J
Jian-Kang Zhu
Researcher at Chinese Academy of Sciences
Publications - 596
Citations - 124170
Jian-Kang Zhu is an academic researcher from Chinese Academy of Sciences. The author has contributed to research in topics: Arabidopsis & DNA methylation. The author has an hindex of 161, co-authored 550 publications receiving 105551 citations. Previous affiliations of Jian-Kang Zhu include Beijing Planetarium & University of California.
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Salt and drought stress signal transduction in plants
TL;DR: Salt and drought stress signal transduction consists of ionic and osmotic homeostasis signaling pathways, detoxification (i.e., damage control and repair) response pathways, and pathways for growth regulation.
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Plant cellular and molecular responses to high salinity.
TL;DR: Evidence for plant stress signaling systems is summarized, some of which have components analogous to those that regulate osmotic stress responses of yeast, some that presumably function in intercellular coordination or regulation of effector genes in a cell-/tissue-specific context required for tolerance of plants.
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Plant salt tolerance
TL;DR: A recently discovered halophytic plant species, Thellungiella halophila, now promises to help in the detection of new tolerance determinants and operating pathways in a model system that is not limited to Arabidopsis traits or ecotype variations.
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Abiotic Stress Signaling and Responses in Plants
TL;DR: Core stress-signaling pathways involve protein kinases related to the yeast SNF1 and mammalian AMPK, suggesting that stress signaling in plants evolved from energy sensing.
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Abscisic Acid Inhibits Type 2C Protein Phosphatases via the PYR/PYL Family of START Proteins
Sang-Youl Park,Pauline Fung,Noriyuki Nishimura,Davin R. Jensen,Hiroaki Fujii,Yang Zhao,Shelley Lumba,Julia Santiago,Americo Rodrigues,Tsz-fung Freeman Chow,Simon E. Alfred,Dario Bonetta,Ruth R. Finkelstein,Nicholas J. Provart,Darrell Desveaux,Pedro L. Rodriguez,Peter McCourt,Jian-Kang Zhu,Julian I. Schroeder,Brian F. Volkman,Sean R. Cutler +20 more
TL;DR: PYR/PYLs are ABA receptors functioning at the apex of a negative regulatory pathway that controls ABA signaling by inhibiting PP2Cs, illustrating the power of the chemical genetic approach for sidestepping genetic redundancy.