Example of Cell Communication and Signaling format
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Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format Example of Cell Communication and Signaling format
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open access Open Access

Cell Communication and Signaling — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Biochemistry #132 of 415 down down by 72 ranks
Molecular Biology #151 of 382 down down by 70 ranks
Cell Biology #122 of 279 down down by 59 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 501 Published Papers | 2919 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 07/07/2020
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Related Journals

open access Open Access
recommended Recommended

American Association for the Advancement of Science

Quality:  
High
CiteRatio: 10.6
SJR: 3.659
SNIP: 1.504
open access Open Access

Springer

Quality:  
High
CiteRatio: 6.8
SJR: 1.329
SNIP: 1.08
open access Open Access

Portland Press

Quality:  
High
CiteRatio: 6.7
SJR: 1.706
SNIP: 1.117
open access Open Access

American Society for Biochemistry and Molecular Biology (ASBMB)

Quality:  
High
CiteRatio: 7.7
SJR: 2.361
SNIP: 1.18

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.344

15% from 2018

Impact factor for Cell Communication and Signaling from 2016 - 2019
Year Value
2019 4.344
2018 5.111
2017 5.324
2016 3.943
graph view Graph view
table view Table view

5.8

26% from 2019

CiteRatio for Cell Communication and Signaling from 2016 - 2020
Year Value
2020 5.8
2019 4.6
2018 6.1
2017 7.8
2016 8.4
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 15% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 26% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.618

8% from 2019

SJR for Cell Communication and Signaling from 2016 - 2020
Year Value
2020 1.618
2019 1.762
2018 2.4
2017 2.211
2016 1.985
graph view Graph view
table view Table view

1.141

4% from 2019

SNIP for Cell Communication and Signaling from 2016 - 2020
Year Value
2020 1.141
2019 1.1
2018 1.271
2017 1.15
2016 1.177
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 8% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 4% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Cell Communication and Signaling

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Springer

Cell Communication and Signaling

Approved by publishing and review experts on SciSpace, this template is built as per for Cell Communication and Signaling formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 145 authors to write and format their manuscripts to this journal.

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Last updated on
07 Jul 2020
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ISSN
1606-8610
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder, G.E., Tinkham, M., Klapwijk, T.M.: Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25(7), 4515–4532 (1982)

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1186/1478-811X-9-12
Different populations and sources of human mesenchymal stem cells (MSC): A comparison of adult and neonatal tissue-derived MSC
Ralf Hass, Cornelia Kasper1, Stefanie Böhm1, Roland Jacobs2

Abstract:

The mesenchymal stroma harbors an important population of cells that possess stem cell-like characteristics including self renewal and differentiation capacities and can be derived from a variety of different sources. These multipotent mesenchymal stem cells (MSC) can be found in nearly all tissues and are mostly located in p... The mesenchymal stroma harbors an important population of cells that possess stem cell-like characteristics including self renewal and differentiation capacities and can be derived from a variety of different sources. These multipotent mesenchymal stem cells (MSC) can be found in nearly all tissues and are mostly located in perivascular niches. MSC have migratory abilities and can secrete protective factors and act as a primary matrix for tissue regeneration during inflammation, tissue injuries and certain cancers. These functions underlie the important physiological roles of MSC and underscore a significant potential for the clinical use of distinct populations from the various tissues. MSC derived from different adult (adipose tissue, peripheral blood, bone marrow) and neonatal tissues (particular parts of the placenta and umbilical cord) are therefore compared in this mini-review with respect to their cell biological properties, surface marker expression and proliferative capacities. In addition, several MSC functions including in vitro and in vivo differentiation capacities within a variety of lineages and immune-modulatory properties are highlighted. Differences in the extracellular milieu such as the presence of interacting neighbouring cell populations, exposure to proteases or a hypoxic microenvironment contribute to functional developments within MSC populations originating from different tissues, and intracellular conditions such as the expression levels of certain micro RNAs can additionally balance MSC function and fate. read more read less

Topics:

Clinical uses of mesenchymal stem cells (61%)61% related to the paper, Stem cell transplantation for articular cartilage repair (60%)60% related to the paper, Mesenchymal stem cell (55%)55% related to the paper, Cellular differentiation (53%)53% related to the paper, Population (51%)51% related to the paper
View PDF
1,369 Citations
open accessOpen access Journal Article DOI: 10.1186/S12964-020-0530-4
Tumor microenvironment complexity and therapeutic implications at a glance.

Abstract:

The dynamic interactions of cancer cells with their microenvironment consisting of stromal cells (cellular part) and extracellular matrix (ECM) components (non-cellular) is essential to stimulate the heterogeneity of cancer cell, clonal evolution and to increase the multidrug resistance ending in cancer cell progression and m... The dynamic interactions of cancer cells with their microenvironment consisting of stromal cells (cellular part) and extracellular matrix (ECM) components (non-cellular) is essential to stimulate the heterogeneity of cancer cell, clonal evolution and to increase the multidrug resistance ending in cancer cell progression and metastasis. The reciprocal cell-cell/ECM interaction and tumor cell hijacking of non-malignant cells force stromal cells to lose their function and acquire new phenotypes that promote development and invasion of tumor cells. Understanding the underlying cellular and molecular mechanisms governing these interactions can be used as a novel strategy to indirectly disrupt cancer cell interplay and contribute to the development of efficient and safe therapeutic strategies to fight cancer. Furthermore, the tumor-derived circulating materials can also be used as cancer diagnostic tools to precisely predict and monitor the outcome of therapy. This review evaluates such potentials in various advanced cancer models, with a focus on 3D systems as well as lab-on-chip devices. read more read less

Topics:

Tumor microenvironment (57%)57% related to the paper, Cancer cell (56%)56% related to the paper, Cancer (55%)55% related to the paper, Stromal cell (55%)55% related to the paper, Somatic evolution in cancer (54%)54% related to the paper
View PDF
693 Citations
open accessOpen access Journal Article DOI: 10.1186/1478-811X-8-31
PARP-1 cleavage fragments: signatures of cell-death proteases in neurodegeneration

Abstract:

The normal function of poly (ADP-ribose) polymerase-1 (PARP-1) is the routine repair of DNA damage by adding poly (ADP ribose) polymers in response to a variety of cellular stresses. Recently, it has become widely appreciated that PARP-1 also participates in diverse physiological and pathological functions from cell survival ... The normal function of poly (ADP-ribose) polymerase-1 (PARP-1) is the routine repair of DNA damage by adding poly (ADP ribose) polymers in response to a variety of cellular stresses. Recently, it has become widely appreciated that PARP-1 also participates in diverse physiological and pathological functions from cell survival to several forms of cell death and has been implicated in gene transcription, immune responses, inflammation, learning, memory, synaptic functions, angiogenesis and aging. In the CNS, PARP inhibition attenuates injury in pathologies like cerebral ischemia, trauma and excitotoxicity demonstrating a central role of PARP-1 in these pathologies. PARP-1 is also a preferred substrate for several 'suicidal' proteases and the proteolytic action of suicidal proteases (caspases, calpains, cathepsins, granzymes and matrix metalloproteinases (MMPs)) on PARP-1 produces several specific proteolytic cleavage fragments with different molecular weights. These PARP-1 signature fragments are recognized biomarkers for specific patterns of protease activity in unique cell death programs. This review focuses on specific suicidal proteases active towards PARP-1 to generate signature PARP-1 fragments that can identify key proteases and particular forms of cell death involved in pathophysiology. The roles played by some of the PARP-1 fragments and their associated binding partners in the control of different forms of cell death are also discussed. read more read less

Topics:

Proteases (57%)57% related to the paper, Calpain (55%)55% related to the paper, Poison control (53%)53% related to the paper, Granzyme (53%)53% related to the paper, Poly ADP ribose polymerase (53%)53% related to the paper
View PDF
688 Citations
open accessOpen access Journal Article DOI: 10.1186/1478-811X-8-23
Rac and Rho GTPases in cancer cell motility control
Matteo Parri, Paola Chiarugi1

Abstract:

Rho GTPases represent a family of small GTP-binding proteins involved in cell cytoskeleton organization, migration, transcription, and proliferation. A common theme of these processes is a dynamic reorganization of actin cytoskeleton which has now emerged as a major switch control mainly carried out by Rho and Rac GTPase subf... Rho GTPases represent a family of small GTP-binding proteins involved in cell cytoskeleton organization, migration, transcription, and proliferation. A common theme of these processes is a dynamic reorganization of actin cytoskeleton which has now emerged as a major switch control mainly carried out by Rho and Rac GTPase subfamilies, playing an acknowledged role in adaptation of cell motility to the microenvironment. Cells exhibit three distinct modes of migration when invading the 3 D environment. Collective motility leads to movement of cohorts of cells which maintain the adherens junctions and move by photolytic degradation of matrix barriers. Single cell mesenchymal-type movement is characterized by an elongated cellular shape and again requires extracellular proteolysis and integrin engagement. In addition it depends on Rac1-mediated cell polarization and lamellipodia formation. Conversely, in amoeboid movement cells have a rounded morphology, the movement is independent from proteases but requires high Rho GTPase to drive elevated levels of actomyosin contractility. These two modes of cell movement are interconvertible and several moving cells, including tumor cells, show an high degree of plasticity in motility styles shifting ad hoc between mesenchymal or amoeboid movements. This review will focus on the role of Rac and Rho small GTPases in cell motility and in the complex relationship driving the reciprocal control between Rac and Rho granting for the opportunistic motile behaviour of aggressive cancer cells. In addition we analyse the role of these GTPases in cancer progression and metastatic dissemination. read more read less

Topics:

Lamellipodium (56%)56% related to the paper, Amoeboid movement (56%)56% related to the paper, GTPase (56%)56% related to the paper, Actin cytoskeleton (56%)56% related to the paper, Cell polarity (56%)56% related to the paper
View PDF
574 Citations
open accessOpen access Journal Article DOI: 10.1186/S12964-015-0125-7
Phase separation in biology; functional organization of a higher order
Diana M. Mitrea1, Richard W. Kriwacki2, Richard W. Kriwacki1

Abstract:

Inside eukaryotic cells, macromolecules are partitioned into membrane-bounded compartments and, within these, some are further organized into non-membrane-bounded structures termed membrane-less organelles. The latter structures are comprised of heterogeneous mixtures of proteins and nucleic acids and assemble through a phase... Inside eukaryotic cells, macromolecules are partitioned into membrane-bounded compartments and, within these, some are further organized into non-membrane-bounded structures termed membrane-less organelles. The latter structures are comprised of heterogeneous mixtures of proteins and nucleic acids and assemble through a phase separation phenomenon similar to polymer condensation. Membrane-less organelles are dynamic structures maintained through multivalent interactions that mediate diverse biological processes, many involved in RNA metabolism. They rapidly exchange components with the cellular milieu and their properties are readily altered in response to environmental cues, often implicating membrane-less organelles in responses to stress signaling. In this review, we discuss: (1) the functional roles of membrane-less organelles, (2) unifying structural and mechanistic principles that underlie their assembly and disassembly, and (3) established and emerging methods used in structural investigations of membrane-less organelles. read more read less
View PDF
556 Citations
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With SciSpace, you do not need a word template for Cell Communication and Signaling.

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Frequently asked questions

1. Can I write Cell Communication and Signaling in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Cell Communication and Signaling guidelines and auto format it.

2. Do you follow the Cell Communication and Signaling guidelines?

Yes, the template is compliant with the Cell Communication and Signaling guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Cell Communication and Signaling?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Cell Communication and Signaling citation style.

4. Can I use the Cell Communication and Signaling templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Cell Communication and Signaling.

5. Can I use a manuscript in Cell Communication and Signaling that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Cell Communication and Signaling that you can download at the end.

6. How long does it usually take you to format my papers in Cell Communication and Signaling?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Cell Communication and Signaling.

7. Where can I find the template for the Cell Communication and Signaling?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cell Communication and Signaling's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Cell Communication and Signaling's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Cell Communication and Signaling an online tool or is there a desktop version?

SciSpace's Cell Communication and Signaling is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Cell Communication and Signaling?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Cell Communication and Signaling?”

11. What is the output that I would get after using Cell Communication and Signaling?

After writing your paper autoformatting in Cell Communication and Signaling, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Cell Communication and Signaling's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Cell Communication and Signaling?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Cell Communication and Signaling. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Cell Communication and Signaling?

The 5 most common citation types in order of usage for Cell Communication and Signaling are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Cell Communication and Signaling?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cell Communication and Signaling's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Cell Communication and Signaling in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Cell Communication and Signaling Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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