Example of Journal of Applied Toxicology format
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Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format
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Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format Example of Journal of Applied Toxicology format
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open access Open Access

Journal of Applied Toxicology — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Toxicology #27 of 122 down down by 8 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 556 Published Papers | 3444 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 30/06/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.997

2% from 2018

Impact factor for Journal of Applied Toxicology from 2016 - 2019
Year Value
2019 2.997
2018 3.065
2017 2.909
2016 3.159
graph view Graph view
table view Table view

6.2

5% from 2019

CiteRatio for Journal of Applied Toxicology from 2016 - 2020
Year Value
2020 6.2
2019 5.9
2018 5.9
2017 6.1
2016 5.9
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 2% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.784

2% from 2019

SJR for Journal of Applied Toxicology from 2016 - 2020
Year Value
2020 0.784
2019 0.796
2018 0.829
2017 0.958
2016 0.922
graph view Graph view
table view Table view

0.976

10% from 2019

SNIP for Journal of Applied Toxicology from 2016 - 2020
Year Value
2020 0.976
2019 0.89
2018 0.921
2017 1.041
2016 0.964
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 2% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 10% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Journal of Applied Toxicology

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Wiley

Journal of Applied Toxicology

Journal of Applied Toxicology publishes peer-reviewed original reviews and hypothesis-driven research articles on mechanistic, fundamental and applied research relating to the toxicity of drugs and chemicals at the molecular, cellular, tissue, target organ and whole body level...... Read More

Toxicology

Pharmacology, Toxicology and Pharmaceutics

i
Last updated on
30 Jun 2020
i
ISSN
0260-437X
i
Impact Factor
High - 1.046
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
apa
i
Citation Type
Author Year
(Blonder et al., 1982)
i
Bibliography Example
Blonder, G.E., Tinkham, M., Klapwijk, T.M., 1982. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25, 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1002/JAT.727
A good practice guide to the administration of substances and removal of blood, including routes and volumes

Abstract:

This article is the result of an initiative between the European Federation of Pharmaceutical Industries Associations (EFPIA) and the European Centre for the Validation of Alternative Methods (ECVAM). Its objectives are to provide the researcher in the safety evaluation laboratory with an up-to-date, easy-to-use set of data s... This article is the result of an initiative between the European Federation of Pharmaceutical Industries Associations (EFPIA) and the European Centre for the Validation of Alternative Methods (ECVAM). Its objectives are to provide the researcher in the safety evaluation laboratory with an up-to-date, easy-to-use set of data sheets to aid in the study design process whilst at the same time affording maximum welfare considerations to the experimental animals. Although this article is targeted at researchers in the European Pharmaceutical Industry, it is considered that the principles underpinning the data sets and refinement proposals are equally applicable to all those who use these techniques on animals in their research, whether in research institutes, universities or other sectors of industry. The implications of this article may lead to discussion with regulators, such as those responsible for pharmacopoeial testing. There are numerous publications dealing with the administration of test substances and the removal of blood samples, and many laboratories also have their own "in-house" guidelines that have been developed by custom and practice over many years. Within European Union Directive 86/609EEC1 we have an obligation to refine experiments to cause the minimum amount of stress. We hope that this article will provide background data useful to those responsible for protocol design and review. This guide is based on peer-reviewed publications whenever possible, but where this is not possible we have used "in-house" data and the experience of those on the working party (as well as helpful comments submitted by the industry) for a final opinion. The guide also addresses the continuing need to refine the techniques associated with the administration of substances and the withdrawal of blood, and suggests ways of doing so. Data-sharing between laboratories should be encouraged to avoid duplication of animal work, as well as sharing practical skills concerning animal welfare and scientific problems caused by "overdosing" in some way or another. The recommendations in this guide refer to the "normal" animal, and special consideration is needed, for instance, during pregnancy and lactation. Interpretation of studies may be confounded when large volumes are administered or excessive sampling employed, particularly if anaesthetics are used. read more read less

Topics:

European union (59%)59% related to the paper
View PDF
1,238 Citations
Journal Article DOI: 10.1002/JAT.1649
Arsenic: toxicity, oxidative stress and human disease.

Abstract:

Arsenic (As) is a toxic metalloid element that is present in air, water and soil. Inorganic arsenic tends to be more toxic than organic arsenic. Examples of methylated organic arsenicals include monomethylarsonic acid [MMA(V)] and dimethylarsinic acid [DMA(V)]. Reactive oxygen species (ROS)-mediated oxidative damage is a comm... Arsenic (As) is a toxic metalloid element that is present in air, water and soil. Inorganic arsenic tends to be more toxic than organic arsenic. Examples of methylated organic arsenicals include monomethylarsonic acid [MMA(V)] and dimethylarsinic acid [DMA(V)]. Reactive oxygen species (ROS)-mediated oxidative damage is a common denominator in arsenic pathogenesis. In addition, arsenic induces morphological changes in the integrity of mitochondria. Cascade mechanisms of free radical formation derived from the superoxide radical, combined with glutathione-depleting agents, increase the sensitivity of cells to arsenic toxicity. When both humans and animals are exposed to arsenic, they experience an increased formation of ROS/RNS, including peroxyl radicals (ROO•), the superoxide radical, singlet oxygen, hydroxyl radical (OH•) via the Fenton reaction, hydrogen peroxide, the dimethylarsenic radical, the dimethylarsenic peroxyl radical and/or oxidant-induced DNA damage. Arsenic induces the formation of oxidized lipids which in turn generate several bioactive molecules (ROS, peroxides and isoprostanes), of which aldehydes [malondialdehyde (MDA) and 4-hydroxy-nonenal (HNE)] are the major end products. This review discusses aspects of chronic and acute exposures of arsenic in the etiology of cancer, cardiovascular disease (hypertension and atherosclerosis), neurological disorders, gastrointestinal disturbances, liver disease and renal disease, reproductive health effects, dermal changes and other health disorders. The role of antioxidant defence systems against arsenic toxicity is also discussed. Consideration is given to the role of vitamin C (ascorbic acid), vitamin E (α-tocopherol), curcumin, glutathione and antioxidant enzymes such as superoxide dismutase, catalase and glutathione peroxidase in their protective roles against arsenic-induced oxidative stress. read more read less

Topics:

Arsenic toxicity (68%)68% related to the paper, Arsenic biochemistry (66%)66% related to the paper, Free-radical theory of aging (61%)61% related to the paper, Reactive oxygen species (60%)60% related to the paper, Arsenic (58%)58% related to the paper
View PDF
1,040 Citations
Journal Article DOI: 10.1002/JAT.1385
Comparative study of cytotoxicity, oxidative stress and genotoxicity induced by four typical nanomaterials: the role of particle size, shape and composition.
Hui Yang1, Chao Liu1, Danfeng Yang1, Huashan Zhang1, Zhuge Xi1

Abstract:

Although the biological effects of some nanomaterials have already been assessed, information on toxicity and possible mechanisms of various particle types are insufficient. Moreover, the role of particle properties in the toxic reaction remains to be fully understood. In this paper, we aimed to explore the interrelationship ... Although the biological effects of some nanomaterials have already been assessed, information on toxicity and possible mechanisms of various particle types are insufficient. Moreover, the role of particle properties in the toxic reaction remains to be fully understood. In this paper, we aimed to explore the interrelationship between particle size, shape, chemical composition and toxicological effects of four typical nanomaterials with comparable properties: carbon black (CB), single wall carbon nanotube, silicon dioxide (SiO(2)) and zinc dioxide (ZnO) nanoparticles. We investigated the cytotoxicity, genotoxicity and oxidative effects of particles on primary mouse embryo fibroblast cells. As observed in the methyl thiazolyl tetrazolium (MTT) and water-soluble tetrazolium (WST) assays, ZnO induced much greater cytotoxicity than non-metal nanoparticles. This was significantly in accordance with intracellular oxidative stress levels measured by glutathione depletion, malondialdehyde production, superoxide dismutase inhibition as well as reactive oxygen species generation. The results indicated that oxidative stress may be a key route in inducing the cytotoxicity of nanoparticles. Compared with ZnO nanoparticles, carbon nanotubes were moderately cytotoxic but induced more DNA damage determined by the comet assay. CB and SiO(2) seemed to be less effective. The comparative analysis demonstrated that particle composition probably played a primary role in the cytotoxic effects of different nanoparticles. However, the potential genotoxicity might be mostly attributed to particle shape. read more read less

Topics:

Genotoxicity (53%)53% related to the paper, Oxidative stress (51%)51% related to the paper, Reactive oxygen species (50%)50% related to the paper
963 Citations
Journal Article DOI: 10.1002/JAT.1660
Triclosan: environmental exposure, toxicity and mechanisms of action
Andrea B. Dann1, Alice Hontela1

Abstract:

Triclosan [5-chloro-2-(2,4-dichlorophenoxy)phenol; TCS] is a broad spectrum antibacterial agent used in personal care, veterinary, industrial and household products. TCS is commonly detected in aquatic ecosystems, as it is only partially removed during the wastewater treatment process. Sorption, biodegradation and photolytic ... Triclosan [5-chloro-2-(2,4-dichlorophenoxy)phenol; TCS] is a broad spectrum antibacterial agent used in personal care, veterinary, industrial and household products. TCS is commonly detected in aquatic ecosystems, as it is only partially removed during the wastewater treatment process. Sorption, biodegradation and photolytic degradation mitigate the availability of TCS to aquatic biota; however the by-products such as methyltriclosan and other chlorinated phenols may be more resistant to degradation and have higher toxicity than the parent compound. The continuous exposure of aquatic organisms to TCS, coupled with its bioaccumulation potential, have led to detectable levels of the antimicrobial in a number of aquatic species. TCS has been also detected in breast milk, urine and plasma, with levels of TCS in the blood correlating with consumer use patterns of the antimicrobial. Mammalian systemic toxicity studies indicate that TCS is neither acutely toxic, mutagenic, carcinogenic, nor a developmental toxicant. Recently, however, concern has been raised over TCS's potential for endocrine disruption, as the antimicrobial has been shown to disrupt thyroid hormone homeostasis and possibly the reproductive axis. Moreover, there is strong evidence that aquatic species such as algae, invertebrates and certain types of fish are much more sensitive to TCS than mammals. TCS is highly toxic to algae and exerts reproductive and developmental effects in some fish. The potential for endocrine disruption and antibiotic cross-resistance highlights the importance of the judicious use of TCS, whereby the use of TCS should be limited to applications where it has been shown to be effective. read more read less

Topics:

Environmental exposure (54%)54% related to the paper, Antibacterial agent (52%)52% related to the paper
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733 Citations
Journal Article DOI: 10.1002/JAT.958
Concentrations of parabens in human breast tumours
Philippa D. Darbre1, A. Aljarrah2, W. R. Miller2, Nick G. Coldham3, Maurice J. Sauer3, G. S. Pope1

Abstract:

Parabens are used as preservatives in many thousands of cosmetic, food and pharmaceutical products to which the human population is exposed. Although recent reports of the oestrogenic properties of parabens have challenged current concepts of their toxicity in these consumer products, the question remains as to whether any of... Parabens are used as preservatives in many thousands of cosmetic, food and pharmaceutical products to which the human population is exposed. Although recent reports of the oestrogenic properties of parabens have challenged current concepts of their toxicity in these consumer products, the question remains as to whether any of the parabens can accumulate intact in the body from the long-term, low-dose levels to which humans are exposed. Initial studies reported here show that parabens can be extracted from human breast tissue and detected by thin-layer chromatography. More detailed studies enabled identification and measurement of mean concentrations of individual parabens in samples of 20 human breast tumours by high-pressure liquid chromatography followed by tandem mass spectrometry. The mean concentration of parabens in these 20 human breast tumours was found to be 20.6 +/- 4.2 ng x g(-1) tissue. Comparison of individual parabens showed that methylparaben was present at the highest level (with a mean value of 12.8 +/- 2.2 ng x g(-1) tissue) and represents 62% of the total paraben recovered in the extractions. These studies demonstrate that parabens can be found intact in the human breast and this should open the way technically for more detailed information to be obtained on body burdens of parabens and in particular whether body burdens are different in cancer from those in normal tissues. read more read less

Topics:

Methylparaben (59%)59% related to the paper, Paraben (58%)58% related to the paper, Ethylparaben (57%)57% related to the paper, Population (51%)51% related to the paper, Environmental exposure (50%)50% related to the paper
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714 Citations
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Journal of Applied Toxicology format uses apa citation style.

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Frequently asked questions

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Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Applied Toxicology guidelines and auto format it.

2. Do you follow the Journal of Applied Toxicology guidelines?

Yes, the template is compliant with the Journal of Applied Toxicology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Applied Toxicology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Applied Toxicology citation style.

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Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Applied Toxicology.

5. Can I use a manuscript in Journal of Applied Toxicology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Journal of Applied Toxicology that you can download at the end.

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12. Is Journal of Applied Toxicology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Applied Toxicology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Applied Toxicology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Applied Toxicology?

The 5 most common citation types in order of usage for Journal of Applied Toxicology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Journal of Applied Toxicology?

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16. Can I download Journal of Applied Toxicology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Applied Toxicology Endnote style according to Elsevier guidelines.

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