G
Giuseppina Rea
Researcher at National Research Council
Publications - 64
Citations - 2912
Giuseppina Rea is an academic researcher from National Research Council. The author has contributed to research in topics: Chlamydomonas reinhardtii & Internal medicine. The author has an hindex of 26, co-authored 56 publications receiving 2435 citations. Previous affiliations of Giuseppina Rea include Roma Tre University & Tuscia University.
Papers
More filters
Journal ArticleDOI
Functions of amine oxidases in plant development and defence
TL;DR: In plants, the production of hydrogen peroxide deriving from polyamine oxidation has been correlated with cell wall maturation and lignification during development as well as with wound-healing and cell wall reinforcement during pathogen invasion.
Journal ArticleDOI
Nanotechnology in Agriculture: Which Innovation Potential Does It Have?
Leonardo Fernandes Fraceto,Renato Grillo,Gerson Araujo de Medeiros,Viviana Scognamiglio,Giuseppina Rea,Cecilia Bartolucci +5 more
TL;DR: In this article, the authors select and report on recent trends in nanomaterial-based systems and nanodevices that could provide benefits on the food supply chain specifically on sustainable intensification, and management of soil and waste.
Journal ArticleDOI
Biosensing technology for sustainable food safety
TL;DR: This review highlights current and future trends in analytical diagnostic tools focused on the food industry and target analytes to support healthier nutrition.
Journal ArticleDOI
Copper amine oxidase expression in defense responses to wounding and Ascochyta rabiei invasion.
TL;DR: It is demonstrated that local and systemic CuAO induction is essential for H2O2 production in response to wounding and indicate the relevance of these enzymes in protection against pathogens.
Journal ArticleDOI
Involvement of Polyamine Oxidase in Wound Healing
Riccardo Angelini,Alessandra Tisi,Giuseppina Rea,Martha M. Chen,Maurizio Botta,Rodolfo Federico,Alessandra Cona +6 more
TL;DR: It is demonstrated that G3 inhibits wound-induced H2O2 production and strongly reduces lignin and suberin polyphenolic domain deposition along the wound, while it is ineffective in inhibiting the deposition of suber in aliphatic domain.