S
Sang-Won Park
Researcher at Sungkyunkwan University
Publications - 25
Citations - 3416
Sang-Won Park is an academic researcher from Sungkyunkwan University. The author has contributed to research in topics: Nitric oxide synthase & Copolyester. The author has an hindex of 11, co-authored 25 publications receiving 3009 citations. Previous affiliations of Sang-Won Park include Jaseng Hospital of Korean Medicine.
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6.5% efficient perovskite quantum-dot-sensitized solar cell
TL;DR: Perovskite QD-sensitized 3.6 μm-thick TiO(2) film shows maximum external quantum efficiency (EQE) of 78.6% at 530 nm and solar-to-electrical conversion efficiency of 6.54% at AM 1.5G 1 sun intensity (100 mW cm(-2)), which is by far the highest efficiency among the reported inorganic quantum dot sensitizers.
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Protective mechanism of glycyrrhizin on acute liver injury induced by carbon tetrachloride in mice.
TL;DR: It is suggested that glycyrrhizin alleviates CCl(4)-induced liver injury, and this protection is likely due to the induction of heme oxygenase-1 and the downregulation of proinflammatory mediators.
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Protective Effect of Baicalin Against Carbon Tetrachloride–Induced Acute Hepatic Injury in Mice
TL;DR: It is suggested that baicalin protects hepatocytes from the oxidative damage caused by CCl(4), and this protection is likely due to the induction of HO-1 expression and the inhibition of the proinflammatory mediators.
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Effect of GCSB-5, a Herbal Formulation, on Monosodium Iodoacetate-Induced Osteoarthritis in Rats.
Joon-Ki Kim,Sang-Won Park,Jung-Woo Kang,Yu-Jin Kim,Sung Youl Lee,Joon-Shik Shin,Sang-Ho Lee,Sun-Mee Lee +7 more
TL;DR: Results indicate that GCSB-5 is a potential therapeutic agent for the protection of articular cartilage against progression of osteoarthritis through inhibition of MMPs activity, inflammatory mediators, and NF-κB activation.
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Protective Effects of Human Placenta Extract on Cartilage Degradation in Experimental Osteoarthritis
TL;DR: The results indicate that HPE has therapeutic effects on OA by protecting cartilage by inhibiting MMP-2 activity in MG-63 cells and reducing deformity of knee joints.