Example of Pediatric Nephrology format
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Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format
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Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format Example of Pediatric Nephrology format
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open access Open Access

Pediatric Nephrology — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Pediatrics, Perinatology and Child Health #48 of 294 down down by 10 ranks
Nephrology #17 of 62 down down by 2 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 1004 Published Papers | 4368 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 07/07/2020
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FAQ

Related Journals

open access Open Access

SAGE

Quality:  
High
CiteRatio: 4.6
SJR: 2.107
SNIP: 2.487
open access Open Access

SAGE

Quality:  
High
CiteRatio: 4.5
SJR: 1.185
SNIP: 1.607
open access Open Access

SAGE

Quality:  
High
CiteRatio: 4.3
SJR: 1.395
SNIP: 2.063
open access Open Access

Nature

Quality:  
High
CiteRatio: 3.6
SJR: 0.912
SNIP: 1.249

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.676

5% from 2018

Impact factor for Pediatric Nephrology from 2016 - 2019
Year Value
2019 2.676
2018 2.816
2017 2.627
2016 2.516
graph view Graph view
table view Table view

4.4

2% from 2019

CiteRatio for Pediatric Nephrology from 2016 - 2020
Year Value
2020 4.4
2019 4.3
2018 4.0
2017 4.5
2016 4.8
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 5% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 2% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.831

2% from 2019

SJR for Pediatric Nephrology from 2016 - 2020
Year Value
2020 0.831
2019 0.813
2018 0.949
2017 0.938
2016 0.988
graph view Graph view
table view Table view

1.249

5% from 2019

SNIP for Pediatric Nephrology from 2016 - 2020
Year Value
2020 1.249
2019 1.187
2018 1.069
2017 1.169
2016 1.098
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 2% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 5% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Pediatric Nephrology

Guideline source: View

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Springer

Pediatric Nephrology

Pediatric Nephrology publishes original laboratory or clinical research and new or important clinical observations pertaining to any aspect of the wide spectrum of acute and chronic diseases that affect renal function in children as well as on hypertension and fluid and electr...... Read More

Pediatrics, Perinatology, and Child Health

Nephrology

Medicine

i
Last updated on
07 Jul 2020
i
ISSN
0931-041X
i
Impact Factor
High - 1.447
i
Acceptance Rate
45%
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
i
Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1007/S00467-011-1939-1
Epidemiology of chronic kidney disease in children
Jérôme Harambat1, Karlijn J. van Stralen1, Jon Jin Kim2, E. Jane Tizard2
01 Mar 2012 - Pediatric Nephrology

Abstract:

In the past 30 years there have been major improvements in the care of children with chronic kidney disease (CKD). However, most of the available epidemiological data stem from end-stage renal disease (ESRD) registries and information on the earlier stages of pediatric CKD is still limited. The median reported incidence of re... In the past 30 years there have been major improvements in the care of children with chronic kidney disease (CKD). However, most of the available epidemiological data stem from end-stage renal disease (ESRD) registries and information on the earlier stages of pediatric CKD is still limited. The median reported incidence of renal replacement therapy (RRT) in children aged 0–19 years across the world in 2008 was 9 per million of the age-related population (4–18 years). The prevalence of RRT in 2008 ranged from 18 to 100 per million of the age-related population. Congenital disorders, including congenital anomalies of the kidney and urinary tract (CAKUT) and hereditary nephropathies, are responsible for about two thirds of all cases of CKD in developed countries, while acquired causes predominate in developing countries. Children with congenital disorders experience a slower progression of CKD than those with glomerulonephritis, resulting in a lower proportion of CAKUT in the ESRD population compared with less advanced stages of CKD. Most children with ESRD start on dialysis and then receive a transplant. While the survival rate of children with ERSD has improved, it remains about 30 times lower than that of healthy peers. Children now mainly die of cardiovascular causes and infection rather than from renal failure. read more read less

Topics:

End stage renal disease (62%)62% related to the paper, Kidney disease (61%)61% related to the paper, Renal replacement therapy (57%)57% related to the paper, Population (53%)53% related to the paper, Nephrology (53%)53% related to the paper
View PDF
714 Citations
Journal Article DOI: 10.1007/S004670000461
Molecular basis of renal fibrosis.
Allison A. Eddy1
01 Dec 2000 - Pediatric Nephrology

Abstract:

All progressive renal diseases are the consequence of a process of destructive fibrosis This review will focus on tubulointerstitial fibrosis, the pathophysiology of which will be divided into four arbitrary phases First is the cellular activation and injury phase The tubules are activated, the peritubular capillary endotheli... All progressive renal diseases are the consequence of a process of destructive fibrosis This review will focus on tubulointerstitial fibrosis, the pathophysiology of which will be divided into four arbitrary phases First is the cellular activation and injury phase The tubules are activated, the peritubular capillary endothelium facilitates migration of mononuclear cells into the interstitium where they mature into macrophages, and myofibroblasts/activated fibroblasts begin to populate the interstitium Each of these cells releases soluble products that contribute to ongoing inflammation and ultimately fibrosis The second phase, the fibrogenic signaling phase, is characterized by the release of soluble factors that have fibrosis-promoting effects Several growth factors and cytokines have been implicated, with primary roles suggested for transforming growth factor-beta, connective tissue growth factor, angiotensin II and endothelin-1 Additional factors may participate including platelet-derived growth factor, basic fibroblast growth factor, tumor necrosis factor-alpha and interleukin-1, while interferon-gamma and hepatocyte growth factor may elicit antifibrotic responses Third is the fibrogenic phase when matrix proteins, both normal and novel to the renal interstitium, begin to accumulate During this time both increased matrix protein synthesis and impaired matrix turnover are evident The latter is due to the renal production of protease inhibitors such as the tissue inhibitors of metalloproteinases and plasminogen activator inhibitors which inactivate the renal proteases that normally regulate matrix turnover Fourth is the phase of renal destruction, the ultimate sequel to excessive matrix accumulation During this time the tubules and peritubular capillaries are obliterated The number of intact nephrons progressively declines resulting in a continuous reduction in glomerular filtration read more read less

Topics:

Renal fibrosis (63%)63% related to the paper, Fibrosis (59%)59% related to the paper, Tubulointerstitial fibrosis (57%)57% related to the paper, Growth factor (56%)56% related to the paper, Peritubular capillaries (55%)55% related to the paper
613 Citations
open accessOpen access Journal Article DOI: 10.1007/S00467-010-1731-7
Ciliopathies: an expanding disease spectrum
Aoife M. Waters1, Philip L. Beales
06 Jan 2011 - Pediatric Nephrology

Abstract:

Ciliopathies comprise a group of disorders associated with genetic mutations encoding defective proteins, which result in either abnormal formation or function of cilia. As cilia are a component of almost all vertebrate cells, cilia dysfunction can manifest as a constellation of features that include characteristically, retin... Ciliopathies comprise a group of disorders associated with genetic mutations encoding defective proteins, which result in either abnormal formation or function of cilia. As cilia are a component of almost all vertebrate cells, cilia dysfunction can manifest as a constellation of features that include characteristically, retinal degeneration, renal disease and cerebral anomalies. Additional manifestations include congenital fibrocystic diseases of the liver, diabetes, obesity and skeletal dysplasias. Ciliopathic features have been associated with mutations in over 40 genes to date. However, with over 1,000 polypeptides currently identified within the ciliary proteome, several other disorders associated with this constellation of clinical features will likely be ascribed to mutations in other ciliary genes. The mechanisms underlying many of the disease phenotypes associated with ciliary dysfunction have yet to be fully elucidated. Several elegant studies have crucially demonstrated the dynamic ciliary localisation of components of the Hedgehog and Wnt signalling pathways during signal transduction. Given the critical role of the cilium in transducing “outside-in” signals, it is not surprising therefore, that the disease phenotypes consequent to ciliary dysfunction are a manifestation of aberrant signal transduction. Further investigation is now needed to explore the developmental and physiological roles of aberrant signal transduction in the manifestation of ciliopathy phenotypes. Utilisation of conditional and inducible murine models to delete or overexpress individual ciliary genes in a spatiotemporal and organ/cell-specific manner should help clarify some of the functional roles of ciliary proteins in the manifestation of phenotypic features. read more read less

Topics:

Ciliopathy (61%)61% related to the paper, Cilium (59%)59% related to the paper, Ciliopathies (58%)58% related to the paper, Bardet–Biedl syndrome (55%)55% related to the paper, Senior–Løken syndrome (54%)54% related to the paper
605 Citations
open accessOpen access Journal Article DOI: 10.1007/S00467-006-0410-1
Chronic kidney disease in children: the global perspective
Bradley A. Warady1, Bradley A. Warady2, Vimal Chadha3
20 Feb 2007 - Pediatric Nephrology

Abstract:

In contrast to the increasing availability of information pertaining to the care of children with chronic kidney disease (CKD) from large-scale observational and interventional studies, epidemiological information on the incidence and prevalence of pediatric CKD is currently limited, imprecise, and flawed by methodological di... In contrast to the increasing availability of information pertaining to the care of children with chronic kidney disease (CKD) from large-scale observational and interventional studies, epidemiological information on the incidence and prevalence of pediatric CKD is currently limited, imprecise, and flawed by methodological differences between the various data sources. There are distinct geographic differences in the reported causes of CKD in children, in part due to environmental, racial, genetic, and cultural (consanguinity) differences. However, a substantial percentage of children develop CKD early in life, with congenital renal disorders such as obstructive uropathy and aplasia/hypoplasia/dysplasia being responsible for almost one half of all cases. The most favored end-stage renal disease (ESRD) treatment modality in children is renal transplantation, but a lack of health care resources and high patient mortality in the developing world limits the global provision of renal replacement therapy (RRT) and influences patient prevalence. Additional efforts to define the epidemiology of pediatric CKD worldwide are necessary if a better understanding of the full extent of the problem, areas for study, and the potential impact of intervention is desired. read more read less

Topics:

End stage renal disease (63%)63% related to the paper, Kidney disease (59%)59% related to the paper, Renal replacement therapy (56%)56% related to the paper, Transplantation (52%)52% related to the paper, Nephrology (52%)52% related to the paper
View PDF
469 Citations
Journal Article DOI: 10.1007/S00467-007-0601-4
NGAL is an early predictive biomarker of contrast-induced nephropathy in children.
14 Sep 2007 - Pediatric Nephrology

Abstract:

We hypothesized that neutrophil gelatinase-associated lipocalin (NGAL) is an early predictive biomarker of contrast-induced nephropathy (CIN). We prospectively enrolled 91 children (age 0–18 years) with congenital heart disease undergoing elective cardiac catheterization and angiography with contrast administration (CC; Iover... We hypothesized that neutrophil gelatinase-associated lipocalin (NGAL) is an early predictive biomarker of contrast-induced nephropathy (CIN). We prospectively enrolled 91 children (age 0–18 years) with congenital heart disease undergoing elective cardiac catheterization and angiography with contrast administration (CC; Ioversol). Serial urine and plasma samples were analyzed in a double-blind fashion by NGAL enzyme-linked immunosorbent assay (ELISA). CIN, defined as a 50% increase in serum creatinine from baseline, was found in 11 subjects (12%), but detection using increase in serum creatinine was only possible 6–24 h after CC. In contrast, significant elevation of NGAL concentrations in urine (135 ± 32 vs. 11.6 ± 2 ng/ml without CIN, p < 0.001) and plasma (151 ± 34 vs. 36 ± 4 without CIN, p < 0.001) were noted within 2 h after CC in those subjects. Using a cutoff value of 100 ng/ml, sensitivity, specificity, and area under the receiver-operating characteristic (ROC) curve for prediction of CIN were excellent for the 2-h urine NGAL (73%, 100%, and 0.92, respectively) and 2-h plasma NGAL (73%, 100%, and 0.91, respectively). By multivariate analysis, the 2-h NGAL concentrations in the urine (R2 = 0.52, p < 0.0001) and plasma (R2 = 0.72, p < 0.0001) were found to be powerful independent predictors of CIN. Patient demographics and contrast volume were not predictive of CIN. read more read less

Topics:

Contrast-induced nephropathy (52%)52% related to the paper
432 Citations
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Pediatric Nephrology format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write Pediatric Nephrology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Pediatric Nephrology guidelines and auto format it.

2. Do you follow the Pediatric Nephrology guidelines?

Yes, the template is compliant with the Pediatric Nephrology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Pediatric Nephrology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Pediatric Nephrology citation style.

4. Can I use the Pediatric Nephrology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Pediatric Nephrology.

5. Can I use a manuscript in Pediatric Nephrology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Pediatric Nephrology that you can download at the end.

6. How long does it usually take you to format my papers in Pediatric Nephrology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Pediatric Nephrology.

7. Where can I find the template for the Pediatric Nephrology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Pediatric Nephrology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Pediatric Nephrology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Pediatric Nephrology an online tool or is there a desktop version?

SciSpace's Pediatric Nephrology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Pediatric Nephrology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Pediatric Nephrology?”

11. What is the output that I would get after using Pediatric Nephrology?

After writing your paper autoformatting in Pediatric Nephrology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Pediatric Nephrology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Pediatric Nephrology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Pediatric Nephrology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Pediatric Nephrology?

The 5 most common citation types in order of usage for Pediatric Nephrology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Pediatric Nephrology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Pediatric Nephrology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Pediatric Nephrology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Pediatric Nephrology Endnote style according to Elsevier guidelines.

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