Example of Drug and Chemical Toxicology format
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Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format
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Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format Example of Drug and Chemical Toxicology format
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open access Open Access

Drug and Chemical Toxicology — Template for authors

Publisher: Taylor and Francis
Categories Rank Trend in last 3 yrs
Public Health, Environmental and Occupational Health #136 of 526 up up by 26 ranks
Chemical Health and Safety #4 of 11 -
Health, Toxicology and Mutagenesis #61 of 134 up up by 4 ranks
Pharmacology #141 of 297 up up by 34 ranks
Toxicology #68 of 122 up up by 6 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 288 Published Papers | 1116 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 13/07/2020
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Related Journals

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Quality:  
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SJR: 0.633
SNIP: 1.433
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Quality:  
High
CiteRatio: 9.1
SJR: 0.842
SNIP: 0.9

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.405

24% from 2018

Impact factor for Drug and Chemical Toxicology from 2016 - 2019
Year Value
2019 2.405
2018 1.946
2017 1.531
2016 1.732
graph view Graph view
table view Table view

3.9

30% from 2019

CiteRatio for Drug and Chemical Toxicology from 2016 - 2020
Year Value
2020 3.9
2019 3.0
2018 3.0
2017 2.9
2016 3.1
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 24% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 30% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.422

SJR for Drug and Chemical Toxicology from 2016 - 2020
Year Value
2020 0.422
2019 0.422
2018 0.479
2017 0.46
2016 0.542
graph view Graph view
table view Table view

0.689

8% from 2019

SNIP for Drug and Chemical Toxicology from 2016 - 2020
Year Value
2020 0.689
2019 0.75
2018 0.813
2017 0.642
2016 0.792
graph view Graph view
table view Table view

insights Insights

  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 8% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Drug and Chemical Toxicology

Guideline source: View

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Taylor and Francis

Drug and Chemical Toxicology

Drug and Chemical Toxicology publishes full-length research papers, review articles and short communications that encompass a broad spectrum of toxicological data surrounding risk assessment and harmful exposure. Manuscripts are considered according to their relevance to the j...... Read More

Medicine

i
Last updated on
12 Jul 2020
i
ISSN
0148-0545
i
Impact Factor
High - 1.732
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Acceptance Rate
Not provided
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Taylor and Francis Custom Citation
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Citation Type
Author Year
(Blonder et al., 1982)
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Bibliography Example
Blonder, G. E., Tinkham, M.,, Klapwijk, T. M. 1982. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B, 25(7), 4515–4532.

Top papers written in this journal

Journal Article DOI: 10.1081/DCT-120020404
Thymoquinone is a potent superoxide anion scavenger
Osama A. Badary1, Ragia A. Taha1, Ayman M. Gamal El-Din1, Mohamed H. Abdel-Wahab1

Abstract:

The antioxidant and pro-oxidant effects of thymoquinone (TQ), a natural main constituent of the volatile oil of Nigella saliva seeds, and a synthetic structurally-related tert-butylhydroquinone (TBHQ), were examined in vitro. Both TQ and TBHQ efficiently inhibited iron-dependent microsomal lipid peroxidation in a concentratio... The antioxidant and pro-oxidant effects of thymoquinone (TQ), a natural main constituent of the volatile oil of Nigella saliva seeds, and a synthetic structurally-related tert-butylhydroquinone (TBHQ), were examined in vitro. Both TQ and TBHQ efficiently inhibited iron-dependent microsomal lipid peroxidation in a concentration-dependent manner with median inhibitory concentration (IC50) values of 16.8 and 14.9 microM, respectively. TBHQ was stronger than TQ as a scavenger of 2,2'-diphenyl-p-picrylhydrazyl radical (DPPH) (IC50 = 5 microM, 200 times more active than TQ) and as a scavenger of hydroxyl radical (OH*) with an IC50 of 4.6 microM (approximately 10 times more active than TQ). TQ was more active than TBHQ as a superoxide anion scavenger with IC50 of 3.35 microM compared to 18.1 microM for TBHQ. Only TBHQ significantly promoted DNA damage in the bleomycin-Fe(III) system. The results suggest that both TQ and TBHQ have strong antioxidant potentials through scavenging ability of different free radicals. Moreover, the data indicate that TQ is acting mainly as a potent superoxide anion scavenger. read more read less

Topics:

Thymoquinone (51%)51% related to the paper, Superoxide (51%)51% related to the paper
403 Citations
Journal Article DOI: 10.3109/01480547809034433
Serum enzymes as indicators of chemically induced liver damage.
R. B. Drotman1, G. T. Lawhorn1

Abstract:

Rats were dosed with CCl4 or diethylamine to induce liver injury. The time and magnitude of peak liver injury were assessed by histopathological examination of liver specimens taken at intervals after dosing. Serum enzymes were measured at the same intervals. Serum ornithine carbamyl transferase (SOCT) activity increased at l... Rats were dosed with CCl4 or diethylamine to induce liver injury. The time and magnitude of peak liver injury were assessed by histopathological examination of liver specimens taken at intervals after dosing. Serum enzymes were measured at the same intervals. Serum ornithine carbamyl transferase (SOCT) activity increased at least 6-fold in animals that showed liver damage by histopathology, and fell again as the injuries resolved. Measurements of other enzymes were less sensitive. SOCT measurements appear to be as sensitive a method as histopathology for detecting liver damage caused by administering xenobiotics. Since serum enzyme measurements do not require that the animals be sacrificed, they can be used for repeated examinations of the same animals, thus increasing the likelihood of detecting transient injury. read more read less

Topics:

Liver injury (57%)57% related to the paper
383 Citations
Journal Article DOI: 10.1081/DCT-120014789
Nmr-based metabolomics
Nicholas V. Reo1

Abstract:

Similar to genomics and proteomics which yield vast amounts of data about the expression of genes and proteins, metabolomics refers to the whole metabolic profile of the cell. The focus of this report concerns the use of nuclear magnetic resonance (NMR) spectroscopy for metabolic analyses and, in particular, its use in toxico... Similar to genomics and proteomics which yield vast amounts of data about the expression of genes and proteins, metabolomics refers to the whole metabolic profile of the cell. The focus of this report concerns the use of nuclear magnetic resonance (NMR) spectroscopy for metabolic analyses and, in particular, its use in toxicology for examining the metabolic profile of biofluids. Examples from the literature will demonstrate how 1H NMR and pattern recognition methods are used to obtain the urinary metabolic profile, and how this profile is affected by exposure to various toxicants. These particular studies which focus on the metabolic profiles of biofluids, specifically urine, are referred to as metabonomics. NMR-based metabonomics provides a means to categorize organ-specific toxicity, monitor the onset and progression of toxicological effects, and identify biomarkers of toxicity. A future challenge, however, is to describe the cellular metabolome for purposes of understanding cellular functions (i.e., metabolomics). Thus the capabilities and advantages of multinuclear NMR to provide metabolic information in cells and tissues will also be discussed. Such information is essential if metabolomics is to provide a complementary dataset which together with genomics and proteomics can be used to construct computer network models to describe cellular functions. read more read less

Topics:

Metabolomics (56%)56% related to the paper, Proteomics (52%)52% related to the paper, Metabolome (52%)52% related to the paper
298 Citations
Journal Article DOI: 10.3109/01480545.2013.866134
Effects of sub-acute exposure to TiO2, ZnO and Al2O3 nanoparticles on oxidative stress and histological changes in mouse liver and brain.
Rupal Shrivastava1, Saimah Raza1, Abhishek Yadav1, Pramod Kushwaha1, Swaran J.S. Flora1

Abstract:

Nanomaterials are at the leading edge of the rapidly developing field of nanotechnology. However the information regarding toxicity of these nanoparticles on humans and environment is still deficient. The present study investigated the toxic effects of three metal oxide nanoparticles, TiO2, ZnO and Al2O3 on mouse erythrocytes... Nanomaterials are at the leading edge of the rapidly developing field of nanotechnology. However the information regarding toxicity of these nanoparticles on humans and environment is still deficient. The present study investigated the toxic effects of three metal oxide nanoparticles, TiO2, ZnO and Al2O3 on mouse erythrocytes, brain and liver. Male mice were administered a single oral dose of 500 mg/kg of each nanoparticles for 21 consecutive days. The results suggest that exposure to these nano metallic particles produced a significant oxidative stress in erythrocyte, liver and brain as evident from enhanced levels of Reactive Oxygen Species (ROS) and altered antioxidant enzymes activities. A significant increase in dopamine and norepinephrine levels in brain cerebral cortex and increased brain oxidative stress suggest neurotoxic potential of these nanoparticles. Transmission electron microscopic (TEM) analysis indicated the presence of these nanoparticles inside the cytoplasm and nucleus. These changes were also supported by the inhibition of CuZnSOD and MnSOD, considered as important biomarkers of oxidative stress. The toxic effects produced by these nanoparticles were more pronounced in the case of zinc oxide, followed by aluminum oxide and titanium dioxide, respectively. The present results further suggest the involvement of oxidative stress as one of the main mechanisms involved in nanoparticles induced toxic manifestations. read more read less

Topics:

Nanotoxicology (58%)58% related to the paper, Oxidative stress (56%)56% related to the paper, Reactive oxygen species (51%)51% related to the paper
157 Citations
Journal Article DOI: 10.1081/DCT-120005891
Testicular gametogenic and steroidogenic activities in cyclophosphamide treated rat: a correlative study with testicular oxidative stress.
Debidas Ghosh1, Ujjal Baran Das1, Sampa Ghosh1, M. Mallick1, Jogendra Mohan Debnath1

Abstract:

The present work examined the changes in testicular activities in relation to testicular oxidative stress in cyclophosphamide as well as human chorionic gonadotrophin (hCG) co-treated cyclophosphamide treated Wistar strain rats. Testicular activities were evaluated by the quantification of spermatogenesis and by the measureme... The present work examined the changes in testicular activities in relation to testicular oxidative stress in cyclophosphamide as well as human chorionic gonadotrophin (hCG) co-treated cyclophosphamide treated Wistar strain rats. Testicular activities were evaluated by the quantification of spermatogenesis and by the measurement of steroidogenic key enzyme activities along with plasma levels of testosterone. Testicular oxidative stress in relation to cyclophosphamide treatment was monitored by the study of products of free radicals like conjugated dienes and malondialdehyde (MDA) as well as the activity of testicular antioxidant enzymes like peroxidase and catalase. Cyclophosphamide treatment at the dose of 5 mg/kg body weight/day for 28 days resulted a significant diminution in the activities of testicular Δ5, 3β-hydroxysteroid dehydrogenase (Δ5, 3β-HSD), 17β-hydroxysteroid dehydrogenase (17β-HSD) activities, plasma level of testosterone along with significant reduction in the number of germ cells... read more read less

Topics:

Malondialdehyde (51%)51% related to the paper, Oxidative stress (50%)50% related to the paper
150 Citations
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Frequently asked questions

1. Can I write Drug and Chemical Toxicology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Drug and Chemical Toxicology guidelines and auto format it.

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Yes, the template is compliant with the Drug and Chemical Toxicology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Drug and Chemical Toxicology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Drug and Chemical Toxicology citation style.

4. Can I use the Drug and Chemical Toxicology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Drug and Chemical Toxicology.

5. Can I use a manuscript in Drug and Chemical Toxicology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Drug and Chemical Toxicology that you can download at the end.

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It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Drug and Chemical Toxicology.

7. Where can I find the template for the Drug and Chemical Toxicology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Drug and Chemical Toxicology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Drug and Chemical Toxicology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Drug and Chemical Toxicology an online tool or is there a desktop version?

SciSpace's Drug and Chemical Toxicology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Drug and Chemical Toxicology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Drug and Chemical Toxicology?”

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After writing your paper autoformatting in Drug and Chemical Toxicology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Drug and Chemical Toxicology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Drug and Chemical Toxicology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Drug and Chemical Toxicology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Drug and Chemical Toxicology?

The 5 most common citation types in order of usage for Drug and Chemical Toxicology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Drug and Chemical Toxicology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Drug and Chemical Toxicology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Drug and Chemical Toxicology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Drug and Chemical Toxicology Endnote style according to Elsevier guidelines.

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