Example of Cancer Microenvironment format
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Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format
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Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format Example of Cancer Microenvironment format
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open access Open Access

Cancer Microenvironment — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Oncology #124 of 340 down down by 33 ranks
Cancer Research #110 of 207 down down by 30 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 39 Published Papers | 191 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 28/06/2020
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Related Journals

open access Open Access
recommended Recommended

Nature

Quality:  
High
CiteRatio: 16.0
SJR: 4.539
SNIP: 2.28
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American Association for Cancer Research

Quality:  
High
CiteRatio: 15.8
SJR: 4.103
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American Association for Cancer Research

Quality:  
High
CiteRatio: 18.2
SJR: 5.427
SNIP: 2.243
open access Open Access

American Association for Cancer Research

Quality:  
High
CiteRatio: 8.7
SJR: 2.273
SNIP: 1.157

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.9

4% from 2019

CiteRatio for Cancer Microenvironment from 2016 - 2020
Year Value
2020 4.9
2019 4.7
2018 6.6
2017 5.8
2016 6.5
graph view Graph view
table view Table view

0.626

29% from 2019

SJR for Cancer Microenvironment from 2016 - 2020
Year Value
2020 0.626
2019 0.877
2018 1.759
2017 1.457
2016 1.439
graph view Graph view
table view Table view

0.631

8% from 2019

SNIP for Cancer Microenvironment from 2016 - 2020
Year Value
2020 0.631
2019 0.685
2018 1.061
2017 0.608
2016 0.822
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 4% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 29% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 8% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Cancer Microenvironment

Guideline source: View

All company, product and service names used in this website are for identification purposes only. All product names, trademarks and registered trademarks are property of their respective owners.

Use of these names, trademarks and brands does not imply endorsement or affiliation. Disclaimer Notice

Springer

Cancer Microenvironment

Cancer Microenvironment is the official journal of the International Cancer Microenvironment Society (ICMS). It publishes original studies in all aspects of basic, clinical and translational research devoted to the study of cancer microenvironment. Topics include: Regulation o...... Read More

Oncology

Cancer Research

Medicine

i
Last updated on
28 Jun 2020
i
ISSN
1875-2292
i
Impact Factor
Medium - 0.806
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
i
Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1007/S12307-014-0147-5
The Multifaceted Roles Neutrophils Play in the Tumor Microenvironment
Ronit Vogt Sionov1, Zvi G. Fridlender1, Zvi Granot1
01 Dec 2015 - Cancer Microenvironment

Abstract:

Neutrophils are myeloid cells that constitute 50–70 % of all white blood cells in the human circulation. Traditionally, neutrophils are viewed as the first line of defense against infections and as a major component of the inflammatory process. In addition, accumulating evidence suggest that neutrophils may also play a key ro... Neutrophils are myeloid cells that constitute 50–70 % of all white blood cells in the human circulation. Traditionally, neutrophils are viewed as the first line of defense against infections and as a major component of the inflammatory process. In addition, accumulating evidence suggest that neutrophils may also play a key role in multiple aspects of cancer biology. The possible involvement of neutrophils in cancer prevention and promotion was already suggested more than half a century ago, however, despite being the major component of the immune system, their contribution has often been overshadowed by other immune components such as lymphocytes and macrophages. Neutrophils seem to have conflicting functions in cancer and can be classified into anti-tumor (N1) and pro-tumor (N2) sub-populations. The aim of this review is to discuss the varying nature of neutrophil function in the cancer microenvironment with a specific emphasis on the mechanisms that regulate neutrophil mobilization, recruitment and activation. read more read less

Topics:

Tumor microenvironment (52%)52% related to the paper, Chemokine (50%)50% related to the paper
View PDF
311 Citations
open accessOpen access Journal Article DOI: 10.1007/S12307-010-0052-5
Tumor-associated Macrophages (TAM) and Inflammation in Colorectal Cancer
Marco Erreni, Alberto Mantovani1, Paola Allavena
01 Aug 2011 - Cancer Microenvironment

Abstract:

Experimental and epidemiological studies indicate a strong link between chronic inflammation and tumor progression. Human colorectal cancer (CRC), a major cause of cancer-related death in Western countries, represents a paradigm for this link. Key features of cancer-related inflammation in CRC are the activation of transcript... Experimental and epidemiological studies indicate a strong link between chronic inflammation and tumor progression. Human colorectal cancer (CRC), a major cause of cancer-related death in Western countries, represents a paradigm for this link. Key features of cancer-related inflammation in CRC are the activation of transcription factors (e.g. NF-κB, STAT3), the expression of inflammatory cytokines and chemokines (e.g. TNFα, IL-6, CCL2, CXCL8) as well as a prominent leukocyte infiltrate. While considerable evidence indicates that the presence of lymphocytes of adaptive immunity may positively influence patient survival and clinical outcome in CRC, the role of tumor-associated macrophages (TAM) and of other lymphoid populations (e.g. Th17, Treg) is still unclear. In this review we will summarize the different and controversial effects that TAM play in CRC-related inflammation and progression of disease. The characterization of the most relevant inflammatory pathways in CRC is instrumental for the identification of new target molecules that could lead to improved diagnosis and treatment. read more read less

Topics:

Tumor progression (55%)55% related to the paper, Proinflammatory cytokine (54%)54% related to the paper, Tumor necrosis factor alpha (53%)53% related to the paper, Inflammation (52%)52% related to the paper
View PDF
300 Citations
open accessOpen access Journal Article DOI: 10.1007/S12307-009-0022-Y
Cancer Cells Expressing Toll-like Receptors and the Tumor Microenvironment
15 Aug 2009 - Cancer Microenvironment

Abstract:

Toll-like receptors (TLRs) play a crucial role in the innate immune response and the subsequent induction of adaptive immune responses against microbial infection or tissue injury. Recent findings show that functional TLRs are expressed not only on immune cells but also on cancer cells. TLRs play an active role in carcinogene... Toll-like receptors (TLRs) play a crucial role in the innate immune response and the subsequent induction of adaptive immune responses against microbial infection or tissue injury. Recent findings show that functional TLRs are expressed not only on immune cells but also on cancer cells. TLRs play an active role in carcinogenesis and tumor progression during chronic inflammation that involves the tumor microenvironment. Damage-associated molecular patterns (DAMPs) derived from injured normal epithelial cells and necrotic cancer cells appear to be present at significant levels in the tumor microenvironment, and their stimulation of specific TLRs can foster chronic inflammation. This review discusses how carcinogenesis, cancer progression, and site-specific metastasis are related to interactions between cancer cells, immune cells, and DAMPs through TLR activation in the tumor microenvironment. read more read less

Topics:

Tumor microenvironment (63%)63% related to the paper, Toll-like receptor (62%)62% related to the paper, Innate immune system (59%)59% related to the paper, Tumor progression (59%)59% related to the paper, Immune system (57%)57% related to the paper
View PDF
284 Citations
open accessOpen access Journal Article DOI: 10.1007/S12307-012-0127-6
Effector CD4 and CD8 T Cells and Their Role in the Tumor Microenvironment
Sine Reker Hadrup1, Marco Donia1, Per thor Straten1
01 Aug 2013 - Cancer Microenvironment

Abstract:

T cells in tumors—the so-called tumor infiltrating lymphocytes (TIL) have been studied intensively over the past years Compelling evidence point to a clinical relevance for high numbers of T cells at the tumor site with CD8 memory T cells as a key denominator for overall survival (OS) in patients with colo-rectal cancer (CRC)... T cells in tumors—the so-called tumor infiltrating lymphocytes (TIL) have been studied intensively over the past years Compelling evidence point to a clinical relevance for high numbers of T cells at the tumor site with CD8 memory T cells as a key denominator for overall survival (OS) in patients with colo-rectal cancer (CRC), and also for others solid cancers These data goes hand in hand with studies of clonality of TIL showing the T cells among TIL are expanded clonally, and also that tumor specific T cells of CD4 as well as CD8 type are enriched at the tumor site The tumor microenvironment is hostile to T cell function eg, due to expression of enzymes that depletes the amino acids tryptophan and arginine, high concentration of tumor secreted lactate, and presence innate cells or regulatory T cells both with suppressive activity Analyses of the specificity of TILs in melanoma demonstrate that quite few known antigens are in fact recognized by these cultures underscoring patient unique and/or mutated antigens may represent important target for recognition read more read less

Topics:

T cell (68%)68% related to the paper, Cytotoxic T cell (65%)65% related to the paper, Antigen-presenting cell (64%)64% related to the paper, IL-2 receptor (63%)63% related to the paper, Interleukin 21 (63%)63% related to the paper
View PDF
259 Citations
open accessOpen access Journal Article DOI: 10.1007/S12307-011-0069-4
Epithelial-Mesenchymal Transition Induced by Senescent Fibroblasts
Remi-Martin Laberge1, Pierre Awad1, Judith Campisi1, Pierre-Yves Desprez2, Pierre-Yves Desprez1
01 Apr 2012 - Cancer Microenvironment

Abstract:

Depending on the cell type and tissue environment, epithelial and mesenchymal cell phenotypes are not static and can be highly dynamic. Epithelial-mesenchymal transitions (EMTs) and reverse EMTs provide flexibility during embryogenesis. While EMTs are a critical normal process during development and wound healing, properties ... Depending on the cell type and tissue environment, epithelial and mesenchymal cell phenotypes are not static and can be highly dynamic. Epithelial-mesenchymal transitions (EMTs) and reverse EMTs provide flexibility during embryogenesis. While EMTs are a critical normal process during development and wound healing, properties of the EMT have been implicated in human pathology, particularly cancer metastasis. A normal undamaged epithelium does not typically exhibit features of an EMT. However, particularly under the influence of the surrounding microenvironment, cancer cells may reactivate developmental phenotypes out of context in the adult. This reactivation, such as the EMT, can facilitate tumor cell invasion and metastasis, and therefore is a major mechanism of tumor progression. Conversely, cellular senescence, which is associated with aging, is a process by which cells enter a state of permanent cell cycle arrest, thereby constituting a potent tumor suppressive mechanism. However, accumulating evidence shows that senescent cells can have deleterious effects on the tissue microenvironment. The most significant of these effects is the acquisition of a senescence-associated secretory phenotype (SASP) that turns senescent fibroblasts into pro-inflammatory cells having the ability to promote tumor progression, in part by inducing an EMT in nearby epithelial cells. Here, we summarize the potential impacts of SASP factors, particularly interleukins, on tissue microenvironments and their ability to stimulate tumor progression through induction of an EMT. read more read less

Topics:

Epithelial–mesenchymal transition (60%)60% related to the paper, Tumor progression (57%)57% related to the paper, Cancer cell (54%)54% related to the paper, Mesenchymal stem cell (52%)52% related to the paper, Cell type (51%)51% related to the paper
View PDF
216 Citations
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Cancer Microenvironment format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write Cancer Microenvironment in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Cancer Microenvironment guidelines and auto format it.

2. Do you follow the Cancer Microenvironment guidelines?

Yes, the template is compliant with the Cancer Microenvironment guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Cancer Microenvironment?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Cancer Microenvironment citation style.

4. Can I use the Cancer Microenvironment templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Cancer Microenvironment.

5. Can I use a manuscript in Cancer Microenvironment that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Cancer Microenvironment that you can download at the end.

6. How long does it usually take you to format my papers in Cancer Microenvironment?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Cancer Microenvironment.

7. Where can I find the template for the Cancer Microenvironment?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cancer Microenvironment's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Cancer Microenvironment's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Cancer Microenvironment an online tool or is there a desktop version?

SciSpace's Cancer Microenvironment is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Cancer Microenvironment?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Cancer Microenvironment?”

11. What is the output that I would get after using Cancer Microenvironment?

After writing your paper autoformatting in Cancer Microenvironment, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Cancer Microenvironment's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Cancer Microenvironment?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Cancer Microenvironment. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Cancer Microenvironment?

The 5 most common citation types in order of usage for Cancer Microenvironment are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Cancer Microenvironment?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cancer Microenvironment's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Cancer Microenvironment in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Cancer Microenvironment Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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