Example of neurogenetics format
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Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format Example of neurogenetics format
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open access Open Access

neurogenetics — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Genetics #131 of 325 down down by 39 ranks
Genetics (clinical) #36 of 87 down down by 7 ranks
Cellular and Molecular Neuroscience #53 of 88 down down by 16 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 103 Published Papers | 513 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 30/06/2020
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Related Journals

open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 7.3
SJR: 2.628
SNIP: 1.713
open access Open Access
recommended Recommended

PLOS

Quality:  
High
CiteRatio: 9.0
SJR: 3.587
SNIP: 1.457
open access Open Access

Springer

Quality:  
High
CiteRatio: 7.7
SJR: 1.945
SNIP: 0.996
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 15.2
SJR: 5.564
SNIP: 2.245

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.774

8% from 2018

Impact factor for neurogenetics from 2016 - 2019
Year Value
2019 2.774
2018 3.017
2017 3.09
2016 3.269
graph view Graph view
table view Table view

5.0

7% from 2019

CiteRatio for neurogenetics from 2016 - 2020
Year Value
2020 5.0
2019 5.4
2018 5.4
2017 5.9
2016 6.0
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 8% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has decreased by 7% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.934

36% from 2019

SJR for neurogenetics from 2016 - 2020
Year Value
2020 0.934
2019 1.457
2018 1.377
2017 1.593
2016 2.188
graph view Graph view
table view Table view

0.846

4% from 2019

SNIP for neurogenetics from 2016 - 2020
Year Value
2020 0.846
2019 0.882
2018 0.832
2017 0.755
2016 0.937
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 36% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 4% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

neurogenetics

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Springer

neurogenetics

Neurogenetics publishes findings that contribute to a better understanding of the genetic basis of normal and abnormal function of the nervous system. Neurogenetic disorders are the main focus of the journal. Neurogenetics therefore includes findings in humans and other organi...... Read More

Genetics

Genetics(clinical)

Cellular and Molecular Neuroscience

Biochemistry, Genetics and Molecular Biology

i
Last updated on
30 Jun 2020
i
ISSN
1364-6745
i
Impact Factor
Medium - 0.982
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
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Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1007/S10048-006-0032-6
Gene expression analyses reveal molecular relationships among 20 regions of the human CNS
Richard B. Roth1, Peter Hevezi1, Jerry Lee1, Dorian Willhite1, Sandra M. Lechner1, Alan C. Foster1, Albert Zlotnik1
30 Mar 2006 - Neurogenetics

Abstract:

Transcriptional profiling was performed to survey the global expression patterns of 20 anatomically distinct sites of the human central nervous system (CNS). Forty-five non-CNS tissues were also profiled to allow for comparative analyses. Using principal component analysis and hierarchical clustering, we were able to show tha... Transcriptional profiling was performed to survey the global expression patterns of 20 anatomically distinct sites of the human central nervous system (CNS). Forty-five non-CNS tissues were also profiled to allow for comparative analyses. Using principal component analysis and hierarchical clustering, we were able to show that the expression patterns of the 20 CNS sites profiled were significantly different from all non-CNS tissues and were also similar to one another, indicating an underlying common expression signature. By focusing our analyses on the 20 sites of the CNS, we were able to show that these 20 sites could be segregated into discrete groups with underlying similarities in anatomical structure and, in many cases, functional activity. These findings suggest that gene expression data can help define CNS function at the molecular level. We have identified subsets of genes with the following patterns of expression: (1) across the CNS, suggesting homeostatic/housekeeping function; (2) in subsets of functionally related sites of the CNS identified by our unsupervised learning analyses; and (3) in single sites within the CNS, indicating their participation in distinct site-specific functions. By performing network analyses on these gene sets, we identified many pathways that are upregulated in particular sites of the CNS, some of which were previously described in the literature, validating both our dataset and approach. In summary, we have generated a database of gene expression that can be used to gain valuable insight into the molecular characterization of functions carried out by different sites of the human CNS. read more read less

Topics:

Gene expression profiling (54%)54% related to the paper
326 Citations
Journal Article DOI: 10.1007/S10048-005-0218-3
Correlations between genotype, ultrastructural morphology and clinical phenotype in the neuronal ceroid lipofuscinoses.
Sara E. Mole1, Ruth E Williams2, Hans H. Goebel3
18 Jun 2005 - Neurogenetics

Abstract:

The neuronal ceroid lipofuscinoses (NCLs) are a group of severe neurodegenerative diseases with onset usually in childhood and characterised by the intracellular accumulation of autofluorescent storage material. Within the last decade, mutations that cause NCL have been found in six human genes (CLN1, CLN2, CLN3, CLN5, CLN6 a... The neuronal ceroid lipofuscinoses (NCLs) are a group of severe neurodegenerative diseases with onset usually in childhood and characterised by the intracellular accumulation of autofluorescent storage material. Within the last decade, mutations that cause NCL have been found in six human genes (CLN1, CLN2, CLN3, CLN5, CLN6 and CLN8). Mutations in two additional genes cause disease in animal models that share features with NCL-CTSD in sheep and mice and PPT2 in mice. Approximately 160 NCL disease-causing mutations have now been described (listed and fully cited in the NCL Mutation Database, http://www.ucl.ac.uk/ncl/). Most mutations result in a classic morphology and disease phenotype, but some mutations are associated with disease that is of later onset, less severe or protracted in its course, or with atypical morphology. Seven common mutations exist, some having a worldwide distribution and others associated with families originating from specific geographical regions. This review attempts to correlate the gene, disease-causing mutation, morphology and clinical phenotype for each type of NCL. read more read less

Topics:

Batten disease (57%)57% related to the paper, Jansky–Bielschowsky disease (55%)55% related to the paper, Tripeptidyl peptidase I (55%)55% related to the paper, CLN8 (54%)54% related to the paper, Kufs disease (52%)52% related to the paper
276 Citations
Journal Article DOI: 10.1007/S10048-008-0133-5
Heterogeneous dysregulation of microRNAs across the autism spectrum
01 Jul 2008 - Neurogenetics

Abstract:

microRNAs (miRNAs) are ~21 nt transcripts capable of regulating the expression of many mRNAs and are abundant in the brain. miRNAs have a role in several complex diseases including cancer as well as some neurological diseases such as Tourette’s syndrome and Fragile x syndrome. As a genetically complex disease, dysregulation o... microRNAs (miRNAs) are ~21 nt transcripts capable of regulating the expression of many mRNAs and are abundant in the brain. miRNAs have a role in several complex diseases including cancer as well as some neurological diseases such as Tourette’s syndrome and Fragile x syndrome. As a genetically complex disease, dysregulation of miRNA expression might be a feature of autism spectrum disorders (ASDs). Using multiplex quantitative polymerase chain reaction (PCR), we compared the expression of 466 human miRNAs from postmortem cerebellar cortex tissue of individuals with ASD (n = 13) and a control set of non-autistic cerebellar samples (n = 13). While most miRNAs levels showed little variation across all samples suggesting that autism does not induce global dysfunction of miRNA expression, some miRNAs among the autistic samples were expressed at significantly different levels compared to the mean control value. Twenty-eight miRNAs were expressed at significantly different levels compared to the non-autism control set in at least one of the autism samples. To validate the finding, we reversed the analysis and compared each non-autism control to a single mean value for each miRNA across all autism cases. In this analysis, the number of dysregulated miRNAs fell from 28 to 9 miRNAs. Among the predicted targets of dysregulated miRNAs are genes that are known genetic causes of autism such Neurexin and SHANK3. This study finds that altered miRNA expression levels are observed in postmortem cerebellar cortex from autism patients, a finding which suggests that dysregulation of miRNAs may contribute to autism spectrum phenotype. read more read less

Topics:

Gene silencing (57%)57% related to the paper, Autism (57%)57% related to the paper, Cerebellar cortex (56%)56% related to the paper, Causes of autism (56%)56% related to the paper, Fragile X syndrome (52%)52% related to the paper
View PDF
256 Citations
Journal Article DOI: 10.1007/S10048-006-0041-5
A common missense variant in the LRRK2 gene, Gly2385Arg, associated with Parkinson's disease risk in Taiwan.
22 Apr 2006 - Neurogenetics

Abstract:

Mutations in the LRRK2 gene are a cause of autosomal dominant Parkinson's disease (PD). Whether LRRK2 variants influence susceptibility to the commoner, sporadic forms of PD remains largely unknown. Data are particularly limited concerning the Asian population. In search for novel, biologically relevant variants, we sequenced... Mutations in the LRRK2 gene are a cause of autosomal dominant Parkinson's disease (PD). Whether LRRK2 variants influence susceptibility to the commoner, sporadic forms of PD remains largely unknown. Data are particularly limited concerning the Asian population. In search for novel, biologically relevant variants, we sequenced the LRRK2 coding region in Taiwanese patients with PD. Four newly identified variants and another variant recently found in a Taiwanese PD family were tested for association with the disease in a sample of 608 PD cases and 373 ethnically matched controls. Heterozygosity for the Gly2385Arg variant was significantly more frequent among PD patients than controls (nominal p value=0.004, corrected for multiple comparisons=0.012, gender- and age-adjusted odds ratio=2.24, 95% C.I.: 1.29-3.88); this variant was uniformly distributed across genders and age strata. Two novel variants, Met1869Val and Glu1874Stop, were found in one PD case each; their pathogenic role remains, therefore, uncertain. The remaining two novel variants (Ala419Val and Pro755Leu) were present with similar frequency in cases and controls, and were therefore, interpreted as disease-unrelated polymorphisms. Our findings suggest that the LRRK2 Gly2385Arg is the first identified, functionally relevant variant, which acts as common risk factor for sporadic PD in the population of Chinese ethnicity. read more read less

Topics:

Common disease-common variant (61%)61% related to the paper, Population (52%)52% related to the paper, LRRK2 (51%)51% related to the paper
251 Citations
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SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

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With SciSpace, you do not need a word template for neurogenetics.

It automatically formats your research paper to Springer formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

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neurogenetics format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write neurogenetics in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the neurogenetics guidelines and auto format it.

2. Do you follow the neurogenetics guidelines?

Yes, the template is compliant with the neurogenetics guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in neurogenetics?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the neurogenetics citation style.

4. Can I use the neurogenetics templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for neurogenetics.

5. Can I use a manuscript in neurogenetics that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper neurogenetics that you can download at the end.

6. How long does it usually take you to format my papers in neurogenetics?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in neurogenetics.

7. Where can I find the template for the neurogenetics?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per neurogenetics's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the neurogenetics's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. neurogenetics an online tool or is there a desktop version?

SciSpace's neurogenetics is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like neurogenetics?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like neurogenetics?”

11. What is the output that I would get after using neurogenetics?

After writing your paper autoformatting in neurogenetics, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is neurogenetics's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for neurogenetics?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for neurogenetics. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In neurogenetics?

The 5 most common citation types in order of usage for neurogenetics are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the neurogenetics?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per neurogenetics's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download neurogenetics in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in neurogenetics Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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