Example of Inflammation and Regeneration format
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Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format Example of Inflammation and Regeneration format
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open access Open Access

Inflammation and Regeneration — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Cell Biology #74 of 279 down down by None rank
Immunology and Allergy #52 of 182 down down by None rank
Immunology #59 of 202 down down by None rank
journal-quality-icon Journal quality:
Good
calendar-icon Last 4 years overview: 110 Published Papers | 813 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 02/06/2020
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Journal Performance & Insights

CiteRatio

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

A measure of average citations received per peer-reviewed paper published in the journal.

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

7.4

CiteRatio for Inflammation and Regeneration from 2016 - 2020
Year Value
2020 7.4
graph view Graph view
table view Table view

1.962

SJR for Inflammation and Regeneration from 2020 - 2020
Year Value
2020 1.962
graph view Graph view
table view Table view

1.423

Year Value
2020 1.423
graph view Graph view
table view Table view

insights Insights

  • This journal’s CiteRatio is in the top 10 percentile category.

insights Insights

  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • This journal’s SNIP is in the top 10 percentile category.

Inflammation and Regeneration

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Springer

Inflammation and Regeneration

Approved by publishing and review experts on SciSpace, this template is built as per for Inflammation and Regeneration formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 824 authors to write and format their manuscripts to this journal.

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Last updated on
02 Jun 2020
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ISSN
1606-8610
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
White faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder, G.E., Tinkham, M., Klapwijk, T.M.: Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25(7), 4515–4532 (1982)

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1186/S41232-020-00146-3
How COVID-19 induces cytokine storm with high mortality
Shintaro Hojyo1, Mona Uchida1, Kumiko Tanaka1, Rie Hasebe1, Yuki Tanaka1, Masaaki Murakami1, Toshio Hirano1

Abstract:

The newly emerging coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) was first reported in Wuhan, China, but has rapidly spread all over the world. Some COVID-19 patients encounter a severe symptom of acute respiratory distress syndrome (ARDS) with high mortality. This ... The newly emerging coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) was first reported in Wuhan, China, but has rapidly spread all over the world. Some COVID-19 patients encounter a severe symptom of acute respiratory distress syndrome (ARDS) with high mortality. This high severity is dependent on a cytokine storm, most likely induced by the interleukin-6 (IL-6) amplifier, which is hyper-activation machinery that regulates the nuclear factor kappa B (NF-κB) pathway and stimulated by the simultaneous activation of IL-6-signal transducer and activator of transcription 3 (STAT3) and NF-κB signaling in non-immune cells including alveolar epithelial cells and endothelial cells. We hypothesize that IL-6-STAT3 signaling is a promising therapeutic target for the cytokine storm in COVID-19, because IL-6 is a major STAT3 stimulator, particularly during inflammation. We herein review the pathogenic mechanism and potential therapeutic targets of ARDS in COVID-19 patients. read more read less

Topics:

Cytokine storm (63%)63% related to the paper, ARDS (55%)55% related to the paper
View PDF
455 Citations
open accessOpen access Journal Article DOI: 10.1186/S41232-019-0101-5
The role of interleukin-1 in general pathology
Naoe Kaneko1, Mie Kurata1, Toshihiro Yamamoto1, Shinnosuke Morikawa1, Junya Masumoto1

Abstract:

Interleukin-1, an inflammatory cytokine, is considered to have diverse physiological functions and pathological significances and play an important role in health and disease. In this decade, interleukin-1 family members have been expanding and evidence is accumulating that highlights the importance of interleukin-1 in linkin... Interleukin-1, an inflammatory cytokine, is considered to have diverse physiological functions and pathological significances and play an important role in health and disease. In this decade, interleukin-1 family members have been expanding and evidence is accumulating that highlights the importance of interleukin-1 in linking innate immunity with a broad spectrum of diseases beyond inflammatory diseases. In this review, we look back on the definition of “inflammation” in traditional general pathology and discuss new insights into interleukin-1 in view of its history and the molecular bases of diseases, as well as current progress in therapeutics. read more read less

Topics:

Disease (50%)50% related to the paper
View PDF
262 Citations
open accessOpen access Journal Article DOI: 10.1186/S41232-018-0059-8
Piezoelectric smart biomaterials for bone and cartilage tissue engineering.

Abstract:

Tissues like bone and cartilage are remodeled dynamically for their functional requirements by signaling pathways. The signals are controlled by the cells and extracellular matrix and transmitted through an electrical and chemical synapse. Scaffold-based tissue engineering therapies largely disturb the natural signaling pathw... Tissues like bone and cartilage are remodeled dynamically for their functional requirements by signaling pathways. The signals are controlled by the cells and extracellular matrix and transmitted through an electrical and chemical synapse. Scaffold-based tissue engineering therapies largely disturb the natural signaling pathways, due to their rigidity towards signal conduction, despite their therapeutic advantages. Thus, there is a high need of smart biomaterials, which can conveniently generate and transfer the bioelectric signals analogous to native tissues for appropriate physiological functions. Piezoelectric materials can generate electrical signals in response to the applied stress. Furthermore, they can stimulate the signaling pathways and thereby enhance the tissue regeneration at the impaired site. The piezoelectric scaffolds can act as sensitive mechanoelectrical transduction systems. Hence, it is applicable to the regions, where mechanical loads are predominant. The present review is mainly concentrated on the mechanism related to the electrical stimulation in a biological system and the different piezoelectric materials suitable for bone and cartilage tissue engineering. read more read less

Topics:

Tissue engineering (55%)55% related to the paper
View PDF
205 Citations
open accessOpen access Journal Article DOI: 10.1186/S41232-018-0063-Z
Maintenance of intestinal homeostasis by mucosal barriers.
Ryu Okumura1, Ryu Okumura2, Kiyoshi Takeda2, Kiyoshi Takeda1

Abstract:

The intestine is inhabited by a tremendous number of microorganisms, which provide many benefits to nutrition, metabolism and immunity. Mucosal barriers by intestinal epithelial cells make it possible to maintain the symbiotic relationship between the gut microbiota and the host by separating them. Recent evidence indicates t... The intestine is inhabited by a tremendous number of microorganisms, which provide many benefits to nutrition, metabolism and immunity. Mucosal barriers by intestinal epithelial cells make it possible to maintain the symbiotic relationship between the gut microbiota and the host by separating them. Recent evidence indicates that mucosal barrier dysfunction contributes to the development of inflammatory bowel disease (IBD). In this review, we focus on the mechanisms by which mucosal barriers maintain gut homeostasis. Gut mucosal barriers are classified into chemical and physical barriers. Chemical barriers, including antimicrobial peptides (AMPs), are chemical agents that attack invading microorganisms, and physical barriers, including the mucus layer and the cell junction, are walls that physically repel invading microorganisms. These barriers, which are ingeniously modulated by gut microbiota and host immune cells, spatially segregate gut microbiota and the host immunity to avoid unnecessary immune responses to gut commensal microbes. Therefore, mucosal barrier dysfunction allows gut bacteria to invade gut mucosa, inducing excessive immune responses of the host immune cells, which result in intestinal inflammation. Gut mucosal barriers constructed by intestinal epithelial cells maintain gut homeostasis by segregating gut microbiota and host immune cells. Impaired mucosal barrier function contributes to the development of IBD. However, the mechanism by which the mucosal barrier is regulated by gut microbiota remains unclear. Thus, it should be further elucidated in the future to develop a novel therapeutic approach to IBD by targeting the mucosal barrier. read more read less

Topics:

Gut flora (61%)61% related to the paper, Immune system (50%)50% related to the paper
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203 Citations
open accessOpen access Journal Article DOI: 10.1186/S41232-019-0111-3
RANKL biology: bone metabolism, the immune system, and beyond

Abstract:

Receptor activator of NF-κB (RANK) ligand (RANKL) induces the differentiation of monocyte/macrophage–lineage cells into the bone–resorbing cells called osteoclasts. Because abnormalities in RANKL, its signaling receptor RANK, or decoy receptor osteoprotegerin (OPG) lead to bone diseases such as osteopetrosis, the RANKL/RANK/O... Receptor activator of NF-κB (RANK) ligand (RANKL) induces the differentiation of monocyte/macrophage–lineage cells into the bone–resorbing cells called osteoclasts. Because abnormalities in RANKL, its signaling receptor RANK, or decoy receptor osteoprotegerin (OPG) lead to bone diseases such as osteopetrosis, the RANKL/RANK/OPG system is essential for bone resorption. RANKL was first discovered as a T cell-derived activator of dendritic cells (DCs) and has many functions in the immune system, including organogenesis, cellular development. The essentiality of RANKL in the bone and the immune systems lies at the root of the field of “osteoimmunology.” Furthermore, this cytokine functions beyond the domains of bone metabolism and the immune system, e.g., mammary gland and hair follicle formation, body temperature regulation, muscle metabolism, and tumor development. In this review, we will summarize the current understanding of the functions of the RANKL/RANK/OPG system in biological processes. read more read less

Topics:

RANKL (65%)65% related to the paper, Osteoimmunology (60%)60% related to the paper, Osteoprotegerin (57%)57% related to the paper, Bone resorption (54%)54% related to the paper, Immune system (51%)51% related to the paper
View PDF
195 Citations
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Frequently asked questions

1. Can I write Inflammation and Regeneration in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Inflammation and Regeneration guidelines and auto format it.

2. Do you follow the Inflammation and Regeneration guidelines?

Yes, the template is compliant with the Inflammation and Regeneration guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Inflammation and Regeneration?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Inflammation and Regeneration citation style.

4. Can I use the Inflammation and Regeneration templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Inflammation and Regeneration.

5. Can I use a manuscript in Inflammation and Regeneration that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Inflammation and Regeneration that you can download at the end.

6. How long does it usually take you to format my papers in Inflammation and Regeneration?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Inflammation and Regeneration.

7. Where can I find the template for the Inflammation and Regeneration?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Inflammation and Regeneration's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Inflammation and Regeneration's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Inflammation and Regeneration an online tool or is there a desktop version?

SciSpace's Inflammation and Regeneration is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Inflammation and Regeneration?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Inflammation and Regeneration?”

11. What is the output that I would get after using Inflammation and Regeneration?

After writing your paper autoformatting in Inflammation and Regeneration, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Inflammation and Regeneration's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Inflammation and Regeneration?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Inflammation and Regeneration. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Inflammation and Regeneration?

The 5 most common citation types in order of usage for Inflammation and Regeneration are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Inflammation and Regeneration?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Inflammation and Regeneration's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Inflammation and Regeneration in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Inflammation and Regeneration Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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