Example of ASN Neuro format
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Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format
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Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format Example of ASN Neuro format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

ASN Neuro — Template for authors

Publisher: SAGE
Categories Rank Trend in last 3 yrs
Neurology (clinical) #104 of 343 down down by 45 ranks
Neuroscience (all) #52 of 110 down down by 25 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 87 Published Papers | 433 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 21/06/2020
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Related Journals

open access Open Access

Taylor and Francis

Quality:  
High
CiteRatio: 6.4
SJR: 1.187
SNIP: 1.192
open access Open Access

Cambridge University Press

Quality:  
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CiteRatio: 4.4
SJR: 1.074
SNIP: 1.257
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Springer

Quality:  
High
CiteRatio: 8.4
SJR: 1.756
SNIP: 1.825
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 8.4
SJR: 1.549
SNIP: 1.479

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.167

54% from 2018

Impact factor for ASN Neuro from 2016 - 2019
Year Value
2019 4.167
2018 2.707
2017 3.617
2016 3.03
graph view Graph view
table view Table view

5.0

16% from 2019

CiteRatio for ASN Neuro from 2016 - 2020
Year Value
2020 5.0
2019 4.3
2018 6.8
2017 5.7
2016 5.6
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 54% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 16% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.039

13% from 2019

SJR for ASN Neuro from 2016 - 2020
Year Value
2020 1.039
2019 1.2
2018 1.398
2017 1.534
2016 1.491
graph view Graph view
table view Table view

0.755

10% from 2019

SNIP for ASN Neuro from 2016 - 2020
Year Value
2020 0.755
2019 0.843
2018 0.937
2017 0.935
2016 0.844
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 13% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 10% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

ASN Neuro

Guideline source: View

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SAGE

ASN Neuro

Approved by publishing and review experts on SciSpace, this template is built as per for ASN Neuro formatting guidelines as mentioned in SAGE author instructions. The current version was created on 21 Jun 2020 and has been used by 239 authors to write and format their manuscripts to this journal.

Clinical Neurology

General Neuroscience

Medicine

i
Last updated on
21 Jun 2020
i
ISSN
1759-0914
i
Impact Factor
Medium - 0.767
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SageV
i
Citation Type
Numbered (Superscripted)
25
i
Bibliography Example
Blonder GE, Tinkham M and Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 1982; 25(7): 4515–4532. URL 10.1103/PhysRevB.25.4515.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1042/AN20100019
Mitochondria and neuroplasticity
Aiwu Cheng1, Yan Hou1, Mark P. Mattson1, Mark P. Mattson2
04 Oct 2010 - Asn Neuro

Abstract:

The production of neurons from neural progenitor cells, the growth of axons and dendrites and the formation and reorganization of synapses are examples of neuroplasticity. These processes are regulated by cell-autonomous and intercellular (paracrine and endocrine) programs that mediate responses of neural cells to environment... The production of neurons from neural progenitor cells, the growth of axons and dendrites and the formation and reorganization of synapses are examples of neuroplasticity. These processes are regulated by cell-autonomous and intercellular (paracrine and endocrine) programs that mediate responses of neural cells to environmental input. Mitochondria are highly mobile and move within and between subcellular compartments involved in neuroplasticity (synaptic terminals, dendrites, cell body and the axon). By generating energy (ATP and NAD+), and regulating subcellular Ca2+ and redox homoeostasis, mitochondria may play important roles in controlling fundamental processes in neuroplasticity, including neural differentiation, neurite outgrowth, neurotransmitter release and dendritic remodelling. Particularly intriguing is emerging data suggesting that mitochondria emit molecular signals (e.g. reactive oxygen species, proteins and lipid mediators) that can act locally or travel to distant targets including the nuc... read more read less

Topics:

Neurite (55%)55% related to the paper, Neurogenesis (51%)51% related to the paper, Axon (50%)50% related to the paper
View PDF
317 Citations
open accessOpen access Journal Article DOI: 10.1042/AN20090007
Leucine-rich repeat kinase 2 mutations and Parkinson's disease: three questions.
Elisa Greggio1, Mark R. Cookson1
14 Apr 2009 - Asn Neuro

Abstract:

Mutations in the gene encoding LRRK2 (leucine-rich repeat kinase 2) were first identified in 2004 and have since been shown to be the single most common cause of inherited Parkinson's disease. The protein is a large GTP-regulated serine/threonine kinase that additionally contains several protein-protein interaction domains. I... Mutations in the gene encoding LRRK2 (leucine-rich repeat kinase 2) were first identified in 2004 and have since been shown to be the single most common cause of inherited Parkinson's disease. The protein is a large GTP-regulated serine/threonine kinase that additionally contains several protein-protein interaction domains. In the present review, we discuss three important, but unresolved, questions concerning LRRK2. We first ask: what is the normal function of LRRK2? Related to this, we discuss the evidence of LRRK2 activity as a GTPase and as a kinase and the available data on protein-protein interactions. Next we raise the question of how mutations affect LRRK2 function, focusing on some slightly controversial results related to the kinase activity of the protein in a variety of in vitro systems. Finally, we discuss what the possible mechanisms are for LRRK2-mediated neurotoxicity, in the context of known activities of the protein. read more read less

Topics:

Kinase activity (66%)66% related to the paper, LRRK2 (66%)66% related to the paper, ASK1 (61%)61% related to the paper, MAP2K7 (61%)61% related to the paper, MAP3K7 (60%)60% related to the paper
View PDF
276 Citations
open accessOpen access Journal Article DOI: 10.1042/AN20110061
Astroglial Excitability and Gliotransmission: An Appraisal of Ca2+ as a Signalling Route
22 Mar 2012 - Asn Neuro

Abstract:

Astroglial cells, due to their passive electrical properties, were long considered subservient to neurons and to merely provide the framework and metabolic support of the brain. Although astrocytes do play such structural and housekeeping roles in the brain, these glial cells also contribute to the brain's computational power... Astroglial cells, due to their passive electrical properties, were long considered subservient to neurons and to merely provide the framework and metabolic support of the brain. Although astrocytes do play such structural and housekeeping roles in the brain, these glial cells also contribute to the brain's computational power and behavioural output. These more active functions are endowed by the Ca(2+)-based excitability displayed by astrocytes. An increase in cytosolic Ca(2+) levels in astrocytes can lead to the release of signalling molecules, a process termed gliotransmission, via the process of regulated exocytosis. Dynamic components of astrocytic exocytosis include the vesicular-plasma membrane secretory machinery, as well as the vesicular traffic, which is governed not only by general cytoskeletal elements but also by astrocyte-specific IFs (intermediate filaments). Gliotransmitters released into the ECS (extracellular space) can exert their actions on neighbouring neurons, to modulate synaptic transmission and plasticity, and to affect behaviour by modulating the sleep homoeostat. Besides these novel physiological roles, astrocytic Ca(2+) dynamics, Ca(2+)-dependent gliotransmission and astrocyte-neuron signalling have been also implicated in brain disorders, such as epilepsy. The aim of this review is to highlight the newer findings concerning Ca(2+) signalling in astrocytes and exocytotic gliotransmission. For this we report on Ca(2+) sources and sinks that are necessary and sufficient for regulating the exocytotic release of gliotransmitters and discuss secretory machinery, secretory vesicles and vesicle mobility regulation. Finally, we consider the exocytotic gliotransmission in the modulation of synaptic transmission and plasticity, as well as the astrocytic contribution to sleep behaviour and epilepsy. read more read less

Topics:

Exocytosis (51%)51% related to the paper
View PDF
266 Citations
open accessOpen access Journal Article DOI: 10.1042/AN20120010
Neurological diseases as primary gliopathies: a reassessment of neurocentrism.
05 Apr 2012 - Asn Neuro

Abstract:

Diseases of the human brain are almost universally attributed to malfunction or loss of nerve cells. However, a considerable amount of work has, during the last decade, expanded our view on the role of astrocytes in CNS (central nervous system), and this analysis suggests that astrocytes contribute to both initiation and prop... Diseases of the human brain are almost universally attributed to malfunction or loss of nerve cells. However, a considerable amount of work has, during the last decade, expanded our view on the role of astrocytes in CNS (central nervous system), and this analysis suggests that astrocytes contribute to both initiation and propagation of many (if not all) neurological diseases. Astrocytes provide metabolic and trophic support to neurons and oligodendrocytes. Here, we shall endeavour a broad overviewing of the progress in the field and forward the idea that loss of homoeostatic astroglial function leads to an acute loss of neurons in the setting of acute insults such as ischaemia, whereas more subtle dysfunction of astrocytes over periods of months to years contributes to epilepsy and to progressive loss of neurons in neurodegenerative diseases. The majority of therapeutic drugs currently in clinical use target neuronal receptors, channels or transporters. Future therapeutic efforts may benefit by a stronger focus on the supportive homoeostatic functions of astrocytes. read more read less

Topics:

Gliosis (51%)51% related to the paper
View PDF
234 Citations
open accessOpen access Journal Article DOI: 10.1042/AN20110014
Current review of in vivo GBM rodent models: emphasis on the CNS-1 tumour model.
Valerie L. Jacobs1, Pablo A. Valdés1, William F. Hickey1, Joyce A. De Leo1, Joyce A. De Leo2
03 Aug 2011 - Asn Neuro

Abstract:

GBM (glioblastoma multiforme) is a highly aggressive brain tumour with very poor prognosis despite multi-modalities of treatment. Furthermore, recent failure of targeted therapy for these tumours highlights the need of appropriate rodent models for preclinical studies. In this review, we highlight the most commonly used roden... GBM (glioblastoma multiforme) is a highly aggressive brain tumour with very poor prognosis despite multi-modalities of treatment. Furthermore, recent failure of targeted therapy for these tumours highlights the need of appropriate rodent models for preclinical studies. In this review, we highlight the most commonly used rodent models (U251, U86, GL261, C6, 9L and CNS-1) with a focus on the pathological and genetic similarities to the human disease. We end with a comprehensive review of the CNS-1 rodent model. read more read less

Topics:

U87 (53%)53% related to the paper
View PDF
221 Citations
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ASN Neuro format uses SageV citation style.

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Frequently asked questions

1. Can I write ASN Neuro in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the ASN Neuro guidelines and auto format it.

2. Do you follow the ASN Neuro guidelines?

Yes, the template is compliant with the ASN Neuro guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in ASN Neuro?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the ASN Neuro citation style.

4. Can I use the ASN Neuro templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for ASN Neuro.

5. Can I use a manuscript in ASN Neuro that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper ASN Neuro that you can download at the end.

6. How long does it usually take you to format my papers in ASN Neuro?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in ASN Neuro.

7. Where can I find the template for the ASN Neuro?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per ASN Neuro's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the ASN Neuro's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. ASN Neuro an online tool or is there a desktop version?

SciSpace's ASN Neuro is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like ASN Neuro?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like ASN Neuro?”

11. What is the output that I would get after using ASN Neuro?

After writing your paper autoformatting in ASN Neuro, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is ASN Neuro's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for ASN Neuro?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for ASN Neuro. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In ASN Neuro?

The 5 most common citation types in order of usage for ASN Neuro are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the ASN Neuro?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per ASN Neuro's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download ASN Neuro in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in ASN Neuro Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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