scispace - formally typeset
G

Galip Zihni Sanus

Researcher at Istanbul University

Publications -  67
Citations -  1364

Galip Zihni Sanus is an academic researcher from Istanbul University. The author has contributed to research in topics: Cerebrospinal fluid & Cranioplasty. The author has an hindex of 20, co-authored 67 publications receiving 1175 citations. Previous affiliations of Galip Zihni Sanus include Ahi Evran University & Montreal Neurological Institute and Hospital.

Papers
More filters
Journal ArticleDOI

Cranioplasty: Review of materials and techniques.

TL;DR: The principle materials and techniques of cranioplasty are reviewed and there is still no consensus about the best material.
Journal ArticleDOI

Intrathecal gadolinium-enhanced MR cisternography in the evaluation of CSF leakage.

TL;DR: MR cisternography after the intrathecal administration of gadopentate dimeglumine represents an effective and minimally invasive method for evaluating suspected CSF fistulas along the skull base and is an excellent approach to depict the anatomy of CSF spaces and CSF Fistulas.
Journal ArticleDOI

Microsurgical anatomy of the superior petrosal venous complex: new classifications and implications for subtemporal transtentorial and retrosigmoid suprameatal approaches.

TL;DR: The proposed modified classification system and its effect on the surgical exposure may aid in planning the approach directed along the petrous apex and may reduce the probability of venous complications.
Journal ArticleDOI

Middle fossa approach: microsurgical anatomy and surgical technique from the neurosurgical perspective

TL;DR: The surgical technique for themiddle fossa approach which includes an anterior-to-posterior elevation of middle fossa dura starting from the foramen ovale and uses the angle between the IAM and the long axis of the GSPN to localize the meatus from above may be an alternative to previously proposed surgical methods.
Journal ArticleDOI

Evaluation of apoptosis in cerebrospinal fluid of patients with severe head injury.

TL;DR: A prolonged activation of pro-apoptotic (sFas, caspase-3) and anti-ap optotic (bcl-2) proteins after severe TBI in humans is indicates a pivotal role of apoptosis in the pathophysiology of post-traumatic brain oedema, secondary cell destruction and chronic cell loss following severe TBO.