Example of Molecular Carcinogenesis format
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Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format
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Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format Example of Molecular Carcinogenesis format
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open access Open Access

Molecular Carcinogenesis — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Molecular Biology #82 of 382 up up by 29 ranks
Cancer Research #54 of 207 up up by 15 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 700 Published Papers | 5648 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 09/06/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.825

12% from 2018

Impact factor for Molecular Carcinogenesis from 2016 - 2019
Year Value
2019 3.825
2018 3.411
2017 3.851
2016 4.185
graph view Graph view
table view Table view

8.1

14% from 2019

CiteRatio for Molecular Carcinogenesis from 2016 - 2020
Year Value
2020 8.1
2019 7.1
2018 7.2
2017 6.5
2016 7.2
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 12% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 14% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.254

2% from 2019

SJR for Molecular Carcinogenesis from 2016 - 2020
Year Value
2020 1.254
2019 1.277
2018 1.176
2017 1.277
2016 1.317
graph view Graph view
table view Table view

0.888

11% from 2019

SNIP for Molecular Carcinogenesis from 2016 - 2020
Year Value
2020 0.888
2019 1.003
2018 0.89
2017 0.903
2016 0.937
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 2% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 11% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Molecular Carcinogenesis

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Wiley

Molecular Carcinogenesis

Molecular Carcinogenesis presents information describing investigations of molecular aspects of the mechanisms involved in chemical, physical, and viral (biological) carcinogenesis. Areas of interest include, but are not limited to, the structure, expression, or function of ge...... Read More

Molecular Biology

Cancer Research

Biochemistry, Genetics and Molecular Biology

i
Last updated on
09 Jun 2020
i
ISSN
0899-1987
i
Impact Factor
High - 1.104
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
apa
i
Citation Type
Numbered
[25]
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Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1002/MC.20465
Molecular Mechanisms of Nrf2-Mediated Antioxidant Response
Wenge Li1, Ah Ng Kong
01 Feb 2009 - Molecular Carcinogenesis

Abstract:

Nrf2 is the key transcription factor regulating the antioxidant response. Nrf2 signaling is repressed by Keap1 at basal condition and induced by oxidative stress. Keap1 is recently identified as a Cullin 3-dependent substrate adaptor protein. A two-sites binding “hinge & latch” model vividly depicts how Keap1 can efficiently ... Nrf2 is the key transcription factor regulating the antioxidant response. Nrf2 signaling is repressed by Keap1 at basal condition and induced by oxidative stress. Keap1 is recently identified as a Cullin 3-dependent substrate adaptor protein. A two-sites binding “hinge & latch” model vividly depicts how Keap1 can efficiently present Nrf2 as substrate for ubiquitination. Oxidative perturbation can impede Keap1-mediated Nrf2 ubiquitination but fail to disrupt Nrf2/Keap1 binding. Nrf2 per se is a redox-sensitive transcripon factor. A new Nrf2-mediated redox signaling model is proposed based on these new discoveries. Free floating Nrf2 protein functions as a redox-sensitive probe. Keap1 instead functions as a gate keeper to control the availability of Nrf2 probes and thus regulates the overall sensitivity of the redox signaling. read more read less

Topics:

KEAP1 (56%)56% related to the paper, Signal transducing adaptor protein (52%)52% related to the paper, Transcription factor (51%)51% related to the paper, Signal transduction (51%)51% related to the paper
View PDF
666 Citations
open accessOpen access Journal Article DOI: 10.1002/(SICI)1098-2744(199903)24:3<153::AID-MC1>3.0.CO;2-P
Microarrays and toxicology: the advent of toxicogenomics.
Emile F. Nuwaysir1, Michael L. Bittner1, Jeffrey M. Trent1, J. Carl Barrett1, Cynthia A. Afshari1
01 Mar 1999 - Molecular Carcinogenesis

Abstract:

The availability of genome-scale DNA sequence information and reagents has radically altered life-science research. This revolution has led to the development of a new scientific subdiscipline derived from a combination of the fields of toxicology and genomics. This subdiscipline, termed toxicogenomics, is concerned with the ... The availability of genome-scale DNA sequence information and reagents has radically altered life-science research. This revolution has led to the development of a new scientific subdiscipline derived from a combination of the fields of toxicology and genomics. This subdiscipline, termed toxicogenomics, is concerned with the identification of potential human and environmental toxicants, and their putative mechanisms of action, through the use of genomics resources. One such resource is DNA microarrays or “chips,” which allow the monitoring of the expression levels of thousands of genes simultaneously. Here we propose a general method by which gene expression, as measured by cDNA microarrays, can be used as a highly sensitive and informative marker for toxicity. Our purpose is to acquaint the reader with the development and current state of microarray technology and to present our view of the usefulness of microarrays to the field of toxicology. Mol. Carcinog. 24:153‐ 159, 1999. © 1999 Wiley-Liss, Inc. read more read less

Topics:

Toxicogenomics (57%)57% related to the paper, Microarray (53%)53% related to the paper, DNA microarray (52%)52% related to the paper
648 Citations
Journal Article DOI: 10.1002/MC.20712
Circulating microRNAs, miR-21, miR-122, and miR-223, in patients with hepatocellular carcinoma or chronic hepatitis.
Jian Xu1, Chen Wu1, Xu Che1, Li Wang1, Dianke Yu1, Tongwen Zhang1, Liming Huang1, Hui Li1, Wen Tan1, Chengfeng Wang1, Dongxin Lin1
01 Feb 2011 - Molecular Carcinogenesis

Abstract:

Numerous studies have shown that aberrant microRNA (miRNA) expression is associated with the development and progression of various types of human cancer and serum miRNAs are potential biomarkers. This study examined whether some commonly deregulated miRNAs in hepatocellular carcinoma (HCC) are presented in serum of patients ... Numerous studies have shown that aberrant microRNA (miRNA) expression is associated with the development and progression of various types of human cancer and serum miRNAs are potential biomarkers. This study examined whether some commonly deregulated miRNAs in hepatocellular carcinoma (HCC) are presented in serum of patients with HCC and can serve as diagnostic markers. Serum miRNAs (miR-21, miR-122, and miR-223) were quantified by real-time quantitative RT-PCR in 101 patients with HCC and 89 healthy controls. In addition, 48 patients with chronic type B hepatitis were also analyzed for comparison. We found that the median levels of miR-21, miR-122, and miR-223 were significantly higher in patients with HCC than those in healthy controls (P = 7.48 × 10−13, P = 6.93 × 10−9, and P = 3.90 × 10−12, respectively). However, these elevated serum miRNAs were also detected in patients with chronic hepatitis (P = 2.05 × 10−12, P = 4.52 × 10−16, and P = 1.65 × 10−11, respectively). Moreover, serum miR-21 and miR-122 in patients with chronic hepatitis were higher than in patients with HCC (P = 3.99 × 10−4 and P = 4.97 × 10−8), although no such significant difference was found for miR-223. Receiver-operator characteristic (ROC) curve analyses suggest that these serum miRNAs may be useful markers for discriminating patients with HCC or chronic hepatitis from healthy controls, but not patients with HCC from patients with chronic hepatitis. Our results indicate that serum miR-21, miR-122 and miR-223 are elevated in patients with HCC or chronic hepatitis and these miRNAs have strong potential to serve as novel biomarkers for liver injury but not specifically for HCC. © 2010 Wiley-Liss, Inc. read more read less

Topics:

Hepatitis (56%)56% related to the paper, Hepatocellular carcinoma (55%)55% related to the paper, MiR-122 (52%)52% related to the paper
508 Citations
Journal Article DOI: 10.1002/MC.2940070303
How many mutations are required for tumorigenesis? implications from human cancer data
01 Jan 1993 - Molecular Carcinogenesis

Topics:

Mutation (genetic algorithm) (56%)56% related to the paper, Carcinogenesis (52%)52% related to the paper
484 Citations
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Molecular Carcinogenesis format uses apa citation style.

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Frequently asked questions

1. Can I write Molecular Carcinogenesis in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Molecular Carcinogenesis guidelines and auto format it.

2. Do you follow the Molecular Carcinogenesis guidelines?

Yes, the template is compliant with the Molecular Carcinogenesis guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Molecular Carcinogenesis?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Molecular Carcinogenesis citation style.

4. Can I use the Molecular Carcinogenesis templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Molecular Carcinogenesis.

5. Can I use a manuscript in Molecular Carcinogenesis that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Molecular Carcinogenesis that you can download at the end.

6. How long does it usually take you to format my papers in Molecular Carcinogenesis?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Molecular Carcinogenesis.

7. Where can I find the template for the Molecular Carcinogenesis?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Molecular Carcinogenesis's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Molecular Carcinogenesis's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Molecular Carcinogenesis an online tool or is there a desktop version?

SciSpace's Molecular Carcinogenesis is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Molecular Carcinogenesis?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Molecular Carcinogenesis?”

11. What is the output that I would get after using Molecular Carcinogenesis?

After writing your paper autoformatting in Molecular Carcinogenesis, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Molecular Carcinogenesis's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Molecular Carcinogenesis?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Molecular Carcinogenesis. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Molecular Carcinogenesis?

The 5 most common citation types in order of usage for Molecular Carcinogenesis are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Molecular Carcinogenesis?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Molecular Carcinogenesis's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Molecular Carcinogenesis in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Molecular Carcinogenesis Endnote style according to Elsevier guidelines.

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