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Richard A. Dixon

Researcher at University of North Texas

Publications -  628
Citations -  77927

Richard A. Dixon is an academic researcher from University of North Texas. The author has contributed to research in topics: Elicitor & Lignin. The author has an hindex of 126, co-authored 603 publications receiving 71424 citations. Previous affiliations of Richard A. Dixon include Fermilab & University of Texas at Austin.

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Stress-Induced Phenylpropanoid Metabolism.

Richard A. Dixon, +1 more
- 01 Jul 1995 - 
TL;DR: Limiting discussion to stress-induced phenylpropanoids eliminates few of the structural classes, because many compounds that are constitutive in one plant species or tissue can be induced by various stresses in another species or in another tissue of the same plant.
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The oxidative burst in plant disease resistance

TL;DR: Emerging data indicate that the oxidative burst reflects activation of a membrane-bound NADPH oxidase closely resembling that operating in activated neutrophils, which underlies the expression of disease-resistance mechanisms.
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Lignin valorization: improving lignin processing in the biorefinery.

TL;DR: Recent developments in genetic engineering, enhanced extraction methods, and a deeper understanding of the structure of lignin are yielding promising opportunities for efficient conversion of this renewable resource to carbon fibers, polymers, commodity chemicals, and fuels.
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H2O2 from the oxidative burst orchestrates the plant hypersensitive disease resistance response

TL;DR: It is reported here that H2O2 from this oxidative burst not only drives the cross-linking of cell wall structural proteins, but also functions as a local trigger of programmed death in challenged cells and as a diffusible signal for the induction in adjacent cells of genes encoding cellular protectants.
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Nitric oxide functions as a signal in plant disease resistance

TL;DR: It is shown that nitric oxide potentiates the induction of hypersensitive cell death in soybean cells by reactive oxygen intermediates and functions independently of such intermediates to induce genes for the synthesis of protective natural products.