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Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format
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Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format Example of Frontiers in Synaptic Neuroscience format
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open access Open Access

Frontiers in Synaptic Neuroscience — Template for authors

Publisher: Frontiers Media
Categories Rank Trend in last 3 yrs
Cell Biology #163 of 279 down down by 73 ranks
Cellular and Molecular Neuroscience #62 of 88 down down by 35 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 112 Published Papers | 503 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 12/06/2020
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Related Journals

open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 12.8
SJR: 2.928
SNIP: 1.815
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Springer

Quality:  
Good
CiteRatio: 7.4
SJR: 1.255
SNIP: 0.945
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Springer

Quality:  
Good
CiteRatio: 5.4
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SNIP: 0.754
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Taylor and Francis

Quality:  
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CiteRatio: 15.1
SJR: 3.934
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.5

31% from 2019

CiteRatio for Frontiers in Synaptic Neuroscience from 2016 - 2020
Year Value
2020 4.5
2019 6.5
2018 6.0
2017 6.7
2016 5.5
graph view Graph view
table view Table view

1.869

17% from 2019

SJR for Frontiers in Synaptic Neuroscience from 2016 - 2020
Year Value
2020 1.869
2019 2.257
2018 2.398
2017 2.14
2016 2.551
graph view Graph view
table view Table view

1.084

5% from 2019

SNIP for Frontiers in Synaptic Neuroscience from 2016 - 2020
Year Value
2020 1.084
2019 1.146
2018 1.135
2017 1.031
2016 1.153
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 31% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • SJR of this journal has decreased by 17% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 5% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Frontiers in Synaptic Neuroscience

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Frontiers Media

Frontiers in Synaptic Neuroscience

Approved by publishing and review experts on SciSpace, this template is built as per for Frontiers in Synaptic Neuroscience formatting guidelines as mentioned in Frontiers Media author instructions. The current version was created on 12 Jun 2020 and has been used by 981 authors to write and format their manuscripts to this journal.

Neuroscience

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Last updated on
12 Jun 2020
i
ISSN
1663-3563
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Impact Factor
High - 1.412
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
frontiersinSCNS_ENG_HUMS
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder GE, Tinkham M, Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25 (1982) 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.3389/FNSYN.2011.00004
A History of Spike-Timing-Dependent Plasticity
Henry Markram1, Wulfram Gerstner1, Per Jesper Sjöström2, Per Jesper Sjöström3

Abstract:

How learning and memory is achieved in the brain is a central question in neuroscience research. Key to today’s research into information storage in the brain is the concept of synaptic plasticity, a notion that has been heavily influenced by Donald Hebb’s 1949 postulate. Hebb conjectured that repeatedly and persistently coac... How learning and memory is achieved in the brain is a central question in neuroscience research. Key to today’s research into information storage in the brain is the concept of synaptic plasticity, a notion that has been heavily influenced by Donald Hebb’s 1949 postulate. Hebb conjectured that repeatedly and persistently coactive cells should increase connective strength among populations of interconnected neurons as a means of storing a memory trace, also known as an engram. Hebb certainly was not the first to make such a conjecture, as we show in this history. Nevertheless, literally thousands of studies into the classical frequency-dependent paradigm of cellular learning rules were directly inspired by the Hebbian postulate. But in more recent years, a novel concept in cellular learning has emerged, where temporal order instead of frequency is emphasized. This new learning paradigm — known as Spike-Timing-Dependent Plasticity, or STDP — has rapidly gained tremendous interest, perhaps because of its combination of elegant simplicity, biological plausibility, and computational power. But what are the roots of today’s STDP concept? Here, we discuss several centuries of diverse thinking, beginning with philosophers such as Aristotle, Locke and Ribot, traversing e.g. Lugaro’s plasticita and Rosenblatt’s Perceptron, and culminating with the discovery of STDP. We highlight interactions between theoretical and experimental fields, showing how discoveries sometimes occurred in parallel, seemingly without much knowledge of the other field, and sometimes via concrete back-and-forth communication. We point out where the future directions may lie, which includes interneuron STDP, the functional impact of STDP, its mechanisms and its neuromodulatory regulation, and the linking of STDP to the developmental formation and continuous plasticity of neuronal networks. read more read less

Topics:

Hebbian theory (59%)59% related to the paper, Spike-timing-dependent plasticity (55%)55% related to the paper
View PDF
377 Citations
open accessOpen access Journal Article DOI: 10.3389/FNSYN.2012.00002
Spike-timing-dependent plasticity: a comprehensive overview
Henry Markram1, Wulfram Gerstner1, Per Jesper Sjöström2

Abstract:

Reference EPFL-ARTICLE-183375doi:10.3389/fnsyn.2012.00002View record in PubMed Record created on 2013-01-28, modified on 2017-05-12
View PDF
326 Citations
open accessOpen access Journal Article DOI: 10.3389/FNSYN.2010.00146
Timing is not Everything: Neuromodulation Opens the STDP Gate
Verena Pawlak1, Jeffery R. Wickens2, Alfredo Kirkwood3, Jason N. D. Kerr1

Abstract:

Spike timing dependent plasticity (STDP) is a temporally specific extension of Hebbian associative plasticity that has tied together the timing of presynaptic inputs relative to the postsynaptic single spike. However, it is difficult to translate this mechanism to in vivo conditions where there is an abundance of presynaptic ... Spike timing dependent plasticity (STDP) is a temporally specific extension of Hebbian associative plasticity that has tied together the timing of presynaptic inputs relative to the postsynaptic single spike. However, it is difficult to translate this mechanism to in vivo conditions where there is an abundance of presynaptic activity constantly impinging upon the dendritic tree as well as ongoing postsynaptic spiking activity that backpropagates along the dendrite. Theoretical studies have proposed that, in addition to this pre- and postsynaptic activity, a ‘third factor’ would enable the association of specific inputs to specific outputs. Experimentally, the picture that is beginning to emerge, is that in addition to the precise timing of pre- and postsynaptic spikes, this third factor involves neuromodulators that have a distinctive influence on STDP rules. Specifically, neuromodulatory systems can influence STDP rules by acting via dopaminergic, noradrenergic, muscarinic and nicotinic receptors. Neuromodulator actions can enable STDP induction or - by increasing or decreasing the threshold - can change the conditions for plasticity induction. Because some of the neuromodulators are also involved in reward, a link between STDP and reward-mediated learning is emerging. However, many outstanding questions concerning the relationship between neuromodulatory systems and STDP rules remain, that once solved, will help make the crucial link from timing-based synaptic plasticity rules to behaviorally-based learning. read more read less

Topics:

Spike-timing-dependent plasticity (65%)65% related to the paper, Hebbian theory (52%)52% related to the paper
View PDF
251 Citations
open accessOpen access Journal Article DOI: 10.3389/FNSYN.2010.00139
Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease
Melissa Vos1, Elsa Lauwers1, Patrik Verstreken1

Abstract:

Cell types rich in mitochondria, including neurons, display a high energy demand and a need for calcium buffering. The importance of mitochondria for proper neuronal function is stressed by the occurrence of neurological defects in patients suffering from a great variety of diseases caused by mutations in mitochondrial genes.... Cell types rich in mitochondria, including neurons, display a high energy demand and a need for calcium buffering. The importance of mitochondria for proper neuronal function is stressed by the occurrence of neurological defects in patients suffering from a great variety of diseases caused by mutations in mitochondrial genes. Genetic and pharmacological evidence also reveal a role of these organelles in various aspects of neuronal physiology and in the pathogenesis of neurodegenerative disorders. Yet the mechanisms by which mitochondria can affect neurotransmission largely remain to be elucidated. In this review we focus on experimental data that suggest a critical function of synaptic mitochondria in the function and organization of synaptic vesicle pools, and in neurotransmitter release during intense neuronal activity. We discuss how calcium handling, ATP production and other mitochondrial mechanisms may influence synaptic vesicle pool organization and synaptic function. Given the link between synaptic mitochondrial function and neuronal communication, efforts towards better understanding mitochondrial biology may lead to novel therapeutic approaches of neurological disorders including Alzheimer’s disease, Parkinson’s disease, amyotrophic lateral sclerosis and psychiatric disorders that are at least in part caused by mitochondrial deficits. read more read less

Topics:

mitochondrial fusion (61%)61% related to the paper, Neurotransmission (55%)55% related to the paper, Synaptic vesicle (54%)54% related to the paper, Neurotransmitter (53%)53% related to the paper, Mitochondrion (51%)51% related to the paper
View PDF
213 Citations
open accessOpen access Journal Article DOI: 10.3389/FNSYN.2010.00004
Genetic controls balancing excitatory and inhibitory synaptogenesis in neurodevelopmental disorder models
Cheryl L Gatto1, Kendal Broadie1

Abstract:

Proper brain function requires stringent balance of excitatory and inhibitory synapse formation during neural circuit assembly. Mutation of genes that normally sculpt and maintain this balance results in severe dysfunction, causing neurodevelopmental disorders including autism, epilepsy and Rett syndrome. Such mutations may r... Proper brain function requires stringent balance of excitatory and inhibitory synapse formation during neural circuit assembly. Mutation of genes that normally sculpt and maintain this balance results in severe dysfunction, causing neurodevelopmental disorders including autism, epilepsy and Rett syndrome. Such mutations may result in defective architectural structuring of synaptic connections, molecular assembly of synapses and/or functional synaptogenesis. The affected genes often encode synaptic components directly, but also include regulators that secondarily mediate the synthesis or assembly of synaptic proteins. The prime example is Fragile X syndrome (FXS), the leading heritable cause of both intellectual disability and autism spectrum disorders. FXS results from loss of mRNA-binding FMRP, which regulates synaptic transcript trafficking, stability and translation in activity-dependent synaptogenesis and plasticity mechanisms. Genetic models of FXS exhibit striking excitatory and inhibitory synapse imbalance, associated with impaired cognitive and social interaction behaviors. Downstream of translation control, a number of specific synaptic proteins regulate excitatory versus inhibitory synaptogenesis, independently or combinatorially, and loss of these proteins is also linked to disrupted neurodevelopment. The current effort is to define the cascade of events linking transcription, translation and the role of specific synaptic proteins in the maintenance of excitatory versus inhibitory synapses during neural circuit formation. This focus includes mechanisms that fine-tune excitation and inhibition during the refinement of functional synaptic circuits, and later modulate this balance throughout life. The use of powerful new genetic models has begun to shed light on the mechanistic bases of excitation/inhibition imbalance for a range of neurodevelopmental disease states. read more read less

Topics:

Synaptogenesis (61%)61% related to the paper, Synaptic plasticity (59%)59% related to the paper, Genetic model (54%)54% related to the paper, Neurodevelopmental disorder (51%)51% related to the paper, Synapse (51%)51% related to the paper
View PDF
193 Citations
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Frontiers in Synaptic Neuroscience format uses frontiersinSCNS_ENG_HUMS citation style.

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Frequently asked questions

1. Can I write Frontiers in Synaptic Neuroscience in LaTeX?

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Yes, the template is compliant with the Frontiers in Synaptic Neuroscience guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Frontiers in Synaptic Neuroscience?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Frontiers in Synaptic Neuroscience citation style.

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5. Can I use a manuscript in Frontiers in Synaptic Neuroscience that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Frontiers in Synaptic Neuroscience that you can download at the end.

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13. What is Sherpa RoMEO Archiving Policy for Frontiers in Synaptic Neuroscience?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Frontiers in Synaptic Neuroscience. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Frontiers in Synaptic Neuroscience?

The 5 most common citation types in order of usage for Frontiers in Synaptic Neuroscience are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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