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Joshua B. Bederson

Researcher at Icahn School of Medicine at Mount Sinai

Publications -  222
Citations -  14803

Joshua B. Bederson is an academic researcher from Icahn School of Medicine at Mount Sinai. The author has contributed to research in topics: Medicine & Subarachnoid hemorrhage. The author has an hindex of 42, co-authored 193 publications receiving 13090 citations. Previous affiliations of Joshua B. Bederson include Albert Einstein College of Medicine & Mount Sinai Hospital.

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Rat middle cerebral artery occlusion: evaluation of the model and development of a neurologic examination.

TL;DR: In this article, the authors examined the incidence and size of infarction after occlusion of different portions of the rat middle cerebral artery (MCA) in order to define the reliability and predictability of this model of brain ischemia.
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Large-Vessel Stroke as a Presenting Feature of Covid-19 in the Young.

TL;DR: Five patients younger than 50 years of age with large-vessel stroke and Covid-19 infection presented to a health system in New York City over a 2-week period with signs of stroke and infection.
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Guidelines for the management of aneurysmal subarachnoid hemorrhage: A statement for healthcare professionals from a special writing group of the stroke council, American heart association

TL;DR: A systematic literature review was conducted based on a search of MEDLINE to identify all relevant randomized clinical trials published between June 30, 1994, and November 1, 2006 to reevaluate the recommendations for management of aneurysmal SAH.
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Evaluation of 2,3,5-triphenyltetrazolium chloride as a stain for detection and quantification of experimental cerebral infarction in rats.

TL;DR: Staining with TTC is a rapid, convenient, inexpensive, and reliable method for the detection and quantification of cerebral infarction in rats 24 hr after the onset of ischemia.
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Cortical Blood Flow and Cerebral Perfusion Pressure in a New Noncraniotomy Model of Subarachnoid Hemorrhage in the Rat

TL;DR: It is demonstrated that although reduced CPP causes the initial decrease in cortical blood flow after SAH, secondary reductions occurring after CPP has reached its nadir are caused by other factors such as acute vasoconstriction.