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Navdeep S. Chandel

Researcher at Northwestern University

Publications -  328
Citations -  59666

Navdeep S. Chandel is an academic researcher from Northwestern University. The author has contributed to research in topics: Mitochondrion & Reactive oxygen species. The author has an hindex of 99, co-authored 286 publications receiving 46249 citations. Previous affiliations of Navdeep S. Chandel include University of Chicago & University of Illinois at Chicago.

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ROS Promotes Cancer Cell Survival through Calcium Signaling

TL;DR: A novel, non-canonical oxidative stress defense mechanism involving TRPA1, a redox-sensitive Ca2+ channel, and the upregulation of anti-apoptotic pathways to promote cancer cell survival is identified.
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Matrix Metalloproteinase (MMP)-1 Induces Lung Alveolar Epithelial Cell Migration and Proliferation, Protects from Apoptosis, and Represses Mitochondrial Oxygen Consumption *

TL;DR: The findings indicate that epithelial expression of MMP-1 inhibits mitochondrial function, increases HIF-1α expression, decreases reactive oxygen species production, and contributes to a proliferative, migratory, and anti-apoptotic AEC phenotype.
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Mitochondrial oxygen sensing: regulation of hypoxia-inducible factor by mitochondrial generated reactive oxygen species.

TL;DR: The model in which mitochondria regulate the stability of HIF through the increased production of ROS (reactive oxygen species) during hypoxia is outlined.
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Cellular energy utilization and supply during hypoxia in embryonic cardiac myocytes.

TL;DR: It is demonstrated that embryonic chick cardiac myocytes can suppress their rates of ATP demand, ATP utilization, and O2 uptake during moderate hypoxia through a mechanism that involves a reversible inhibition of cytochrome-c oxidase.
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Death Induced by CD95 or CD95 Ligand Elimination

TL;DR: It is shown that elimination of CD95 or CD95L results in a form of cell death that is independent of caspase-8, RIPK1/MLKL, and p53, is not inhibited by Bcl-xL expression, and preferentially affects cancer cells.