Example of Inflammation Research format
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Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format
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Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format Example of Inflammation Research format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Inflammation Research — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Pharmacology #67 of 297 up up by 30 ranks
Immunology #78 of 202 up up by 14 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 392 Published Papers | 2495 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 08/06/2020
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SJR: 1.219
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.174

4% from 2018

Impact factor for Inflammation Research from 2016 - 2019
Year Value
2019 3.174
2018 3.061
2017 2.99
2016 2.659
graph view Graph view
table view Table view

6.4

21% from 2019

CiteRatio for Inflammation Research from 2016 - 2020
Year Value
2020 6.4
2019 5.3
2018 4.9
2017 5.0
2016 5.0
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 4% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 21% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.121

14% from 2019

SJR for Inflammation Research from 2016 - 2020
Year Value
2020 1.121
2019 0.98
2018 0.97
2017 1.062
2016 1.025
graph view Graph view
table view Table view

1.13

20% from 2019

SNIP for Inflammation Research from 2016 - 2020
Year Value
2020 1.13
2019 0.941
2018 0.955
2017 0.926
2016 0.934
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 14% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 20% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Inflammation Research

Guideline source: View

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Springer

Inflammation Research

Inflammation Research (IR) publishes peer-reviewed papers on all aspects of inflammation and related fields including histopathology, immunological mechanisms, gene expression, mediators, experimental models, clinical investigations and the effect of drugs. Related fields are ...... Read More

Pharmacology

Immunology

Pharmacology, Toxicology and Pharmaceutics

i
Last updated on
08 Jun 2020
i
ISSN
1023-3830
i
Impact Factor
Medium - 0.828
i
Acceptance Rate
36%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
i
Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1007/BF01973261
Biological effects of cyclosporin A: A new antilymphocytic agent
01 Jul 1976 - Inflammation Research

Abstract:

The fungus metabolite cyclosporin A is a small peptide acting as a novel antilymphocytic agent. It strongly depressed appearance of both direct and indirect plaque-forming cells and produced a clear dose-dependent inhibition of haemagglutinin formation in mice upon oral administration. Skin graft rejection in mice and graft-v... The fungus metabolite cyclosporin A is a small peptide acting as a novel antilymphocytic agent. It strongly depressed appearance of both direct and indirect plaque-forming cells and produced a clear dose-dependent inhibition of haemagglutinin formation in mice upon oral administration. Skin graft rejection in mice and graft-versus-host disease in mice and rats were considerably delayed by cyclosporin A which also prevented the occurrence of paralysis in rats with experimental allergic encephalomyelitis. This compound was not only highly effective in preventing development of Freund's adjuvant arthritis, but in addition improved the symptoms in rats with established arthritis, although it is inactive in acute inflammation. This new agent contrasts with other immunosuppressives and cytostatic drugs in its weak myelotoxicity. Experimental evidence suggests that cyclosporin A, rather than being cytostatic or lympholytic, affects an early stage of mitogenic triggering of the immunocompetent lymphoid cell. read more read less

Topics:

Cyclosporin a (69%)69% related to the paper, Arthritis (51%)51% related to the paper, Encephalomyelitis (51%)51% related to the paper
1,341 Citations
Journal Article DOI: 10.1007/S000110050622
Peroxisome proliferator-activated receptors (PPARs): nuclear receptors at the crossroads between lipid metabolism and inflammation.
Giulia Chinetti1, Jean-Charles Fruchart1, Bart Staels1
01 Oct 2000 - Inflammation Research

Abstract:

Peroxisome proliferator-activated (PPARs) are ligand-activated transcription factors belonging to the nuclear receptor family. PPARs function as regulators of lipid and lipoprotein metabolism and glucose homeostasis and influence cellular proliferation, differentiation and apoptosis. PPARalpha is highly expressed in tissues s... Peroxisome proliferator-activated (PPARs) are ligand-activated transcription factors belonging to the nuclear receptor family. PPARs function as regulators of lipid and lipoprotein metabolism and glucose homeostasis and influence cellular proliferation, differentiation and apoptosis. PPARalpha is highly expressed in tissues such as liver, muscle, kidney and heart, where it stimulates the beta-oxidative degradation of fatty acids. PPARgamma is predominantly expressed in intestine and adipose tissue. PPARgamma triggers adipocyte differentiation and promotes lipid storage. The hypolipidemic fibrates and the antidiabetic glitazones are synthetic ligands for PPARalpha and PPARgamma, respectively. Furthermore, fatty acids and eicosanoids are natural PPAR ligands: PPARalpha is activated by leukotriene B4, whereas prostaglandin J2 is a PPARgamma ligand. These observations suggested a potential role for PPARs not only in metabolic but also in inflammation control. The first evidence for a role of PPARalpha in inflammation control came from the demonstration that PPARalpha deficient mice display a prolonged response to inflammatory stimuli. It was suggested that PPARalpha deficiency results in a reduced beta-oxidative degradation of these inflammatory fatty acid derivatives. More recently, PPAR activators were shown to inhibit the activation of inflammatory response genes (such as IL-2, IL-6, IL-8, TNFalpha and metalloproteases) by negatively interfering with the NF- kappaB, STAT and AP-1 signalling pathways. PPAR activators exert these anti-inflammatory activities in different immunological and vascular wall cell types such as monocyte/macrophages, endothelial, epithelial and smooth muscle cells in which PPARs are expressed. These recent findings indicate a modulatory role for PPARs in the control of the inflammatory response with potential therapeutic applications in inflammation-related diseases, such as atherosclerosis and inflammatory bowel disease. read more read less

Topics:

Peroxisome proliferator-activated receptor (58%)58% related to the paper, Glucose homeostasis (52%)52% related to the paper, Lipid metabolism (52%)52% related to the paper, Inflammation (52%)52% related to the paper, Nuclear receptor (50%)50% related to the paper
895 Citations
Journal Article DOI: 10.1007/S00011-009-0037-3
Flavonoids as anti-inflammatory agents: implications in cancer and cardiovascular disease.
21 Apr 2009 - Inflammation Research

Abstract:

Chronic inflammation is being shown to be increasingly involved in the onset and development of several pathological disturbances such as arteriosclerosis, obesity, diabetes, neurodegenerative diseases and even cancer. Treatment for chronic inflammatory disorders has not been solved, and there is an urgent need to find new an... Chronic inflammation is being shown to be increasingly involved in the onset and development of several pathological disturbances such as arteriosclerosis, obesity, diabetes, neurodegenerative diseases and even cancer. Treatment for chronic inflammatory disorders has not been solved, and there is an urgent need to find new and safe anti-inflammatory compounds. Flavonoids belong to a group of natural substances occurring normally in the diet that exhibit a variety of beneficial effects on health. The anti-inflammatory properties of flavonoids have been studied recently, in order to establish and characterize their potential utility as therapeutic agents in the treatment of inflammatory diseases. Several mechanisms of action have been proposed to explain in vivo flavonoid anti-inflammatory actions, such as antioxidant activity, inhibition of eicosanoid generating enzymes or the modulation of the production of proinflammatory molecules. Recent studies have also shown that some flavonoids are modulators of proinflammatory gene expression, thus leading to the attenuation of the inflammatory response. However, much work remains to be done in order to achieve definitive conclusions about their potential usefulness. This review summarizes the known mechanisms involved in the anti-inflammatory activity of flavonoids and the implications of these effects on the protection against cancer and cardiovascular disease. read more read less

Topics:

Proinflammatory cytokine (51%)51% related to the paper
889 Citations
Journal Article DOI: 10.1007/BF01630479
New insights into the mode of action of anti-inflammatory drugs
John R. Vane1, Regina M. Botting1
01 Jan 1995 - Inflammation Research

Abstract:

The discovery of a second cyclooxygenase has provided fresh impetus to the search for new anti-inflammatory drugs. The second enzyme is effectively absent from healthy tissues but its levels rise dramatically during inflammation. It can be induced in migratory cells by bacterial lipopolysaccharide, cytokines and growth factor... The discovery of a second cyclooxygenase has provided fresh impetus to the search for new anti-inflammatory drugs. The second enzyme is effectively absent from healthy tissues but its levels rise dramatically during inflammation. It can be induced in migratory cells by bacterial lipopolysaccharide, cytokines and growth factors. The constitutive cyclooxygenase-1 (COX-1) can thus be considered a "housekeeping" enzyme, in contrast to cyclooxygenase-2 (COX-2) which is activated by tissue damage. Both enzymes have a molecular weight of around 70 kDa and similar Km and Vmax values for their reaction with arachidonic acid. Several non steroid anti-inflammatory drugs which have more than 1,000 fold selectivity for COX-2 over COX-1 are in the early stages of drug development. read more read less

Topics:

Cyclooxygenase (51%)51% related to the paper, Anti-inflammatory (50%)50% related to the paper
796 Citations
Journal Article DOI: 10.1007/S000110050284
Anti-inflammatory drugs and their mechanism of action
John R. Vane1, Regina M. Botting1
01 Oct 1998 - Inflammation Research

Abstract:

Nonsteroidal anti-inflammatory drugs (NSAIDs) produce their therapeutic activities through inhibition of cyclooxygenase (COX), the enzyme that makes prostaglandins (PGs). They share, to a greater or lesser degree, the same side effects, including gastric and renal toxicity. Recent research has shown that there are at least tw... Nonsteroidal anti-inflammatory drugs (NSAIDs) produce their therapeutic activities through inhibition of cyclooxygenase (COX), the enzyme that makes prostaglandins (PGs). They share, to a greater or lesser degree, the same side effects, including gastric and renal toxicity. Recent research has shown that there are at least two COX isoenzymes. COX-1 is constitutive and makes PGs that protect the stomach and kidney from damage. COX-2 is induced by inflammatory stimuli, such as cytokines, and produces PGs that contribute to the pain and swelling of inflammation. Thus, selective COX-2 inhibitors should be anti-inflammatory without side effects on the kidney and stomach. Of course, selective COX-2 inhibitors may have other side effects and perhaps other therapeutic potential. For instance, COX-2 (and not COX-1) is thought to be involved in ovulation and in labor. In addition, the well-known protective action of aspirin on colon cancer may be through an action on COX-2, which is expressed in this disease. Moreover, NSAIDs delay the progress of Alzheimer's disease. Thus, selective COX-2 inhibitors may demonstrate new important therapeutic benefits as anticancer agents, as well as in preventing premature labor and perhaps even retarding the progression of Alzheimer's disease. read more read less

Topics:

Mechanism of action (53%)53% related to the paper, Aspirin (50%)50% related to the paper, Cyclooxygenase (50%)50% related to the paper
652 Citations
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Inflammation Research format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write Inflammation Research in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Inflammation Research guidelines and auto format it.

2. Do you follow the Inflammation Research guidelines?

Yes, the template is compliant with the Inflammation Research guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Inflammation Research?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Inflammation Research citation style.

4. Can I use the Inflammation Research templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Inflammation Research.

5. Can I use a manuscript in Inflammation Research that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Inflammation Research that you can download at the end.

6. How long does it usually take you to format my papers in Inflammation Research?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Inflammation Research.

7. Where can I find the template for the Inflammation Research?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Inflammation Research's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Inflammation Research's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Inflammation Research an online tool or is there a desktop version?

SciSpace's Inflammation Research is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Inflammation Research?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Inflammation Research?”

11. What is the output that I would get after using Inflammation Research?

After writing your paper autoformatting in Inflammation Research, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Inflammation Research's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Inflammation Research?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Inflammation Research. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Inflammation Research?

The 5 most common citation types in order of usage for Inflammation Research are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Inflammation Research?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Inflammation Research's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Inflammation Research in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Inflammation Research Endnote style according to Elsevier guidelines.

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