. Rev. Lett. 97, 067007 (2006). URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1038/NRNEUROL.2012.263
Apolipoprotein E and Alzheimer disease: risk, mechanisms and therapy
Chia Chan Liu1, Takahisa Kanekiyo2, Huaxi Xu1, Guojun Bu1
01 Feb 2013 - Nature Reviews Neurology

Abstract:

Apolipoprotein E (Apo-E) is a major cholesterol carrier that supports lipid transport and injury repair in the brain. APOE polymorphic alleles are the main genetic determinants of Alzheimer disease (AD) risk: individuals carrying the e4 allele are at increased risk of AD compared with those carrying the more common e3 allele,... Apolipoprotein E (Apo-E) is a major cholesterol carrier that supports lipid transport and injury repair in the brain. APOE polymorphic alleles are the main genetic determinants of Alzheimer disease (AD) risk: individuals carrying the e4 allele are at increased risk of AD compared with those carrying the more common e3 allele, whereas the e2 allele decreases risk. Presence of the APOE e4 allele is also associated with increased risk of cerebral amyloid angiopathy and age-related cognitive decline during normal ageing. Apo-E-lipoproteins bind to several cell-surface receptors to deliver lipids, and also to hydrophobic amyloid-β (Aβ) peptide, which is thought to initiate toxic events that lead to synaptic dysfunction and neurodegeneration in AD. Apo-E isoforms differentially regulate Aβ aggregation and clearance in the brain, and have distinct functions in regulating brain lipid transport, glucose metabolism, neuronal signalling, neuroinflammation, and mitochondrial function. In this Review, we describe current knowledge on Apo-E in the CNS, with a particular emphasis on the clinical and pathological features associated with carriers of different Apo-E isoforms. We also discuss Aβ-dependent and Aβ-independent mechanisms that link Apo-E4 status with AD risk, and consider how to design effective strategies for AD therapy by targeting Apo-E. read more read less

Topics:

Apolipoprotein E (61%)61% related to the paper, Alzheimer's disease (55%)55% related to the paper, Cerebral amyloid angiopathy (53%)53% related to the paper, Cognitive decline (51%)51% related to the paper, Neurodegeneration (51%)51% related to the paper
View PDF
2,463 Citations
Journal Article DOI: 10.1038/NRNEUROL.2010.4
Cerebrospinal fluid and plasma biomarkers in Alzheimer disease.
Kaj Blennow1, Harald Hampel2, Michael W. Weiner3, Henrik Zetterberg1
01 Mar 2010 - Nature Reviews Neurology

Abstract:

Intense multidisciplinary research has provided detailed knowledge of the molecular pathogenesis of Alzheimer disease (AD). This knowledge has been translated into new therapeutic strategies with putative disease-modifying effects. Several of the most promising approaches, such as amyloid-beta immunotherapy and secretase inhi... Intense multidisciplinary research has provided detailed knowledge of the molecular pathogenesis of Alzheimer disease (AD). This knowledge has been translated into new therapeutic strategies with putative disease-modifying effects. Several of the most promising approaches, such as amyloid-beta immunotherapy and secretase inhibition, are now being tested in clinical trials. Disease-modifying treatments might be at their most effective when initiated very early in the course of AD, before amyloid plaques and neurodegeneration become too widespread. Thus, biomarkers are needed that can detect AD in the predementia phase or, ideally, in presymptomatic individuals. In this Review, we present the rationales behind and the diagnostic performances of the core cerebrospinal fluid (CSF) biomarkers for AD, namely total tau, phosphorylated tau and the 42 amino acid form of amyloid-beta. These biomarkers reflect AD pathology, and are candidate markers for predicting future cognitive decline in healthy individuals and the progression to dementia in patients who are cognitively impaired. We also discuss emerging plasma and CSF biomarkers, and explore new proteomics-based strategies for identifying additional CSF markers. Furthermore, we outline the roles of CSF biomarkers in drug discovery and clinical trials, and provide perspectives on AD biomarker discovery and the validation of such markers for use in the clinic. read more read less

Topics:

Alzheimer's disease biomarkers (71%)71% related to the paper, Biomarker discovery (59%)59% related to the paper, Alzheimer's disease (53%)53% related to the paper, Cognitive decline (51%)51% related to the paper
View PDF
1,659 Citations
open accessOpen access Journal Article DOI: 10.1038/NRNEUROL.2009.215
The clinical use of structural MRI in Alzheimer disease
Giovanni B. Frisoni, Nick C. Fox1, Clifford R. Jack2, Philip Scheltens3, Paul M. Thompson4
01 Feb 2010 - Nature Reviews Neurology

Abstract:

Structural imaging based on magnetic resonance is an integral part of the clinical assessment of patients with suspected Alzheimer dementia. Prospective data on the natural history of change in structural markers from preclinical to overt stages of Alzheimer disease are radically changing how the disease is conceptualized, an... Structural imaging based on magnetic resonance is an integral part of the clinical assessment of patients with suspected Alzheimer dementia. Prospective data on the natural history of change in structural markers from preclinical to overt stages of Alzheimer disease are radically changing how the disease is conceptualized, and will influence its future diagnosis and treatment. Atrophy of medial temporal structures is now considered to be a valid diagnostic marker at the mild cognitive impairment stage. Structural imaging is also included in diagnostic criteria for the most prevalent non-Alzheimer dementias, reflecting its value in differential diagnosis. In addition, rates of whole-brain and hippocampal atrophy are sensitive markers of neurodegeneration, and are increasingly used as outcome measures in trials of potentially disease-modifying therapies. Large multicenter studies are currently investigating the value of other imaging and nonimaging markers as adjuncts to clinical assessment in diagnosis and monitoring of progression. The utility of structural imaging and other markers will be increased by standardization of acquisition and analysis methods, and by development of robust algorithms for automated assessment. read more read less

Topics:

Alzheimer's disease (52%)52% related to the paper
View PDF
1,572 Citations
open accessOpen access Journal Article DOI: 10.1038/NRNEUROL.2017.188
Blood–brain barrier breakdown in Alzheimer disease and other neurodegenerative disorders
Melanie D. Sweeney1, Abhay P. Sagare1, Berislav V. Zlokovic1
29 Jan 2018 - Nature Reviews Neurology

Abstract:

The blood-brain barrier (BBB) is a continuous endothelial membrane within brain microvessels that has sealed cell-to-cell contacts and is sheathed by mural vascular cells and perivascular astrocyte end-feet The BBB protects neurons from factors present in the systemic circulation and maintains the highly regulated CNS interna... The blood-brain barrier (BBB) is a continuous endothelial membrane within brain microvessels that has sealed cell-to-cell contacts and is sheathed by mural vascular cells and perivascular astrocyte end-feet The BBB protects neurons from factors present in the systemic circulation and maintains the highly regulated CNS internal milieu, which is required for proper synaptic and neuronal functioning BBB disruption allows influx into the brain of neurotoxic blood-derived debris, cells and microbial pathogens and is associated with inflammatory and immune responses, which can initiate multiple pathways of neurodegeneration This Review discusses neuroimaging studies in the living human brain and post-mortem tissue as well as biomarker studies demonstrating BBB breakdown in Alzheimer disease, Parkinson disease, Huntington disease, amyotrophic lateral sclerosis, multiple sclerosis, HIV-1-associated dementia and chronic traumatic encephalopathy The pathogenic mechanisms by which BBB breakdown leads to neuronal injury, synaptic dysfunction, loss of neuronal connectivity and neurodegeneration are described The importance of a healthy BBB for therapeutic drug delivery and the adverse effects of disease-initiated, pathological BBB breakdown in relation to brain delivery of neuropharmaceuticals are briefly discussed Finally, future directions, gaps in the field and opportunities to control the course of neurological diseases by targeting the BBB are presented read more read less

Topics:

Chronic traumatic encephalopathy (55%)55% related to the paper, Neurodegeneration (55%)55% related to the paper, Blood–brain barrier (53%)53% related to the paper, Human brain (51%)51% related to the paper, Alzheimer's disease (51%)51% related to the paper
View PDF
1,507 Citations
open accessOpen access Journal Article DOI: 10.1038/NRNEUROL.2011.2
Epidemiology of Alzheimer disease
Christiane Reitz1, Carol Brayne2, Richard Mayeux1
01 Mar 2011 - Nature Reviews Neurology

Abstract:

The global prevalence of dementia is estimated to be as high as 24 million, and is predicted to double every 20 years through to 2040, leading to a costly burden of disease. Alzheimer disease (AD) is the leading cause of dementia and is characterized by a progressive decline in cognitive function, which typically begins with ... The global prevalence of dementia is estimated to be as high as 24 million, and is predicted to double every 20 years through to 2040, leading to a costly burden of disease. Alzheimer disease (AD) is the leading cause of dementia and is characterized by a progressive decline in cognitive function, which typically begins with deterioration in memory. Before death, individuals with this disorder have usually become dependent on caregivers. The neuropathological hallmarks of the AD brain are diffuse and neuritic extracellular amyloid plaques-which are frequently surrounded by dystrophic neurites-and intracellular neurofibrillary tangles. These hallmark pathologies are often accompanied by the presence of reactive microgliosis and the loss of neurons, white matter and synapses. The etiological mechanisms underlying the neuropathological changes in AD remain unclear, but are probably affected by both environmental and genetic factors. Here, we provide an overview of the criteria used in the diagnosis of AD, highlighting how this disease is related to, but distinct from, normal aging. We also summarize current information relating to AD prevalence, incidence and risk factors, and review the biomarkers that may be used for risk assessment and in diagnosis. read more read less

Topics:

Dementia (58%)58% related to the paper, Alzheimer's disease (58%)58% related to the paper
View PDF
1,496 Citations
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Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Nature Reviews Neurology guidelines and auto format it.

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Yes, the template is compliant with the Nature Reviews Neurology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Nature Reviews Neurology ?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Nature Reviews Neurology citation style.

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Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Nature Reviews Neurology .

5. Can I use a manuscript in Nature Reviews Neurology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Nature Reviews Neurology that you can download at the end.

6. How long does it usually take you to format my papers in Nature Reviews Neurology ?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Nature Reviews Neurology .

7. Where can I find the template for the Nature Reviews Neurology ?

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Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Nature Reviews Neurology an online tool or is there a desktop version?

SciSpace's Nature Reviews Neurology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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After writing your paper autoformatting in Nature Reviews Neurology , you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Nature Reviews Neurology 's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Nature Reviews Neurology ?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Nature Reviews Neurology . The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Nature Reviews Neurology ?

The 5 most common citation types in order of usage for Nature Reviews Neurology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Nature Reviews Neurology Endnote style according to Elsevier guidelines.

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