Example of Frontiers in Endocrinology format
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Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format
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Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format Example of Frontiers in Endocrinology format
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open access Open Access

Frontiers in Endocrinology — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Endocrinology, Diabetes and Metabolism #72 of 219 down down by 22 ranks
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 2696 Published Papers | 13625 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 21/06/2020
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Related Journals

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Quality:  
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SNIP: 1.036
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Quality:  
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CiteRatio: 5.0
SJR: 1.229
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open access Open Access

Springer

Quality:  
High
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SNIP: 1.155

Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

5.1

50% from 2019

CiteRatio for Frontiers in Endocrinology from 2016 - 2020
Year Value
2020 5.1
2019 3.4
2018 3.3
2017 6.2
2016 8.0
graph view Graph view
table view Table view

1.518

16% from 2019

SJR for Frontiers in Endocrinology from 2016 - 2020
Year Value
2020 1.518
2019 1.31
2018 1.344
2017 1.79
2016 1.8
graph view Graph view
table view Table view

1.389

39% from 2019

SNIP for Frontiers in Endocrinology from 2016 - 2020
Year Value
2020 1.389
2019 1.0
2018 1.035
2017 1.254
2016 1.182
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 50% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has increased by 16% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 39% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Endocrinology

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Frontiers Media

Frontiers in Endocrinology

Approved by publishing and review experts on SciSpace, this template is built as per for Frontiers in Endocrinology formatting guidelines as mentioned in Frontiers Media author instructions. The current version was created on 21 Jun 2020 and has been used by 118 authors to write and format their manuscripts to this journal.

Endocrinology, Diabetes and Metabolism

Medicine

i
Last updated on
21 Jun 2020
i
ISSN
1664-2392
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
frontiersinSCNS_ENG_HUMS
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Citation Type
Numbered
[25]
i
Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FENDO.2018.00402
Overview of MicroRNA Biogenesis, Mechanisms of Actions, and Circulation.
Jacob O’Brien1, Heyam Hayder1, Yara Zayed1, Chun Peng1

Abstract:

MicroRNAs (miRNAs) are a class of non-coding RNAs that play important roles in regulating gene expression. The majority of miRNAs are transcribed from DNA sequences into primary miRNAs and processed into precursor miRNAs, and finally mature miRNAs. In most cases, miRNAs interact with the 3' untranslated region (3' UTR) of tar... MicroRNAs (miRNAs) are a class of non-coding RNAs that play important roles in regulating gene expression. The majority of miRNAs are transcribed from DNA sequences into primary miRNAs and processed into precursor miRNAs, and finally mature miRNAs. In most cases, miRNAs interact with the 3' untranslated region (3' UTR) of target mRNAs to induce mRNA degradation and translational repression. However, interaction of miRNAs with other regions, including the 5' UTR, coding sequence, and gene promoters, have also been reported. Under certain conditions, miRNAs can also activate translation or regulate transcription. The interaction of miRNAs with their target genes is dynamic and dependent on many factors, such as subcellular location of miRNAs, the abundancy of miRNAs and target mRNAs, and the affinity of miRNA-mRNA interactions. miRNAs can be secreted into extracellular fluids and transported to target cells via vesicles, such as exosomes, or by binding to proteins, including Argonautes. Extracellular miRNAs function as chemical messengers to mediate cell-cell communication. In this review, we provide an update on canonical and non-canonical miRNA biogenesis pathways and various mechanisms underlying miRNA-mediated gene regulations. We also summarize the current knowledge of the dynamics of miRNA action and of the secretion, transfer, and uptake of extracellular miRNAs. read more read less

Topics:

Untranslated region (51%)51% related to the paper, Regulation of gene expression (51%)51% related to the paper
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2,538 Citations
open accessOpen access Journal Article DOI: 10.3389/FENDO.2020.00025
The Role of Short-Chain Fatty Acids From Gut Microbiota in Gut-Brain Communication
Ygor Parladore Silva1, Andressa Bernardi1, Rudimar Luiz Frozza1

Abstract:

A substantial body of evidence supports that the gut microbiota plays a pivotal role in the regulation of metabolic, endocrine and immune functions. In recent years, there has been growing recognition of the involvement of the gut microbiota in the modulation of multiple neurochemical pathways through the highly interconnecte... A substantial body of evidence supports that the gut microbiota plays a pivotal role in the regulation of metabolic, endocrine and immune functions. In recent years, there has been growing recognition of the involvement of the gut microbiota in the modulation of multiple neurochemical pathways through the highly interconnected gut-brain axis. Although amazing scientific breakthroughs over the last few years have expanded our knowledge on the communication between microbes and their hosts, the underpinnings of microbiota-gut-brain crosstalk remain to be determined. Short-chain fatty acids (SCFAs), the main metabolites produced in the colon by bacterial fermentation of dietary fibers and resistant starch, are speculated to play a key role in neuro-immunoendocrine regulation. However, the underlying mechanisms through which SCFAs might influence brain physiology and behavior have not been fully elucidated. In this review, we outline the current knowledge about the involvement of SCFAs in microbiota-gut-brain interactions. We also highlight how the development of future treatments for central nervous system (CNS) disorders can take advantage of the intimate and mutual interactions of the gut microbiota with the brain by exploring the role of SCFAs in the regulation of neuro-immunoendocrine function. read more read less

Topics:

Gut–brain axis (56%)56% related to the paper, Gut flora (56%)56% related to the paper
View PDF
966 Citations
open accessOpen access Journal Article DOI: 10.3389/FENDO.2017.00006
Clinical Review of Antidiabetic Drugs: Implications for Type 2 Diabetes Mellitus Management

Abstract:

Type 2 diabetes mellitus (T2DM) is a global pandemic, as evident from the global cartographic picture of diabetes by the International Diabetes Federation (http://www.diabetesatlas.org/). Diabetes mellitus is a chronic, progressive, incompletely understood metabolic condition chiefly characterized by hyperglycemia. Impaired i... Type 2 diabetes mellitus (T2DM) is a global pandemic, as evident from the global cartographic picture of diabetes by the International Diabetes Federation (http://www.diabetesatlas.org/). Diabetes mellitus is a chronic, progressive, incompletely understood metabolic condition chiefly characterized by hyperglycemia. Impaired insulin secretion, resistance to tissue actions of insulin, or a combination of both are thought to be the commonest reasons contributing to the pathophysiology of T2DM, a spectrum of disease originally arising from tissue insulin resistance and gradually progressing to a state characterized by complete loss of secretory activity of the beta cells of the pancreas. T2DM is a major contributor to the very large rise in the rate of non-communicable diseases affecting developed as well as developing nations. In this mini review, we endeavor to outline the current management principles, including the spectrum of medications that are currently used for pharmacologic management, for lowering the elevated blood glucose in T2DM. read more read less

Topics:

Type 2 Diabetes Mellitus (71%)71% related to the paper, Insulin resistance (62%)62% related to the paper, Diabetes mellitus (59%)59% related to the paper, Insulin (55%)55% related to the paper
View PDF
841 Citations
open accessOpen access Journal Article DOI: 10.3389/FENDO.2016.00030
Adipose Tissue Remodeling: Its Role in Energy Metabolism and Metabolic Disorders.
Sung Sik Choe1, Jin Young Huh1, In Jae Hwang1, Jong In Kim1, Jae Bum Kim1

Abstract:

The adipose tissue is a central metabolic organ in the regulation of whole-body energy homeostasis. The white adipose tissue functions as a key energy reservoir for other organs, whereas the brown adipose tissue accumulates lipids for cold-induced adaptive thermogenesis. Adipose tissues secrete various hormones, cytokines, an... The adipose tissue is a central metabolic organ in the regulation of whole-body energy homeostasis. The white adipose tissue functions as a key energy reservoir for other organs, whereas the brown adipose tissue accumulates lipids for cold-induced adaptive thermogenesis. Adipose tissues secrete various hormones, cytokines, and metabolites (termed as adipokines) that control systemic energy balance by regulating appetitive signals from the central nerve system as well as metabolic activity in peripheral tissues. In response to changes in the nutritional status, the adipose tissue undergoes dynamic remodeling, including quantitative and qualitative alterations in adipose tissue-resident cells. A growing body of evidence indicates that adipose tissue remodeling in obesity is closely associated with adipose tissue function. Changes in the number and size of the adipocytes affect the microenvironment of expanded fat tissues, accompanied by alterations in adipokine secretion, adipocyte death, local hypoxia, and fatty acid fluxes. Concurrently, stromal vascular cells in the adipose tissue, including immune cells, are involved in numerous adaptive processes, such as dead adipocyte clearance, adipogenesis, and angiogenesis, all of which are dysregulated in obese adipose tissue remodeling. Chronic overnutrition triggers uncontrolled inflammatory responses, leading to systemic low-grade inflammation and metabolic disorders, such as insulin resistance. This review will discuss current mechanistic understandings of adipose tissue remodeling processes in adaptive energy homeostasis and pathological remodeling of adipose tissue in connection with immune response. read more read less

Topics:

White adipose tissue (78%)78% related to the paper, Adipose tissue (77%)77% related to the paper, Adipose tissue macrophages (73%)73% related to the paper, Brown adipose tissue (70%)70% related to the paper, Adipocyte (66%)66% related to the paper
View PDF
773 Citations
open accessOpen access Journal Article DOI: 10.3389/FENDO.2013.00037
Beta cell dysfunction and insulin resistance.
Marlon E. Cerf1

Abstract:

Beta cell dysfunction and insulin resistance are inherently complex with their interrelation for triggering the pathogenesis of diabetes also somewhat undefined. Both pathogenic states induce hyperglycemia and therefore increase insulin demand. Beta cell dysfunction results from inadequate glucose sensing to stimulate insulin... Beta cell dysfunction and insulin resistance are inherently complex with their interrelation for triggering the pathogenesis of diabetes also somewhat undefined. Both pathogenic states induce hyperglycemia and therefore increase insulin demand. Beta cell dysfunction results from inadequate glucose sensing to stimulate insulin secretion therefore elevated glucose concentrations prevail. Persistently elevated glucose concentrations above the physiological range result in the manifestation of hyperglycemia. With systemic insulin resistance, insulin signaling within glucose recipient tissues is defective therefore hyperglycemia perseveres. Beta cell dysfunction supersedes insulin resistance in inducing diabetes. Both pathological states influence each other and presumably synergistically exacerbate diabetes. Preserving beta cell function and insulin signaling in beta cells and insulin signaling in the glucose recipient tissues will maintain glucose homeostasis. read more read less

Topics:

Insulin resistance (73%)73% related to the paper, Insulin receptor (66%)66% related to the paper, Insulin (66%)66% related to the paper, Insulin oscillation (65%)65% related to the paper, Glucose homeostasis (62%)62% related to the paper
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592 Citations
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Frontiers in Endocrinology format uses frontiersinSCNS_ENG_HUMS citation style.

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Frequently asked questions

1. Can I write Frontiers in Endocrinology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Frontiers in Endocrinology guidelines and auto format it.

2. Do you follow the Frontiers in Endocrinology guidelines?

Yes, the template is compliant with the Frontiers in Endocrinology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Endocrinology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Endocrinology citation style.

4. Can I use the Frontiers in Endocrinology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Frontiers in Endocrinology.

5. Can I use a manuscript in Frontiers in Endocrinology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Endocrinology that you can download at the end.

6. How long does it usually take you to format my papers in Frontiers in Endocrinology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Frontiers in Endocrinology.

7. Where can I find the template for the Frontiers in Endocrinology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Endocrinology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Frontiers in Endocrinology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Frontiers in Endocrinology an online tool or is there a desktop version?

SciSpace's Frontiers in Endocrinology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Frontiers in Endocrinology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Frontiers in Endocrinology?”

11. What is the output that I would get after using Frontiers in Endocrinology?

After writing your paper autoformatting in Frontiers in Endocrinology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Frontiers in Endocrinology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Frontiers in Endocrinology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Endocrinology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Endocrinology?

The 5 most common citation types in order of usage for Frontiers in Endocrinology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Frontiers in Endocrinology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Frontiers in Endocrinology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Frontiers in Endocrinology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Frontiers in Endocrinology Endnote style according to Elsevier guidelines.

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