Example of Avian Pathology format
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Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format Example of Avian Pathology format
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open access Open Access

Avian Pathology — Template for authors

Publisher: Taylor and Francis
Categories Rank Trend in last 3 yrs
Animal Science and Zoology #62 of 416 up up by 7 ranks
Food Animals #7 of 32 -
Immunology and Microbiology (all) #20 of 45 up up by 3 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 300 Published Papers | 1184 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 23/06/2020
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Related Journals

open access Open Access
recommended Recommended

Elsevier

Quality:  
High
CiteRatio: 4.2
SJR: 0.816
SNIP: 1.354
open access Open Access

Elsevier

Quality:  
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CiteRatio: 4.2
SJR: 0.726
SNIP: 1.171
open access Open Access

Elsevier

Quality:  
High
CiteRatio: 3.2
SJR: 0.573
SNIP: 1.103
open access Open Access

Elsevier

Quality:  
High
CiteRatio: 4.1
SJR: 0.816
SNIP: 1.317

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.338

19% from 2018

Impact factor for Avian Pathology from 2016 - 2019
Year Value
2019 2.338
2018 1.961
2017 2.054
2016 1.596
graph view Graph view
table view Table view

3.9

5% from 2019

CiteRatio for Avian Pathology from 2016 - 2020
Year Value
2020 3.9
2019 3.7
2018 3.2
2017 3.0
2016 2.6
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 19% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.733

9% from 2019

SJR for Avian Pathology from 2016 - 2020
Year Value
2020 0.733
2019 0.805
2018 0.696
2017 0.871
2016 0.637
graph view Graph view
table view Table view

1.375

16% from 2019

SNIP for Avian Pathology from 2016 - 2020
Year Value
2020 1.375
2019 1.181
2018 1.083
2017 1.043
2016 0.762
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 9% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 16% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Avian Pathology

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Taylor and Francis

Avian Pathology

Avian Pathology will consider original material relevant to the entire field of infectious and non-infectious diseases of poultry and all other birds, including infections that may be of zoonotic/food-borne importance. Subject areas include pathology; diagnosis; detection and ...... Read More

Animal Science and Zoology

Food Animals

General Immunology and Microbiology

Agricultural and Biological Sciences

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Last updated on
23 Jun 2020
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ISSN
0307-9457
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Impact Factor
High - 1.266
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Open Access
No
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Taylor and Francis Custom Citation
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys Rev B. 1982; 25(7):4515–4532. Available from: 10.1103/PhysRevB.25.4515.

Top papers written in this journal

Journal Article DOI: 10.1080/03079450400013162
Clostridium perfringens in poultry: an emerging threat for animal and public health
01 Dec 2004 - Avian Pathology

Abstract:

The incidence of Clostridium perfringens-associated necrotic enteritis in poultry has increased in countries that stopped using antibiotic growth promoters. Necrotic enteritis and the subclinical form of C. perfringens infection in poultry are caused by C. perfringens type A, producing the alpha toxin, and to a lesser extent ... The incidence of Clostridium perfringens-associated necrotic enteritis in poultry has increased in countries that stopped using antibiotic growth promoters. Necrotic enteritis and the subclinical form of C. perfringens infection in poultry are caused by C. perfringens type A, producing the alpha toxin, and to a lesser extent type C, producing both alpha toxin and beta toxin. Some strains of C. perfringens type A produce an enterotoxin at the moment of sporulation and are responsible for foodborne disease in humans. The mechanisms of colonization of the avian small intestinal tract and the factors involved in toxin production are largely unknown. It is generally accepted, however, that predisposing factors are required for these bacteria to colonize and cause disease in poultry. The best known predisposing factor is mucosal damage, caused by coccidiosis. Diets with high levels of indigestible, water-soluble non-starch polysaccharides, known to increase the viscosity of the intestinal contents, also predispose to necrotic enteritis. Standardized models are being developed for the reproduction of colonization of poultry by C. perfringens and the C. perfringens-associated necrotic enteritis. One such model is a combined infection with Eimeria species and C. perfringens. Few tools and strategies are available for prevention and control of C. perfringens in poultry. Vaccination against the pathogen and the use of probiotic and prebiotic products has been suggested, but are not available for practical use in the field at the present time. The most cost-effective control will probably be achieved by balancing the composition of the feed. read more read less

Topics:

Clostridium perfringens (65%)65% related to the paper
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566 Citations
Journal Article DOI: 10.1080/03079450050045431
Acute infectious bursal disease in poultry: A review
01 Jun 2000 - Avian Pathology

Abstract:

This review is focused on the acute form of infectious bursal disease (IBD) caused by very virulent IBD virus (vvIBDV). First described in Europe about 10 years ago, this new form of the disease has rapidly spread all over the world, causing dramatic losses; after a decade, it still represents a considerable threat to the pou... This review is focused on the acute form of infectious bursal disease (IBD) caused by very virulent IBD virus (vvIBDV). First described in Europe about 10 years ago, this new form of the disease has rapidly spread all over the world, causing dramatic losses; after a decade, it still represents a considerable threat to the poultry industry. Emergence of the acute forms of the disease has drastically changed the epidemiology of IBD. Although their origin is still under investigation, vvIBDVs have spread all over the world in a very explosive but conserved manner. This raises the question of the origin of vvIBDVs, of the possible existence of reservoirs and of the possible emergence of new, distinct lineages in the future. While it has become clear that amino acids within the variable region of virus protein VP2 account for the molecular basis of antigenic variation, no definite hot spot that determines pathogenicity has been identified. Fingerprints of VP2 on vvIBDVs have to be considered more as common evo... read more read less

Topics:

Infectious bursal disease (53%)53% related to the paper
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474 Citations
open accessOpen access Journal Article DOI: 10.1080/03079450500112195
Intercurrent coccidiosis and necrotic enteritis of chickens: rational, integrated disease management by maintenance of gut integrity.
R. B. Williams1
01 Jun 2005 - Avian Pathology

Abstract:

Coccidiosis and necrotic enteritis (NE) are globally common, sometimes intercurrent, diseases of poultry. The risk of NE, due to the Gram-positive bacterium Clostridium perfringens, has increased in recent years because of the voluntary or legally required withdrawal of the use of certain in-feed antibiotic growth promoters w... Coccidiosis and necrotic enteritis (NE) are globally common, sometimes intercurrent, diseases of poultry. The risk of NE, due to the Gram-positive bacterium Clostridium perfringens, has increased in recent years because of the voluntary or legally required withdrawal of the use of certain in-feed antibiotic growth promoters with anticlostridial activity. In-feed ionophorous anticoccidial drugs incidentally also possess anticlostridial activity. Such ionophores, although not banned, are usually precluded when live anticoccidial vaccines are used, potentially increasing yet further the risk of NE. This review provides information for the design of rational, integrated management strategies for the prevention and control of coccidiosis and NE in chickens by maintaining gut integrity. Because of differences in local availability of feed ingredients and national legislations regarding antibiotic growth promoters and anticoccidial vaccine licensing, no universal strategy is applicable. The diseases and their interactions are described under the headings of forms of disease, diagnosis, sources of infection, pathophysiological effects, predisposing factors, and control methods. Elements of gut integrity, which influences host predisposition and clinical responses to disease, include physical development, immune competence, gut enzyme activity, mucin production, gut flora and epithelial damage. Experimental studies of coccidiosis and NE are compared, and where possible reconciled, with field observations. Gaps in knowledge and necessary further experiments are identified. Insights are provided regarding interactions between coccidiosis, NE, and the use of live anticoccidial vaccines. Recent changes in NE prevalence in commercial flocks, and their possible causes, are discussed. The necessarily wide range of topics reviewed emphasizes the enormous complexity of this disease combination, and indicates the importance of a multidisciplinary approach in order to reduce its harmful impact on the world's poultry industry. read more read less
View PDF
446 Citations
Journal Article DOI: 10.1080/03079459308418934
Ascites in poultry
R. J. Julian1
01 Sep 1993 - Avian Pathology

Abstract:

Research on ascites occurring in meat-type chickens reared at moderate and low altitude has shown that the pathogenesis is similar to that of the high altitude disease. Pulmonary hypertension (PH) caused by increased blood flow or increased resistance to flow in the lung results in right ventricular hypertrophy (RVH), valvula... Research on ascites occurring in meat-type chickens reared at moderate and low altitude has shown that the pathogenesis is similar to that of the high altitude disease. Pulmonary hypertension (PH) caused by increased blood flow or increased resistance to flow in the lung results in right ventricular hypertrophy (RVH), valvular insufficiency, increased venous pressure and ascites. The structure of the avian heart, with its thin-walled right ventricle and muscular right atrioventricular valve, allows PH to induce heart failure quickly. The sudden increase in pulmonary hypertension syndrome (PHS) in meat-type chickens in the 1980s was associated with a rapid increase in growth rate and feed conversion. This was a result of a combination of genetic selection for fast-growing, heavy broilers with low feed conversion and a more dense, high caloric, pelleted food that supplied all the nutrients required for rapid growth and encouraged a high nutrient intake. PHS in meat-type chickens is usually primary pulmonary hypertension, that is, PH that occurs without evidence of prior heart or lung disease that could account for the increase in blood flow or resistance to flow that results in the increased pressure in the pulmonary arteries. The lungs of birds are firm and fixed in the thoracic cavity and they do not expand to draw air into the lung. The blood and air capillaries form a rigid network that allows only minimal expansion of the blood capillaries when more blood flow is required. Air is moved through the lung by abdominal movement which draws air in and out of the air sacs. The anatomy and physiology of the avian respiratory system are important in the susceptibility of meat-type chickens to PHS. The small stature of the modern meat-type chicken, the large, heavy breast mass, the pressure from abdominal contents on air sacs, and the small lung volume compared to body weight, may all be involved in the increased incidence of PHS. There is limited space for blood flow in the avian lung. Factors that increase blood flow or increase resistance to flow are additive. Increased blood viscosity caused by the polycythaemia of hypoxia, or increased erythrocyte rigidity of high Na (+), are more likely to produce PH in fast-growing than in slow-growing birds. Increased flow due to cold exposure is also additive. Ascites caused by PH is a production-related disease at low altitude. It can be prevented easily by restricting growth rate. It is possible that some meat-type chickens of the phenotype we have created have reached the limit of blood flow through their lungs and that future improvements in growth rate will only be possible if the lung and abdominal cavity capacities are enlarged. read more read less

Topics:

Pulmonary hypertension (55%)55% related to the paper, Lung volumes (54%)54% related to the paper, Heart failure (54%)54% related to the paper, Blood flow (54%)54% related to the paper, Lung (53%)53% related to the paper
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443 Citations
Journal Article DOI: 10.1080/03079450400003601
Investigation of outbreaks of highly pathogenic H5N1 avian influenza in waterfowl and wild birds in Hong Kong in late 2002
01 Oct 2004 - Avian Pathology

Abstract:

Outbreaks of highly pathogenic H5N1 avian influenza have occurred in Hong Kong in chickens and other gallinaceous poultry in 1997, 2001, twice in 2002 and 2003. High mortality rates were seen in gallinaceous birds but not in domestic or wild waterfowl or other wild birds until late 2002 when highly pathogenic H5N1 avian influ... Outbreaks of highly pathogenic H5N1 avian influenza have occurred in Hong Kong in chickens and other gallinaceous poultry in 1997, 2001, twice in 2002 and 2003. High mortality rates were seen in gallinaceous birds but not in domestic or wild waterfowl or other wild birds until late 2002 when highly pathogenic H5N1 avian influenza occurred in waterfowl (geese, ducks and swans), captive Greater Flamingo (Phoenicopterus ruber) and other wild birds (Little Egret Egretta garzetta) at two waterfowl parks and from two dead wild Grey Heron (Ardea cinerea) and a Black-headed Gull (Larus ridibundus) in Hong Kong. H5N1 avian influenza virus was also isolated from a dead feral pigeon (Columba livia) and a dead tree sparrow (Passer montanus) during the second outbreak. The first waterfowl outbreak was controlled by immediate strict quarantine and depopulation 1 week before the second outbreak commenced. Control measures implemented for the second outbreak included strict isolation, culling, increased sanitation and vaccination. Outbreaks in gallinaceous birds occurred in some live poultry markets concurrently with the second waterfowl outbreak, and infection on a chicken farm was detected 1 week after the second waterfowl park outbreak was detected, on the same day the second grey heron case was detected. Subsequent virus surveillance showed the outbreaks had been contained. read more read less

Topics:

Waterfowl (58%)58% related to the paper, Influenza A virus subtype H5N1 (57%)57% related to the paper, Outbreak (54%)54% related to the paper, Greater flamingo (54%)54% related to the paper, Influenza A virus (54%)54% related to the paper
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436 Citations
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Frequently asked questions

1. Can I write Avian Pathology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Avian Pathology guidelines and auto format it.

2. Do you follow the Avian Pathology guidelines?

Yes, the template is compliant with the Avian Pathology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Avian Pathology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Avian Pathology citation style.

4. Can I use the Avian Pathology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Avian Pathology.

5. Can I use a manuscript in Avian Pathology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Avian Pathology that you can download at the end.

6. How long does it usually take you to format my papers in Avian Pathology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Avian Pathology.

7. Where can I find the template for the Avian Pathology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Avian Pathology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Avian Pathology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Avian Pathology an online tool or is there a desktop version?

SciSpace's Avian Pathology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Avian Pathology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Avian Pathology?”

11. What is the output that I would get after using Avian Pathology?

After writing your paper autoformatting in Avian Pathology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Avian Pathology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Avian Pathology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Avian Pathology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Avian Pathology?

The 5 most common citation types in order of usage for Avian Pathology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Avian Pathology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Avian Pathology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Avian Pathology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Avian Pathology Endnote style according to Elsevier guidelines.

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