S
Shoji Ohno
Researcher at Jichi Medical University
Publications - 59
Citations - 6052
Shoji Ohno is an academic researcher from Jichi Medical University. The author has contributed to research in topics: Bronchoalveolar lavage & Idiopathic interstitial pneumonia. The author has an hindex of 17, co-authored 59 publications receiving 5580 citations. Previous affiliations of Shoji Ohno include International University of Health and Welfare.
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Journal ArticleDOI
Identification of the transforming EML4–ALK fusion gene in non-small-cell lung cancer
Manabu Soda,Young Lim Choi,Munehiro Enomoto,Shuji Takada,Yoshihiro Yamashita,Shunpei Ishikawa,Shin-ichiro Fujiwara,Hideki Watanabe,Kentaro Kurashina,Hisashi Hatanaka,Masashi Bando,Shoji Ohno,Yuichi Ishikawa,Hiroyuki Aburatani,Toshiro Niki,Yasunori Sohara,Yukihiko Sugiyama,Hiroyuki Mano +17 more
TL;DR: It is shown that a small inversion within chromosome 2p results in the formation of a fusion gene comprising portions of the echinoderm microtubule-associated protein-like 4 (EML4) gene and the anaplastic lymphoma kinase (ALK) gene in non-small-cell lung cancer (NSCLC) cells.
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Elevated soluble ST2 protein levels in sera of patients with asthma with an acute exacerbation.
Katsuhisa Oshikawa,Kenji Kuroiwa,Kenji Tago,Hiroyuki Iwahana,Ken Yanagisawa,Shoji Ohno,Shin-ichi Tominaga,Yukihiko Sugiyama +7 more
TL;DR: The results suggest that soluble human ST2 protein in sera may be related to Th2-mediated allergic inflammation inducing acute exacerbation in patients with atopic asthma.
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Evidence that exogenous substances can be phagocytized by alveolar epithelial cells and transported into blood capillaries.
Tomoko Kato,Takashi Yashiro,Yoshio Murata,Damon C. Herbert,Katsuhisa Oshikawa,Masashi Bando,Shoji Ohno,Yukihiko Sugiyama +7 more
TL;DR: It is suggested that alveolar epithelial cells can incorporate exogenous particles, which are then transferred from the alveoli to intravascular spaces by transcytosis.
Journal ArticleDOI
Expression of ST2 in helper T lymphocytes of malignant pleural effusions.
TL;DR: The results suggest that CD4+ T cells in CA shift to Th2, which can produce soluble ST2 protein, resulting in increased concentrations of p-ST2 in malignant pleural effusion.
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Macrolides inhibit epithelial cell-mediated neutrophil survival by modulating granulocyte macrophage colony-stimulating factor release
TL;DR: Results suggest that macrolides inhibit epithelial cell-mediated neutrophil survival by modulating GM-CSF release, which may, at least in part, explain the effectiveness of this family of drugs on DPB.