R
Ronald L. Davis
Researcher at Scripps Research Institute
Publications - 192
Citations - 18684
Ronald L. Davis is an academic researcher from Scripps Research Institute. The author has contributed to research in topics: Mushroom bodies & Olfactory memory. The author has an hindex of 69, co-authored 186 publications receiving 17200 citations. Previous affiliations of Ronald L. Davis include Baylor University & Wake Forest University.
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Spatiotemporal rescue of memory dysfunction in Drosophila
TL;DR: A method for temporal and regional gene expression targeting in Drosophila is developed and shown the simultaneous spatial and temporal rescue of a memory defect, which rules out a developmental brain defect in the etiology of this deficit.
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Spatiotemporal gene expression targeting with the TARGET and gene-switch systems in Drosophila.
TL;DR: In this protocol, guidelines are provided for the use of these two novel systems, which should have general utility in studying Drosophila biology and in using the fly as a model for human disease.
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OLFACTORY MEMORY FORMATION IN DROSOPHILA: From Molecular to Systems Neuroscience
TL;DR: The Olfactory nervous system of insects and mammals exhibits many similarities, which suggests that the mechanisms for olfactory learning may be shared, and the genes identified to participate in Drosophila olfaction learning have conserved roles in mammalian organisms.
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The Drosophila learning and memory gene rutabaga encodes a Ca2+/Calmodulin-responsive adenylyl cyclase.
Lonny R. Levin,Pyung Lim Han,Paul M. Hwang,Paul Feinstein,Ronald L. Davis,Ronald L. Davis,Randall R. Reed +6 more
TL;DR: Data confirm the identity of the rutabaga locus as the structural gene for the Ca2+/CaM-responsive adenylyl cyclase and show that the inactivation of this cyclase leads to a learning and memory defect.
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The role of Drosophila mushroom body signaling in olfactory memory
TL;DR: Inactivation of mushroom body signaling through α/β neurons during different phases of memory processing revealed a requirement for mushrooms body signaling during memory retrieval, but not during acquisition or consolidation.