Example of Clinical Science format
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Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format
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Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format Example of Clinical Science format
Sample paper formatted on SciSpace - SciSpace
This content is only for preview purposes. The original open access content can be found here.
open access Open Access
recommended Recommended

Clinical Science — Template for authors

Publisher: Portland Press
Categories Rank Trend in last 3 yrs
Medicine (all) #30 of 793 down down by 15 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 732 Published Papers | 6683 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 03/07/2020
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Related Journals

open access Open Access

Taylor and Francis

Quality:  
High
CiteRatio: 2.8
SJR: 0.806
SNIP: 1.28
open access Open Access

Springer

Quality:  
High
CiteRatio: 3.1
SJR: 0.859
SNIP: 1.433
open access Open Access

SAGE

Quality:  
High
CiteRatio: 4.5
SJR: 0.914
SNIP: 1.191
open access Open Access
recommended Recommended

SAGE

Quality:  
High
CiteRatio: 8.6
SJR: 1.669
SNIP: 1.889

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

5.223

0% from 2018

Impact factor for Clinical Science from 2016 - 2019
Year Value
2019 5.223
2018 5.237
2017 5.22
2016 4.936
graph view Graph view
table view Table view

9.1

3% from 2019

CiteRatio for Clinical Science from 2016 - 2020
Year Value
2020 9.1
2019 8.8
2018 8.9
2017 8.3
2016 8.8
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 0% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 3% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.91

6% from 2019

SJR for Clinical Science from 2016 - 2020
Year Value
2020 1.91
2019 1.794
2018 1.946
2017 2.166
2016 2.161
graph view Graph view
table view Table view

1.393

1% from 2019

SNIP for Clinical Science from 2016 - 2020
Year Value
2020 1.393
2019 1.376
2018 1.291
2017 1.264
2016 1.303
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 6% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 1% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Clinical Science

Guideline source: View

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Use of these names, trademarks and brands does not imply endorsement or affiliation. Disclaimer Notice

Portland Press

Clinical Science

The Journal publishes papers in the field of translational science and medicine, defined as the whole range of biochemical, physiological, immunological and other approaches that may have relevance to disease in humans, particularly those which explore integrative biology of s...... Read More

Medicine

i
Last updated on
03 Jul 2020
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ISSN
0143-5221
i
Impact Factor
High - 1.534
i
Acceptance Rate
20%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
unsrt
i
Citation Type
Numbered
[25]
i
Bibliography Example
C. W. J. Beenakker. Specular andreev reflection in graphene. Phys. Rev. Lett., 97(6):067007, 2006.

Top papers written in this journal

Journal Article DOI: 10.1042/CS0840407
A Novel Method for Measuring Antioxidant Capacity and its Application to Monitoring the Antioxidant Status in Premature Neonates
01 Apr 1993 - Clinical Science

Abstract:

1. A new method has been developed for measuring the total antioxidant capacity of body fluids and drug solutions, based on the absorbance of the ABTS.+ radical cation. 2. An automated method for use on a centrifugal analyser, as well as a manual method, is described. 3. The procedure has been applied to physiological antioxi... 1. A new method has been developed for measuring the total antioxidant capacity of body fluids and drug solutions, based on the absorbance of the ABTS.+ radical cation. 2. An automated method for use on a centrifugal analyser, as well as a manual method, is described. 3. The procedure has been applied to physiological antioxidant compounds and radical-scavenging drugs, and an antioxidant ranking was established based on their reactivity relative to a 1.0 mmol/l Trolox standard. 4. The Trolox equivalent antioxidant capacity of plasma from an adult reference population has been measured, and the method optimized and validated. 5. The method has been applied to investigate the total plasma antioxidant capacity of neonates and how this may be compromised in prematurity. read more read less

Topics:

Trolox equivalent antioxidant capacity (75%)75% related to the paper, Trolox (65%)65% related to the paper
2,844 Citations
Journal Article DOI: 10.1042/CS0950115
In utero programming of chronic disease
David J.P. Barker1
01 Aug 1998 - Clinical Science

Abstract:

1 Many human fetuses have to adapt to a limited supply of nutrients In doing so they permanently change their structure and metabolism 2 These 'programmed' changes may be the origins of a number of diseases in later life, including coronary heart disease and the related disorders stroke, diabetes and hypertension 3 This revie... 1 Many human fetuses have to adapt to a limited supply of nutrients In doing so they permanently change their structure and metabolism 2 These 'programmed' changes may be the origins of a number of diseases in later life, including coronary heart disease and the related disorders stroke, diabetes and hypertension 3 This review examines the evidence linking these diseases to fetal undernutrition and provides an overview of previous studies in this area read more read less
1,563 Citations
Journal Article DOI: 10.1042/CS0940557
Anti-inflammatory actions of glucocorticoids : molecular mechanisms
Peter J. Barnes1
01 Jun 1998 - Clinical Science

Abstract:

1. Glucocorticoids are widely used for the suppression of inflammation in chronic inflammatory diseases such as asthma, rheumatoid arthritis, inflammatory bowel disease and autoimmune diseases, all of which are associated with increased expression of inflammatory genes. The molecular mechanisms involved in this anti-inflammat... 1. Glucocorticoids are widely used for the suppression of inflammation in chronic inflammatory diseases such as asthma, rheumatoid arthritis, inflammatory bowel disease and autoimmune diseases, all of which are associated with increased expression of inflammatory genes. The molecular mechanisms involved in this anti-inflammatory action of glucocorticoids is discussed, particularly in asthma, which accounts for the highest clinical use of these agents. 2. Glucocorticoids bind to glucocorticoid receptors in the cytoplasm which then dimerize and translocate to the nucleus, where they bind to glucocorticoid response elements (GRE) on glucocorticoid-responsive genes, resulting in increased transcription. Glucocorticoids may increase the transcription of genes coding for anti-inflammatory proteins, including lipocortin-1, interleukin-10, interleukin-1 receptor antagonist and neutral endopeptidase, but this is unlikely to account for all of the widespread anti-inflammatory actions of glucocorticoids. 3. The most striking effect of glucocorticoids is to inhibit the expression of multiple inflammatory genes (cytokines, enzymes, receptors and adhesion molecules). This cannot be due to a direct interaction between glucocorticoid receptors and GRE, as these binding sites are absent from the promoter regions of most inflammatory genes. It is more likely to be due to a direct inhibitory interaction between activated glucocorticoid receptors and activated transcription factors, such as nuclear factor-kappa B and activator protein-1, which regulate the inflammatory gene expression. 4. It is increasingly recognized that glucocorticoids change the chromatin structure. Glucocorticoid receptors also interact with CREB-binding protein (CBP), which acts as a co-activator of transcription, binding several other transcription factors that compete for binding sites on this molecule. Increased transcription is associated with uncoiling of DNA wound around histone and this is secondary to acetylation of the histone residues by the enzymic action of CBP. Glucocorticoids may lead to deacetylation of histone, resulting in tighter coiling of DNA and reduced access of transcription factors to their binding sites, thereby suppressing gene expression. 5. Rarely patients with chronic inflammatory diseases fail to respond to glucocorticoids, although endocrine function of steroids is preserved. This may be due to excessive formation of activator protein-1 at the inflammatory site, which consumes activated glucocorticoid receptors so that they are not available for suppressing inflammatory genes. 6. This new understanding of glucocorticoid mechanisms may lead to the development of novel steroids with less risk of side effects (which are due to the endocrine and metabolic actions of steroids). 'Dissociated' steroids which are more active in transrepression (interaction with transcription factors) than transactivation (GRE binding) have now been developed. Some of the transcription factors that are inhibited by glucocorticoid, such as nuclear factor-kappa B, are also targets for novel anti-inflammatory therapies. read more read less

Topics:

Glucocorticoid receptor (67%)67% related to the paper, Transrepression (63%)63% related to the paper, PELP-1 (62%)62% related to the paper, Glucocorticoid (59%)59% related to the paper, Transcription factor (59%)59% related to the paper
1,500 Citations
open accessOpen access Journal Article DOI: 10.1042/CS20110386
Cellular and molecular mechanisms of metformin: an overview
01 Mar 2012 - Clinical Science

Abstract:

Considerable efforts have been made since the 1950s to better understand the cellular and molecular mechanisms of action of metformin, a potent antihyperglycaemic agent now recommended as the first-line oral therapy for T2D (Type 2 diabetes). The main effect of this drug from the biguanide family is to acutely decrease hepati... Considerable efforts have been made since the 1950s to better understand the cellular and molecular mechanisms of action of metformin, a potent antihyperglycaemic agent now recommended as the first-line oral therapy for T2D (Type 2 diabetes). The main effect of this drug from the biguanide family is to acutely decrease hepatic glucose production, mostly through a mild and transient inhibition of the mitochondrial respiratory chain complex I. In addition, the resulting decrease in hepatic energy status activates AMPK (AMP-activated protein kinase), a cellular metabolic sensor, providing a generally accepted mechanism for the action of metformin on hepatic gluconeogenesis. The demonstration that respiratory chain complex I, but not AMPK, is the primary target of metformin was recently strengthened by showing that the metabolic effect of the drug is preserved in liver-specific AMPK-deficient mice. Beyond its effect on glucose metabolism, metformin has been reported to restore ovarian function in PCOS (polycystic ovary syndrome), reduce fatty liver, and to lower microvascular and macrovascular complications associated with T2D. Its use has also recently been suggested as an adjuvant treatment for cancer or gestational diabetes and for the prevention in pre-diabetic populations. These emerging new therapeutic areas for metformin will be reviewed together with recent findings from pharmacogenetic studies linking genetic variations to drug response, a promising new step towards personalized medicine in the treatment of T2D. read more read less

Topics:

Metformin (61%)61% related to the paper, Mitochondrial respiratory chain (57%)57% related to the paper, Biguanide (55%)55% related to the paper, Polycystic ovary (55%)55% related to the paper, Type 2 diabetes (52%)52% related to the paper
View PDF
1,449 Citations
Journal Article DOI: 10.1042/CS20000335
The UKPDS risk engine: a model for the risk of coronary heart disease in Type II diabetes (UKPDS 56)
Richard Stevens1, Viti Kothari1, Amanda I Adler1, Irene M Stratton1, Rury R. Holman1
01 Dec 2001 - Clinical Science

Abstract:

A definitive model for predicting absolute risk of coronary heart disease (CHD) in male and female people with Type II diabetes is not yet available. This paper provides an equation for estimating the risk of new CHD events in people with Type II diabetes, based on data from 4540 U.K. Prospective Diabetes Study male and femal... A definitive model for predicting absolute risk of coronary heart disease (CHD) in male and female people with Type II diabetes is not yet available. This paper provides an equation for estimating the risk of new CHD events in people with Type II diabetes, based on data from 4540 U.K. Prospective Diabetes Study male and female patients. Unlike previously published risk equations, the model is diabetes-specific and incorporates glycaemia, systolic blood pressure and lipid levels as risk factors, in addition to age, sex, ethnic group, smoking status and time since diagnosis of diabetes. All variables included in the final model were statistically significant (P<0.001, except smoking for which P=0.0013) in likelihood ratio testing. This model provides the estimates of CHD risk required by current guidelines for the primary prevention of CHD in Type II diabetes. read more read less

Topics:

Risk factor (59%)59% related to the paper, United Kingdom Prospective Diabetes Study (58%)58% related to the paper, Absolute risk reduction (57%)57% related to the paper, Risk assessment (55%)55% related to the paper, Diabetes mellitus (52%)52% related to the paper
View PDF
1,188 Citations
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SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

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What to expect from SciSpace?

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With SciSpace, you do not need a word template for Clinical Science.

It automatically formats your research paper to Portland Press formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

Time comparison

Time taken to format a paper and Compliance with guidelines

Plagiarism Reports via Turnitin

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Using this service, researchers can compare submissions against more than 170 million scholarly articles, a database of 70+ billion current and archived web pages. How Turnitin Integration works?

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Clinical Science format uses unsrt citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Clinical Science in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Clinical Science guidelines and auto format it.

2. Do you follow the Clinical Science guidelines?

Yes, the template is compliant with the Clinical Science guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Clinical Science?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Clinical Science citation style.

4. Can I use the Clinical Science templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Clinical Science.

5. Can I use a manuscript in Clinical Science that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Clinical Science that you can download at the end.

6. How long does it usually take you to format my papers in Clinical Science?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Clinical Science.

7. Where can I find the template for the Clinical Science?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Clinical Science's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Clinical Science's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Clinical Science an online tool or is there a desktop version?

SciSpace's Clinical Science is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Clinical Science?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Clinical Science?”

11. What is the output that I would get after using Clinical Science?

After writing your paper autoformatting in Clinical Science, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Clinical Science's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Clinical Science?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Clinical Science. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Clinical Science?

The 5 most common citation types in order of usage for Clinical Science are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Clinical Science?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Clinical Science's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Clinical Science in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Clinical Science Endnote style according to Elsevier guidelines.

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Typset automatically formats your research paper to Clinical Science formatting guidelines and citation style.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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Researcher & Ex MS Word user
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