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Sergio Pecorelli

Researcher at University of Brescia

Publications -  314
Citations -  28079

Sergio Pecorelli is an academic researcher from University of Brescia. The author has contributed to research in topics: Cancer & Ovarian cancer. The author has an hindex of 74, co-authored 311 publications receiving 25853 citations. Previous affiliations of Sergio Pecorelli include University of California, Irvine & University of Milan.

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Overexpression of claudin-3 and claudin-4 receptors in uterine serous papillary carcinoma: novel targets for a type-specific therapy using Clostridium perfringens enterotoxin (CPE).

TL;DR: Using gene expression profiling, high expression of the claudin‐3 and claud in‐4 receptors in a limited set of USPC is identified, which are sufficient to mediate CPE binding and trigger subsequent toxin‐mediated cytolysis.
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Human epididymis protein 4 as a serum marker for diagnosis of endometrial carcinoma and prediction of clinical outcome

TL;DR: HE4 significant correlation with decreased Overall Survival, Disease Free Survival and Progression Free Survival suggests its potential role as a novel prognostic marker for endometrial cancer.
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Histological features of uteroplacental vessels in normal and hypertensive patients in relation to birthweight

TL;DR: Placental bed biopsies obtained during caesarean section from normal, pre‐eclamptic and hypertensive pregnancies showed normal vascular physiological changes in the decidua and in the myometrium, and the mean birthweight centile was lower in the group with atherosis than in the groups with limited and thegroup with normal physiological changes.
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Phenotypic and functional analysis of tumor-infiltrating lymphocytes compared with tumor-associated lymphocytes from ascitic fluid and peripheral blood lymphocytes in patients with advanced ovarian cancer.

TL;DR: Data demonstrate the recruitment and accumulation of high concentrations of antigen-experienced T lymphocytes in TAL and TIL compared to PBL, and suggests that the impaired antitumor function commonly detected in these lymphocyte populations may be secondary to an acquired dysregulation of the IL-2 pathway.