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Andreas Pollreisz

Researcher at Medical University of Vienna

Publications -  85
Citations -  1618

Andreas Pollreisz is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Medicine & Diabetic retinopathy. The author has an hindex of 17, co-authored 69 publications receiving 1146 citations.

Papers
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Journal ArticleDOI

Diabetic cataract-pathogenesis, epidemiology and treatment.

TL;DR: An overview of the pathogenesis of diabetic cataracts, clinical studies investigating the association between diabetes and cataract development, and current treatment ofCataract in diabetics is provided.
Journal Article

Elevated HSP27, HSP70 and HSP90 alpha in chronic obstructive pulmonary disease: markers for immune activation and tissue destruction.

TL;DR: This is the first study to demonstrate elevated serum levels of the described heat shock proteins in patients with COPD, and showed sensitivity and specificity of serum HSP27 and HSP70 as diagnostic markers for COPD.
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Efficacy, durability, and safety of intravitreal faricimab with extended dosing up to every 16 weeks in patients with diabetic macular oedema (YOSEMITE and RHINE): two randomised, double-masked, phase 3 trials

Charles C. Wykoff, +385 more
- 01 Jan 2022 - 
TL;DR: Robust vision gains and anatomical improvements with faricimab were achieved with adjustable dosing up to every 16 weeks, demonstrating the potential for faricIMab to extend the durability of treatment for patients with diabetic macular oedema.
Journal ArticleDOI

Identification and clinical role of choroidal neovascularization characteristics based on optical coherence tomography angiography.

TL;DR: To suggest a novel classification of neovascular age‐related macular degeneration (AMD) based on optical coherence tomography angiography (OCTA) and to correlate morphological characteristics based on Optical Coherence Tomography/OCTA with clinical criteria of disease activity.
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T cell senescence and contraction of T cell repertoire diversity in patients with chronic obstructive pulmonary disease.

TL;DR: The finding of increased CD4+CD28null T cells in COPD indicates that chronic antigen exposure, e.g. through contents of smoke, leads to loss of CD28 and up‐regulation of NK cell receptors expression on T cells on susceptible patients.