Example of Basic Research in Cardiology format
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Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format
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Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format Example of Basic Research in Cardiology format
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open access Open Access
recommended Recommended

Basic Research in Cardiology — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Cardiology and Cardiovascular Medicine #8 of 317 up up by 20 ranks
Physiology (medical) #4 of 98 up up by 8 ranks
Physiology #7 of 169 up up by 11 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 222 Published Papers | 3826 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 30/06/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

11.981

85% from 2018

Impact factor for Basic Research in Cardiology from 2016 - 2019
Year Value
2019 11.981
2018 6.47
2017 5.723
2016 5.306
graph view Graph view
table view Table view

17.2

18% from 2019

CiteRatio for Basic Research in Cardiology from 2016 - 2020
Year Value
2020 17.2
2019 14.6
2018 10.7
2017 8.8
2016 10.2
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 85% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 18% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.596

14% from 2019

SJR for Basic Research in Cardiology from 2016 - 2020
Year Value
2020 1.596
2019 1.397
2018 1.659
2017 2.468
2016 2.32
graph view Graph view
table view Table view

1.811

6% from 2019

SNIP for Basic Research in Cardiology from 2016 - 2020
Year Value
2020 1.811
2019 1.716
2018 1.158
2017 1.346
2016 1.424
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 14% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 6% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Basic Research in Cardiology

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Springer

Basic Research in Cardiology

Basic Research in Cardiology is an international journal for cardiovascular research. It provides a forum for original and review articles related to experimental cardiology that meet its stringent scientific standards. Thus, it comprises all aspects related to the physiology ...... Read More

Medicine

i
Last updated on
29 Jun 2020
i
ISSN
0300-8428
i
Impact Factor
High - 1.454
i
Acceptance Rate
27%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
i
Bibliography Name
SPBASIC
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Citation Type
Author Year
(Blonder et al, 1982)
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Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1007/BF01907940
The role of lipid peroxidation in pathogenesis of ischemic damage and the antioxidant protection of the heart.

Abstract:

A working hypothesis on pathogenesis of ischemic heart damage has been proposed. According to this hypothesis, a crucial role in conversion of reversible damage into irreversible damage is played by cardiomyocyte membrane destruction caused by the so-called “lipid triad”. The latter comprises activation of lipid peroxidation,... A working hypothesis on pathogenesis of ischemic heart damage has been proposed. According to this hypothesis, a crucial role in conversion of reversible damage into irreversible damage is played by cardiomyocyte membrane destruction caused by the so-called “lipid triad”. The latter comprises activation of lipid peroxidation, activation of phospholipases, and the degergentlike action of excessive amounts of free fatty acids and lysophospholipids. Marked activation of lipid peroxidation in experimental myocardial infarction, as well as reoxygenation following transitory ischemia, have been demonstrated. The proposed hypothesis and experimental data underly successful application of synthetic free radical scavengers (antioxidants) for heart protection against experimental myocardial infarction, transitory ischemia, and emotional, painful stress. read more read less

Topics:

Lipid peroxidation (57%)57% related to the paper, Ischemia (53%)53% related to the paper
501 Citations
Journal Article DOI: 10.1007/BF00788278
Quantitative assessment of myocardial collagen with picrosirius red staining and circularly polarized light.
Peter Whittaker1, Robert A. Kloner1, Derek R. Boughner2, J. G. Pickering2

Abstract:

Collagen plays a major role in the structural organization of the heart and therefore direct visualization of collagen fibers is a crucial component of cardiac analysis. Although linearly polarized light has proven an effective tool for the examination of myocardial collagen in histologic sections, the use of circularly polar... Collagen plays a major role in the structural organization of the heart and therefore direct visualization of collagen fibers is a crucial component of cardiac analysis. Although linearly polarized light has proven an effective tool for the examination of myocardial collagen in histologic sections, the use of circularly polarized light may offer advantages and additional possibilities. We examined the potential enhancement of collagen analysis using circularly polarized light in two ways. We first measured the brightness, and hence indirectly assessed the birefringence, of collagen fibers in scars examined at different times after myocardial infarction. Secondly, we measured collagen content in myocardial tissue and compared results obtained from brightfield analysis of trichrome stained sections with those obtained from circularly polarized light analysis of picrosirius red stained sections. We observed a progressive increase in the maximum brightness of collagen fibers in the scar with time, and a time-dependent shift in the relative distribution of collagen fiber brightness from lower to higher levels. We found consistently lower values of collagen content in trichrome stained versus picrosirius red stained tissue, and concluded that trichrome staining underestimated collagen content. The information provided by these studies could not be obtained by brightfield analysis and could be only partially obtained from linearly polarized light analysis. Thus, analysis using circularly polarized light has the ability to enhance histologic assessment of tissue and can provide additional insights into the composition and structure of myocardial collagen. read more read less

Topics:

Trichrome (58%)58% related to the paper, Masson's trichrome stain (53%)53% related to the paper, Polarized light microscopy (51%)51% related to the paper
485 Citations
open accessOpen access Journal Article DOI: 10.1007/S00395-008-0760-X
Collateral circulation: past and present.

Abstract:

Following an arterial occlusion outward remodeling of pre-existent inter-connecting arterioles occurs by proliferation of vascular smooth muscle and endothelial cells. This is initiated by deformation of the endothelial cells through increased pulsatile fluid shear stress (FSS) caused by the steep pressure gradient between th... Following an arterial occlusion outward remodeling of pre-existent inter-connecting arterioles occurs by proliferation of vascular smooth muscle and endothelial cells. This is initiated by deformation of the endothelial cells through increased pulsatile fluid shear stress (FSS) caused by the steep pressure gradient between the high pre-occlusive and the very low post-occlusive pressure regions that are interconnected by collateral vessels. Shear stress leads to the activation and expression of all NOS isoforms and NO production, followed by endothelial VEGF secretion, which induces MCP-1 synthesis in endothelium and in the smooth muscle of the media. This leads to attraction and activation of monocytes and T-cells into the adventitial space (peripheral collateral vessels) or attachment of these cells to the endothelium (coronary collaterals). Mononuclear cells produce proteases and growth factors to digest the extra-cellular scaffold and allow motility and provide space for the new cells. They also produce NO from iNOS, which is essential for arteriogenesis. The bulk of new tissue production is carried by the smooth muscles of the media, which transform their phenotype from a contractile into a synthetic and proliferative one. Important roles are played by actin binding proteins like ABRA, cofilin, and thymosin beta 4 which determine actin polymerization and maturation. Integrins and connexins are markedly up-regulated. A key role in this concerted action which leads to a 2-to-20 fold increase in vascular diameter, depending on species size (mouse versus human) are the transcription factors AP-1, egr-1, carp, ets, by the Rho pathway and by the Mitogen Activated Kinases ERK-1 and -2. In spite of the enormous increase in tissue mass (up to 50-fold) the degree of functional restoration of blood flow capacity is incomplete and ends at 30% of maximal conductance (coronary) and 40% in the vascular periphery. The process of arteriogenesis can be drastically stimulated by increases in FSS (arterio-venous fistulas) and can be completely blocked by inhibition of NO production, by pharmacological blockade of VEGF-A and by the inhibition of the Rho-pathway. Pharmacological stimulation of arteriogenesis, important for the treatment of arterial occlusive diseases, seems feasible with NO donors. read more read less

Topics:

Arteriogenesis (60%)60% related to the paper, Vascular smooth muscle (59%)59% related to the paper, Endothelium (57%)57% related to the paper, Growth factor (51%)51% related to the paper, Actin (51%)51% related to the paper
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341 Citations
open accessOpen access Journal Article DOI: 10.1007/S00395-008-0733-0
The role of inflammatory cytokines in endothelial dysfunction
Cuihua Zhang1

Abstract:

Clinical and experimental data support a link between endothelial dysfunction and inflammation. Inflammatory cytokines are important protagonists in formation of atherosclerotic plaque, eliciting effects throughout the atherosclerotic vessel. Importantly, the development of atherosclerotic lesions, regardless of the risk fact... Clinical and experimental data support a link between endothelial dysfunction and inflammation. Inflammatory cytokines are important protagonists in formation of atherosclerotic plaque, eliciting effects throughout the atherosclerotic vessel. Importantly, the development of atherosclerotic lesions, regardless of the risk factor, e.g., diabetes, hypertension, obesity, is characterized by disruption in normal function of the endothelial cells. Endothelial cells, which line the internal lumen of the vasculature, are part of a complex system that regulates vasodilation and vasoconstriction, growth of vascular smooth muscle cells, inflammation, and hemostasis, maintaining a proper blood supply to tissues and regulating inflammation and coagulation. Current concepts suggest that the earliest event in atherogenesis is endothelial dysfunction, manifested by deficiencies in the production of nitric oxide (NO) and prostacyclin. The focus of this review is to summarize recent evidence showing the effects of inflammation on vascular dysfunction in ischemic-heart disease, which may prompt new directions for targeting inflammation in future therapies. read more read less

Topics:

Endothelial dysfunction (60%)60% related to the paper, Endothelium (60%)60% related to the paper, Inflammation (57%)57% related to the paper, Proinflammatory cytokine (56%)56% related to the paper, Hemostasis (54%)54% related to the paper
View PDF
339 Citations
open accessOpen access Journal Article DOI: 10.1007/S00395-010-0124-1
Inhibition of permeability transition pore opening by mitochondrial STAT3 and its role in myocardial ischemia/reperfusion
Kerstin Boengler, Denise Hilfiker-Kleiner1, Gerd Heusch, Rainer Schulz

Abstract:

The signal transducer and activator of transcription 3 (STAT3) contributes to cardioprotection by ischemic pre- and postconditioning. Mitochondria are central elements of cardioprotective signaling, most likely by delaying mitochondrial permeability transition pore (MPTP) opening, and STAT3 has recently been identified in mit... The signal transducer and activator of transcription 3 (STAT3) contributes to cardioprotection by ischemic pre- and postconditioning. Mitochondria are central elements of cardioprotective signaling, most likely by delaying mitochondrial permeability transition pore (MPTP) opening, and STAT3 has recently been identified in mitochondria. We now characterized the mitochondrial localization of STAT3 and its impact on respiration and MPTP opening. STAT3 was mainly present in the matrix of subsarcolemmal and interfibrillar cardiomyocyte mitochondria. STAT1, but not STAT5 was also detected in mitochondria under physiological conditions. ADP-stimulated respiration was reduced in mitochondria from mice with a cardiomyocyte-specific deletion of STAT3 (STAT3-KO) versus wildtypes and in rat mitochondria treated with the STAT3 inhibitor Stattic (STAT3 inhibitory compound, 6-Nitrobenzo[b]thiophene 1,1-dioxide). Mitochondria from STAT3-KO mice and Stattic-treated rat mitochondria tolerated less calcium until MPTP opening occurred. STAT3 co-immunoprecipitated with cyclophilin D, the target of the cardioprotective agent and MPTP inhibitor cyclosporine A (CsA). However, CsA reduced infarct size to a similar extent in wildtype and STAT3-KO mice in vivo. Thus, STAT3 possibly contributes to cardioprotection by stimulation of respiration and inhibition of MPTP opening. read more read less

Topics:

Mitochondrial permeability transition pore (61%)61% related to the paper, Mitochondrial membrane transport protein (56%)56% related to the paper, Cardioprotection (55%)55% related to the paper, MPTP (53%)53% related to the paper, Cellular respiration (52%)52% related to the paper
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330 Citations
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Frequently asked questions

1. Can I write Basic Research in Cardiology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Basic Research in Cardiology guidelines and auto format it.

2. Do you follow the Basic Research in Cardiology guidelines?

Yes, the template is compliant with the Basic Research in Cardiology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Basic Research in Cardiology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Basic Research in Cardiology citation style.

4. Can I use the Basic Research in Cardiology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Basic Research in Cardiology.

5. Can I use a manuscript in Basic Research in Cardiology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Basic Research in Cardiology that you can download at the end.

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7. Where can I find the template for the Basic Research in Cardiology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Basic Research in Cardiology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

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Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Basic Research in Cardiology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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11. What is the output that I would get after using Basic Research in Cardiology?

After writing your paper autoformatting in Basic Research in Cardiology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Basic Research in Cardiology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Basic Research in Cardiology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Basic Research in Cardiology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Basic Research in Cardiology?

The 5 most common citation types in order of usage for Basic Research in Cardiology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Basic Research in Cardiology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Basic Research in Cardiology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Basic Research in Cardiology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Basic Research in Cardiology Endnote style according to Elsevier guidelines.

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