Example of Free Radical Research format
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Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format
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Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format Example of Free Radical Research format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access

Free Radical Research — Template for authors

Publisher: Taylor and Francis
Categories Rank Trend in last 3 yrs
Biochemistry #149 of 415 down down by 51 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 381 Published Papers | 2163 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 13/07/2020
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Related Journals

open access Open Access
recommended Recommended

Taylor and Francis

Quality:  
High
CiteRatio: 13.9
SJR: 4.634
SNIP: 2.046
open access Open Access

Springer

Quality:  
High
CiteRatio: 4.3
SJR: 0.633
SNIP: 1.433
open access Open Access

De Gruyter

Quality:  
High
CiteRatio: 6.5
SJR: 1.246
SNIP: 0.854

Royal Society of Chemistry

Quality:  
High
CiteRatio: 6.0
SJR: 0.923
SNIP: 0.776

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.839

0% from 2018

Impact factor for Free Radical Research from 2016 - 2019
Year Value
2019 2.839
2018 2.825
2017 3.038
2016 3.188
graph view Graph view
table view Table view

5.7

16% from 2019

CiteRatio for Free Radical Research from 2016 - 2020
Year Value
2020 5.7
2019 4.9
2018 5.5
2017 6.2
2016 5.8
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 0% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 16% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.861

12% from 2019

SJR for Free Radical Research from 2016 - 2020
Year Value
2020 0.861
2019 0.772
2018 0.844
2017 1.044
2016 1.114
graph view Graph view
table view Table view

0.977

12% from 2019

SNIP for Free Radical Research from 2016 - 2020
Year Value
2020 0.977
2019 0.872
2018 0.881
2017 0.959
2016 1.027
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 12% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 12% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Free Radical Research

Guideline source: View

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Use of these names, trademarks and brands does not imply endorsement or affiliation. Disclaimer Notice

Taylor and Francis

Free Radical Research

Free Radical Research aims to publish high-quality research papers, hypotheses and reviews in all areas in the fields of Free radicals and other reactive species in biological, clinical, environmental and other systems; Redox signalling; Antioxidants, including diet-derived an...... Read More

Medicine

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Last updated on
13 Jul 2020
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ISSN
1071-5762
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Impact Factor
High - 1.049
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Taylor and Francis Custom Citation
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys Rev B. 1982; 25(7):4515–4532. Available from: 10.1103/PhysRevB.25.4515.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3109/10715761003667554
Reactive oxygen species in cancer
Geou Yarh Liou1, Peter Storz1
19 Apr 2010 - Free Radical Research

Abstract:

Elevated rates of reactive oxygen species (ROS) have been detected in almost all cancers, where they promote many aspects of tumour development and progression. However, tumour cells also express increased levels of antioxidant proteins to detoxify from ROS, suggesting that a delicate balance of intracellular ROS levels is re... Elevated rates of reactive oxygen species (ROS) have been detected in almost all cancers, where they promote many aspects of tumour development and progression. However, tumour cells also express increased levels of antioxidant proteins to detoxify from ROS, suggesting that a delicate balance of intracellular ROS levels is required for cancer cell function. Further, the radical generated, the location of its generation, as well as the local concentration is important for the cellular functions of ROS in cancer. A challenge for novel therapeutic strategies will be the fine tuning of intracellular ROS signalling to effectively deprive cells from ROS-induced tumour promoting events, towards tipping the balance to ROS-induced apoptotic signalling. Alternatively, therapeutic antioxidants may prevent early events in tumour development, where ROS are important. However, to effectively target cancer cells specific ROS-sensing signalling pathways that mediate the diverse stress-regulated cellular functions need to be identified. This review discusses the generation of ROS within tumour cells, their detoxification, their cellular effects, as well as the major signalling cascades they utilize, but also provides an outlook on their modulation in therapeutics. read more read less

Topics:

Cancer cell (52%)52% related to the paper
View PDF
2,625 Citations
Journal Article DOI: 10.3109/10715769509145649
The relative antioxidant activities of plant-derived polyphenolic flavonoids
Catherine Rice-Evans1, Nicholas J. Miller1, Paul G. Bolwell2, Peter M. Bramley2, J.B. Pridham2
01 Jan 1995 - Free Radical Research

Abstract:

The relative antioxidant activities, against radicals generated in the aqueous phase, of a range of plant-derived polyphenolic flavonoids, constituents of fruit, vegetables, tea and wine, have been assessed. The results show that compounds such as quercetin and cyanidin, with 3′,4′ dihydroxy substituents in the B ring and con... The relative antioxidant activities, against radicals generated in the aqueous phase, of a range of plant-derived polyphenolic flavonoids, constituents of fruit, vegetables, tea and wine, have been assessed. The results show that compounds such as quercetin and cyanidin, with 3′,4′ dihydroxy substituents in the B ring and conjugation between the A and B rings, have antioxidant potentials four times that of Trolox, the vitamin E analogue. Removing the ortho-dihydroxy substitution, as in kaempferol, or the potential for electron deloculisation by reducing the 2.3 double bond in the C ring, as in catechin and epicatechin, decreases the antioxidant activity by more than 50%. but these structures are still more effective than α-tocopherol or ascorbate. The relative significance of the positions and extents of hydroxylation of the A and B rings to the total antioxidant activity of these plant polyphenols is demonstrated. read more read less

Topics:

Trolox (61%)61% related to the paper, Catechin (54%)54% related to the paper, Polyphenol (54%)54% related to the paper, Kaempferol (54%)54% related to the paper, Antioxidant (53%)53% related to the paper
2,101 Citations
Journal Article DOI: 10.1080/10715769900300851
Glutathione and glutathione-dependent enzymes represent a co-ordinately regulated defence against oxidative stress
John D. Hayes1, Lesley I. McLellan1
01 Oct 1999 - Free Radical Research

Abstract:

Increases in the intracellular levels of reactive oxygen species (ROS), frequently referred to as oxidative stress, represents a potentially toxic insult which if not counteracted will lead to membrane dysfunction, DNA damage and inactivation of proteins. Chronic oxidative stress has numerous pathological consequences includi... Increases in the intracellular levels of reactive oxygen species (ROS), frequently referred to as oxidative stress, represents a potentially toxic insult which if not counteracted will lead to membrane dysfunction, DNA damage and inactivation of proteins. Chronic oxidative stress has numerous pathological consequences including cancer, arthritis and neurodegenerative disease. Glutathione-associated metabolism is a major mechanism for cellular protection against agents which generate oxidative stress. It is becoming increasingly apparent that the glutathione tripeptide is central to a complex multifaceted detoxification system, where there is substantial inter-dependence between separate component members. Glutathione participates in detoxification at several different levels, and may scavenge free radicals, reduce peroxides or be conjugated with electrophilic compounds. Thus, glutathione provides the cell with multiple defences not only against ROS but also against their toxic products. This article discusses how glutathione biosynthesis, glutathione peroxidases, glutathione S-transferases and glutathione S-conjugate efflux pumps function in an integrated fashion to allow cellular adaption to oxidative stress. Co-ordination of this response is achieved, at least in part, through the antioxidant responsive element (ARE) which is found in the promoters of many of the genes that are inducible by oxidative and chemical stress. Transcriptional activation through this enhancer appears to be mediated by basic leucine zipper transcription factors such as Nrf and small Maf proteins. The nature of the intracellular sensor(s) for ROS and thiol-active chemicals which induce genes through the ARE is described. Gene activation through the ARE appears to account for the enhanced antioxidant and detoxification capacity of normal cells effected by many cancer chemopreventive agents. In certain instances it may also account for acquired resistance of tumours to cancer chemotherapeutic drugs. It is therefore clear that determining the mechanisms involved in regulation of ARE-driven gene expression has enormous medical implications. read more read less

Topics:

Glutathione synthetase (67%)67% related to the paper, Glutathione reductase (65%)65% related to the paper, GPX4 (63%)63% related to the paper, GPX1 (63%)63% related to the paper, Glutathione peroxidase (62%)62% related to the paper
1,476 Citations
open accessOpen access Journal Article DOI: 10.1080/10715760600918142
Protein oxidation and aging
Earl R. Stadtman1
01 Dec 2006 - Free Radical Research

Abstract:

Organisms are constantly exposed to various forms of reactive oxygen species (ROS) that lead to oxidation of proteins, nucleic acids, and lipids. Protein oxidation can involve cleavage of the polypeptide chain, modification of amino acid side chains, and conversion of the protein to derivatives that are highly sensitive to pr... Organisms are constantly exposed to various forms of reactive oxygen species (ROS) that lead to oxidation of proteins, nucleic acids, and lipids. Protein oxidation can involve cleavage of the polypeptide chain, modification of amino acid side chains, and conversion of the protein to derivatives that are highly sensitive to proteolytic degradation. Unlike other types of modification (except cysteine oxidation), oxidation of methionine residues to methionine sulfoxide is reversible; thus, cyclic oxidation and reduction of methionine residues leads to consumption of ROS and thereby increases the resistance of proteins to oxidation. The importance of protein oxidation in aging is supported by the observation that levels of oxidized proteins increase with animal age. The age-related accumulation of oxidized proteins may reflect age-related increases in rates of ROS generation, decreases in antioxidant activities, or losses in the capacity to degrade oxidized proteins. read more read less

Topics:

Protein oxidation (74%)74% related to the paper, Methionine sulfoxide (61%)61% related to the paper, Methionine (57%)57% related to the paper, Cysteine (54%)54% related to the paper, Antioxidant (54%)54% related to the paper
View PDF
1,464 Citations
Journal Article DOI: 10.1080/10715769900300841
Antioxidant defence mechanisms: from the beginning to the end (of the beginning).
Barry Halliwell1
01 Oct 1999 - Free Radical Research

Abstract:

When life first evolved on Earth, there was little oxygen in the atmosphere. Evolution of antioxidant defences must have been closely associated with the evolution of photosynthesis and of O2-dependent electron transport mechanisms. Studies with mice lacking antioxidant defences confirm the important roles of MnSOD and transf... When life first evolved on Earth, there was little oxygen in the atmosphere. Evolution of antioxidant defences must have been closely associated with the evolution of photosynthesis and of O2-dependent electron transport mechanisms. Studies with mice lacking antioxidant defences confirm the important roles of MnSOD and transferrin in maintaining health, but show that glutathione peroxidase (GPX) and CuZnSOD are not essential for everyday life (at least in mice). Superoxide can be cytotoxic by several mechanisms: one is the formation of hydroxyl radicals. There is good evidence that OH* formation occurs in vivo. Other important antioxidants may include thioredoxin, and selenoproteins other than GPX. Nitric oxide may be an important antioxidant in the vascular system. Diet-derived antioxidants are important in maintaining human health, but recent studies employing "biomarkers" of oxidative DNA damage are questioning the "antioxidant" roles of beta-carotene and ascorbate. An important area of future research will be elucidation of the reasons why levels of steady-state oxidative damage to DNA and lipids vary so much between individuals, and their predictive value for the later development of human disease. read more read less

Topics:

Antioxidant (52%)52% related to the paper, Superoxide dismutase (51%)51% related to the paper, Oxidative stress (51%)51% related to the paper
946 Citations
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With SciSpace, you do not need a word template for Free Radical Research.

It automatically formats your research paper to Taylor and Francis formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

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Free Radical Research format uses Taylor and Francis Custom Citation citation style.

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Frequently asked questions

1. Can I write Free Radical Research in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Free Radical Research guidelines and auto format it.

2. Do you follow the Free Radical Research guidelines?

Yes, the template is compliant with the Free Radical Research guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Free Radical Research?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Free Radical Research citation style.

4. Can I use the Free Radical Research templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Free Radical Research.

5. Can I use a manuscript in Free Radical Research that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Free Radical Research that you can download at the end.

6. How long does it usually take you to format my papers in Free Radical Research?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Free Radical Research.

7. Where can I find the template for the Free Radical Research?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Free Radical Research's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Free Radical Research's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Free Radical Research an online tool or is there a desktop version?

SciSpace's Free Radical Research is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Free Radical Research?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Free Radical Research?”

11. What is the output that I would get after using Free Radical Research?

After writing your paper autoformatting in Free Radical Research, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Free Radical Research's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Free Radical Research?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Free Radical Research. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Free Radical Research?

The 5 most common citation types in order of usage for Free Radical Research are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Free Radical Research?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Free Radical Research's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Free Radical Research in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Free Radical Research Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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