Example of Current Heart Failure Reports format
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Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format
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Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format Example of Current Heart Failure Reports format
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Current Heart Failure Reports — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Emergency Medicine #4 of 80 down down by 2 ranks
Cardiology and Cardiovascular Medicine #63 of 317 down down by 18 ranks
Physiology (medical) #31 of 98 down down by 11 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 179 Published Papers | 992 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 10/07/2020
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Related Journals

open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 17.2
SJR: 1.596
SNIP: 1.811
open access Open Access
recommended Recommended

Elsevier

Quality:  
High
CiteRatio: 7.0
SJR: 2.366
SNIP: 1.736
open access Open Access
recommended Recommended

Elsevier

Quality:  
High
CiteRatio: 10.0
SJR: 2.768
SNIP: 2.855
open access Open Access

Springer

Quality:  
Good
CiteRatio: 2.8
SJR: 0.75
SNIP: 0.813

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

5.5

17% from 2019

CiteRatio for Current Heart Failure Reports from 2016 - 2020
Year Value
2020 5.5
2019 4.7
2017 6.8
2016 5.2
graph view Graph view
table view Table view

1.036

6% from 2019

SJR for Current Heart Failure Reports from 2016 - 2020
Year Value
2020 1.036
2019 1.102
2017 1.468
2016 1.295
graph view Graph view
table view Table view

1.264

59% from 2019

SNIP for Current Heart Failure Reports from 2016 - 2020
Year Value
2020 1.264
2019 0.796
2017 0.97
2016 0.923
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 17% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 6% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 59% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Current Heart Failure Reports

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Springer

Current Heart Failure Reports

Approved by publishing and review experts on SciSpace, this template is built as per for Current Heart Failure Reports formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 195 authors to write and format their manuscripts to this journal.

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Last updated on
09 Jul 2020
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ISSN
1606-8610
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Open Access
Hybrid
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Sherpa RoMEO Archiving Policy
White faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Author Year
(Blonder et al, 1982)
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Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1007/S11897-017-0337-9
Inflammation – Cause or Consequence of Heart Failure or Both?
Sophie Van Linthout1, Carsten Tschöpe1

Abstract:

With the intention to summarize the currently available evidence on the pathophysiological relevance of inflammation in heart failure, this review addresses the question whether inflammation is a cause or consequence of heart failure, or both. This review discusses the diversity (sterile, para-inflammation, chronic inflammati... With the intention to summarize the currently available evidence on the pathophysiological relevance of inflammation in heart failure, this review addresses the question whether inflammation is a cause or consequence of heart failure, or both. This review discusses the diversity (sterile, para-inflammation, chronic inflammation) and sources of inflammation and gives an overview of how inflammation (local versus systemic) can trigger heart failure. On the other hand, the review is outlined how heart failure-associated wall stress and signals released by stressed, malfunctioning, or dead cells (DAMPs: e.g., mitochondrial DNA, ATP, S100A8, matricellular proteins) induce cardiac sterile inflammation and how heart failure provokes inflammation in various peripheral tissues in a direct (inflammatory) and indirect (hemodynamic) manner. The crosstalk between the heart and peripheral organs (bone marrow, spleen, gut, adipose tissue) is outlined and the importance of neurohormonal mechanisms including the renin angiotensin aldosteron system and the s-adrenergic nervous system in inflammation and heart failure is discussed. Inflammation and heart failure are strongly interconnected and mutually reinforce each other. This indicates the difficulty to counteract inflammation and heart failure once this chronic vicious circle has started and points out the need to control the inflammatory process at an early stage avoiding chronic inflammation and heart failure. The diversity of inflammation further addresses the need for a tailored characterization of inflammation enabling differentiation of inflammation and subsequent target-specific strategies. It is expected that the characterization of the systemic and/or cardiac immune profile will be part of precision medicine in the future of cardiology. read more read less

Topics:

Heart failure (56%)56% related to the paper, Inflammation (53%)53% related to the paper
View PDF
305 Citations
open accessOpen access Journal Article DOI: 10.1007/S11897-013-0155-7
The Emerging Epidemic of Heart Failure with Preserved Ejection Fraction
A. Afşin Oktay1, Jonathan D. Rich1, Sanjiv J. Shah1

Abstract:

Heart failure with preserved ejection fraction (HFpEF), which currently represents approximately 50 % of heart failure (HF) cases, is common and associated with high morbidity and mortality. Understanding the epidemiology of HFpEF has been difficult due to the challenges in HFpEF diagnosis and the heterogeneous etiologies and... Heart failure with preserved ejection fraction (HFpEF), which currently represents approximately 50 % of heart failure (HF) cases, is common and associated with high morbidity and mortality. Understanding the epidemiology of HFpEF has been difficult due to the challenges in HFpEF diagnosis and the heterogeneous etiologies and pathophysiologies that underlie HFpEF. Nevertheless, several high-quality epidemiology and observational registry studies of HFpEF demonstrate that an increasing prevalence of HFpEF in both the outpatient and inpatient settings, coupled with a lack of evidence-based effective treatments for HFpEF, is resulting in an emerging epidemic of HFpEF. In this review, we discuss the emerging HFpEF epidemic, focusing on: (1) reasons for the rising prevalence of HFpEF; (2) the abnormalities in cardiac structure and function that dictate the transition from risk factors to HFpEF; (3) novel HFpEF mechanisms that may underlie the increase in HFpEF prevalence; (4) prognosis of HFpEF; and (5) risk prediction in HFpEF. We conclude with 10 unanswered questions on HFpEF epidemiology that will be important areas for future investigation. read more read less
264 Citations
open accessOpen access Journal Article DOI: 10.1007/S11897-010-0004-X
Galectin-3 in Cardiac Remodeling and Heart Failure
Rudolf A. de Boer1, Lili Yu1, Dirk J. van Veldhuisen1

Abstract:

Galectin-3 is a member of the galectin family, which consists of animal lectins that bind β-galactosides. Recently, a role for galectin-3 in the pathophysiology of heart failure has been suggested. It was observed that galectin-3 is specifically upregulated in decompensated heart failure compared with compensated heart failur... Galectin-3 is a member of the galectin family, which consists of animal lectins that bind β-galactosides. Recently, a role for galectin-3 in the pathophysiology of heart failure has been suggested. It was observed that galectin-3 is specifically upregulated in decompensated heart failure compared with compensated heart failure in animal models of heart failure. This has been associated with activation of fibroblasts and macrophages, which are a hallmark of cardiac remodeling. Therefore, galectin-3 may be a culprit biomarker in heart failure. Initial clinical observations indicate that galectin-3 may be a useful biomarker for decompensated heart failure, with incremental value over well-used “pressure-dependent” biomarkers, such as B-type natriuretic peptide. Future studies should focus on galectin-3 biology to better address the usefulness of galectin-3 as a biomarker and probe the usefulness of anti-galectin-3 therapy in treating heart failure. read more read less

Topics:

Heart failure (62%)62% related to the paper, Ventricular remodeling (58%)58% related to the paper, Biomarker (medicine) (51%)51% related to the paper, Galectin-3 (51%)51% related to the paper
205 Citations
open accessOpen access Journal Article DOI: 10.1007/S11897-017-0343-Y
From Inflammation to Fibrosis—Molecular and Cellular Mechanisms of Myocardial Tissue Remodelling and Perspectives on Differential Treatment Opportunities
Navin Suthahar1, Wouter C. Meijers1, Herman H W Silljé1, Rudolf A. de Boer1

Abstract:

In this review, we highlight the most important cellular and molecular mechanisms that contribute to cardiac inflammation and fibrosis. We also discuss the interplay between inflammation and fibrosis in various precursors of heart failure (HF) and how such mechanisms can contribute to myocardial tissue remodelling and develop... In this review, we highlight the most important cellular and molecular mechanisms that contribute to cardiac inflammation and fibrosis. We also discuss the interplay between inflammation and fibrosis in various precursors of heart failure (HF) and how such mechanisms can contribute to myocardial tissue remodelling and development of HF. Recently, many research articles attempt to elucidate different aspects of the interplay between inflammation and fibrosis. Cardiac inflammation and fibrosis are major pathophysiological mechanisms operating in the failing heart, regardless of HF aetiology. Currently, novel therapeutic options are available or are being developed to treat HF and these are discussed in this review. A progressive disease needs an aggressive management; however, existing therapies against HF are insufficient. There is a dynamic interplay between inflammation and fibrosis in various precursors of HF such as myocardial infarction (MI), myocarditis and hypertension, and also in HF itself. There is an urgent need to identify novel therapeutic targets and develop advanced therapeutic strategies to combat the syndrome of HF. Understanding and describing the elements of the inflammatory and fibrotic pathways are essential, and specific drugs that target these pathways need to be evaluated. read more read less

Topics:

Fibrosis (52%)52% related to the paper
View PDF
196 Citations
open accessOpen access Journal Article DOI: 10.1007/S11897-017-0351-Y
Acute Heart Failure: Definition, Classification and Epidemiology.
Sameer Kurmani1, Iain B. Squire2

Abstract:

The purpose of this review is to describe the extent and scope of acute heart failure (AHF), place it within its clinical context and highlight some of the difficulties in defining it as a pathophysiological entity. A diagnosis of AHF is made when patients present acutely with signs and symptoms of heart failure, often with d... The purpose of this review is to describe the extent and scope of acute heart failure (AHF), place it within its clinical context and highlight some of the difficulties in defining it as a pathophysiological entity. A diagnosis of AHF is made when patients present acutely with signs and symptoms of heart failure, often with decompensation of pre-existing cardiomyopathy. The most current guidelines classify based on clinical features at initial presentation and are used to both risk stratify and guide the management of haemodynamic compromise. Despite this, AHF remains a diagnosis with a poor prognosis and there is no therapy proven to have long-term mortality benefits. We provide an introduction to AHF and discuss its definition, causes and precipitants. We also present epidemiological and demographic data to suggest that there is significant patient heterogeneity and that AHF is not a single pathology, but rather a range of pathophysiological entities. This poses a challenge when designing clinical trials and may, at least in part, explain why the results in this area have been largely disappointing. read more read less
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196 Citations
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Frequently asked questions

1. Can I write Current Heart Failure Reports in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Current Heart Failure Reports guidelines and auto format it.

2. Do you follow the Current Heart Failure Reports guidelines?

Yes, the template is compliant with the Current Heart Failure Reports guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Current Heart Failure Reports?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Current Heart Failure Reports citation style.

4. Can I use the Current Heart Failure Reports templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Current Heart Failure Reports.

5. Can I use a manuscript in Current Heart Failure Reports that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Current Heart Failure Reports that you can download at the end.

6. How long does it usually take you to format my papers in Current Heart Failure Reports?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Current Heart Failure Reports.

7. Where can I find the template for the Current Heart Failure Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Current Heart Failure Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Current Heart Failure Reports's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Current Heart Failure Reports an online tool or is there a desktop version?

SciSpace's Current Heart Failure Reports is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Current Heart Failure Reports?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Current Heart Failure Reports?”

11. What is the output that I would get after using Current Heart Failure Reports?

After writing your paper autoformatting in Current Heart Failure Reports, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Current Heart Failure Reports's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Current Heart Failure Reports?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Current Heart Failure Reports. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Current Heart Failure Reports?

The 5 most common citation types in order of usage for Current Heart Failure Reports are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Current Heart Failure Reports?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Current Heart Failure Reports's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Current Heart Failure Reports in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Current Heart Failure Reports Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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